ライフサイエンスコーパス: EB

1 ecially those from fragmented amylopectin in EB).

2 /rested, untrained/EB, and endurance trained/EB).

3 col at rest or after an acute exercise bout (EB).

4 s, such as junctional epidermolysis bullosa (EB).

5 les in the presence of end binding proteins (EBs).

6 al differentiation of daughter enteroblasts (EBs).

7 initive subcompartments of the AOTU, BU, and EB.

8 ected by dystrophic, junctional, and simplex EB.

9 ared with patients with recessive dystrophic EB.

10 bends to adopt the ring shape of the mature EB.

11 ularly dominant in dystrophic and junctional EB.

12 ccessions indicating their potential role in EB.

13 n, have been involved in the pathogenesis of EB.

14 mpared with free EB and non-BNPs loaded with EB.

15 burns on >30% of total body surface area and EB.

16 ular surface anomalies seen in children with EB.

17 bring these new tools to effectively tackle EB.

18 same core plasticity mechanisms as CO(2) and EB.

19 ex, junctional EB, dystrophic EB and Kindler EB.

20 e defects highlight the concept of syndromic EB.

21 rastructure and mitochondrial damage in ISCs/EBs.

22 was not significantly expressed in the whole EBs.

23 rmal lineage progression are synchronized in EBs.

24 active intermediates that quickly react with EBS.

25 B and tested on UKB: 0.6133, DE: 0.5143, and EB: 0.6049.

26 s in independent testing sets in DE: 0.6752, EB: 0.6156, and UKB: 0.5989; trained on EB and tested on

27 and UKB: 0.5989; trained on EB and tested on EB: 0.6565, DE: 0.5407, and UKB: 0.6043; trained on UKB

28 Ebselen (EBS), 2-phenyl-1,2-benzisoselenazol-3(2H)-one, is an org

29 was significantly higher in patients with CR-EB (28 mo, P = 0.01) and CR-NEB resections (24 mo, P = 0

30 og-rank P = .09); median PFS was longer with EBd (9.7 months) vs Bd (6.9 months).

31 Epidermolysis bullosa (EB), a group of complex heritable blistering diseases, i

32 he heritable forms of epidermolysis bullosa (EB), a phenotypically heterogeneous group of skin fragil

33 of these compartments is the ellipsoid body (EB), a structure formed largely by the axons of ring (R)

34 vertebrate stocks in the eastern Bering Sea (EBS), a data-rich ecosystem.

35 ventually a cure for patients suffering from EB, a currently intractable disease.

36 uclear translocation of transcription factor EB, a known activator of lysosomal gene transcription.

37 key autophagy genes and transcription factor EB, a master regulator of lysosomal biogenesis.

38 The EBD accumulation in the heated tumors increased by nearl

39 ocytes confirmed substantial infiltration of EB-affected skin with resting (CD45RA(+)) and activated

40 Here, we describe AAV-PHP.eB and AAV-PHP.S, capsids that efficiently transduce the

41 3 dopaminergic neurons, which project to the EB and are normally co-active with EB-RNs.

42 xtension of neuropil glia around the nascent EB and BU, and analyze the relationship of primary and s

43 no differences in overall liking between MB, EB and control, validating feasibility of including bana

44 pathologic and treatment characteristics, CR-EB and CR-NEB margin status were found to be independent

45 ality: EB simplex, junctional EB, dystrophic EB and Kindler EB.

46 cinoma-derived xenografts compared with free EB and non-BNPs loaded with EB.

47 752, EB: 0.6156, and UKB: 0.5989; trained on EB and tested on EB: 0.6565, DE: 0.5407, and UKB: 0.6043

48 nucleus, ACSS2 binds to transcription factor EB and translocates to lysosomal and autophagy gene prom

49 ysosomal stress-sensing transcription factor EB and, eventually, cell death.

50 nt years, the living marine resources of the EBS and Aleutian Islands have supported fisheries worth

51 Klu is induced in Notch-positive EBs and its activity restricts cell fate towards the ent

52 This bias is further enhanced by EBs and Lis1.

53 Endoreplication occurs exclusively in EBs and newborn ECs that inherit EGFR and active MAPK fr

54 form a complex via the C-terminal region of EBs and the microtubule-binding sites of tau.

55 broaden our knowledge about the mechanism of EBS and the neuromodulation of the human brain.

56 elationship between the circuitry engaged by EBS and the types of neural responses elicited by sensor

57 We collected endobronchial brush (EB) and bronchoalveolar lavage (BAL) samples from 39 ast

58 FU enhanced delivery of both Evans blue dye (EBD) and Gadolinium-chelated N-(2-hydroxypropyl)methacry

59 size- and shape-controlled embryoid bodies (EBs) and can be easily modified to control EB self-assem

60 testinal stem cells to produce enteroblasts (EBs) and enterocytes (ECs) that regenerate the gut.

61 that ERbeta was induced in embryoid bodies (EBs) and neural precursor cells (NPCs) during developmen

62 g during differentiation to embryoid bodies (EBs) and to fibroblast-like cells was driven by the huma

63 /rested, endurance trained/rested, untrained/EB, and endurance trained/EB).

64 A-A receptors to their axon terminals in the EB, and optogenetic stimulation coupled with electrophys

65 x cancer incidence in young white US men has ebbed, and modest increases are occurring/anticipated in

66 Cell apoptosis was detected by EB/AO staining, and cell cycle was analyzed by flow cyto

67 EBs are essential for 480AnkG localization and stabiliza

68 Knockout mutants lacking all EBs are viable and fertile and display normal pulling fo

69 use data from UK and Estonian biobanks (UKB, EB) as well as case-control data from the German populat

70 se discussed in a talk presented at the 2019 EB/ASPET meeting, which was organized by Professor Joe B

71 control the recruitment of leukocytes to the EB-associated skin lesions.

72 The role of native (NB) and extruded (EB) banana starch, and a 1:1 native:extruded banana star

73 ere, a live stem cell derived embryoid body (EB) based cardiac cell syncytium served as a biorecognit

74 Expression of the reporter construct EBS: beta-glucuronidase (GUS) was detected in Arabidopsi

75 blocked GTP hydrolysis, we demonstrate that EBs bind with high affinity to the GTP conformation of m

76 Whether the EB binding region is identical to the GTP cap is unclear

77 ctron microscopy has recently identified the EB binding site as the interface of four tubulin dimers

78 stablish that the GTP cap coincides with the EB-binding region.

79 rowing MTs, but information about additional EB-binding zones on MTs has been limited.

80 The EBD binds to the suppressor ligand UDP-N-acetyl-beta-d-m

81 Endothelial barrier (EB) breaching is a frequent event during inflammation, a

82 XE) and elastic backscattering spectrometry (EBS), can be used to explore potential matrix effects in

83 We find that brain accumulation of the PHP.eB capsid 1) exceeds that reported in any previous PET s

84 The PHP.eB capsid brain endothelial receptor affinity is nearly

85 Both CD38(+) hPGCLCs and CD38(-) EB cells significantly expressed PRDM1 and TFAP2C, altho

86 roximately 43% of FACS-sorted embryoid body (EB) cells] from primed-state induced pluripotent stem ce

87 the further investigation of the microtubule-EB-Cep104-tubulin-CP110-Cep97 network of proteins.

88 The CR-EB cohort comprised 749 (76%) patients, CR-NEB 159 patie

89 Electrical brain stimulation (EBS) complements neural measurements by probing the caus

90 ups: complete resection achieved en-bloc (CR-EB), complete resection achieved non-en-bloc (CR-NEB), a

91 mutagenesis of the effector-binding domain (EBD) coupled with various toggled selection regimes, we

92 ontrol) to 4.2, 6.6, and 7.76% in NB, MB and EB crumbs (fully gelatinized), respectively.

93 The EBD crystallizes in a "closed" conformation, in contrast

94 7 per mille (SD)), and human exhaled breath (EB; deltaD = -119.63 +/- 7.27 per mille (SD), delta(18)O

95 tracking dynamic microtubule plus-ends in an EB-dependent manner or moving processively towards minus

96 The sign of EB depends on the magnitude of the cooling field.

97 , CXCR2(+), and CCR2(+) myeloid cells toward EB-derived blister fluids.

98 As EBs differentiate into ECs, they become postmitotic, but

99 mutant promoters were rapidly silenced upon EB differentiation, indicating that transcription factor

100 The crystal structure of the EBD dimer was solved to 2.2 A resolution.

101 lycopersicum L.) yield causing early blight (EB) disease in tropical environment.

102 The EBD does not bind to 1,6-anhydro-N-acetyl-beta-d-muramyl

103 EBS does not undergo acid dissociation, but pH-dependent

104 e adverse effect of diabetes on SCI, reduced EB dye extravasation, and limited the loss of endothelia

105 howed increased extravasation of Evans Blue (EB) dye, and loss of endothelial cells and pericytes 1 d

106 olecular abnormality: EB simplex, junctional EB, dystrophic EB and Kindler EB.

107 uired and sufficient to drive damage-induced EB/EC growth.

108 of the AF layer results in a control of the EB effect which has potential for device applications.

109 near field responses and an unusual in-plane EB effect.

110 A notable example is the exchange bias (EB) effect between an antiferromagnet or ferrimagnet and

111 y role in the transfer of the exchange bias (EB) effect from the FM/AF interface to the AF/pinned-FM

112 in 16 distinct genes have been implicated in EB, encoding proteins influencing cellular integrity and

113 Consequently, the transcription factor EB enters the nucleus, and autophagy is up-regulated.

114 AMPK(-/-) EBs exhibited reduced levels of Tfeb, a master transcrip

115 We document that the EBD exists exclusively as a dimer, even at a concentrati

116 BERKO EBs expressed higher levels of key ectodermal and neural

117 lary ultrastructure, significantly decreased EB extravasation into spinal cord parenchyma, meaningful

118 ed by autoradiography, by histology, and for EBD fluorescence to assess BBB permeability.

119 = 0.004) in BBB permeability, as assessed by EBD fluorescence.

120 polynomial and calculate the energy barrier (EB) for direct domain switching between two variants of

121 rs with predicted effector binding elements (EBEs) for AvrHah1.

122 Tau and EBs form a complex via the C-terminal region of EBs and

123 xpense of the endoderm during embryoid body (EB) formation.

124 , whereas a distinct 4E-BP1 phospho-isoform, EB-gamma, phosphorylated at Thr-70, Ser-83, and Ser-101,

125 Over seven days of differentiation VIM -/- EBs had altered morphology compared to WT EBs, with a ri

126 The primordium of the EB has a complex composition.

127 The study of EB has led to seminal advances in our understanding of c

128 ow that patients with the junctional form of EB have significantly more cells infiltrating their woun

129 ently, tissue-specific AAV capsids (e.g. PHP.eB) have been shown to enhance brain delivery in rodents

130 redictors of improved OS (relative to IR, CR-EB hazard ratio [HR] 0.65, 95% confidence interval [CI]

131 s interaction is dependent on the C-terminal EB homology region of EB1 and partially dependent on an

132 In EBs, hPGCLCs were identified exclusively in the outermos

133 Remarkably, PHP.eB-iMecp2 administration was well tolerated in female Me

134 Intravenous injections of the PHP.eB-iMecp2 virus in symptomatic Mecp2 mutant mice signifi

135 icantly attenuated the percentage of beating EBs in culture and expression of early and late myocardi

136 measured neural responses and the effects of EBS in primary visual cortex in four patients implanted

137 is required for the specific accumulation of EBs in the proximal axon.

138 dy has evaluated the role of energy balance (EB) in modulating metabolic adaptation.

139 we report the control of the exchange bias (EB) in single-phase manganite thin films with nominallyu

140 The ellipsoid body (EB) in the Drosophila brain is a central complex (CX) su

141 effectors bind to effector-binding elements (EBEs) in SWEET gene promoters and induce SWEET genes.

142 lu) in restricting the fate of enteroblasts (EBs) in the Drosophila intestine.

143 In addition, the observed rate constant of EBS increased in the presence of sorbic acid, isopropano

144 bulk Brillouin zone and 6 eV binding-energy (EB) interval was acquired in approximately 3 h thanks to

145 Loss of Klu results in differentiation of EBs into EE cells.

146 jugating molecular vaccines with Evans blue (EB) into albumin-binding vaccines (AlbiVax), here we dev

147 EB is primarily a disorder affecting the epithelial laye

148 The sign of EB is related to the frustration of antiferromagnetic co

149 We show here that the EB is sequentially innervated by small-field and large-f

150 Furthermore, EBS is an excellent scavenger of reactive oxygen species

151 rinsic differentiation program common to all EBs is unknown.

152 Epidermolysis bullosa (EB) is an inherited, heterogeneous group of rare genetic

153 Hereditary epidermolysis bullosa (EB) is associated with skin blistering and the developme

154 Importance: Epidermolysis bullosa simplex (EBS) is a group of clinically and genetically diverse me

155 The 4D spectral function rho(EB; k) in the entire bulk Brillouin zone and 6 eV bindin

156 read of synaptic inhibition between adjacent EB lamina.

157 neurons play an important regulatory role in EB laminae formation.

158 These results provide direct evidence that EB lamination is critical for local pre-synaptic inhibit

159 EB levels in spinal cord parenchyma determined capillary

160 o showed significantly decreased parenchymal EB levels.

161 Structural analyses revealed an EB-like +TIP-binding motif within the capsid major homol

162 We show that aggregation method instructs EB lineage bias, with faster aggregation promoting pluri

163 quired for the inhibitory activity of tau on EB localization to microtubule ends.

164 We also find that aggregation kinetics of EBs markedly influence EB structure, with slower kinetic

165 Overall, PHP.eB-mediated delivery of iMecp2 provided widespread and e

166 o this problem is to use an Empirical Bayes (EB) method that assumes the variances among genes follow

167 The carbon analogue of EBS, namely, 2-phenyl-3H-isoindol-1-one, was included as

168 with energy-based prior encoding (Btrfly(pe-eb)) network performed as well as current state-of-the-a

169 Patch-clamp analysis indicates these EB neurons are highly sensitive to sleep loss, switching

170 Strikingly, the synaptic plasticity of these EB neurons is both necessary and sufficient for generati

171 arge-field neurons and that early developing EB neurons play an important regulatory role in EB lamin

172 The size of EB-NS can be tailored to diameters <20 nm and heights do

173 EB-NS can be uptaken by plants and remotely detected in

174 Furthermore, EB-NS display no bleaching and high brightness compared

175 EB-NS fluoresce at 910 nm and the fluorescence intensity

176 In summary, EB-NS have the potential for a wide range of bioimaging

177 The versatility of EB-NS is demonstrated by in-vivo single-particle trackin

178 sonication into NIR fluorescent nanosheets (EB-NS).

179 f the GabR effector-binding/oligomerization (Eb/O) domain suggest that binding a monocarboxylic gamma

180 A comparison between the structures of the Eb/O-PLP-AFPA complex and Asp-AT-PLP-AFPA complex reveal

181 yers and the trophoblast was abnormal in the EBs of tetraploid ESCs compared with diploid ESCs.

182 (AOTU) and bulb (BU) to the ellipsoid body (EB) of the central complex.

183 The effector-binding domain (EBD) of AmpR was purified to homogeneity.

184 effectors can drive plant transcription from EBEs on either strand and in both directions.

185 regulator of autophagy transcription factor EB or treatments with the autophagy enhancers rapamycin

186 ctural plasticity induced by ethyl butyrate (EB) or carbon dioxide (CO(2)) closes within 48 h after e

187 lotuzumab with bortezomib and dexamethasone (EBd) or bortezomib and dexamethasone (Bd) until disease

188 plasticity could be extended beyond 48 h if EB- or CO(2)-responsive olfactory sensory neurons (OSNs)

189 Furthermore, with EBs originating from patient's stem cells, this biosenso

190 al, Vienna, Austria, the network of national EB patient advocacy organizations.

191 t negative effect that cosegregates with the EB phenotype in an extended family.

192 o confirm the impact of the selenium atom on EBS photochemical reactivity.

193 We found that both AB and EB populations of DGCs recorded from epileptic rats rece

194 with slower kinetics resulting in increased EB porosity and growth factor signaling.

195 e retrovirally birthdated either early-born [EB; postnatal day (P)7] or adult-born (AB; P60) DGCs.

196 tive ophthalmic examination of children with EB presenting over seventeen months including meibomian

197 he fan-shaped body primordium, the posterior EB primordium moves forward and merges with the anterior

198 DALv2 neurons form the anterior EB primordium, which starts out as a bilateral structure

199 e type II lineages DM1-4, form the posterior EB primordium.

200 m moves forward and merges with the anterior EB primordium.

201 gitudinal lattice compaction associated with EB protein binding and GTP hydrolysis.

202 However, EB protein-dependent dynein plus end tracking was found

203 Previously EB proteins have been shown to recognize a stabilizing G

204 as a scaffold interacting with End-Binding (EB) proteins and membrane proteins such as Neurofascin-1

205 End-binding (EB) proteins associate with the growing tips of microtub

206 n GDP-tubulin and competes with end-binding (EB) proteins for binding to the microtubule plus end.

207 at MT plus ends and depends on end-binding (EB) proteins.

208 tions with plus-end microtubule tip-binding (EB) proteins.

209 nsity profiles of one of the three mammalian EB-proteins, EB3, fused with red fluorescent protein (RF

210 sion of cell division through its binding to EB-proteins.

211 nson's disease and type 2 diabetes show that EB-PRS achieved 307.1%, 42.8%, 25.5%, 3.1%, 74.3% and 49

212 to LDpred that makes use of LD information, EB-PRS also achieved 37.9%, 33.6%, 8.6%, 36.2%, 40.6% an

213 In this paper, we introduce EB-PRS, a novel method that leverages information for ef

214 gs suggest that Dop1R1 signaling in E-PG and EB R2/4 m circuits are compared against each other, ther

215 We finally show EB R2/R4m neurons are in contact with E-PG neurons that

216 ridge gall (E-PG) neuron and ellipsoid body (EB) R2/R4m ring neuron circuits both negatively gate mot

217 that the excitability of SLC5A11-expressing EB R4 neurons increases dramatically during starvation a

218 erved in RB-sensitized solutions, suggesting EBS reaction with active intermediates ((3)RB(2-)*, O(2)

219 Effector binding elements (EBEs) recognized by native TAL effectors in plants have

220 The molecular mechanisms of EB recovery are poorly understood.

221 Using this model, we show that EB recovery requires a CD31 receptor-induced, robust gly

222 End Binding proteins (EBs) recruit a diverse range of regulators of microtubul

223 his study highlights a novel role for tau in EB regulation, which might be impaired in neurodegenerat

224 Many fundamental questions about EBS remain unanswered, including the spatial extent of c

225 neuron classes connecting the AOTU, BU, and EB represent discrete lineages, genetically and developm

226 Many in the field who have participated in EB research for many years were especially enthusiastic

227 tructures are written with EBL on a positive EB-resist coated GaAs and developed followed by shallow

228 on unrevealing possible mechanistic basis of EB resistance in wild tomato.

229 canum Peralta could be a potential source of EB resistance; however, its underlying molecular mechani

230 in the peripheral and central domains of the EB, respectively.

231 ular platform for the therapeutic control of EB response.

232 dministration of 17-beta-estradiol benzoate (EB) restored this escalated anxiety-like behavior in TTC

233 Further, M and E cells regulate EB-RNs via identified PPM3 dopaminergic neurons, which p

234 how that ring neurons of the ellipsoid body (EB-RNs) display spontaneous morning and evening neural a

235 ct to the EB and are normally co-active with EB-RNs.

236 Fungal diversity was significantly lower in EB samples from patients with T2-high compared with T2-l

237 y for BAL fluid samples and 80% accuracy for EB samples.

238 The EB scheme thus unifies a body of observations on biologi

239 furcation with limited short-circuiting (the EB scheme).

240 (EBs) and can be easily modified to control EB self-assembly kinetics.

241 potent chemotherapeutic agent [epothilone B (EB)] showed significantly lower systemic toxicity and hi

242 ecting the underlying molecular abnormality: EB simplex, junctional EB, dystrophic EB and Kindler EB.

243 ISC/EB-specific knockdown of the mitophagy-related genes Pin

244 tion at the level of RMR is dependent on the EB status of the participants, being reduced to half aft

245 ic rate (RMR), is modulated by participants' EB status.

246 The sensitivity of EBS stocks to climate change ranged from "low" to "high,

247 gregation kinetics of EBs markedly influence EB structure, with slower kinetics resulting in increase

248 dicate that the structural MAP tau modulates EB subcellular localization in neurons.

249 ary changes in this rare autosomal recessive EBS subtype.

250 ysis using the doxycycline-inducible Etv2 ES/EB system showed increased levels of cell cycle genes in

251 However, the molecular determinants of EB/tau interaction remain unknown, as is the effect of t

252 he nuclear abundance of transcription factor EB (TFEB) and nuclear factor of activated T cells (NFAT)

253 ssion is upregulated by transcription factor EB (TFEB) and other members of the MiTF/TFE family of tr

254 factors (TFs), such as transcription factor EB (TFEB) and transcription factor E3 (TFE3), are emergi

255 mTOR complex 1 (mTORC1)-transcription factor EB (TFEB) axis in the host cells.

256 Transcription factor EB (TFEB) functions as a master controller of lysosomal

257 y overexpression of the transcription factor EB (TFEB) gene was effective in improving muscle patholo

258 Because transcription factor EB (TFEB) has recently emerged as a master regulator of

259 Transcriptional factor EB (TFEB) is a nutrient and stress-sensing transcription

260 egradation and promoted transcription factor EB (TFEB) nuclear localization.

261 iptionally regulated by transcription factor EB (TFEB) through the induction of genes involved in lys

262 Recently, transcription factor EB (TFEB) was implicated in expression of autophagy and

263 More recently, transcription factor EB (TFEB), a major regulator of autophagy and lysosomal

264 Transcription factor EB (TFEB), a master regulator of autophagy and lysosome

265 s known to activate the transcription factor EB (TFEB), a master regulator of lipid metabolism and ly

266 Here we show that transcription factor EB (TFEB), a master regulator of lysosomal biogenesis an

267 ia dephosphorylation of transcription factor EB (TFEB), a master regulator of lysosomal biogenesis an

268 Transcription Factor EB (TFEB), a master regulator of lysosomal biogenesis an

269 cid export based on the transcription factor EB (TFEB), a master regulator of lysosomal biogenesis th

270 We report here that the transcription factor EB (TFEB), a master regulator of lysosomal biogenesis, p

271 es an activation of the transcription factor EB (TFEB), a master regulator of lysosomal functions and

272 e via Akt modulation of transcription factor EB (TFEB), a master regulator of lysosomal pathways.

273 criptional regulator of transcription factor EB (TFEB), a master transcription factor of lysosomal bi

274 by up-regulation of the transcription factor EB (TFEB), which was down-regulated in cystinosis.

275 Here we show that transcription factor EB (TFEB)-a master transcriptional regulator of lysosoma

276 uclear translocation of transcription factor EB (TFEB).

277 ar translocation of the transcription factor EB (TFEB).

278 ar translocation of the transcription factor EB (TFEB).

279 In summary, our study delineates findings in EB that have potential relevance for all chronic wounds,

280 er, Met) were significantly reduced after an EB; that metabolites associated with skeletal muscle glu

281 Importantly, by targeting only the EBD, the evolved biosensors display DNA-binding affiniti

282 and genotypic features of syndromic forms of EB to delineate the concept of syndromic versus nonsyndr

283 Herein, we use the AAV-PHP.eB to deliver an instability-prone Mecp2 (iMecp2) transg

284 n of the mTOR substrate transcription factor EB to the nucleus and activation of autophagy-related ge

285 of 1 x 10(11) vector genomes (vg) of AAV-PHP.eB transduced 69% of cortical and 55% of striatal neuron

286 In an updated analysis, EBd-treated patients homozygous for the high-affinity Fc

287 d median PFS of 22.3 months vs 9.8 months in EBd-treated patients homozygous for the low-affinity all

288 Majority of the EB-triggered metabolic changes were active from steroida

289 AS events observed in mouse embryoid bodies (EBs) undergoing a neuroectoderm-like state.

290 yrin-G (480AnkG) selectively associates with EBs via its specific tail domain and that this interacti

291 yered pigment CaCuSi(4)O(10) (Egyptian Blue, EB) via ball milling and facile tip sonication into NIR

292 PFS was greater with EBd vs Bd (HR, 0.72; 70% confidence interval [CI], 0.59-

293 lly significant adverse events occurred with EBd vs Bd.

294 this study, the photochemical reactivity of EBS was investigated in the presence of (1) (1)O(2) and

295 After PET scanning, Evans blue dye (EBD) was injected into animals, and cryosections of the

296 in-blistering disease epidermolysis bullosa (EB), we show that large numbers of cells, often in exces

297 thylene glycol (HO(3)SO-CH(2)-CH(2)-OSO(3)H, EBS), whereas for state-of-the-art platinum-based cataly

298 and autophagy genes via transcription factor EB, which increased lysosomal biogenesis and activation

299 /- EBs had altered morphology compared to WT EBs, with a rippled outer surface and a smaller size due

300 ally distinct primordia of the AOTU, BU, and EB within the late larval brain.