1                                              ELAM-1 expression was induced in normal cells by ultraso

2 e endothelial leukocyte adhesion molecule 1 (ELAM-1; E-selectin) was primarily expressed in the pulmo

3 thelial cell-leukocyte adherence molecule-1 (ELAM-1), intracellular leukocyte adherence molecule-1 (I

4 t endothelial leukocyte adhesion molecule-1 (ELAM-1), the earliest marker for the atherosclerotic pla

5 cyte-EC attachment and expression of ICAM-1, ELAM-1 and VCAM-1.

6 ell surface expression of ICAM-1, VCAM-1 and ELAM-1.

7  the promoter regions of ICAM-1, VCAM-1, and ELAM-1 contain NF-kappaB binding sites and these adhesio

8 lates adhesion molecules ICAM-1, VCAM-1, and ELAM-1.

9 not other cytokine-induced molecules such as ELAM-1 or ICAM-1.

10                                           NF-ELAM-1 contributes to maximal promoter activity, but doe

11 uclear factor (NF)-kappaB and an element, NF-ELAM-1 (endothelial leukocyte adhesion molecule-1), cons

12                  We determined expression of ELAM-1 to be controlled by activation of an interleukin-

13           PLC strongly induced expression of ELAM-1, ICAM-1, and IL-8, while PFO stimulated early ICA

14  early ICAM-1 expression but did not promote ELAM-1 expression or IL-8 synthesis.

15  neutrophil chemoattractant, and E-selectin (ELAM-1) but not intercellular adhesion molecule-1 mRNAs

16 ant leukocyte adhesion molecule, E-selectin (ELAM-1, CD62E).