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1 n healthy EBV carriers, transcription of the EBV nuclear antigen 1 (EBNA-1) gene is mediated by the p
4 n permissive proliferating cell lines due to EBV nuclear antigen 1 (EBNA-1) protein-mediated replicat
5 dependent on genome maintenance functions of EBV nuclear antigen 1 (EBNA-1), one of six EBNAs express
6 ry cells in the tonsil express the genes for EBV nuclear antigen 1 (EBNA1) (from the Qp promoter), la
7 of 9 tumors from American patients expressed EBV nuclear antigen 1 (EBNA1) and contained standard epi
9 o DCs, but also efficiently present targeted EBV nuclear antigen 1 (EBNA1) and EBV-latent membrane pr
10 mid origin of replication, oriP, and express EBV nuclear antigen 1 (EBNA1) are stably maintained extr
11 riP) in the Epstein-Barr virus (EBV) genome, EBV nuclear antigen 1 (EBNA1) enables persistence and en
12 s episome includes two elements from EBV: an EBV nuclear antigen 1 (EBNA1) expression cassette and an
16 Ls) carry a wild-type EBV genome and express EBV nuclear antigen 1 (EBNA1) selectively from the BamHI
17 elective increase of T cell responses to the EBV nuclear antigen 1 (EBNA1), the most consistently rec
18 se processes require a single viral protein, EBV nuclear antigen 1 (EBNA1), which binds two clusters
20 n from an integrase mutant reporter virus in EBV nuclear antigen 1-expressing cells, simian virus 40
23 tified binding sites for Epstein-Barr virus (EBV) nuclear antigen 1 (EBNA1) in the human genome using
28 , we show that targeting Epstein-Barr virus (EBV) nuclear antigen 1 (EBNA1) to one of them, the human
29 - and promoter-regulated Epstein-Barr virus (EBV) nuclear antigen 1 (EBNA1) was evaluated in three tr
32 AdE1-LMPpoly has been generated that encodes EBV nuclear antigen-1 (EBNA1) fused to multiple CD8(+) T
33 gen 1 (LANA1) is functionally similar to the EBV nuclear antigen-1 (EBNA1) protein expressed during v
34 clonal antibody (2B4-1) reactive against the EBV nuclear antigen-1, we noted strong staining of tumor
35 ansgenic mice expressing Epstein-Barr virus (EBV) nuclear antigen-1 (EBNA-1) in B-cells which show a
39 V transformation of primary B cells requires EBV nuclear antigen 2 (EBNA-2) to interact with RBP-Jk t
45 membrane protein 1 and, to a lesser extent, EBV nuclear antigen 2 mediated the increase in p53 level
52 To evaluate the role of Epstein-Barr Virus (EBV) nuclear antigen 3A (EBNA3A) in the continuous proli
54 in vitro and in vivo interaction between the EBV nuclear antigen 3C (EBNA3C) and the metastatic suppr
57 The product of one of these viral genes, the EBV nuclear antigen 3C (EBNA3C), is essential for the gr
58 V small RNAs, latent membrane protein 2, and EBV nuclear antigen 3C expression in peripheral blood su
63 interaction between the Epstein-Barr virus (EBV) nuclear antigen 3C (EBNA3C) and the metastatic supp
64 interaction between the Epstein-Barr virus (EBV) nuclear antigen 3C (EBNA3C) and the metastatic supp
71 -seronegative recipients was associated with EBV nuclear antigen antibody deficiency, polymorphic dis
72 mphoblastoid Cell Lines (LCLs) requires four EBV nuclear antigen (EBNA) oncoproteins: EBNA2, EBNALP,
73 ohistochemistry, two cases were positive for EBV nuclear antigen (EBNA)-1 (5%), one was positive for
75 s positive for latent membrane protein-1 and EBV nuclear antigen (EBNA)-4 DNAs by polymerase chain re
76 cells, and only a single viral protein, the EBV nuclear antigen (EBNA)1, is expressed via the altern
77 gamma secretion assays, was specific for the EBV nuclear antigen (EBNA)3A and latent membrane protein
80 0), kappa and lambda light chains as well as EBV nuclear antigens (EBNA2) and latent membrane protein
81 nk between the essential Epstein-Barr virus (EBV) nuclear antigen EBNA3C and the SCFSkp2 complex, pro
85 p and Cp, resulting in the expression of six EBV nuclear antigens (EBNAs) and the viral Bcl2 homologu
86 ent infection of B lymphocytes in vitro, six EBV nuclear antigens (EBNAs) are expressed from one of t
87 his may ensure against overexpression of the EBV nuclear antigens (EBNAs) prior to the transcriptiona
88 s of EBV latent membrane proteins (LMPs) and EBV nuclear antigens (EBNAs), as well as nontranslated v
91 ody to both the EBV viral capsid antigen and EBV nuclear antigen, followed by a more rapid rise in an
92 /E7), EBV latent membrane protein-1 and -2A, EBV nuclear antigen, HBV-encoded X antigen, and nonstruc
93 trated that the expression of this essential EBV nuclear antigen is capable of releasing the G2/M che
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