ライフサイエンスコーパス: DTH

1 DTH lesions were initiated in adult rats by intracerebra

2 DTH reactions were elicited within 48 h in 16 of 29 untr

3 DTH response increased rapidly, to peak at 31-90 days af

4 DTH responses and CNS infiltration were reduced in prote

5 DTH responses to intradermal injections of HPV E7 antige

6 DTH responses to unmodified autologous melanoma were ind

7 DTH sites had significantly larger blood flow as measure

8 DTH was assessed at 24, 48, 72, and 96 hours post bite i

9 A DTH response to bites was observed in 75% of individuals

10 rovision of exogenous TGF-beta or IL-10 at a DTH challenge site of allograft rejector mice caused a s

11 nhanced the ability of the mice to develop a DTH response after immunization with CneF-CFA, while ani

12 es feeding at sites in mouse ears that had a DTH response also fed faster than at normal sites.

13 DnaK and GroEL2 for the capacity to induce a DTH response in the guinea pig model.

14 rst, rescue with either a wild-type DTH or a DTH mutant lacking neural expression leads to weak circa

15 om L. longipalpis saliva, does not produce a DTH response in these mice, suggesting that structural o

16 ype was defined as the ability to suppress a DTH response to a recall antigen in the presence of dono

17 Our findings demonstrate that a DTH reaction to a non-CNS antigen within a CNS white mat

18 dence, 19.8%) and are all caused by allergic DTH reactions.

19 ntibody significantly inhibited alloreactive DTH but did not prevent T cell priming or interferon-gam

20 (-) hosts failed to demonstrate allospecific DTH (P < 0.001).

21 ceiving B10.D2 grafts developed allospecific DTH.

22 T mice, as was the induction of allospecific DTH.

23 uvant in that myosin-specific antibodies and DTH responses both develop by 21 days postinfection or p

24 h necessary and sufficient for gastritis and DTH due to H. pylori in mice; 2) high expression of CD45

25 Gastritis and DTH were present in recipients of unfractionated splenoc

26 repertoires between individuals with IH and DTH reactions to Tri r 2.

27 pregulated in multiple sclerosis plaques and DTH lesions.

28 ing the relationships between protection and DTH reactions.

29 aive animals showed graft survival rates and DTH responses that were indistinguishable from those of

30 sured by de novo target-cell recognition and DTH priming, indicating that IN may be the preferred ROA

31 in cutaneous inflammation and angiogenesis, DTH reactions were investigated in the ear skin of wild-

32 sly that the presence of an anticryptococcal DTH response in mice is not always indicative of protect

33 occal cells (HKC), enhanced anticryptococcal DTH reactivity.

34 rotective and nonprotective anticryptococcal DTH responses are induced by different immunogens and ha

35 Even when the anticryptococcal DTH response was augmented by blocking the down-regulato

36 use, depending on the nature of the antigen, DTH reactions mediate numerous protective (e.g., resista

37 nt protein antigen tetanus toxoid as well as DTH responses were preserved in rituximab-treated RA pat

38 e than background levels of TGF-beta mRNA at DTH challenge sites.

39 ts and showed the largest difference between DTH and IH responders in proliferation (mean standardize

40 induced within 2 weeks of immunization both DTH and IgG antibodies to guinea pig xenoantigens.

41 ossible to stimulate antigen-specific bovine DTH responses by using ESAT-6 in combination with a synt

42 Results from functional analyses by DTH, enzyme-linked immunospot, and immunohistofluorescen

43 inguished based on Th2 cytokine induction by DTH-associated major epitopes localizing to the amino-te

44 lografts and in donor alloantigen-challenged DTH sites in mice that have either accepted or rejected

45 microglial activation surrounding a chronic DTH EAE lesion.

46 The chronic DTH EAE lesion led to increased ligand binding in the ip

47 icroglial activation surrounding the chronic DTH EAE lesion.

48 the protective immunogen undergo a classical DTH response characterized by mononuclear cell and neutr

49 This treatment reduced T. cruzi DTH, although there was no effect on parasite-specific A

50 ferative responses in subjects with delayed (DTH), but not immediate (IH) hypersensitivity skin tests

51 The ability to depress DTH was transferred by naive spleen cells from transgeni

52 uPA(-/-) mice failed to develop DTH to SEA; did not polarize Ig production to IgE; did n

53 with 200 to 300 microg of peptide, developed DTH and EAU with scores comparable to those induced by 1

54 An alternative assay uses dithionite (DTH) to provide reduced Fd.

55 elated with the level of systemic anti-donor DTH, which varied over a 6-year interval.

56 ells into Bim-/- mice prevented an effective DTH response, thereby suggesting a causal link between a

57 ased type 1 cytokine production, an enhanced DTH response, and elevated production of CII-specific Ig

58 T cells at 5 days after infection, enhanced DTH, and increased mRNA levels for IFN-gamma in cornea a

59 cytokines thus leads to a state of enhanced DTH and depressed immediate-type hypersensitivity, which

60 A in inflammation, we evaluated experimental DTH reactions induced in the ear skin of transgenic mice

61 FN-gamma (IFN-gamma:IL-5 > or = 4:1) in five DTH subjects, even in the presence of Th2-dominated resp

62 splantation by challenging the recipient for DTH responses to donor alloantigen and evaluating the cy

63 guinea pigs that were indistinguishable from DTH responses driven by a PPD injection.

64 osure clearly distinguish this reaction from DTH.

65 By contrast, no 48-h DTH reactions occurred among 25 high risk and 32 low ris

66 hile nadroparin treatment exhibited a higher DTH risk than dalteparin (hazard ratio [HR], 26.7; 95% C

67 UAS-mediated rescue of tyrosine hydroxylase (DTH) mutant (ple) flies to study the roles of dopamine i

68 , RT-PCR, and delayed-type hypersensitivity (DTH) analyses were used to examine the effects on bacter

69 osin-specific delayed-type hypersensitivity (DTH) and antibody production at 21 days postinfection.

70 osin-specific delayed-type hypersensitivity (DTH) and autoantibodies in the absence of detectable car

71 s in allergic delayed-type hypersensitivity (DTH) and bacterial chancroid skin lesions express both C

72 ermined using delayed-type hypersensitivity (DTH) and collagen-induced arthritis (CIA) models in mice

73 uppression of delayed type hypersensitivity (DTH) and in vivo lymphoproliferation correlated with MMc

74 had depressed delayed-type hypersensitivity (DTH) and splenocyte proliferative responses relative to

75 14 patients, delayed-type hypersensitivity (DTH) and/or CD4 proliferative responses developed after

76 Delayed-type hypersensitivity (DTH) assays demonstrate that CJ83193 can elicit durable

77 of measuring delayed-type hypersensitivity (DTH) following intradermal injection of recombinant solu

78 rum IgG2a, or delayed-type hypersensitivity (DTH) footpad reactions were detected.

79 DPPD elicited delayed-type hypersensitivity (DTH) in 100% of Mycobacterium tuberculosis-infected guin

80 gastritis and delayed-type hypersensitivity (DTH) in mice.

81 ry edema, and delayed-type hypersensitivity (DTH) in mice.

82 nor-specific, delayed-type hypersensitivity (DTH) in tolerant organ transplant recipients.

83 revaccination delayed-type hypersensitivity (DTH) on the time course of the DTH response over 1-36 mo

84 nt model of a delayed-type hypersensitivity (DTH) reaction directed against the mycobacterium bacille

85 ve (anergic), delayed-type hypersensitivity (DTH) reaction to intradermal injection of purified prote

86 ty, toxicity, delayed-type hypersensitivity (DTH) reaction, and induction of serological and cellular

87 n a classical delayed-type hypersensitivity (DTH) reaction.

88 d by allergic delayed-type hypersensitivity (DTH) reactions and not by HIT or other rare conditions.

89 Delayed-type hypersensitivity (DTH) reactions elicited by the immunization were signifi

90 licitation of delayed-type hypersensitivity (DTH) reactions in vivo.

91 redominate in delayed-type hypersensitivity (DTH) reactions of the skin.

92 PAF inhibited delayed-type hypersensitivity (DTH) reactions to the chemical dinitrofluorobenzene only

93 ng psoriasis, delayed-type hypersensitivity (DTH) reactions, and rheumatoid arthritis.

94 and cutaneous delayed-type hypersensitivity (DTH) reactions.

95 that induces delayed-type hypersensitivity (DTH) reactions.

96 e humoral and delayed-type hypersensitivity (DTH) reactions.

97 es defined by delayed-type hypersensitivity (DTH) reactivity to cryptococcal culture filtrate antigen

98 d inhibit the delayed-type hypersensitivity (DTH) response caused by skin-homing effector memory T ce

99 duces a T(H)1 delayed-type hypersensitivity (DTH) response conferring protection against leishmaniasi

100 nduction of a delayed type hypersensitivity (DTH) response following exposure to L. longipalpis saliv

101 the effect of delayed-type hypersensitivity (DTH) response on clinical outcome.

102 a significant delayed-type hypersensitivity (DTH) response to adjuvant vaccine therapy (P =.0001), an

103 M) attenuated delayed-type hypersensitivity (DTH) response to methylated BSA and generation of Th17 c

104 op a vigorous delayed-type hypersensitivity (DTH) response upon intradermal virus antigen challenge.

105 Delayed-type hypersensitivity (DTH) response was detected in 7 of 10 patients for KLH a

106 se as being a delayed-type hypersensitivity (DTH) response with increased lung macrophage and Th1 cel

107 in vitro, the delayed-type hypersensitivity (DTH) response, and serum levels of CII-specific antibodi

108 s an impaired delayed-type hypersensitivity (DTH) response, manifested by a loss of skin testing to r

109 t resembles a delayed-type hypersensitivity (DTH) response.

110 onor-reactive delayed-type hypersensitivity (DTH) responses and donor-reactive alloantibody productio

111 to stimulate delayed-type hypersensitivity (DTH) responses in cattle experimentally infected with M.

112 ity to induce delayed-type hypersensitivity (DTH) responses in H37Rv-infected or BCG-vaccinated guine

113 dLAN dampened delayed type hypersensitivity (DTH) responses in male offspring.

114 ced increased delayed-type hypersensitivity (DTH) responses to autologous tumor cells in three patien

115 ine sites and delayed-type hypersensitivity (DTH) responses to autologous tumor cells indicative of T

116 stvaccination delayed-type hypersensitivity (DTH) responses to autologous tumor in 3 out of 14 treate

117 al effects on delayed type hypersensitivity (DTH) responses to donor antigens and type V collagen, an

118 The delayed-type hypersensitivity (DTH) responses to intradermal tuberculin were significan

119 titers and no delayed-type hypersensitivity (DTH) responses to Leishmania antigens.

120 We have used delayed-type hypersensitivity (DTH) responses to probe the mechanisms of drug-induced c

121 ecreased skin delayed-type hypersensitivity (DTH) responses to recall Ags in affected individuals wou

122 Delayed-type hypersensitivity (DTH) responses to recall antigens (CANDIDA:, mumps, and

123 hat cutaneous delayed type hypersensitivity (DTH) responses to recall antigens are significantly decr

124 cific CTL and delayed-type hypersensitivity (DTH) responses were tested at the time of graft rejectio

125 allospecific delayed-type hypersensitivity (DTH) responses, and leukocytic infiltration of grafts we

126 and suppress delayed-type hypersensitivity (DTH) responses.

127 measure human delayed type hypersensitivity (DTH) responses.

128 n assays, and delayed-type hypersensitivity (DTH) responses.

129 igen-specific delayed-type hypersensitivity (DTH) responses.

130 Delayed-type hypersensitivity (DTH) responsiveness was assessed using the ear-swelling

131 at a distant delayed-type hypersensitivity (DTH) site would force the immune system to reveal its cu

132 PB83 elicited delayed-type hypersensitivity (DTH) skin test responses in 78% of naturally infected tu

133 by PV, and by delayed-type hypersensitivity (DTH) skin testing with PV (PV-DTH).

134 Delayed-type hypersensitivity (DTH) testing in vivo and Elispot analysis in vitro sugge

135 n of systemic delayed-type hypersensitivity (DTH) that is induced when Ags are introduced into the an

136 osin-specific delayed-type hypersensitivity (DTH) that normally develops in infected mice, although i

137 re tested for delayed-type hypersensitivity (DTH) to autologous melanoma cells, both DNP-modified and

138 ntibodies and delayed type hypersensitivity (DTH) to cardiac myosin.

139 magnitude of delayed-type hypersensitivity (DTH) to PPD in the skin.

140 ice developed delayed-type hypersensitivity (DTH) to SEA; high levels of serum immunoglobulin E (IgE)

141 Allospecific delayed-type hypersensitivity (DTH) was evaluated after transplantation in high-risk gr

142 ity as myosin delayed-type hypersensitivity (DTH) was reduced, while anti-myosin Ab production was no

143 ked cutaneous delayed-type hypersensitivity (DTH) was significantly enhanced in VPAC(2)R-null mice co

144 allospecific delayed-type hypersensitivity (DTH) were used as measures of alloreactivity.

145 e disease and delayed-type hypersensitivity (DTH), a convenient model for two-photon imaging of Tem c

146 allospecific delayed-type hypersensitivity (DTH), and cytokine expression were compared among the re

147 mal models of delayed-type hypersensitivity (DTH), contact hypersensitivity (CHS), and arthritis.

148 e in reducing delayed-type hypersensitivity (DTH), even in RAGE(-/-) mice by 50% (p < 0.001).

149 ctions, i.e., delayed-type hypersensitivity (DTH), in response to an intradermal injection of specifi

150 ling test for delayed-type hypersensitivity (DTH), in vitro proliferation assays, and cytokine assays

151 Delayed-type hypersensitivity (DTH), suggesting T-cell responses, was seen in 14 of 19

152 resulted in a delayed-type hypersensitivity (DTH)-like EAE lesion.

153 rine model of delayed-type hypersensitivity (DTH).

154 s measured by delayed-type hypersensitivity (DTH).

155 allospecific delayed-type hypersensitivity (DTH).

156 nd suppressed delayed-type hypersensitivity (DTH).

157 uppression of delayed-type hypersensitivity (DTH).

158 s, a modified delayed-type-hypersensitivity (DTH) protocol was developed to induce innate and adaptiv

159 ion (positive delayed-type hypersensitivity [DTH+]), or (iii) no evidence of infection (DTH-).

160 iferation and delayed type hypersensitivity [DTH]) and B-cell (antibody) responses specific to CM wer

161 displayed an only slight reduction of 16% in DTH, explained by compensatory reciprocal upregulation o

162 otpad, CD4(+) T cells were hyporesponsive in DTH to donor type HLA-B Ags and derivative allopeptides.

163 was accompanied by a progressive increase in DTH responsiveness.

164 Concomitant with increases in DTH reactivity in mice treated with anti-CTLA-4 Fab frag

165 lly, P5 induced IL-5 and IL-10 production in DTH, but not IH subjects (p = 0.003 (IL-5), p = 0.024 (I

166 double-knockout mice show a 27% reduction in DTH reaction.

167 h Leishmania major as well as a reduction in DTH responses to Leishmania Ag.

168 mall interfering RNA show a 35% reduction in DTH, and ALCAM(-/-) RAGE(-/-) double-knockout mice show

169 aa 41-60) induced the strongest response in DTH subjects and showed the largest difference between D

170 and DnaK/GroEL2/Rv0685, were found to induce DTH responses in H37Rv-infected or BCG-vaccinated guinea

171 BCG-induced DTH responsiveness appears to decline more rapidly in tr

172 in response to dinitrofluorobenzene-induced DTH that correlated with a reduced number of T cells in

173 leas and bed bugs, the strong saliva-induced DTH response may reflect an adaptation of the fly to man

174 y in the three patients with vaccine-induced DTH responses.

175 [DTH+]), or (iii) no evidence of infection (DTH-).

176 ShK-186, a specific Kv1.3 blocker, inhibited DTH and suppressed Tem cell enlargement and motility in

177 ereby providing a possible mechanism for its DTH-enhancing properties.

178 for a cell-mediated immune defect with lower DTH responses and macrophages that have a decreased abil

179 Using this new protocol, we measured DTH responses induced by killed bacteria or PHA in the p

180 In mice treated with imatinib mesylate, DTH was reduced in comparison to sham-injected controls.

181 Significantly more DTH-positive patients developed a recurrence than DTH-ne

182 WYE-151650 was efficacious in mouse DTH and CIA models.

183 ion of primed allogeneic T cells in a murine DTH model.

184 C57BL/6 mice did not develop cardiac myosin DTH upon immunization with T. cruzi extract.

185 els of myosin IgG and did not exhibit myosin DTH or myocarditis upon T. cruzi infection.

186 osin-immunized mice, the magnitude of myosin DTH in the two groups was statistically equivalent.

187 ly into the test site also suppressed myosin DTH.

188 14 of 19 volunteers receiving LT and CS6; no DTH was seen in subjects receiving CS6 alone.

189 0%] of six IV patients; P =.005) and de novo DTH (seven [87.5%] of eight IN patients v two [33.3%] of

190 ervations was demonstrated by attenuation of DTH in WT and RAGE(-/-) animals pretreated with CD166/AL

191 Finally, the development of DTH was dependent on the schedule of administration of t

192 te population responsible for improvement of DTH responses were identified as being NK1.1 positive.

193 The inhibition of DTH reactions was abrogated by the addition of neutraliz

194 f pertussis toxin reversed the inhibition of DTH.

195 ligation and puncture demonstrated a loss of DTH for the 7 d following cecal ligation and puncture; h

196 sion or Bim deficiency prevented the loss of DTH.

197 ents who generated the greatest magnitude of DTH response and also displayed a strong clinical respon

198 monstrate the durability and T(H)1 nature of DTH to sand fly bites in humans living in a cutaneous le

199 Here, we describe the duration and nature of DTH to sand fly saliva in humans from an endemic area of

200 the thymus did not affect down-regulation of DTH.

201 The relevance of DTH to sand fly bites in humans living in a leishmaniasi

202 e findings raise questions about the role of DTH as an important mediator of cardiac allograft injury

203 we tested the hypothesis that suppression of DTH is mediated by tolerogenic properties of the apoptot

204 er, in contrast to the linked suppression of DTH seen when a given B-LCL expressed donor-type HLA-B a

205 on) and expression (efferent suppression) of DTH.

206 f this DTH-positive group was double that of DTH-negative patients (59.3% v 29.3%; P <.001).

207 Using a local adoptive transfer of DTH assay, we found that gamma delta T cells were requir

208 Uncoupling of DTH reactivity and protection has been demonstrated in o

209 cy for classification of subjects into IH or DTH groups.

210 ptide recognition after > or =20 mo in IH or DTH subjects.

211 unized mice also exhibited a decrease in OVA DTH.

212 uency of cellular responses to the parasite (DTH).

213 bility of some persons to mount a persistent DTH response probably reflects genetic background and/or

214 The ability to maintain a positive DTH response to the C albicans skin test was comparable

215 cognition pattern correlated with a positive DTH test to normal kidney cells despite no evidence of i

216 In contrast, positive DTH responses to DNP-modified autologous melanoma cells

217 ly longer in patients who developed positive DTH to unmodified tumor cells (25.2% v 12.3%; P <.001).

218 ity as measured by increased postvaccination DTH responses against autologous tumors.

219 the single best predictor of postvaccination DTH.

220 mismatched heart transplant exhibited potent DTH, T-cell proliferation and antibody responses to CM a

221 vival with vaccine-DTH responses but not PPD-DTH indicates a treatment-specific effect.

222 Prevaccination DTH was the single best predictor of postvaccination DTH

223 more, RTL201 inhibited T cell proliferation, DTH responses, and cytokine mRNA expression in the eye,

224 range, 10.7 to 93.6 months), an increased PV-DTH response seemed to be associated with improved 5-yea

225 ersensitivity (DTH) skin testing with PV (PV-DTH).

226 ta mAb for the restoration of donor-reactive DTH responses in allograft acceptor mice.

227 The weakness of donor-reactive DTH responses in these patients is due to donor alloanti

228 Donor-reactive DTH responses were not detected in reconstituted SCID mi

229 (4) Donor-reactive DTH sites of allograft rejector mice displayed a broad a

230 he ability of intestinal helminths to reduce DTH responses may have clinical implications for the use

231 ternal and paternal exposure to dLAN reduced DTH responses in female offspring.

232 All three patients manifested regulated DTH responses to HA-1H peptide.

233 ty, indicating a role for strictly regulated DTH and dopamine in robust circadian rhythmicity.

234 individuals developed induration resembling DTH, and the cellular infiltrates contained monocytes an

235 ignificantly increased spleen Th1 responses, DTH reaction, and serum IgG2a levels along with decrease

236 are important for the induction of a robust DTH response.

237 cultures established from seven IH and seven DTH subjects.

238 h swine, which failed to mount a significant DTH response to CM and displayed low and transient anti-

239 e with donor alloantigens at a distant site (DTH challenge).

240 tween both mDC allogeneic responses and skin DTH responses with clinical disease severity as measured

241 rectly corresponded to the magnitude of skin DTH reactions to recall Ags in both sarcoidosis subjects

242 duced the stress-induced enhancement of skin DTH.

243 The CD8+ TE cells mediated HA-1-specific DTH and produced interferon-gamma.

244 pulation that suppressed an antigen-specific DTH response.

245 h cardiac myosin developed T. cruzi-specific DTH and antibodies.

246 O mice adoptively transferred donor-specific DTH and induced apoptosis of BALB/c corneal endothelial

247 l grafts and exhibited strong donor-specific DTH.

248 ively transfer suppression of donor-specific DTH.

249 ients tested with positive envelope-specific DTH.

250 In this case, myosin-specific DTH and Ab production were significantly reduced.

251 eatment also reduced cardiac myosin-specific DTH and antibody production.

252 status and induced mild guinea pig-specific DTH only after 5 weeks.

253 interleukin-10, which could restore a strong DTH response to donor B-LCL.

254 the protein cocktails tested induced strong DTH responses in H37Rv-infected guinea pigs.

255 expression of CD8, and capacity to suppress DTH, which is mediated by previously immunized T cells.

256 COL-3 suppressed DTH responses to donor antigens and type V collagen, abr

257 s in ICAM-1-/- mice, along with a suppressed DTH response to donor alloantigens after transplantation

258 In addition, Th2 clones suppressed DTH responses mediated by Th1 clones in vivo and blocked

259 P-1alpha but not KC significantly suppressed DTH and sharply reduced neutrophil accumulation in the e

260 ced a CD8+ regulatory T cell that suppressed DTH by TRAIL production.

261 ositive patients developed a recurrence than DTH-negative patients.

262 We conclude that DTH analysis can readily detect donor antigen-linked sup

263 These results indicate that DTH response against saliva provides most or all of the

264 This suggests that DTH regulation in allograft acceptor mice may involve TG

265 The DTH response to C albicans was measured 2-3 days followi

266 Although TNF-alpha is increased at the DTH reaction site in mice immunized with the nonprotecti

267 cells, and amount of gamma interferon at the DTH reaction site.

268 mor necrosis factor alpha (TNF-alpha) at the DTH reaction site.

269 Ex vivo phenotyping of T cells at the DTH site indicated that this response is mediated by act

270 ating the cytokine profiles displayed at the DTH site.

271 us retinal beta-gal expression depressed the DTH response and proliferation assays after beta-gal imm

272 In contrast, FICZ exacerbated the DTH response and promoted Th17 cells.

273 injury, there was a further decrease in the DTH response of aged injured mice, compared with aged sh

274 d what role, if any, neutrophils play in the DTH to a viral antigen.

275 e main leukocyte population infiltrating the DTH reaction site is the neutrophil.

276 l allow a more thorough understanding of the DTH response and may provide a platform for more rapid a

277 rsensitivity (DTH) on the time course of the DTH response over 1-36 months after vaccination were stu

278 neutrophils are a critical component of the DTH response to viral antigen.

279 , but not interleukin-10, at the site of the DTH response.

280 cytokine mRNA expression at the site of the DTH response.

281 Second, the DTH rescue strain deficient in neural dopamine selective

282 They are recruited to the DTH test site by MIP-2 and MIP-1alpha, where they can be

283 a broad array of cytokine mRNAs, whereas the DTH sites of allograft acceptor mice displayed only IL-4

284 2-week-old thymocytes did not improve their DTH response after immunization.

285 had significant apoptosis but retained their DTH responses.

286 This DTH was also antigen specific, since immunization with s

287 tokines establishes the T(H)1 nature of this DTH response.

288 The OS of this DTH-positive group was double that of DTH-negative patie

289 y than expected from heterozygous parents to DTH+ offspring (P = 0.0006), and haplotypes containing T

290 Four or eight weeks after transfer, DTH to H. pylori Ags was determined by footpad injection

291 First, rescue with either a wild-type DTH or a DTH mutant lacking neural expression leads to w

292 GFbeta) or interleukin-10 (IL10) can uncover DTH responses to donor alloantigens in cardiac allograft

293 The correlation of survival with vaccine-DTH responses but not PPD-DTH indicates a treatment-spec

294 MPB83, and MPB64 failed to stimulate in vivo DTH in cattle that had been experimentally infected with

295 Using the trans vivo DTH test, we identified four regulators, and four nonreg

296 The mechanism by which DTH responses were induced was elucidated by histologic

297 1), and OS was significantly correlated with DTH to vaccine (P =.0001) but not with DTH to purified p

298 profile in draining lymph nodes of mice with DTH, treatment with I3C and DIM decreased the expression

299 with DTH to vaccine (P =.0001) but not with DTH to purified protein derivative (PPD), a control anti

300 d between individuals with VL and those with DTH+ phenotypes.