ライフサイエンスコーパス: EOG

8 (OCT), Arden ratio, and electrooculography (EOG) light peak.

9 s of walking tasks using electrooculography (EOG) synchronised with 3D motion analysis (VICON).

10 Interestingly, electroolfactogram (EOG) responses stimulated by either cAMP- or inositol 1,

11 ory field potential, the electroolfactogram (EOG).

12 ing finding is that the profile of intrinsic EOG response measured in surgically opened nose without

13 Octanal and other aldehydes induce large EOG responses in the rodent olfactory epithelium, sugges

14 mon Cl(-)/HCO(3)(-) exchanger AE2 had normal EOGs.

15 l produces changes in the electro-oculogram (EOG) similar to those caused by light, but indirect evid

16 ic examination, including electro-oculogram (EOG), short-wavelength FAF, near-infrared FAF, spectral-

17 roencephalograms (EEGs), electro-oculograms (EOGs), submental electromyogram (EMG), GG EMG (intramusc

18 D patients having normal electro-oculograms (EOGs), to examine the hypothesis that the severity of di

19 uals exhibit a reduced electro-oculographic (EOG) response to changes in light exposure and have sign

20 oretinography (ERG) and electro-oculography (EOG), and color vision testing were performed.

21 ctroretinography (ERG), electro-oculography (EOG), and Goldmann perimetry were performed.

22 retinography (ERG), and electro-oculography (EOG).

23 isual field testing and electro-oculography (EOG).

24 ents were measured with electro-oculography (EOG).

25 he timing of the slow oscillatory changes of EOG voltage.

26 Light caused small increments of EOG voltage (termed light rises), again related to the f

27 s tested there was significant inhibition of EOG function; however, over time there was at least a pa

28 om the olfactory organ [electro-olfactogram (EOG) or integrated neural activity], local field potenti

29 We performed electro-olfactogram (EOG) recordings on the double-mutant mice, NCKX4(-/-);CN

30 he effect of airflow on electro-olfactogram (EOG) responses and found that the MOE of mice can sense

31 activities measured by electro-olfactogram (EOG) under same flow conditions.

32 siological technique, electro-olfactography (EOG), was employed to determine the level of olfactory d

33 s for the measured retronasal and orthonasal EOG response at all flow rates and positions.

34 amplitude of the olfactory evoked potential (EOG) recorded inside the nose; (2) the EOG amplitude is

35 , we present electro-olfactogram recordings (EOG) demonstrating that NKCC1-deficient mice exhibit sig

36 A bilateral reduced EOG response was detected in 1 patient.

37 B1(DeltaCaM) mice displayed markedly reduced EOG amplitude accompanied by alterations in other respon

38 The airflow-sensitive EOG response in the MOE was attenuated when cAMP was inc

39 Standard EOGs were recorded after oral administration of alcohol

40 The EOG results were abnormal in all patients.

41 tial (EOG) recorded inside the nose; (2) the EOG amplitude is correlated with the amplitude of the ol

42 The slower decrease in the EOG voltage was evident in patients with small fields an

43 gly suggest that the light peak phase of the EOG is temporally related to a decreased OSEL in normal

44 rt the suggestion that the light peak of the EOG may not be generated solely by hBest1.

45 However, because the light peak of the EOG of some patients with the DeltaI295, D312N, E119Q, a

46 nge in OS length corresponded to that of the EOG waveform.

47 combination with an absent light rise on the EOG that outweighs the full-field ERG abnormalities, whi

48 and Cl(-)/HCO(3)(-) exchangers, reduced the EOG in epithelia from both wild-type and NKCC1 knockout

49 NKCC cotransport with bumetanide reduced the EOG in epithelia from wild-type mice but had no effect i

50 ubjects, and to compare these changes to the EOG.

51 d for androstenone and, as they do so, their EOG and OERP increase.

52 ensitivity, as determined by their unchanged EOG response amplitude.