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1 (DPPA)2]2+ (1) and [Pt3(NH3)6{NH2(CH2)6NH2}2(DPPS)2]2+ (2), respectively.
2    The estimated free energy of binding of a DPPS lipid to the second binding site is around -8.8 kca
3 s of the incubation of BBR3464 with DPPA and DPPS correspond to chloride displacement and formation o
4 ity relative to net negative DPPA, DPPG, and DPPS.
5 tached with nearly equal coverage to OPN and DPPS surfaces alone, suggesting that the preferential se
6 s demonstrated that lateral proton fluxes at DPPS are significant, but the lateral proton diffusion c
7 DL-alpha-phosphatidyl-L-serine, dipalmitoyl (DPPS) or zwitterionic L-alpha-phosphatidylcholine, dipal
8 e (DPPC), and dipalmitoylphosphatidylserine (DPPS) are reported.
9 holine (DPPC)/dipalmitoylphosphatidylserine (DPPS) (3:1:1 molar ratio) bilayers using (2)H-NMR and sp
10 simulation of dipalmitoylphosphatidylserine (DPPS) bilayer with Na+ counterions.
11  one and two dipalmitoylphosphatidylserines (DPPS) in a dipalmitoylphosphatidylcholine (DPPC) bilayer
12 es to negatively charged phospholipids DPPA, DPPS, and DPPG were slightly lower in a 100 mM chloride
13 f the negatively charged phospholipids DPPA, DPPS, and DPPG with the highly positively charged platin
14           Liposomes were formulated as DPPC, DPPS, cholesterol, GM(1) ganglioside; 7/4/7/1.57 molar r
15 r DPPA was the phosphate oxygen, whereas for DPPS, a binding site of the nitrogen of the serine side
16 id monolayer, decreasing in the order DPPG > DPPS > DPPC.
17                               In the case of DPPS-OPN patterns, micrometer-sized COM crystals dispers
18 ing body posture, we show that the extent of DPPS asymmetry is defined in an earth-centred coordinate
19 e structural and electrostatic properties of DPPS bilayer.
20 line (DPPC), dipalmitoyl phosphatidylserine (DPPS), dipalmitoyl phosphatidylethanolamine (DPPE), or d
21              Patterns of phosphatidylserine (DPPS) bilayers and osteopontin (OPN) were fabricated on
22  entropy of negatively charged phospholipids DPPS, DPPA, and DPPG were changed upon reaction with the
23   In contrast, the two acidic phospholipids, DPPS and DPPA, caused a dose-dependent increase in both
24                                The resulting DPPS has the shape of a bubble, elongated asymmetrically
25 ized the fine-grained geometry of the face's DPPS by recording the enhancement of the blink reflex el
26 ol (DPPG), and dipalmitoyl phospho-l-serine (DPPS).
27  binding of the PT1-Ca2+ complex to a single DPPS to be around -11.5 kcal/mol.
28 ion termed the defensive peripersonal space (DPPS) [1,2].
29  STATEMENT The defensive peripersonal space (DPPS) has a crucial role for survival, and its modulatio
30            The defensive peripersonal space (DPPS) is a vital "safety margin" surrounding the body.
31 ned inside the defensive peripersonal space (DPPS) of the face.
32  pain, affects defensive peripersonal space (DPPS), the portion of space surrounding the body within
33        Geometric modelling revealed (1) that DPPS was larger on the side of space ipsilateral to TN,
34  when the hand of the participant enters the DPPS of another individual, either in egocentric or in a
35 and approached to (and not receded from) the DPPS of the face.
36 and approached to (and not receded from) the DPPS of the face.
37  when a static hand is stimulated inside the DPPS of the face.
38 threatening stimulus is delivered inside the DPPS, subcortical defensive responses like the hand-blin
39 en that TN is unilateral, in TN patients the DPPS of the face might not be horizontally symmetric as
40 All these findings provide evidence that the DPPS is dynamically shaped by predictive mechanisms run
41  by the interpersonal interaction within the DPPS of the face.
42 on COM crystals and suppresses attachment to DPPS, suggesting a link between OPN and reduced attachme
43 omes was higher than the ratio of BBR3464 to DPPS liposomes, and similar differences were seen for BB

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