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1                                              E. risticii was cultured from 95% (20 of 21) of seroposi
2                                              E. risticii was detected in the blood by PCR in 81% (17
3                                              E. risticii was detected in the blood of subclinically i
4                                              E. risticii was found to be transmittable from trematode
5 g with antiphosphotyrosine antibody and anti-E. risticii antibody revealed colocalization of tyrosine
6  in murine monocytic cells and identified as E. risticii ultrastructurally and by characterization of
7 identical to those of the genes of an equine E. risticii strain from a property near the snail collec
8 re of the blood of the pony was positive for E. risticii starting on day 1 and was positive through d
9 two clinical specimens were seropositive for E. risticii by the IFA test, with titers ranging from 1:
10   Genetic information was also obtained from E. risticii strains from horses with PHF.
11 genes indicated the presence of geographical E. risticii strain clusters.
12 ggests that the mouse can be used to isolate E. risticii from the infected trematode.
13 t prevent binding of [35S]methionine-labeled E. risticii to P388D1 cells but did prevent internalizat
14 t internalization of [35S]methionine-labeled E. risticii.
15                           Studies of the new E. risticii isolates from the field cases indicated that
16 rmined to be apparent protective antigens of E. risticii.
17        Our findings suggest the evolution of E. risticii in the natural reservoir in separate geograp
18 to the PHF vaccines and the heterogeneity of E. risticii isolates may be associated with the vaccine
19 or 4 postinfection, further proliferation of E. risticii was prevented.
20 inhibition was reversible, since regrowth of E. risticii occurred upon the removal of genistein.
21             Genistein prevented spreading of E. risticii from P388D1 cells to THP-1 cells.
22  was identical to that of the type strain of E. risticii, and the sequence of the gene identified in
23                           By the nested PCR, E. risticii was detectable in the blood and feces of the
24 utamine in Percoll density gradient-purified E. risticii was not inhibited by genistein or herbimycin
25 d that the source organism closely resembled E. risticii, and the sequences of all three genes were v
26                     The heterogeneity of the E. risticii isolates obtained from the vaccinated horses
27 ted from a pony experimentally infected with E. risticii Maryland, blood specimens serially collected
28 tive as culture for detecting infection with E. risticii.
29 serially collected from mice inoculated with E. risticii Ohio 380, and blood and/or fecal specimens c
30 tent infection of trematode populations with E. risticii during summer and early fall.

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