ライフサイエンスコーパス: EC

1 EC differentiation, capillary engraftment, reduced capil

2 EC N1ICD expression in melanoma correlated with shorter

3 EC-specific deletion in mice of glutaminase, the initial

4 ECs derived from cardiogenic and hemogenic mesoderm can

5 ECs from LAL-deficient (lal(-/-)) mice possess enhanced

6 d by three cycles of docetaxel (100 mg/m(2); EC-D).

7 rol epoxide hydrolase (ChEH) metabolizes 5,6-EC into cholestane-3beta,5alpha,6beta-triol, which is tr

8 on the European Commission Decision 2002/657/EC.

9 of angiopoietins with Tie2 in cis and across EC-EC junctions in trans Except for the binding of the C

10 s a difference in their capacity to activate EC and support the secondary recruitment of flowing neut

11 g tumor response to nCRT in locally advanced EC.

12 sive injury in mice to demonstrate that aged ECs impair the repopulating activity of young HSCs and i

13 all ECs in wild-type cells, and 5-7% of all ECs in cells lacking proofreading factors are, in fact,

14 omyces cerevisiae and found that 1-3% of all ECs in wild-type cells, and 5-7% of all ECs in cells lac

15 However, how DF alters EC metabolism, and whether resulting metabolic changes i

16 turbulence and temporal replication allow an EC tower to provide statistically robust flux estimates

17 ectric and the optical characteristics of an EC cell can be engineered to suit a variety of applicati

18 n, a commonly IV-administered antibiotic and EC-SERS to detect dobutamine, a drug commonly administer

19 vity per weight of CNTs than those of CA and EC, respectively.

20 of MCAK expression on MT growth dynamics and EC branching morphology.

21 1%, and 52.8% for plain aspirin, PL2200, and EC aspirin, respectively (p < 0.001 for both comparisons

22 es, increasing the concentration of FAEs and ECs through the inhibition of degrading enzymes has been

23 e strikingly different response of VSMCs and ECs to cAMP elevation.

24 Aortic EC were stimulated with low-dose TNFalpha (0.3 ng/ml) in

25 ondrial respiratory capacity in human aortic ECs.

26 adenosine 5'-phosphosulfate reductase (APR, EC 1.8.99.2), was significantly higher in Salicornia tha

27 monstrate that 43% of detected enhancers are EC-specific and contain SNPs associated to cardiovascula

28 by the passaging of porcine coronary artery ECs from passage P1 to P4, and was associated with a pro

29 n blood-brain barrier endothelial cells (BBB-ECs) that was previously shown to be involved in leukocy

30 isease multiplied their probability of being EC (versus NC) 4.23 times with respect to not having a h

31 iod from baseline in the EC users or between EC users and control subjects in any of the health outco

32 , lacking the extracellular calcium binding (EC) domain, inhibited BMP2 signaling, whereas SMOC-EC (E

33 fy the rejuvenating factors elaborated by BM ECs that restore HSC function and the immune repertoire

34 Under increased electric fields, both EC entropy and adiabatic temperature changes increase qu

35 n of 5-LOX from nucleus to cytoplasm in both ECs and VSMCs, potentially facilitating SPM biosynthesis

36 at reported for uptake of similar PEG NPs by ECs in a static culture, where the smaller disks were fo

37 Furthermore, platelet removal by ECs resulted in a corresponding decrease in platelet-leu

38 m power densities of biofuel cells using CA, EC and EPC electrodes without BQ were 41, 47 and 53micro

39 BQ were 41, 47 and 53microW/cm(2), while CA, EC and EPC electrodes with BQ showed 260, 330 and 500mic

40 battery (LIB) and electrochemical capacitor (EC) are considered as the most widely used energy storag

41 ty (PS)) and emulsifying (emulsion capacity (EC), droplet size, polydispersity (PDI), emulsifying act

42 LP) vaccines that mimic the "empty" capsids (ECs) normally produced in viral infection.

43 ic carbon (TOC) and/or the elemental carbon (EC) contents in the atmospheric aerosol may have a stron

44 ns than PFIs due to higher elemental carbon (EC) emissions.

45 Embryonic carcinoma (EC) cells are malignant counterparts of embryonic stem (

46 Human embryonic carcinoma (EC) cells are shown to restrict the expression of murine

47 Our data highlight the significance of CCN1-EC and CCN1-pericyte communication signals in driving ph

48 associated with classical endothelial cell (EC) activation signs, such as Vegfa/VEGFR1+2 upregulatio

49 s by which molecules cross endothelial cell (EC) barriers in normal tissues and in solid tumors, incl

50 To examine its role in endothelial cell (EC) biology, we generated mice with catalytic inactivati

51 ll (SMC) proliferation and endothelial cell (EC) dysfunction are critical in the pathogenesis of athe

52 Chronic endothelial cell (EC) loss, percentage of eyes with a decrease of >/=25% i

53 fects of PGA2 on pulmonary endothelial cell (EC) permeability and inflammatory activation and identif

54 shear stress, which alters endothelial cell (EC) physiology.

55 fact that the spontaneous endothelial cell (EC) regeneration is a slow and insufficient process, it

56 and functionally distinct endothelial cell (EC) subtypes from cardiogenic versus hemogenic mesoderm

57 fects are fully rescued by endothelial cell (EC)-specific overexpression of Kir2.1.

58 we generated a mouse with endothelial cell (EC)-specific overexpression of the human insulin recepto

59 resulting in increased interepithelial cell (EC) scanning, expression of antimicrobial genes, and gly

60 vivo hematopoietic stem and progenitor cell/EC (HSPC/EC) coculture system as well as in vivo EC infu

61 ever, it is not known how endothelial cells (EC) from large vessels process a postprandial surge of F

62 Endothelial cells (EC) play a key role in atherosclerosis.

63 Cytokine activation of endothelial cells (EC) upregulates VCAM-1 receptors that target monocyte re

64 RPE), fenestrated choroid endothelial cells (ECs) and Bruch's membrane, a highly organized basement m

65 Endothelial cells (ECs) are critical determinants of vascular homeostasis a

66 ression level and role in endothelial cells (ECs) are still unclear.

67 Human endothelial cells (ECs) are widely used to study mechanisms of angiogenesis

68 om the perspective of how endothelial cells (ECs) coordinate migration and proliferation in response

69 col (PEG) hydrogel NPs by endothelial cells (ECs) cultured in a microchannel compared to uptake of ei

70 Microvascular endothelial cells (ECs) display a high degree of phenotypic and functional

71 Endothelial cells (ECs) express O-glycoproteins that are believed to play i

72 strate that microvascular endothelial cells (ECs) from Anxa2(-/-) mice display reduced barrier functi

73 Glutamine metabolism in endothelial cells (ECs) has been poorly studied.

74 on within CD31-expressing endothelial cells (ECs) in affected glomeruli.

75 an important role for BM endothelial cells (ECs) in regulating hematopoietic aging and support furth

76 Endothelial cells (ECs) in the tumor microenvironment have been reported to

77 to Angpt-2 suppression in endothelial cells (ECs) in vitro.

78 calized on the surface of endothelial cells (ECs) is a key determinant of the magnitude and duration

79 We cultured endothelial cells (ECs) on collagen-coupled stiff or compliant polyacrylami

80 ial of MPs from senescent endothelial cells (ECs) or from patients with acute coronary syndrome (ACS)

81 elet disposal mediated by endothelial cells (ECs) through phosphatidylserine (PS) and examined the ef

82 ximity ligation assays in endothelial cells (ECs) to demonstrate compound-induced inhibition of Pfn1-

83 Transplanting vascular endothelial cells (ECs) to support metabolism and express regenerative para

84 nsitizes retinal vascular endothelial cells (ECs) to VEGF-A, leading to upregulation of angiopoietin-

85 determined that aging of endothelial cells (ECs), a critical component of the BM microenvironment, w

86 benign tumors of vascular endothelial cells (ECs), characterized by three distinct stages: proliferat

87 tect against apoptosis of endothelial cells (ECs), one of the hallmarks of endothelial dysfunction le

88 as decreased in mutant DA endothelial cells (ECs), which ectopically expressed the venous marker Coup

89 in retinal and choroidal endothelial cells (ECs).

90 hysiological responses of endothelial cells (ECs).

91 alian cells via choline dehydrogenase (CHDH; EC 1.1.99.1), we assessed whether this enzyme was expres

92 awn a search for additional roles of choroid ECs in RPE physiology and disease.

93 Chronic EC loss was calculated from 6 months postoperatively to

94 A significant linear chronic EC loss was reported after implantation with myopic or t

95 Although electronic cigarettes (ECs) are a much less harmful alternative to tobacco ciga

96 and Wnt7b-specific signals in mammalian CNS ECs to promote angiogenesis and regulate the BBB.

97 irin), and a delayed-release enteric-coated (EC) aspirin.

98 y regulation of E-selectin on the cocultured EC.

99 c acid components of the elongation complex (EC).

100 actively transcribing elongation complexes (ECs) in Escherichia coli and Saccharomyces cerevisiae an

101 (RC) (>/=2 PIMT/year); 2) erratic compliers (EC) (<2 PIMT/year); and 3) non-compliers (NC) (no PIMT).

102 inorganic Conductor/WO3/LiNbO3/NiO/Conductor EC cell isaccompanied by the modulation of its high freq

103 ereas Sox17 expression is required to confer EC function.

104 ing (equalisation of parental contributions (EC)) and the other forcing it (circular sib mating (CM))

105 fected individuals termed elite controllers (ECs) maintain CD4(+) T cell counts and control viral rep

106 Our results uncover a coordinated EC-IEL response to enteric infections that modulates lym

107 ch paired activation of entorhinal cortical (EC) and Schaffer collateral (SC) inputs to hippocampal C

108 rom in vivo angiogenesis models and cultured EC document that TSP-4 mediates upregulation of angiogen

109 effects of low intensity electrical current (EC) and ultrasound (US) treatments on the reduction of s

110 into >90% CD31(+)/VE-cadherin(+) definitive ECs.

111 e abundance was elevated in ECSHIP2(Delta/+) ECs and was suppressed by PI3K and NADPH oxidase 2 inhib

112 further proved crucial in glutamine-deprived ECs to restore protein synthesis, suppress ER stress, an

113 However, patient-derived ECs demonstrate a greater increase in permeability and i

114 zed by diacylglycerol acyltransferase (DGAT, EC 2.3.1.20).

115 ystemic cytokine responses may differentiate ECs from subjects with uncontrolled viral replication or

116 osis factor (TNF) compared with normal donor EC cultures.

117 , inhibited BMP2 signaling, whereas SMOC-EC (EC domain only) enhanced BMP2 signaling.

118 By applying an electrochemical (EC) gate voltage to the molecule, we switch the redox gr

119 In online electrochemistry (EC)/LS DESI MS, when 0 V was applied to the EC cell Tyr

120 work, we show that Fe(0) electrocoagulation (EC) permits the oxidative removal of Mn(II) from solutio

121 Endoplasmic reticulum (ER) stress elicits EC dysregulation in metabolic syndrome.

122 array scan patterns, only found on high-end EC-SPM setups such as hopping mode scan, were easily imp

123 d ethanolamides (FAEs) and endocannabinoids (ECs) have been shown to alleviate pain and inflammation,

124 produce enteroblasts (EBs) and enterocytes (ECs) that regenerate the gut.

125 dium channel (ENaC) subunits in enterocytes (ECs) to maintain osmotic and ISC homeostasis.

126 LPMOs are copper-dependent enzymes (EC 1.14.99.53-56) that, with glycoside hydrolases, parti

127 entation of contact precautions for all ESBL-EC carriers remains weak.

128 of and against contact precautions for ESBL-EC carriers.

129 t precautions for preventing nosocomial ESBL-EC transmission in nonepidemic settings, without harming

130 of these enzymes converts 5-ethynylcytosine (EC) to 5-ethynyluridine monophosphate that is subsequent

131 c patches were created from spheroids (CM:FB:EC = 70:15:15, 70:0:30, 45:40:15) using a 3D bioprinter.

132 n internal networks that are responsible for EC stability.

133 etation gas exchange parameters derived from EC data are subject to scale-dependent and method-specif

134 ross primary productivity (GPP) derived from EC data to calculate a measure of iWUE (G1 , "stomatal s

135 ns have been reported in HIV-1 isolated from EC.

136 packaged in the extracellular vesicles from EC-CM.

137 is incorporated into synaptic vesicles, from EC synaptic terminals using two photon microscopy in sli

138 ellular vesicles could transfer miR-195 from ECs to SMCs to inhibit the expression of 5-HTT in SMCs a

139 (SMCs) and promoted the tube formation from ECs.

140 MPs were prepared from ECs and venous blood from patients with ACS (n=30) and f

141 ell sources capable of generating functional ECs.

142 of protein kinase B was increased in hIRECO EC as was Nox2 NADPH oxidase-dependent generation of sup

143 atopoietic stem and progenitor cell/EC (HSPC/EC) coculture system as well as in vivo EC infusions fol

144 st machinery required for silencing in human EC cells is distinct from that in murine EC cell lines:

145 In human EC cells, MLV integration occurs normally, but no viral

146 We tested the susceptibility of human EC cells to transduction by retroviral vectors derived f

147 The findings reveal novel features of human EC gene regulation not present in their murine counterpa

148 esults demonstrate that neurons in the human EC represent space with an enhanced flexibility relative

149 d contactless conductivity detection (hybrid EC-CE-C(4)D) is herein described.

150 trogen and aspartate to asparagine) impaired EC sprouting even in the presence of glutamine and aspar

151 ith a dipolar interaction exponent of 2/3 in EC materials and the well-proven fractional dimensionali

152 ing pathway were found to be dysregulated in EC.

153 Snail was also expressed in EC overlying atherosclerotic plaques in coronary arterie

154 EndMT) and spontaneous liver fibrogenesis in EC-specific constitutive hemi-deficient (Erg (cEC-Het) )

155 characterize lipid droplet (LD) formation in EC by manipulating pathways leading to the formation and

156 aorta, oleic acid treatment generates LD in EC ex vivo.

157 e non-canonical Wnt5a in pericyte but not in EC cultures.

158 We found that ADAR2 is overexpressed in EC and that the increase in expression positively correl

159 critical for generating action potentials in EC cells, but it was also important for regulating 5-HT

160 METHODS AND LD were present in EC lining the aorta after the peak in plasma triglycerid

161 ly recognized as a focal adhesion protein in EC, was not anticipated to have a role in vascular barri

162 nce of Cav-1 increased cAMP/PKA signaling in EC, as indicated by elevated phosphorylation of hormone-

163 dMT markers (Slug, N-cadherin, alpha-SMA) in EC exposed to low shear stress.

164 nhancing insulin sensitivity specifically in EC leads to a paradoxical decline in endothelial functio

165 findings define critical roles for Stac3 in EC coupling and human disease.

166 In ECs, MKL1 was constitutively nuclear and MKL2 cytoplasmi

167 tion with ECs induced the entry of Ca(2+) in ECs via the EC-expressed TRPM2.

168 ensitive TRPM2 mutant channel (C1008-->A) in ECs suppressed the Ca(2+) entry response.

169 results suggest blocking TRPM2 activation in ECs is a potentially important means of therapeutically

170 ECs of human carcinomas and melanoma, and in ECs of the pre-metastatic niche in mice.

171 to upregulation of angiopoietin-2 (Ang2) in ECs through FOXO1 activation and triggering a positive f

172 essential for the discovery of biomarkers in ECs to further investigate the non-SMGs that were highly

173 Loss of Bmp6 in ECs recapitulated the hemochromatosis phenotype of globa

174 tify a set of cytokines that are elevated in ECs and define their effects on cellular activation, HIV

175 ot M2-polarized macrophages induced EndMT in ECs.

176 d factor-1 (SDF-1) levels were 43% higher in ECs than in NCs.

177 Inhibition of glutamine metabolism in ECs did not cause energy distress, but impaired tricarbo

178 on of phosphodefective STIM1-Y361F mutant in ECs prevented the increase in vascular permeability indu

179 tory MKL1/2 target genes in VSMCs but not in ECs.

180 receptors (N1ICD) are frequently observed in ECs of human carcinomas and melanoma, and in ECs of the

181 r silencing of poly ADP-ribose polymerase in ECs of mice prevented PMN transmigration.

182 at Y361 via proline-rich kinase 2 (Pyk2) in ECs.

183 in IL-37 induced pro-angiogenic response in ECs and in the mouse model of Matrigel plug and oxygen-i

184 ctivation of one SHIP2 allele selectively in ECs (ECSHIP2(Delta/+)).

185 (E2) treatment activates Notch signaling in ECs.

186 t mediate the key actions of Hh signaling in ECs.

187 entify significantly mutated genes (SMGs) in ECs and use them as biomarkers for the classification of

188 that MCAK promotes fast MT growth speeds in ECs cultured on compliant 2D ECMs but promotes slow MT g

189 D ECMs but promotes slow MT growth speeds in ECs cultured on compliant 3D ECMs, and these effects are

190 Increased EC insulin sensitivity leads to a proatherosclerotic imb

191 While individual EC towers are well suited to providing fluxes from the f

192 d whether resulting metabolic changes induce EC activation, is unknown.

193 ion, miR-424 and miR-503 reduced LPS induced EC sprouting, migration and tube formation.

194 Thus, tumor-induced EC-to-OSB conversion is one mechanism that leads to oste

195 Wnt5a inhibits CCN1 gene expression, induces EC proliferation and increases hypersprouting.

196 er and describe one solution: an inexpensive EC system that could help by making spatial replication

197 nt stem cell-derived endothelial cells (iPSC-ECs) from three families with unaffected mutation carrie

198 (PGI2, also called prostacyclin) in Cav-1 KO EC, and this PGI2 increase appeared to stimulate cAMP/PK

199 somal proteins that are involved in lal(-/-) EC dysfunctions.

200 Rab7 inhibition reversed lal(-/-) EC dysfunctions, including decreasing their enhanced mig

201 Here we report that lal(-/-) ECs facilitated in vivo tumor angiogenesis, growth, and

202 longitudinal analyses, higher HEI and lower EC scores were observed in Hispanic compared with white

203 itionally, jagged-2 expressed in bone marrow ECs regulated HSPC cell cycle and quiescence during rege

204 Electro-chromic materials (EC) are a new class of electronically reconfigurable thi

205 Mature ECs lack EGF receptors and are refractory to growth sign

206 HTT or endothelial cells conditional medium (EC-CM).

207 t electrochemical-scanning probe microscope (EC-SPM) is presented.

208 Microvascular ECs instruct neighboring cells in their organ-specific v

209 Human dermal microvascular ECs (HDMECs) treated with TLR3 [Poly(I:C)], TLR4 (LPS),

210 pecifically expressed by the human and mouse EC cells.

211 man EC cells is distinct from that in murine EC cell lines: the histone methyltransferase SETDB1 is r

212 cation occurs exclusively in EBs and newborn ECs that inherit EGFR and active MAPK from fast-dividing

213 Our work demonstrates that nontransformed ECs respond differently to excess centrosomes than do mo

214 proliferation and migration in VSMCs but not EC.

215 ow shear stress induces dedifferentiation of EC through a process termed endothelial-to-mesenchymal t

216 ta lay the groundwork for the exploration of EC therapies that can serve as adjuvant modalities to en

217 utive Akt signalling increases expression of EC morphogenesis genes, including Sox17, shifts the geno

218 However, the molecular mechanism of EC cell mechanotransduction is unknown.

219 lso play important roles in the prognosis of EC patients, has not been extensively studied.

220 ignificant gap in the metabolomic studies of EC.

221 factor-alpha (TNF-alpha) the supernatants of EC cultures were subjected to differential centrifugatio

222 teractions in the enhanced susceptibility of EC HIV-1-infected cells to ADCC responses.IMPORTANCE Att

223 th concerns associated with long-term use of EC in relatively young users who did not also smoke toba

224 ion, and inhibited the angiogenic ability of ECs both in vitro and ex vivo In summary, this study pro

225 from LAL deficiency enhances the ability of ECs to stimulate tumor cell proliferation and metastasis

226 carbons in the tricyclic acid (TCA) cycle of ECs and contributes to lipid biosynthesis via reductive

227 ulating pausing, which increases the flux of ECs into the termination pathway.

228 for blood flow; however, the involvement of ECs in the process of triple-negative breast cancer (TNB

229 family member and is localized in nuclei of ECs.

230 ced migratory and proliferative potential of ECs during the initiation of inflammation.

231 After stimulation of ECs with tumor-necrosis factor-alpha (TNF-alpha) the sup

232 -cranial injury or extracranial injury only (EC) (n = 10), 92 inflammation-associated proteins in ser

233 itatory post-synaptic currents (mEPSCs) onto EC layer III pyramidal neurons, suggesting that these ch

234 tinal symptoms with mycophenolate mofetil or EC-MPS in combination with Tac and cyclosporin, but this

235 lic and cytoskeleton changes in target organ ECs in gene array and proteomic analyses.

236 T mutants and in HHT patient blood outgrowth ECs.

237 Exposure of P1 ECs to MPs shed from senescent P3 cells or circulating M

238 rms a complex with VEC and its phosphatases, EC-specific protein tyrosine phosphatase (VE-PTP) and Sr

239 coronary syndrome (ACS) to promote premature EC aging and thrombogenicity.

240 revealed a twofold increase in proliferative ECs and a 1.35-fold increase in proliferative alpha-smoo

241 entified in PDX-conditioned medium, promoted EC-to-OSB conversion of 2H11 endothelial cells.

242 Prosenescent MPs promoted EC thrombogenicity through tissue factor upregulation, s

243 al monolayers to macromolecules by promoting EC proliferation and migration.

244 ssary to promote angiogenesis and to protect ECs against hydrogen peroxide-induced oxidative stress.

245 Moreover, Flunarizine protected ECs from TNFalpha-induced increase in Angpt-2 transcript

246 Patients receiving EC-D reported significantly more stomatitis, myalgia or

247 x-enhanced electrochemical capacitors (redox ECs) have, however, performed poorly due to cross-diffus

248 the inherent cross-diffusion issue of redox ECs and has the added benefit of greatly stabilizing the

249 The Commission Regulation (EC) Regulation N.

250 s where we use NRS and electrochemical SERS (EC-SERS) to detect IV therapy analytes within their clin

251 el of cellular filamentous (F) actin, slowed EC migration and proliferation, and inhibited the angiog

252 SMOC deletion constructs we found that SMOC-EC, lacking the extracellular calcium binding (EC) domai

253 The SMOC-EC domain bound HSPGs with a similar affinity to BMP2 an

254 main, inhibited BMP2 signaling, whereas SMOC-EC (EC domain only) enhanced BMP2 signaling.

255 (Tac), enteric-coated mycophenolate sodium (EC-MPS) and sirolimus (SRL) in oral dosage forms was wel

256 Stabilized ECs, expressed recombinantly as VLPs, could be ideal can

257 In this study, we selected thermally stable ECs of type 1 PV (PV-1).

258 or-stimulatory myeloid cells, and suppressed EC-mediated stimulation of in vitro tumor cell transmigr

259 es, there is concern as to whether long-term ECs use may cause risks to human health.

260 as a result, to lower genetic diversity than EC.

261 Collectively, these data demonstrate that EC-derived jagged-2 activates Notch2 signaling in HSPCs

262 Here, we found that EC cells in mouse primary cultures fired spontaneous bur

263 Here we report that EC lacking Cav-1 exhibit impaired LD formation.

264 We show that EC cells express specific chemosensory receptors, are el

265 Here, we demonstrate that ECs-when mixed with TNBC cells-could increase TNBC cell

266 We therefore hypothesize that ECs can directly activate basophils via cell-to-cell int

267 Taken together, our results suggest that ECs enhance EMT-induced TNBC cell metastasis via PAI-1 a

268 The EC cell model released 5-HT in response to stretch, and

269 athway of cytokine-mediated signaling at the EC interface, and we propose that this is a mechanism fo

270 g blank wheat and wheat spiked either at the EC regulated levels (100microg/kg for ZEA and T-2 toxin)

271 PGA2 enhanced the EC barrier and protected against barrier dysfunction cau

272 the observation period from baseline in the EC users or between EC users and control subjects in any

273 FS was 87.9% (95% CI, 85.6% to 89.8%) in the EC-D arm and 88.3% (95% CI, 86.1% to 90.1%) in the DC ar

274 In addition, in the EC-H2O2 system, Mn(II) removal efficiency increased as p

275 h domain 3) is an essential component of the EC coupling apparatus and a mutation in human STAC3 caus

276 or ZEA and T-2 toxin) or at one third of the EC level (for DON: 400microg/kg).

277 gets to ameliorate and, perhaps, reverse the EC plexiform phenotype in severe human PAH.

278 We conclude that the EC cells in the human and mouse small bowel GI epitheliu

279 (EC)/LS DESI MS, when 0 V was applied to the EC cell Tyr ion signal was detected only at low pH (2).

280 s induced the entry of Ca(2+) in ECs via the EC-expressed TRPM2.

281 ochrome B (phyB) at multiple sites where the EC is bound provides a mechanism for integrating environ

282 charide challenge in adult mice in which the EC expressed TRPM2 is conditionally deleted (Trpm2(iDelt

283 et of cortical synaptic terminals within the EC and in this way, constrain non-action potential-depen

284 The ECs are antigenically stable at temperatures above the c

285 how that some capsid mutations stabilize the ECs and could be applied as candidates to synthesize sta

286 Furthermore, silencing of ADAR2 in three EC cell lines resulted in a decreased proliferation rate

287 tors specify non-vascular, amniotic cells to EC-like cells, whereas Sox17 expression is required to c

288 or AP activation that in turn contributed to EC damage.

289 ) groups, respectively, resulting in a total EC loss of 16.6% and 21.5% from 6 months to 10 years pos

290 ecombinant AAV serotype 1 (rAAV1) transduces ECs of pathologic vessels, and that editing of genomic V

291 (MAECs) from these mice, and human umbilical ECs (HUVEC) treated with Kindlin-2 siRNA showed enhanced

292 nstability in tumor cells, how untransformed ECs respond to excess centrosomes is poorly understood.

293 HSPC/EC) coculture system as well as in vivo EC infusions following myelosuppressive injury in mice t

294 ectively (p < 0.001 for both comparisons vs. EC aspirin; p = 0.30 for comparison between plain aspiri

295 gs highlight an important mechanism by which EC contribute to atherogenesis under hyperlipidemic cond

296 fects of TL alterations on the rate at which ECs terminate from effects of the TL on the nucleotide a

297 While ECs can take up asparagine, silencing asparagine synthet

298 PMN interaction with ECs induced the entry of Ca(2+) in ECs via the EC-expres

299 on of growth persistence specifically within EC branched protrusions.

300 munoreactivity localized specifically within EC cells of human and mouse small bowel epithelium.