ライフサイエンスコーパス: E-box site

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1 NA-binding affinity of the antibody with the E box site.

2 4 transcription through their binding to the E-box site.

3 ding and transactivation from MyoD-dependent E-box sites.

4 f genes, preferentially at noncanonical (NC) E-box sites.

5 nd binding of MyoD complexes to oligomerized E-box sites.

6 LOCK and up-regulates the PAI-1 gene through E-box sites.

7 hat pTa promoter contains a conserved tandem E box site activated by E protein, HEB.

8 e promoter of Tbeta4 at consensus Thing1 and E-Box sites and identify both activation and repression

9 eins since they bind the classic palindromic E-box site as well as the direct repeat SRE-1 element.

10 protein is nuclear and binds efficiently to E-box sites as a homodimer.

11 ues to dissect the functions of two proximal E-box sites at -165 and +44 nt in regulating the hTERT p

12 raction with Max for specific DNA binding at E-box sites, but have opposing transcriptional activitie

13 gh several mechanisms including occupancy of E box sites by mSharp-1 homodimers and by direct physica

14 -GGTCAC-3', which differs from the canonical E-box site, CANNTG, to which most other bHLH proteins bi

15 e octamer motifs and a single muE5 motif (an E-box site, consensus CANNTG) interact for its function.

16 They bind to the E-box site, found in regulatory regions of B cell-specif

17 r, the possibility remains that the GATA and E box sites function in enhancer independent DV101S1 ger

18 gation, we determined the means by which the E-box site functions in this basal repression.

19 quence specificity as a factor binding to an E box site in the CD4 enhancer; thus, a member of the bH

20 s heterodimers with Max protein that bind to E-box sites in DNA and stimulate genes required for prol

21 with the CLOCK:BMAL1 complex for binding to E-box sites in target genes.

22 SNAI1, SNAI2, TWIST1 and ZEB1, which bind to E-box sites in the EpCAM promoter.

23 significantly regulated through the AP-1 or E-box sites in the promoter.

24 readily homodimerizes and binds to identical E-box sites in vitro.

25 rgy was shown to be GATA site-dependent, but E-box site-independent.

26 ssociation with USF1 and accumulation at the E-box site of the C/EBPalpha promoter in differentiated

27 ted in thymocytes and is thought to regulate E-box sites present in many T cell-specific gene enhance

28 er, a nuclear factor one (NFI) site, and two E-box sites, suggesting a conserved mode of regulation.

29 ChREBP-Mlx binding discriminated between E box sites that are glucose-responsive and those that a

30 Igkappa gene locus, including NF-kappaB and E-box sites that are important for the activity.

31 finity of the CLOCKDelta19:BMAL1 complex for E-box sites, thereby permitting increased USF1 occupancy

32 7 reporter construct, containing a hexameric E-box site, was used.

33 Two E-box sites, which bind E protein transcription factors

34 nds the TFEB recognition sequence CACGTG (an E box site) with a 5-10-fold lower affinity than TFEB.

35 of the protein and on the sequences of three E-box sites within PHM's first intron; the sites make di

36 s are identified that interact with specific E-box sites within the Dll3-promoter in vitro.

37 Mutations of E-box sites within this enhancer did not result in an ap

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