ライフサイエンスコーパス: ENT2

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1 ty of an alternative nucleoside transporter, ENT2.

2 ide transporter ENT1 and thus was designated ENT2.

3 ient expression studies with the full-length ENT2 and a 5'-truncated construct that lacks the first s

4 r newly identified crosstalk pathway between ENT2 and alveolar epithelial Adora2b in lung protection

5 Comparison of the ENT2 and HNP36 nucleotide sequences suggested that HNP36

6 y response gene HNP36 is a truncated form of ENT2 and that the full-length open reading frame of ENT2

7 in the equilibrative nucleoside transporter ENT2, and reconstitution of ENT2 into ENT2-deficient cel

8 bits genetic interactions with both ENT1 and ENT2, and that the clathrin adaptors and Sla2p together

9 east endocytic proteins (the epsins Ent1 and Ent2, and the Eps15 homolog Ede1).

10 nhibitors suppressed the expression of ENT1, ENT2, and TK1 and thus decreased (18)F-FLT PET activity.

11 orylation of known Akl1 substrates (Sla1 and Ent2) confirmed that Akl1 is hyperactive when not phosph

12 porter ENT2, and reconstitution of ENT2 into ENT2-deficient cells restores 3E10 Fv transport into cel

13 wo redundant genes encoding epsins, ENT1 and ENT2; deleting both genes is lethal.

14 Interestingly, pan1-20 ent2 Delta but not pan1-20 ent1 Delta double mutants had

15 Studies in gene-targeted mice for Ent1 or Ent2 demonstrated selective protection from liver injury

16 able to interact with the endocytic adaptor Ent2 in a CBM-dependent manner, and HCs encoded by chc1-

17 rved higher expressional levels of ENT1 than ENT2, in conjunction with repression of ENT1 and ENT2 tr

18 ve nucleoside transporters 1 and 2 (ENT1 and ENT2) inhibitory activity albeit less potent than the pr

19 side transporter ENT2, and reconstitution of ENT2 into ENT2-deficient cells restores 3E10 Fv transpor

20 ne) in tissues such as skeletal muscle where ENT2 is predominantly expressed.

21 d that the full-length open reading frame of ENT2 is required for production of a functional plasma m

22 t1 or Ent2 revealed a selective phenotype in Ent2(-/-) mice, including attenuated pulmonary edema and

23 nslated from a second start codon within the ENT2 open reading frame.

24 singly, the carboxyl-terminal portion of the ENT2 protein was nearly identical to a smaller protein i

25 nt studies in gene-targeted mice for Ent1 or Ent2 revealed a selective phenotype in Ent2(-/-) mice, i

26 However, in contrast to ENT1 and ENT2, the endogenous and green fluorescent protein-tagge

27 trated time-dependent repression of ENT1 and ENT2 transcript and protein levels during ALI.

28 , in conjunction with repression of ENT1 and ENT2 transcript and protein levels following warm ischem

29 FLT is transported into the cell by ENT1 and ENT2, where it is phosphorylated by TK1 and trapped intr

30 the clathrin-binding adaptor proteins Ent1, Ent2, Yap1801, and Yap1802, we identify a second endocyt

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