ライフサイエンスコーパス: ECG

1 ECG analysis was conducted using a novel, proprietary T

2 ECG analysis was conducted using a novel, proprietary T-

3 ECG and 24-hour Holter monitoring were performed biweekl

4 ECG and echocardiography were used to evaluate possible

5 ECG imaging could differentiate between BrS and right bu

6 ECG metrics were independent risk markers for cardiovasc

7 ECG pad was successful in 50 of 52 (96%) patients.

8 ECG T-wave alternans (ECG ALT) and Ca2+ transient altern

9 ECG-derived complexity parameters were determined from a

10 ECG-gated MDCT seems to be currently a method of choice

11 ECGs were collected at baseline, after reperfusion, and

12 ECGs with LPF-VT were also collected from patients who u

13 e expected from patients displaying a type 1 ECG pattern (P<0.001).

14 the root mean square error sum across all 12 ECG leads (E12).

15 Overall, 183 ECGs of LPF-VT were compared with 61 ECGs showing RBBB a

16 al function by using measurements in 289,297 ECGs from 62,974 individuals.

17 er a median follow-up of 5 months (3.8-5.3), ECG remained normal despite flecainide.

18 ll, 183 ECGs of LPF-VT were compared with 61 ECGs showing RBBB and LAHB.

19 rall, 1,072 (21.8%) athletes had an abnormal ECG on the basis of 2010 ESC recommendations; 11.2% requ

20 lassification are two main tasks in abnormal ECG beat recognition.

21 None of the abnormal ECGs recorded in 21 patients were indicative of acute my

22 Furthermore, adding ECG metrics to the model adjusted for standard risk fact

23 ECG T-wave alternans (ECG ALT) and Ca2+ transient alternans (Ca2+ALT) were ind

24 follow-up, only 2.6% and 9.7% had ambulatory ECG monitoring in the 7 days and 12 months post-stroke,

25 ICDs were interrogated and ambulatory ECGs monitored for NSVT episodes, with associations betw

26 PEX group were more likely to demonstrate an ECG abnormality than in the non-PEX group (odds ratio, 1

27 tio (TS/A) was used as starting point for an ECG-based [K(+)] estimate (KECG).

28 Retrospective review of an ECG database at a single pediatric institution.

29 The P wave on an ECG is a measure of atrial electric function, and its ch

30 Conclusions and Relevance: An ECG risk score based on age, sex, heart rate, frontal T

31 possible acute coronary syndrome in whom an ECG and hs-cTnT measurements were obtained and AMI outco

32 he relationship between visual behaviour and ECG interpretation accuracy, providing information that

33 d approach that incorporated contrast CT and ECG-gated PET.

34 Classical analysis of EEG and ECG recordings separately showed significantly decreased

35 Novel analysis of the EEG and ECG together revealed a significant reduction in EEG-ECG

36 ated with history, physical examination, and ECG (interpreted with the 2010 ESC recommendations).

37 When the FRS and ECG scores were combined in a single model, the C statis

38 the association between measured height and ECG parameters.

39 ysician's assessments of patient history and ECG were collected.

40 sion with electrocardiographical imaging and ECG recordings.

41 ich synchronously measured cuff pressure and ECG.

42 Using coregistration of eye recording and ECG in humans, we tested the hypothesis that microsaccad

43 Ventilation (Ve), renal SNA (RSNA) and ECG were measured at rest and during CBC activation in s

44 diverse associations between AF variants and ECG measurements suggest fundamentally different categor

45 pants (35%) with designated variants had any ECG or arrhythmia phenotype, and only 2 had corrected QT

46 Incident SMI was defined as ECG evidence of MI without clinically documented MI (CMI

47 ndations for ECG interpretation in athletes, ECG standards have evolved quickly over the last decade;

48 The current status of automated ECG interpretation is reviewed, with suggestions for imp

49 rdiac workup, including ECG, signal averaged ECG, exercise testing, cardiac imaging, Holter-monitorin

50 ), exercise ECG (n=304), and signal-averaged ECG (n=118) when performed.

51 We sought to correlate the signal-averaged ECG (SAECG) with the endocardial scar characteristics in

52 atients, 36.9% had Q waves on their baseline ECG.

53 of whom 3575 (24%) had AF on their baseline ECG.

54 efforts have employed the time delay between ECG and finger photoplethysmography (PPG) waveforms as a

55 dels were used to assess differences between ECG and cuff pressure timings and to investigate the eff

56 f prior myocardial infarction underwent both ECG and cardiac magnetic resonance imaging.

57 irmed elimination of abnormal substrate, BrS ECG pattern, and ventricular tachycardia/ventricular fib

58 ysiologic basis of the ST-segment in the BrS-ECG with data from various epicardial and endocardial ri

59 A type I Brugada ECG was never noticed.

60 stimulation and spontaneously as assessed by ECG telemetry after isoproterenol injection.

61 - 13.1 years) with spontaneous type 1 BrS by ECG parameters but with no history of cardiac arrest (in

62 e ratio were measured on images generated by ECG gating from pads and standard leads.

63 rategy using left ventricular hypertrophy by ECG, coronary artery calcium, N-terminal pro B-type natr

64 asurement of left ventricular hypertrophy by ECG, coronary artery calcium, N-terminal pro B-type natr

65 Imaging of capillary ECG in human labial mucosa was achieved by the use of a

66 anisms that contribute to the characteristic ECG pattern of Brugada syndrome (BrS) are still debated.

67 n of Diseases, Ninth Revision (ICD-9) codes, ECG data, and manual EMR review.

68 of clinical notes, structured billing codes, ECG reports, and procedure codes, we identified 1056 AF

69 We evaluated whether a panel of common ECG parameters are independent predictors of mortality r

70 These data suggest common ECG metrics are independent risk factors for cardiovascu

71 Contemporary ECG interpretation criteria decrease costs for de novo s

72 Two of three mutant mice under continuous ECG telemetry recording experienced death, with severe b

73 d Patient Outcomes (PLATO) trial, continuous ECGs were performed during the first 7 days after ACS (n

74 Conversely, ECG telemetry in heart-specific phosphodiesterase 2-tran

75 Treatment with ALLO significantly decreased ECG ALT (-77+/-9%, P<0.05) and Ca2+ ALT (-56+/-7%, P<0.0

76 positioner held four ECG leads and detected ECG signals without requiring shaving, adhesive, or remo

77 RV/LV structural disease and newly developed ECG TFC.

78 The overall cost of the devices, ECG interpretation, and patient management were captured

79 METHODS AND Digital ECGs from 328 638 primary care patients were collected d

80 effective method to extract low-dimensional ECG beat feature vectors.

81 The presence of AF on baseline or discharge ECG was a predictor of 1-year mortality (adjusted HR=2.1

82 in 1879 patients with baseline and discharge ECGs who underwent transcatheter aortic valve replacemen

83 and incorrect interpretation groups for each ECG.

84 The top 3 discriminating features in each ECG lead were determined and the lead with the best disc

85 ther revealed a significant reduction in EEG-ECG association in Kcna1-/- mice compared with wild type

86 The degree of EEG-ECG association was also proportional to the survival ra

87 th patient history and an electrocardiogram (ECG) (the extended algorithm) for predicting 30-day majo

88 12-lead smartphone-based electrocardiogram (ECG) acquisition and monitoring system (called "cvrPhone

89 G), Electromyogram (EMG), Electrocardiogram (ECG) and parameters along with other symptoms.

90 essed the resting 12-lead electrocardiogram (ECG) as a screening test in intermediate risk population

91 The standard 12-lead electrocardiogram (ECG) provides a method for non-invasive identification o

92 ties on a resting 12-lead electrocardiogram (ECG).

93 crog/L) and a nonischemic electrocardiogram (ECG) to rule out AMI in adults presenting to the emergen

94 eart surface based on the electrocardiogram (ECG) data from the distributed sensor network placed on

95 ect interpretation of the electrocardiogram (ECG), facilitating health care decision making and reduc

96 n ([K(+)]) influences the electrocardiogram (ECG), particularly T-wave morphology.

97 esults to features of the electrocardiogram (ECG).

98 of the QRS complex on the electrocardiogram (ECG).

99 nts affect AF risk, using electrocardiogram (ECG) measurements.

100 Interpretation of electrocardiograms (ECGs) is a complex task involving visual inspection.

101 ibrillation diagnosed by electrocardiograph (ECG) during routine Framingham examinations, age-adjuste

102 ctural progression and electrocardiographic (ECG) changes in patients with ARVD/C.

103 ial repolarisation and electrocardiographic (ECG) QT interval, associated with increased age-dependen

104 Although continuous electrocardiographic (ECG) monitoring is ubiquitous in hospitals, monitoring p

105 Current electrocardiographic (ECG) criteria for the diagnosis of left ventricular hype

106 (TV) from analysis of electrocardiographic (ECG) signals only.

107 Arrhythmia or electrocardiographic (ECG) phenotypes defined by International Classification

108 and scar with surface electrocardiographic (ECG) parameters in individuals free of prior coronary he

109 ons are an obstacle to electrocardiographic (ECG) screening of young athletes for cardiac disease.

110 morbidity (defined by electrocardiographic [ECG] abnormalities).

111 cephalography (EEG) and electrocardiography (ECG) recordings.

112 clinical findings, and electrocardiography (ECG).

113 = 84) were evaluated by electrocardiography (ECG), Holter monitoring, late-enhancement cardiac magnet

114 Importance: Electrocardiography (ECG) may detect subclinical cardiovascular disease (CVD)

115 ave inversion (ATWI) on electrocardiography (ECG) in young white adults raises the possibility of car

116 Women underwent electrocardiography (ECG).

117 onomic dysfunction with electrocardiography (ECG)-based measures in MetS subjects.

118 ical role were shown 11 commonly encountered ECGs on a computer screen.

119 Contrast-enhanced ECG-gated PET-CT permitted localization of 18F-fluoride

120 EEG, EOG, ECG and NIRS signals have been measured during a simulat

121 were investigated with clinical examination, ECG, echocardiography, exercise testing, 24h Holter ECG,

122 from ajmaline provocation (n=332), exercise ECG (n=304), and signal-averaged ECG (n=118) when perfor

123 dard functional testing strategies (exercise ECG, stress nuclear methods, or stress echocardiography)

124 r-reading and confirmation by an experienced ECG reader are essential and are repeatedly recommended

125 ch encompasses familial and genetic factors, ECG abnormalities, arrhythmias, and structural/functiona

126 Five ECG markers of global electric heterogeneity (GEH; sum a

127 cular septal sharpness (1/slope) was 165 for ECG pad and 152 for standard leads (P = .3).

128 nt represents an international consensus for ECG interpretation in athletes and provides expert opini

129 an Society of Cardiology recommendations for ECG interpretation in athletes, ECG standards have evolv

130 2010 European consensus recommendations for ECG interpretation in young athletes state that ATWI bey

131 The qualitative image quality score for ECG pad (3.9 +/- 0.19) was comparable to ECG leads (3.8

132 ington, to update contemporary standards for ECG interpretation in athletes.

133 can Heart Association practice standards for ECG monitoring on nurses' knowledge, quality of care, an

134 pliant study, a silicon positioner held four ECG leads and detected ECG signals without requiring sha

135 er systolic blood pressure and more frequent ECG abnormalities but not with higher blood glucose, ser

136 rate compared with RR intervals derived from ECG.

137 5-amine (MSNBA) and (-)-epicatechin-gallate (ECG).

138 ty, and drug use were collected, and general ECG variables (heart rate (HR), PQ and QRS intervals) as

139 ents was derived based on computer-generated ECG data, including frontal P, R, and T axes; heart rate

140 scular fragility and endothelial glycocalyx (ECG) in images of the healthy gingival crevice (GC).

141 res of the hypertrophic cardiomyopathy (HCM) ECG make it a challenge for subcutaneous implantable car

142 hocardiography, exercise testing, 24h Holter ECG, and cardiac magnetic resonance.

143 Follow-up included 3-day Holter ECG recordings and office visits at 3, 6, and 12 months.

144 F events detected via continuous in-hospital ECG/telemetry monitoring.

145 However, ECG screening costs have never been systematically asses

146 h spontaneous or drug-induced Brugada type I ECG and no symptoms at our institution were considered e

147 risk factor for sudden death underwent S-ICD ECG screening at rest and on exercise.

148 rest and on exercise to inform optimal S-ICD ECG vector development.

149 On days 10-62 after immunization, ECGs were recorded and blood was sampled for the detecti

150 ile mdx:utr mice displayed markedly improved ECG scores.

151 tes should be knowledgeable and competent in ECG interpretation in athletes.

152 till persist, despite ongoing improvement in ECG algorithms.

153 it led to prolongation of the PR interval in ECG recordings in rodents.

154 nation by radiofrequency ablation results in ECG normalization and VT/VF noninducibility.

155 nderwent extensive cardiac workup, including ECG, signal averaged ECG, exercise testing, cardiac imag

156 Initial ECG findings are not reliable in detecting patients with

157 ion by 7-day Holter monitoring; intermittent ECG event monitoring was also undertaken after ablation

158 lthcare practitioners (n = 31) who interpret ECGs as part of their clinical role were shown 11 common

159 There was a decrease in intracoronary ECG ST-elevation during RCA occlusion from baseline to f

160 ssel patency, the quantitative intracoronary ECG ST-segment elevation, and angina pectoris during the

161 In this large ECG study, individuals with preexcitation had higher haz

162 Use of strict LBBB ECG criteria was not independently associated with outco

163 mptom-limited cycle ergometry during 12-lead ECG monitoring.

164 was directly measured, and a resting 12-lead ECG obtained under standardized conditions.

165 uring in-vivo swine studies (n = 6), 12-lead ECG signals were recorded at baseline and following coro

166 Clinical data, 12-lead ECG, 12-hour Holter recordings, coronary angiography, an

167 were determined from a baseline 10-s 12-lead ECG.

168 tionnaire, physical examination, and 12-lead ECG.

169 al infarction diagnosed on paramedic 12-lead ECG.

170 ach allows its application to any multi-lead ECG system.

171 ting in the morning with ambulatory one-lead ECG monitors.

172 ood pressure monitor (ABPM) with single lead ECG.

173 ity parameters derived from standard 12-lead ECGs for AF termination and long-term success of cathete

174 We analysed 12-lead ECGs from 45 haemodialysis and 12 LQT2 patients.

175 easured from high-resolution digital 12-lead ECGs independently predicts ventricular tachyarrhythmias

176 We analyzed 12-lead ECGs of 401 first-degree relatives of individuals who ha

177 ltage criteria derived from standard 12-lead ECGs recorded at baseline and biannually were compared b

178 High-resolution (1024 Hz) digital 12-lead ECGs were recorded during intrinsic rhythm.

179 ing VMHDVCG extracted from intra-MRI 12-lead ECGs, providing a means to enhance patient monitoring du

180 G azimuth) were measured on standard 12-lead ECGs.

181 easured at baseline (1987-1989) from 12-lead ECGs.

182 edline search was used to identify or locate ECG tracings from patients with LPF-VTs.

183 Currently accepted LVH ECG criteria such as Cornell voltage and Sokolow-Lyon we

184 ether with pseudo-body surface potential map ECGs in 2 of them.

185 The mean ECG signal qualitative scores were also comparable (pad

186 In MI, ECG ALT (2.32+/-0.41%) and Ca2+ ALT (22.3+/-4.5%) were s

187 eed exists for physician education in modern ECG interpretation that distinguishes normal physiologic

188 r data that are commonly available on modern ECGs may offer independent prognostic information that i

189 resent study was to define the morphological ECG characteristics of LPF-VT and attempt to differentia

190 The morphological ECG characteristics of LPF-VT were defined, and a high a

191 Most ECG measures were stronger markers of risk for cardiovas

192 Implementation of new ECG knowledge is also important.

193 n was not associated with development of new ECG TFC.

194 additional criteria (ischemic symptoms, new ECG changes, or imaging evidence of loss of viable myoca

195 e and sex, were incorporated into the NHANES ECG risk equation.

196 t left bundle branch block morphology and no ECG or echocardiographic abnormalities.

197 detection in combination with a nonischemic ECG may successfully rule out AMI in patients presenting

198 or attached to a WiFi-enabled iPod to obtain ECGs (iECGs) in ambulatory patients.

199 This study investigated the costs of ECG screening in athletes according to the 2010 European

200 op a clear guide to the proper evaluation of ECG abnormalities in athletes.

201 Loss of ECG might be part of the inflammatory process in these t

202 Using a combined model of ECG and clinical parameters, sinus rhythm at long-term f

203 The predictive performance of ECG-derived AF complexity parameters for AF termination

204 a exercise test with continuous recording of ECG and physiological measurements before a sojourn abov

205 Worldwide, millions of ECGs are recorded annually, with the majority automatica

206 nts with and without (n=90) abnormalities on ECG (median 28 ng/L in both groups).

207 ve and validate a CVD risk equation based on ECG metrics and to determine its incremental benefit in

208 neral antioxidant treatment had no effect on ECG ALT and Ca2+ ALT.

209 te coronary syndrome without ST-elevation on ECG (n=282) were randomized to the HEART Pathway or usua

210 d (P<0.001) and the corrected QT interval on ECG was prolonged (P<0.001) in HFpEF rats.

211 ber of low-risk patients (no new ischemia on ECG and hs-cTnT measurements <0.005 microg/L) and the nu

212 ype in anaesthetised mice modelling LQTS3 on ECG phenotypes before and following beta-agonist challen

213 nized guinea pigs showed QTc prolongation on ECG after developing high titers of anti-Ro Abs, which i

214 Online ECG monitoring education and strategies to change practi

215 e 2-part intervention consisted of an online ECG monitoring education program and strategies to imple

216 ion of AF termination was similar using only ECG (cross-validated mean area under the curve [AUC], 0.

217 upper limit of normal, ischemic symptoms, or ECG changes (OR, 0.63; 95% CI, 0.48-0.84).

218 ization of 2D real-time imaging with patient ECG allowed for different beats to be categorized by the

219 understanding of how practitioners perceive ECGs, and determine whether visual behaviour can indicat

220 ined criteria reduced the number of positive ECGs to 6.0% and 4.3%, respectively.

221 rrespective of their first postresuscitation ECG and to determine whether this ECG is useful to selec

222 enter, initial heart rhythm, and prehospital ECG information was performed.

223 Blood pressure, ECG, oxygen levels, heart rate, CBC, and metabolic panel

224 The proposed ECG criteria involved measuring the amplitude of the dee

225 differences in durations to apply and remove ECG pad and standard leads.

226 of such QRS peaks (QRSp) in high-resolution ECGs would predict arrhythmic events in implantable card

227 ries, left ventricular function, and resting ECG.

228 tive of the meeting was to define and revise ECG interpretation standards based on new and emerging r

229 matic, thereby making detection with routine ECG methods difficult.

230 reased R:T wave ratio in the S-ICD screening ECG (odds ratio, 4.0; confidence interval, 3.0-5.3; P<0.

231 by the increased R:T ratio on the screening ECG and lead aVF.

232 tients were instructed to record a 30-second ECG everyday between the 2 procedures using a portable m

233 anch block morphology, 13 of 27 (48%) showed ECG repolarization abnormalities, and 5 of 27 (19%) show

234 rected QT interval were the most significant ECG factors in the NHANES I cohort.

235 inion-based recommendations linking specific ECG abnormalities and the secondary evaluation for condi

236 ither AF or AFL as diagnosed by serial study ECGs or by Medicare claims data.

237 Surface ECG measurements showed less prominent J waves in Hey2(+

238 tandard catheter (0.016+/-0.019) and surface ECG (0.02+/-0.01; P<0.05).

239 voltage and shorter QRS duration at surface ECG, whereas clinically unrecognized myocardial scar is

240 orrected QT interval was measured by surface ECG.

241 quantitative analysis on the 12-lead surface ECG provides an effective, novel tool to distinguish pat

242 wave morphology in >/=11/12 leads of surface ECG at the successful ablation site, and paced intracard

243 nt elevation was calculated from the surface ECG and compared with the electrocardiographical imaging

244 S-associated cardiac events from the surface ECG were identified: left slope of T wave in lead V6 (ha

245 al and cause QTc prolongation on the surface ECG.

246 as on a standard catheter and on the surface ECG.

247 adverse event monitoring, laboratory tests, ECG, and psychiatric measures.

248 The ECG is a surface layer lining the luminal walls of blood

249 The ECG signal quality obtained from pad and leads was rated

250 Although the ECG risk equation is low cost, further research is neede

251 test was used to assess agreement among the ECG criteria against the left ventricular mass index.

252 ing from the left ventricular summit and the ECG features associated with successful ablation.

253 METHODS AND The ECG was assessed before primary PCI for the presence of

254 emonstrating that the gaze shift between the ECG leads is different between the groups making correct

255 The proposed criteria for the ECG diagnosis of LVH improved the sensitivity and overal

256 g with their first STEMI and early QW in the ECG had smaller myocardial salvage index and more extens

257 tients with ventricular preexcitation in the ECG originates from tertiary centers.

258 ce of arrhythmia, due to irregularity in the ECG signal associated with atrial activation compared to

259 slow eye movements around the R peak in the ECG.

260 ring the early phase after the R peak in the ECG.

261 During a median follow-up of 10 months, the ECG remained normal even after ajmaline in all except 2

262 an expertise limits wider application of the ECG in the care of the athlete.

263 he correlation between the morphology of the ECG signals from a 64-lead vest and the location of the

264 the action potential (and the J-wave of the ECG) as an additional important biomarker for arrhythmog

265 ed by abnormally 'short' QT intervals on the ECG and increased susceptibility to cardiac arrhythmias

266 nce at baseline of conduction defects on the ECG and left ventricular systolic dysfunction.

267 Algorithms based on the ECG signals in multiple leads (e.g. a 64-lead vest) may

268 ne diseases and with QTc prolongation on the ECG target the human ether-a-go-go-related gene (HERG) K

269 tion seen as QT interval prolongation on the ECG.

270 ately, inexperienced physicians ordering the ECG may fail to recognize interpretation mistakes and ac

271 Volunteers preferred the ECG pad and reported that it was comfortable and conveni

272 A cell-free plasma gap representing the ECG could be imaged in the superficial tissues lining th

273 red cell and endothelial wall represents the ECG.

274 me intervals, PAWP was measured gated to the ECG QRS complex to calculate the QRS-gated DPD (diastoli

275 We used the ECG to gate late-diastolic PAWP measurements.

276 Similar improvements were noted when the ECG score was added to the pooled cohort equation.

277 ard leads compared with 296 seconds with the ECG pad, and mean difference in setup time was 148 secon

278 vement data were recorded as they viewed the ECGs and attempted interpretation.

279 These ECGs were compared with ECGs of consecutive patients wit

280 uscitation ECG and to determine whether this ECG is useful to select patients with no need of ICA.

281 ansition matrices (visual shifts from and to ECG leads) of the correct and incorrect interpretation g

282 This simplified approach to ECG gating is faster to set up and more convenient and c

283 for ECG pad (3.9 +/- 0.19) was comparable to ECG leads (3.8 +/- 0.45; P = .47).

284 frequency ablation eliminated AES leading to ECG normalization and VT/VF noninducibility in all patie

285 tification and elimination of AES leading to ECG pattern normalization and VT/VF noninducibility.

286 item online test, quality of care related to ECG monitoring (N=4587 patients) by on-site observation,

287 w-up was based on daily 30-s transtelephonic ECG transmissions.

288 syndrome (BrS) is characterized by a typical ECG pattern.

289 with MetS and 226 healthy controls underwent ECG recordings of at least 4 hours starting in the morni

290 METHODS AND We used ECG measurements from the ARIC study (Atherosclerosis Ri

291 , limb (I, aVr), and precordial (V1, V2, V6) ECG leads.

292 098Ser in PLEC significantly affects various ECG measurements in the absence of AF.

293 o on images generated by cardiac gating with ECG pad was 38 +/- 12 (standard deviation) compared with

294 Two harbor porpoises, instrumented with ECG recording tags, were trained to perform 20- and 80-s

295 aphy is the initial diagnostic modality with ECG-gated CT and MRI being non-invasive imaging modaliti

296 LV diffuse fibrosis or myocardial scar with ECG parameters (QRS voltage, QRS duration, and corrected

297 6-hour 75-g oral glucose tolerance test with ECG recording and blood sampling for measurements of glu

298 In the 1270 (63%) with ECGs, corrected QT intervals were not different in varia

299 These ECGs were compared with ECGs of consecutive patients with RBBB and LAHB and no o

300 72 (group 2) having inducible VT/VF without ECG documentation at the time of symptoms.