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1 E. faecalis activates EGFR through production of H(2)O(2
2 E. faecalis co-exists with Escherichia coli and other pa
3 E. faecalis infections are associated with a high mortal
4 E. faecalis MN1 inhibited interleukin-8 production from
5 E. faecalis MPO binding is reportedly weak compared to b
6 E. faecalis must be able to adapt its physiology based o
7 E. faecalis tolerates high concentrations of GIT antimic
8 E. faecalis translocation and subsequent sepsis may be c
10 ocus is very highly conserved and only 1/473 E. faecalis isolates tested lacked ebpABC, while only 1.
11 Each of the alleles was analyzed using 50 E. faecalis isolates representing 42 diverse multilocus
15 showed complete bacteriocin activity against E. faecalis, but neither BacL1 nor BacA protein alone sh
17 esent study, we show that antibodies against E. faecalis LTA also bind to type 1 LTA from other gram-
18 immunization with rabbit antibodies against E. faecalis LTA promoted the clearance of bacteremia by
19 single very major error was obtained against E. faecalis, while vancomycin-intermediate S. aureus (VI
22 prgU gene pairs are widely distributed among E. faecalis isolates and other enterococcal and staphylo
24 crobial peptide activity developed either an E. faecalis or Pseudomonas aeruginosa urinary tract infe
27 (LOD) determined for HAdV 41, Phi X 174, and E. faecalis was 35 GU/muL, 1 GU/muL, and 5 x 10(3) GU/mu
28 stem of primary colonic epithelial cells and E. faecalis-infected macrophages (in vitro), with an int
29 )) and two AR bacterial strains (E. coli and E. faecalis, at 10(5) CFU mL(-1)) were spiked in real WW
31 tion of Staphylococcus spp., E. faecium, and E. faecalis and its ability to ascertain mecA, vanA, and
33 mmon NICU organisms including K. oxytoca and E. faecalis and increases in common adult organisms incl
35 excluding Enterobacter cloacae (98.3% S) and E. faecalis (86.0% S), and MIC(90)s ranged from 0.03 mug
37 n of solar exposure for AR E. coli, while AR E. faecalis was more resistant to the disinfection proce
39 l activity against VanA resistant bacteria ( E. faecalis , VanA VRE) at a level accurately reflecting
40 dy aimed to evaluate the association between E. faecalis and oral cancer and to determine the underly
43 ) and EGFR (gefitinib) significantly blocked E. faecalis-induced EGFR activation and cell proliferati
45 h culture-positive endophthalmitis caused by E. faecalis between January 1, 2002, and December 31, 20
46 o be greatly susceptible to sepsis caused by E. faecalis translocation, while orally infected E. faec
50 se (SprE) contribute to biofilm formation by E. faecalis and provide clues to how the activity of the
52 he mechanism of lectin pathway inhibition by E. faecalis, we purified and characterized cell wall car
53 etramic acid compounds naturally produced by E. faecalis MN1, may be useful in prevention of diseases
54 acteriocin 41 (Bac41) is produced by certain E. faecalis clinical isolates, and it is active against
56 porcine gut in response to diets containing E. faecalis were similar to the response to which contai
59 lineages were predominant among endocarditis E. faecalis isolates recovered during this time period.
60 on prior to urinary catheterization enhanced E. faecalis colonization, suggesting that implant-mediat
61 cally and phylogenetically the environmental E. faecalis were indistinguishable from their enteric co
62 rainwater tank samples followed by AS, esp (E. faecalis variant), and cylA genes which were detected
64 vior of two model FIB Enterococcus faecalis (E. faecalis) and Escherichia coli (E. coli) was examined
65 of microbiota in the distal gut of pigs fed E. faecalis UC-100 substituting antibiotics, this study
68 t identification of genetic determinants for E. faecalis commensal and pathogenic interactions with M
70 PCR/ESI-MS again found genetic evidence for E. faecalis at levels comparable to the pretreatment lev
71 inhibitory concentration was 0.57 mg/ml for E. faecalis and 1.15 mg/ml for all the other strains.
72 aureus ATCC 29213, 0.016 to 0.12 mug/ml for E. faecalis ATCC 29212, 0.008 to 0.03 mug/ml for S. pneu
73 0.1 mM), the toxicity of 10 mg L(-1) PCP for E. faecalis and fresh anaerobic sludge was detected in 1
75 mosomal genetic determinants responsible for E. faecalis biofilm-mediated infection, we used a rabbit
77 ding these observations, colon biopsies from E. faecalis-colonized IL-10(-/-) mice exhibited crypt hy
78 to concentrated conditioned media (CCM) from E. faecalis V583 and E. faecalis lacking the gelatinase
81 ur investigations demonstrate that GelE from E. faecalis can regulate enteric epithelial permeability
85 rial resistance against sepsis stemming from E. faecalis translocation through the conversion of resi
86 ) on the development of sepsis stemming from E. faecalis translocation was studied in SCID-beige (SCI
87 to facilitate intergeneric DNA transfer from E. faecalis is of concern because of extensive multiple
88 brain heart infusion (BHI) plus serum-grown E. faecalis isolates and that strain OG1RF expressed pil
91 lowing disruption of intestinal homeostasis, E. faecalis can overgrow, cross the intestinal barrier,
92 ors required for commensalism, we identified E. faecalis genes that are upregulated in the gut of M.
93 the absence of the silicone tubing implant, E. faecalis induced only minimal inflammation and was ra
94 ry response induced by urinary implantation, E. faecalis produced biofilm and high bladder titers in
97 ying overproduction of PrgB-like adhesins in E. faecalis and other clinically-important Gram-positive
99 cci: (i) the accretion of mobile elements in E. faecalis V583 renders it incompatible with commensal
100 e virulence map that explains enhancement in E. faecalis virulence and contributes to a deeper compre
101 EF2638 represents an authentic Rex factor in E. faecalis that influences the production or detoxifica
102 racytoplasmic function (ECF) sigma factor in E. faecalis, and that the deletion of sigV increases the
103 Proteins that localized to single foci in E. faecalis were found to share a positively charged dom
104 ratio of occurrence of ace and gelE genes in E. faecalis was much higher at 7.96 and 6.40 times, resp
105 rrence of ace, gelE, efaA, and asa1 genes in E. faecalis were found to be much higher compared to the
107 oly(ADP-ribose) polymerase were inhibited in E. faecalis-infected cells, indicating that E. faecalis
110 that EfbA is an important factor involved in E. faecalis endocarditis and that rEfbA immunization is
115 clusion, the epa genes, uniformly present in E. faecalis strains and involved in biosynthesis of poly
117 rmine the role of this putative regulator in E. faecalis pathogenesis, a perA-deficient mutant was cr
119 rocally regulate cephalosporin resistance in E. faecalis, dependent on the kinase activity of IreK.
120 inants of cholate and lysozyme resistance in E. faecalis, IreK was the only one found to be required
124 sults demonstrate the involvement of rnjB in E. faecalis pilin gene expression and provide insight in
125 l teichoic acid (WTA) synthesis gene tagB in E. faecalis V583 that exhibited an increased susceptibil
127 anding of how (p)ppGpp promotes virulence in E. faecalis and other bacterial pathogens is still lacki
128 also been repeatedly linked to virulence in E. faecalis, due in part to the transcriptional inductio
132 aecalis translocation, while orally infected E. faecalis did not spread into sepsis in the same mice
136 FBP, Pi is an activator of S. pyogenes LDH, E. faecalis LDH1, and L. lactis LDH1 and LDH2 at pH 6.
138 faecalis LDH2 </= Lactococcus lactis LDH2 < E. faecalis LDH1 < L. lactis LDH1 </= Streptococcus pyog
139 ssociate CcpA with the production of a major E. faecalis virulence factor, providing new insights int
140 tudy we define important roles for the major E. faecalis autolysin (Atn), eDNA, and sortase A (SrtA)
142 owever, little is known about the mechanisms E. faecalis uses to colonize and compete for stable gast
144 sses, we hypothesized that (p)ppGpp mediates E. faecalis virulence through regulation of metal homeos
145 merous animal models have been used to mimic E. faecalis infections, but none of them is considered i
146 thogenic E. coli isolates and (10(0) CFU/mL) E. faecalis and E. faecium strains were detected within
150 wing exposure to CCM from parental or mutant E. faecalis strains indicated paracellular permeability.
152 bp expression was restricted to 30 to 72% of E. faecalis cells, consistent with a bistability mode of
153 ession significantly improved the ability of E. faecalis to adhere to collagen, which is consistent w
157 ntified signatures suggest that ancestors of E. faecalis resided in extra-enteric habitats, challengi
159 ollection is not a random sample of cases of E. faecalis endocarditis, these results indicate that no
160 ssociated pili and an adhesin to collagen of E. faecalis, respectively, are both important in experim
161 s study, we investigated the contribution of E. faecalis to mixed-species infection when iron availab
164 ults provide the first evidence that EfbA of E. faecalis plays a role in UTIs, probably contributing
167 PCR/ESI-MS detected genetic evidence of E. faecalis in all CSF samples, but the level of detecti
169 role of the capsule as a virulence factor of E. faecalis and provide several mechanisms by which the
170 ollected from the peritoneal lavage fluid of E. faecalis-infected mice showed reduced levels of apopt
175 ith human platelets and found that growth of E. faecalis in BHI plus serum significantly enhanced adh
178 calis isolates underscores the importance of E. faecalis as a reservoir of VGs in the fresh water aqu
181 were observed for daptomycin in isolates of E. faecalis and 2 ME, 1 for high-level gentamicin resist
183 enomes, indicating that the vast majority of E. faecalis strains (unlike Enterococcus faecium and str
184 ne of the important pathogenic mechanisms of E. faecalis, as it elevates the organism's potential to
187 heromone-responsive, conjugative plasmids of E. faecalis have retained Prg-like surface functions ove
190 We undertook whole genome sequencing of E. faecalis associated with bloodstream infection in the
191 ies describing the four capsule serotypes of E. faecalis were based on immunodetection methods; howev
193 Thus, we hypothesized that this strain of E. faecalis may make anti-inflammatory factors which blo
194 f nematodes exposed to pathogenic strains of E. faecalis and Staphylococcus aureus, including multidr
195 ependent cleavage of Ace from the surface of E. faecalis, confirming that GelE specifically reduces A
197 es the in vitro antibiotic susceptibility of E. faecalis isolated from periodontitis patients in the
203 ABC mutants, suggesting involvement of other E. faecalis factors in urinary tract colonization or inf
204 the intestine, pPD1 is transferred to other E. faecalis strains by conjugation, enhancing their surv
205 s investigation indicated that, unlike other E. faecalis virulence traits, phage03-like elements were
208 n and epithelial translocation of pathogenic E. faecalis during severe microbial dysbiosis and was am
210 itro inhibitory activity against periodontal E. faecalis, and may be clinically useful in treatment o
211 able niche for the development of persistent E. faecalis infections in the murine bladders and kidney
212 and teicoplanin-susceptible (VanB phenotype) E. faecalis were responsible for major and minor errors.
216 rophages (RAW 264.7), that capsule-producing E. faecalis strains of either serotype C or D are more r
217 (-/-) mice attenuated infection and promoted E. faecalis colonization resistance by restoring the div
218 Upon hymeglusin inactivation of purified E. faecalis mvaS, the thioester adduct is more stable th
220 sistance by electrostatic repulsion, renders E. faecalis more resistant to killing by defensins and l
221 fer in the evolution of antibiotic resistant E. faecalis strains capable of causing human infection.
222 enesis of opportunistic antibiotic-resistant E. faecalis infections and in the transfer of antibiotic
223 eventing colonization by multidrug-resistant E. faecalis could therefore be a valuable approach towar
224 ogenic potential of the vancomycin-resistant E. faecalis V583 and three isogenic protease mutants (De
228 o bind putative promoter segments of several E. faecalis genes in an NADH-responsive manner, indicati
231 The mobility of the gram-positive species E. faecalis was much more sensitive to solution chemistr
233 fish is a novel, powerful model for studying E. faecalis pathogenesis, enabling us to dissect the mec
235 in vitro activity against human subgingival E. faecalis clinical isolates, and would likely be ineff
236 ure of vancomycin-resistant and -susceptible E. faecalis (VSEfs), which has important implications fo
238 t the clinical findings and demonstrate that E. faecalis can induce EGFR activation and cell prolifer
239 polymicrobial infection, we discovered that E. faecalis and C. albicans negatively impact each other
240 E. faecalis-infected cells, indicating that E. faecalis protects macrophages from apoptosis by inhib
241 ew studies have explored the mechanisms that E. faecalis employs to circumvent the host innate immune
244 under hydrodynamic conditions revealed that E. faecalis produces a DNase I-sensitive fibrous network
248 Interaction energy calculations suggest that E. faecalis retention was largely governed by the combin
251 ietary treatments: the basal diet group, the E. faecalis group, and the antibiotic group on d 0, 14,
253 of Fibrobacteres phylum and 12 genera in the E. faecalis group and antibiotics group were lower than
255 Bacterial abundance and diversity in the E. faecalis group, bacterial diversity in the antibiotic
256 We report here the identification of the E. faecalis bacteriocin, EntV, produced from the entV (e
257 ontaneous mutants that allowed growth of the E. faecalis DeltafabI strain on fatty acid-free medium.
260 The presence of multiple VGs in most of the E. faecalis isolates underscores the importance of E. fa
263 hnology (RIVET) to identify promoters on the E. faecalis OG1RF chromosome that were specifically acti
267 ng site mutant (K156T) fractionated with the E. faecalis membrane and also formed foci, whereas PcfC
272 observations were noted by exposing cells to E. faecalis-infected macrophages in a dual-chamber cocul
273 ity differences among seven chlorophenols to E. faecalis and fresh mixed anaerobic sludge were elucid
275 eolytic modulation of the immune response to E. faecalis, we also demonstrate that GelE can cleave th
280 nsport chains in SCV S. aureus and wild-type E. faecalis results in reduced growth rate but provides
282 uggests that for children with uncomplicated E. faecalis bacteremia, the addition of low-dose gentami
285 18-like vanA plasmids were found in 7% of VR E. faecalis isolates and none of the VR E. faecium isola
286 irradiated mice 1-4 d postinfection, whereas E. faecalis was not isolated from MLNs of normal mice.
287 vide novel insights into mechanisms by which E. faecalis and intestinal commensals can contribute to
289 llular DNA (eDNA), have been associated with E. faecalis biofilm production, the mechanisms underlyin
290 comparing clinical outcomes of children with E. faecalis bacteremia without endocarditis receiving am
293 model of colonization of the mouse gut with E. faecalis, without disrupting the microbiota, to evalu
294 veloped in SCIDbgN mice orally infected with E. faecalis, while the orally infected pathogen spread s
297 cationic human beta-defensins interact with E. faecalis at discrete septal foci, and this exposure d
300 4 (17%), 2 (9%), and 1 (4%) samples yielded E. faecalis, E. mundtii, E. casseliflavus, E. faecium, E
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