ライフサイエンスコーパス: EMS

1 EMS advanced life support rescuers (paramedics, prehospi

2 EMS hospital prenotification is associated with improved

3 EMS personnel care for a substantial and increasing numb

4 EMS transport was independently associated with earlier

5 EMS was used in 53.4% of patients with MI, a proportion

6 EMS-transported patients differed from self-transported

7 (68%; p = .016), and "other" (40%; p < .001) EMS call categories, but when expressed as a percent of

8 8.7%) utilized EMS, and 1,941 of these 7,098 EMS transport patients (27.4%) received a pre-hospital E

9 Fine mapping of the sfr6-1 EMS mutation proved impossible as the locus resides very

10 idence rate of severe sepsis was 3.3 per 100 EMS encounters, greater than for acute myocardial infarc

11 ction or stroke (2.3 per 100 and 2.2 per 100 EMS encounters, respectively).

12 An analysis of 1218 EMS-attended, witnessed, out-of-hospital cardiac arrests

13 ibe the genome-wide mutational profile of 17 EMS-mutagenized genomes as assessed with a bioinformatic

14 Among 407,176 EMS encounters, we identified 13,249 hospitalizations fo

15 lind effectiveness trial in 13 US cities (36 EMS agencies), from December 2006 through July 31, 2011,

16 ) location of arrest (public or private); 4) EMS response interval; 5) arrest witnessed; 6) initial s

17 The study included 44 EMS agencies transporting to 28 hospitals in 2 rural and

18 total study population to 1.6% (P-AED, 7.6%; EMS, 6.0%).

19 We screened approximately 7000 EMS-mutagenized genomes and isolated and mapped seven in

20 neous coronary intervention hospital: 11 765 EMS transported and 6502 self-transported; 5542 transfer

21 onary intervention-capable hospitals and 946 EMS agencies.

22 h severe stroke were more likely to activate EMSs.

23 After EMS mutagenesis of a well-characterized transgenic line,

24 39 of 119 patients (33%) who arrested after EMS arrival, in 73 of 363 (20%) when EMS arrival was <7

25 ns arrived at the emergency department after EMS transport, and 80% of cases were diagnosed on admiss

26 al, which represented 1.33% (50/3754) of all EMS-treated cardiac arrests.

27 A STEMI system allowing EMS to transport patients directly to a primary PCI cent

28 after implementation of MICR as an alternate EMS protocol.

29 Although EMS use is independently associated with more rapid eval

30 Organization of care among EMS and hospitals in 12 regions was associated with sign

31 of hospitalization with severe sepsis among EMS encounters, as well as pre-hospital characteristics,

32 An EMS mutagenized population, consisting of approximately

33 Here we characterized an EMS mutant, gom8, with disrupted Golgi movement and posi

34 Here, we report on GOLD36, an EMS mutant identified through a screen for partial displ

35 In an EMS mutagenesis screen, we isolated four point mutations

36 interest are first isolated by screening an EMS mutant collection generated in the miniature cultiva

37 r 10 units, 95% CI: 1.02-2.10, p = 0.04) and EMS (OR: 1.27 per 10 units, 95% CI: 1.02-1.62, p = 0.04)

38 The ABar and EMS spindles both reorient in response to Wnt signals, b

39 e to first responder arrival among P-AED and EMS was 4.88+/-2.88 minutes (P<0.001), compared with a h

40 Among 420 paired dispatches of P-AED and EMS, the mean+/-SD P-AED time from 9-1-1 call to arrival

41 munication between offsite cardiologists and EMS personnel, allowing for patient triage directly to t

42 ess to automated external defibrillators and EMS personnel who arrive during the electrical phase (i.

43 mes-1 mutants strongly enhance endoderm and EMS spindle rotation defects associated with Wnt pathway

44 The effect of AMS, EpMS, and EMS on the development of allosensitization (calculated

45 AMS, EpMS, and EMS were independently associated with the development o

46 howed linear dose-responses, whereas MMS and EMS had nonlinear curves containing a range of nonmutage

47 escuers (paramedics, prehospital nurses, and EMS physicians) who reported at least one patient contac

48 cumulation of phosphotyrosine between P2 and EMS.

49 hi 1974 data on dominance of spontaneous and EMS-induced mutations.

50 that a missense mutation in the Arabidopsis EMS-mutagenized bri1 suppressor 3 (EBS3) gene suppresses

51 res of a new case of L-tryptophan-associated EMS, along with evidence of activated transforming growt

52 resuscitation but had no AED applied before EMS arrival, and 289 (2.1%) had an AED applied before EM

53 al, and 289 (2.1%) had an AED applied before EMS arrival.

54 distinct CPR groups: no bystander CPR before EMS arrival (no bystander CPR), bystander CPR before EMS

55 ted bystander CPR), and bystander CPR before EMS arrival not requiring dispatcher instruction (bystan

56 val (no bystander CPR), bystander CPR before EMS arrival requiring dispatcher instruction (dispatcher

57 hort, 44.1% received no bystander CPR before EMS arrival, 25.7% received dispatcher-assisted bystande

58 s high as that associated with no CPR before EMS arrival.

59 al cardiac arrest were treated by PAD before EMS arrival, which represented 1.33% (50/3754) of all EM

60 ersus 4.0% when CPR was not performed before EMS arrival (P<0.001).

61 rate was 10.5% when CPR was performed before EMS arrival versus 4.0% when CPR was not performed befor

62 CPR performed before EMS arrival was associated with a 30-day survival rate a

63 ess measures reflecting coordination between EMS and hospitals had improved, including the proportion

64 anges in prognostic factors typically beyond EMS control termed "fate" factors (for example, patient

65 rends in prognostic factors typically beyond EMS control.

66 In C. elegans the 4-cell stage blastomere EMS is an endomesodermal precursor.

67 or blastomere, P2, and a ventral blastomere, EMS, specifies endoderm and orients the division axis of

68 l cell fate and division orientation in both EMS and P2.

69 (BR) signaling by phosphorylating BES1 (bri1 EMS suppressor 1) and BZR1 (brassinazole-resistant 1), t

70 BR-regulated transcription factor BES1 (BRI1-EMS SUPRESSOR 1) directly represses and physically inter

71 r components to modulate the BES1/BZR1 (BRI1-EMS SUPPRESSOR 1/BRASSINAZOLE RESISTANT 1) family of tra

72 stitutive activation of BR signaling in bri1-EMS-suppressor 1-D (bes1-D) plants enhances this respons

73 ar localizations, and/or DNA binding of BRI1-EMS suppressor 1 (BES1)/brassinazole-resistant 1 (BZR1)

74 ion factor in the BR signaling pathway, BRI1-EMS-SUPPRESSOR 1 (BES1), largely depends on its phosphor

75 life history, were substantially affected by EMS mutagenesis: the developmental time increased while

76 staining severe traumatic injury assessed by EMS.

77 no O2 but only N2 evolution was detected by EMS, indicating that the photocurrent was related to pho

78 As expected for alkylation of guanine by EMS, >99% of mutations are G/C-to-A/T transitions.

79 Programs implemented by EMS appeared to counter adverse temporal trends in progn

80 ple, patient age) and factors implemented by EMS termed "program" factors (programs of dispatcher-ass

81 -frequency phenotypic instability induced by EMS treatment is caused by a genetic mechanism.

82 s chest compressions during CPR performed by EMS providers did not result in significantly higher rat

83 ystems that include pre-hospital referral by EMS.

84 67047 injured children and adults served by EMS (1971 rural and 65076 urban) was enrolled.

85 Of the 53487 injured patients transported by EMS (17633 patients in the probability sample), 27535 we

86 ts with acute ischemic stroke transported by EMS and enrolled in Get With The Guidelines-Stroke from

87 es Network) registry who were transported by EMS from January 1, 2007, through December 31, 2007.

88 one-quarter of these patients transported by EMS receive a pre-hospital ECG.

89 Among the 53487 patients transported by EMS, a stratified probability sample of 17633 patients (

90 a-related cardiac arrest who were treated by EMS providers received continuous chest compressions (in

91 that this technology can be readily used by EMS providers to identify patients with ST-segment eleva

92 cathodes for at least 60 min was verified by EMS.

93 in the number of resuscitations withheld by EMS personnel.

94 of-hospital cardiac arrests are witnessed by EMS personnel or bystanders and whether AEDs are applied

95 Hospital arrival by EMSs was observed in 63.7% of patients.

96 en the daughters of an embryonic cell called EMS results in part from a Wnt-like signal provided by a

97 mutant lines revealed >1.8 million canonical EMS-induced mutations, affecting >95% of genes in the so

98 screen using Df(3R)ea(5022rx1) and chemical (EMS) mutagenesis from which we identified seven novel mu

99 Citywide EMS calls and ambulance responses were categorized by 91

100 Citywide EMS calls numbered 5,299 on August 14, 2003, and 5,021 o

101 gration and in novel Wnt pathways to control EMS and T cell polarities.

102 o 1 (WRM-1) also plays a role in controlling EMS division orientation.

103 hort of all nonarrest, nontrauma King County EMS encounters from 2000 to 2009 who were transported to

104 compared for P-AED and concurrently deployed EMS, and both were compared with historical EMS experien

105 regions, increasing from 45% to 57% (direct EMS; P<0.001) and 38% to 50% (transfers; P<0.001).

106 hat there are at least 700 mutations in each EMS-mutagenized Arabidopsis line and that fewer than 50,

107 Here we report ebs2 (EMS-mutagenized bri1 suppressor 2) as an allele-specific

108 he division orientation of the endomesoderm (EMS) blastomere and the endoderm fate of the posterior E

109 cides in a controlled ethylmethanesulfonate (EMS) saturation mutagenesis experiment, allowing a direc

110 s treated with either ethylmethanesulfonate (EMS), mitomycin C, mitoxantrone or doxorubicin, at thera

111 mutants by isolating ethylmethanesulfonate (EMS)-induced lethal and visible mutations that define tw

112 Two large-scale ethylmethanesulfonate (EMS) mutant populations from barley (Hordeum vulgare L.)

113 tracheal cell migration, we performed an F4 EMS mutagenesis screen to generate and characterize new

114 Survival following EMS-initiated CPR and defibrillation was 15.2% (30 of 19

115 ng at about 13% for firefighters and 22% for EMS workers during the subsequent 6 years.

116 18% for firefighters and from 12% to 22% for EMS workers, stabilizing at about 13% for firefighters a

117 esponse time, time from collapse to call for EMS, and year of event), CPR before the arrival of EMS w

118 ild type embryos, SKN-1 functions in EMS for EMS blastomere fate specification.

119 Instruction for EMS personnel in MICR, an approach that includes an init

120 nutes, compared with 7.56+/-3.60 minutes for EMS (P<0.001).

121 nfidence interval [CI], 408 to 471) than for EMS workers who had never smoked (a reduction of 267 ml;

122 year for firefighters and 40 ml per year for EMS workers.

123 tic and cytological characterization of four EMS-induced mutations in teflon (tef), a gene involved i

124 enerated 6400 pedigreed M4 mutant pools from EMS-mutagenized BTx623 seeds through single-seed descent

125 The time from EMS arrival to in-hospital fibrinolytic for 630 control

126 The median time from EMS arrival to initiation of rPA was 31 min (25th to 75t

127 opter transport and 5097 (7.3%) after ground EMS.

128 opter transport and 8161 (7.8%) after ground EMS.

129 orted via helicopters and 174,195 via ground EMS.

130 improved survival, in comparison with ground EMS.

131 patients with TBI, in comparison with ground EMS.

132 EMS, and both were compared with historical EMS experience.

133 However, EMS personnel most often arrive after the electrical pha

134 We have identified EMS-induced mutations in Drosophila Miro (dMiro), an aty

135 as a Wnt-dependent activator of endoderm in EMS and an apparently Wnt-independent repressor of the m

136 In wild type embryos, SKN-1 functions in EMS for EMS blastomere fate specification.

137 nd that GPR-1/2 distribution is symmetric in EMS, where GPR-1/2 had been proposed to function as an a

138 The blackout dramatically increased EMS and hospital activity, with unexpected increases res

139 Interventions aimed at increasing EMS activation should target populations at risk, partic

140 need for initiatives targeted at increasing EMS prenotification rates as a mechanism from improving

141 rage dominance would decline with increasing EMS concentration.

142 ription factors BRASSINOSTEROID INSENSITIVE1-EMS SUPPESSOR1 and MONOPTEROS (MP)/ AUXIN RESPONSE FACTO

143 hese contractions are regulated, we isolated EMS-induced mutations that cause males to execute prolon

144 reporter lines were ethyl methanesulfonate (EMS) mutagenized, and stable M(2) seedlings were screene

145 Ethyl methanesulfonate (EMS) mutants of tomato have already proven their utility

146 ction, we applied an ethyl methanesulfonate (EMS) screen for loci synthetically lethal with scp160 lo

147 heavily treated with ethyl methanesulfonate (EMS), approximately 12,000 lines in which the treated se

148 1 mM octanol, 0.5 mM ethyl methanesulfonate (EMS), or cytoplasmic acidification, with coupling coeffi

149 f approximately 1900 ethyl methanesulfonate (EMS)-induced mutations in 192 Arabidopsis thaliana targe

150 type, observed in an ethyl methanesulfonate (EMS)-mutagenized Caenorhabditis elegans strain.

151 re than one-third of ethyl methanesulfonate (EMS)-treated bal M(1) plants.

152 ng them with 21.2 mm ethyl methanesulfonate (EMS).

153 abidopsis IND since ethyl methanesulphonate (EMS) mutant alleles and silenced transgenic lines have v

154 quired 1.25 microg/mL MMS and 1.40 microg/mL EMS before a mutagenic effect was detected.

155 ns was 0.85 microg/mL MMS and 1.40 microg/mL EMS; point mutations required 1.25 microg/mL MMS and 1.4

156 lated from 9216 plants from the 20 and 30 mm EMS treatments and assembled into 1152 eight-plant pools

157 erval (mean difference, 26 seconds) and more EMS-witnessed arrests (18.7% vs 12.6%) with LDB.

158 idelines-Stroke data, we assessed nationwide EMS use by stroke patients.

159 e evaluated the frequency and outcome of non-EMS AED use in a community experience.

160 enotification occurred in 249 197 (67.0%) of EMS-transported patients.

161 and was not performed before the arrival of EMS in 14,869 cases (48.9%).

162 CPR was performed before the arrival of EMS in 15,512 cases (51.1%) and was not performed before

163 nd that had occurred prior to the arrival of EMS personnel.

164 nd year of event), CPR before the arrival of EMS was associated with an increased 30-day survival rat

165 ed arrest and shockable rhythm on arrival of EMS.

166 We evaluated a population-based cohort of EMS-attended adult cardiac arrests (n=7265) from 1983 th

167 We evaluated a population-based cohort of EMS-treated adult patients with cardiac arrest (n=12 591

168 dentify Ptp52F alleles among a collection of EMS-induced lethal mutations.

169 hromatin of the future posterior daughter of EMS and other dividing cells shortly after mitosis compa

170 Deficiencies of EMS proteins in humans lead to the hemolytic anemia Here

171 ring the extrinsically polarized division of EMS cells.

172 The dose of EMS induced an estimated rate of 1.2-1.5 x 10(-3) mutati

173 An epidemic of EMS in 1989 was linked to consumption of L-tryptophan th

174 d -2 GATA factors specify the entire fate of EMS, which otherwise produces two C-like mesectodermal p

175 Here we report the identification of EMS-induced mutations in 21 essential loci in the 45D-45

176 ensus statement highlights the importance of EMS systems, which employ bundles of care focusing on pr

177 n the sale of L-tryptophan, the incidence of EMS declined rapidly.

178 The median incidence of EMS-assessed severe traumatic injury per 100,000 populat

179 The median incidence of EMS-treated cardiac arrest across sites was 52.1 (interq

180 ities were associated with decreased odds of EMS use.

181 Populations of EMS-mutagenized M(2) seedlings were sprayed with twice t

182 creen of approximately 112,000 F1 progeny of EMS-treated br1 animals, we recovered 26 mutations that

183 nd point was the change in the proportion of EMS-transported patients with first medical contact to d

184 Here we analyze a set of EMS-induced mutations in the egg gene, identify the mole

185 imilar to those found in previous studies of EMS-induced mutations, as well as 2.4% indels (insertion

186 Wider use of EMS by patients with suspected MI may offer considerable

187 stering of patients within hospitals, use of EMS prenotification was independently associated with gr

188 Nevertheless, use of EMS transportation is associated with substantial reduct

189 Use of EMS was independently associated with slightly wider use

190 R, a resuscitation strategy using LDB-CPR on EMS ambulances is associated with improved survival to h

191 velopment of HLA-DR and HLA-DQ DSA, but only EMS correlated with the risk of HLA-A and -B DSA develop

192 r P-element insertion or utilizing X rays or EMS as a mutagen.

193 residue in the RING domain is altered in our EMS-mutagenized allele of nopo, suggesting that E3 ligas

194 eral loci affecting eye color imply that our EMS treatment was equivalent to approximately 100 genera

195 on, no mutants identified to date abolish P2/EMS signaling.

196 Compared with self-transported patients, EMS-transported patients had significantly shorter delay

197 adopted by participating agencies permitted EMS personnel to withhold resuscitation if the patient h

198 NARK, isolated from a Forrest M2 population (EMS-mutated soybean) were studied.

199 omere and the endoderm fate of the posterior EMS daughter cell, called E.

200 een two cells - the endomesodermal precursor EMS and the germline precursor P(2) - and both cells ali

201 pecification of the mesendodermal precursor, EMS, in the 4-cell stage embryo.

202 rior daughter of the mesendoderm progenitor (EMS cell), resulting in activation of endoderm different

203 Of the 5,958 persons who received EMS-attempted resuscitation, 1,001 (16.8%) were discharg

204 molecular lesion responsible for a recessive EMS-induced mutation affecting egg shell morphology by u

205 on myocardial infarction, yet data regarding EMS use and its association with subsequent clinical car

206 ) and conventional emergency medical rescue (EMS) responders are simultaneously deployed to possible

207 We reviewed EMS and hospital records, state death certificates, and

208 e of cardiac arrest, initial cardiac rhythm, EMS response time, time from collapse to call for EMS, a

209 apolating this greater survival from the ROC EMS population base (21 million) to the population of th

210 A large-scale EMS mutagenesis screen on the third chromosome resulted

211 al properties (electrostatic mismatch score, EMS) enables prediction of allosensitization to HLA, and

212 over included 114 emergency medical service (EMS) agencies.

213 The time from emergency medical service (EMS) arrival to administration of a fibrinolytic was com

214 tander CPR before emergency medical service (EMS) arrival.

215 : 1) citywide 911 emergency medical service (EMS) calls and ambulance responses; and 2) emergency dep

216 in a metropolitan emergency medical service (EMS) system.

217 spitals and 1253 emergency medical services (EMS) agencies in 16 regions across the United States.

218 tation; 2) a new emergency medical services (EMS) algorithm; and 3) aggressive post-resuscitation car

219 ing care between emergency medical services (EMS) and hospitals.

220 assessed by the emergency medical services (EMS) and referred for primary PCI: 822 (59.2%) were refe

221 ative efforts by emergency medical services (EMS) and were enrolled in the Cardiac Arrest Registry to

222 e the arrival of emergency medical services (EMS) and whether early CPR was correlated with survival.

223 ere sepsis among emergency medical services (EMS) encounters, relative to acute myocardial infarction

224 Emergency medical services (EMS) hospital prenotification of an incoming patient wit

225 yond traditional emergency medical services (EMS) into the community has not been fully evaluated.

226 Activation of emergency medical services (EMS) is critical for the early triage and treatment of p

227 ransmission from emergency medical services (EMS) personnel in the field to the emergency department

228 Emergency medical services (EMS) personnel often are not permitted to honor requests

229 sed by organized emergency medical services (EMS) personnel, did not have traumatic injury, and recei

230 not witnessed by emergency medical services (EMS) personnel, the rate of bystander CPR increased from

231 Emergency medical services (EMS) providers who administer advanced life support shou

232 eports logged by emergency medical services (EMS) technicians arriving with intentionally injured tra

233 eports logged by emergency medical services (EMS) technicians arriving with intentionally injured tra

234 n transported by emergency medical services (EMS).

235 rrest treated by emergency medical services (EMS).

236 served by 9-1-1 emergency medical services (EMS).

237 was witnessed by emergency-medical-services (EMS) personnel, 35% when it was witnessed by a bystander

238 nd 6.4 years for emergency-medical-services (EMS) workers.

239 at hospitals via emergency medical services (EMSs), yet since then, there have been efforts to increa

240 quantitative electrophoretic mobility shift (EMS) assay.

241 hose encoding erythrocyte membrane skeleton (EMS) proteins.

242 y in situ electrochemical mass spectroscopy (EMS).

243 Standard EMS care for cardiac arrest with an LDB-CPR device (n =

244 dia (VT/VF), compared with 9.0% for standard EMS before P-AED implementation (P=0.047).

245 a strategy in which ethylmethane sulfonate (EMS) mutagenized chromosomes were maintained as heterozy

246 ylnitrosourea (MNU), ethylmethane sulfonate (EMS), and ethylnitrosourea (ENU).

247 scopic analysis of ethyl methane sulphonate (EMS)-mutated Arabidopsis thaliana seedlings expressing G

248 ified in all three phenotypically suppressed EMS-treated bal lines investigated, indicating that the

249 ns that are sensitive (CD-1, FVB/N, 129T2 Sv/EMS, and C57Bl/10).

250 in the KA-sensitive strains FVB/N, 129T2 Sv/EMS, and CD-1 were significantly larger at 56 days posti

251 Eosinophilia-myalgia syndrome (EMS) is characterized by subacute onset of myalgias and

252 The 8p11 myeloproliferative syndrome (EMS) is associated with translocations that disrupt the

253 diac arrest before emergency medical system (EMS) arrival at Resuscitation Outcomes Consortium (ROC)

254 zona documented by emergency medical system (EMS) first-care reports to determine the incidence of ga

255 on and inhibition of emotional motor system (EMS) regions in the brain.

256 tion of the 9-1-1 Emergency Medical Systems (EMS) by patients with symptoms consistent with an acute

257 We found that EMS transport was used in only 60% of ST-segment-elevati

258 The EMS of Seattle and King County developed a voluntary Com

259 Among the EMS transport population, primary percutaneous coronary

260 Their localization patterns at the EMS/P2 cell boundary are complementary, suggesting that

261 derm and much of the mesoderm arise from the EMS cell in the four-cell C. elegans embryo.

262 that med transcripts accumulate both in the EMS cell and in the maternal germline in a SKN-1-depende

263 f POP-1/Tcf in the posterior daughter of the EMS blastomere.

264 y cue required for proper orientation of the EMS cell division axis.

265 ndoderm and orients the division axis of the EMS cell.

266 We examined use of the EMS in the United States and ascertained the factors tha

267 Nonusers of the EMS were on average younger, male, and at relatively low

268 ced rCBF in 5-HT3R containing regions of the EMS, but not in areas activated by pain.

269 r this cell expressing the C, instead of the EMS, fate.

270 ptor BRI1, resulted in identification of the EMS-mutagenized bri1 suppressor 5 (EBS5) gene that encod

271 form basic life support until arrival of the EMS.

272 ryos, the C blastomere is transformed to the EMS blastomere fate, resulting in embryonic lethality.

273 ent such asymmetric division occurs when the EMS blastomere divides to produce MS, a mesoderm precurs

274 Through EMS mutagenesis we have identified three complementation

275 KN-1 protein in the C blastomere relative to EMS is believed to be responsible for this cell expressi

276 stage for all 10 genes and was restricted to EMS-derived lineages for 7 of the 10.

277 is likely responsible for the observed C-to-EMS blastomere fate transformation.

278 Urban EMS system change from manual CPR to LDB-CPR.

279 modynamic compromise were more likely to use EMS transport.

280 han one third of stroke patients fail to use EMSs.

281 amines this loss-of-function phenotype using EMS-induced mutations that specifically alter the nau ge

282 of 7,098 of 12,097 patients (58.7%) utilized EMS, and 1,941 of these 7,098 EMS transport patients (27

283 ping after arrest in relation to the various EMS arrival times.

284 n point mutations were created in planta via EMS treatment of a soft wheat.

285 ime, including those directly presenting via EMS (50% to 55%; P<0.001) and transferred patients (44%-

286 When EMS divides, it gives rise to a mesoderm-restricted prec

287 ival was <7 minutes, in 50 of 360 (14%) when EMS arrival time was 7 to 9 minutes, and in 25 of 338 (7

288 d after EMS arrival, in 73 of 363 (20%) when EMS arrival was <7 minutes, in 50 of 360 (14%) when EMS

289 s 7 to 9 minutes, and in 25 of 338 (7%) when EMS arrival time was >9 minutes.

290 al response time of 7.64+/-3.66 minutes when EMS was the sole responder.

291 l with P-AED was compared with outcomes when EMS was the sole responder.

292 tations are caused by TE insertions, whereas EMS induces mainly point mutations, we predicted that av

293 independent patient factors associated with EMS transportation versus patient self-transportation.

294 yer status was significantly associated with EMS use.

295 esence or absence of gasping correlated with EMS arrival time.

296 n both cases, cortical WRM-1 interferes with EMS spindle rotation without affecting endoderm specific

297 Patients with EMS prenotification had shorter door-to-imaging times (2

298 patients arriving by 2 hours, patients with EMS prenotification were more likely to be treated with

299 hers, in the case of viability) treated with EMS did not reveal any significant epistasis among the i

300 Treatment with EMS frequently prevented the repeat expansion observed d