ライフサイエンスコーパス: E. amylovora

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1 encapsulated biocontrol agent, E325, against E. amylovora, and could serve as a model for further stu

2 An E. amylovora dspE mutant, however, elicited a hypersensi

3 genic and epiphytic Erwinia species, such as E. amylovora; E. carotovora subsp. atroseptica, betavasc

4 observed after the addition of an autoclaved E. amylovora suspension.

5 The response elicited by E. amylovora in its host during disease development is s

6 ction, another exopolysaccharide produced by E. amylovora.

7 homologs in E. carotovora, E. chrysanthemi, E. amylovora, E. herbicola, E. stewartii and E. rhaponti

8 By contrast, E. amylovora produces 430- and 300-base rsmB transcripts

9 produce antibiotics that effectively control E. amylovora, the bacterial pathogen responsible for the

10 ly reduced the induction of Hsr203 following E. amylovora challenge, further demonstrating a role for

11 The CobB(L) isoform from E. amylovora restored growth of as S. enterica cobB muta

12 ivo expression technology system to identify E. amylovora genes that are activated during infection o

13 Conversely, in E. amylovora, the homologous glucosyl transferase activi

14 We show that rsmB(Ea) in E. amylovora positively regulates extracellular polysacc

15 e secretion of the main exopolysaccharide in E. amylovora, amylovoran, leading to increased biofilm f

16 hat three of the five predicted DGC genes in E. amylovora (edc genes, for Erwinia diguanylate cyclase

17 the first to describe a role for c-di-GMP in E. amylovora and suggest that downregulation of motility

18 Deletion of hfq in E. amylovora Ea1189 significantly reduced bacterial viru

19 onstrated that CsrA plays a critical role in E. amylovora virulence and suggested that negative regul

20 ringae were identified for the first time in E. amylovora and included HecA hemagglutinin family adhe

21 acA-Csr system regulates virulence traits in E. amylovora remains unknown.

22 saccharide production, and thus virulence in E. amylovora.

23 mmercially available bacterial antagonist of E. amylovora (BlightBan, Pseudomonas fluorescens A506) c

24 r exopolysaccharide, is a virulent factor of E. amylovora.

25 Erwinia species, we cloned the rsmB genes of E. amylovora (rsmB(Ea)) and E. herbicola pv. gypsophilae

26 The genome of apple, an important host of E. amylovora, has been sequenced, creating new opportuni

27 HrpW of E. amylovora consists of two domains connected by a Pro

28 We identified hrpW of E. amylovora, which encodes a protein similar to known h

29 er than that in the wild type (WT) strain of E. amylovora.

30 quence of Ea273, a highly virulent strain of E. amylovora.

31 ns restored pathogenicity to dspE strains of E. amylovora, although restored strains were low in viru

32 egree of genetic uniformity among strains of E. amylovora, suggesting that the pathogen has undergone

33 al proteins confirms that the hrp systems of E. amylovora and P. syringae are closely related to each

34 und to be required for the full virulence of E. amylovora.

35 This study suggests that E. amylovora utilizes a variety of strategies during pla

36 ations of extensive similarities between the E. amylovora and P. syringae hrp systems in pathogenesis

37 In response to inoculation with E. amylovora, the BIS3 gene was expressed in stems of cv

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