ライフサイエンスコーパス: ER retention signal

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1 minal STEL sequence, which is an inefficient ER retention signal.

2 sequence that is ended with SSEL, a putative ER retention signal.

3 e esterase active site and the COOH-terminal ER retention signal.

4 n of a two amino acid endoplasmic reticulum (ER) retention signal.

5 , together with the unique combination of an ER-retention signal, a target sequence for SPCs in the r

6 nserved arginine-based motif functions as an ER retention signal and a lumenal leucine motif is requi

7 The ATA27 protein is predicted to have an ER retention signal and an acidic isoelectric point, sug

8 chimera that contained a C-terminal dilysine ER retention signal and entered the recycling pathway.

9 e active site, the CRP binding site, and the ER retention signal are functionally distinct.

10 phosphorylation and the presence of putative ER retention signals are required for the PKA-mediated i

11 Signal sequences and endoplasmic reticulum (ER) retention signals are known to play central roles in

12 I-II loop, masking an endoplasmic reticulum (ER) retention signal as the dominant mechanism for Ca(V)

13 by the presence of a conserved S. cerevisiae ER retention signal at its C terminus, was confirmed by

14 Interestingly M-Vpu bearing an ER retention signal at the C terminus localizes similarl

15 lasmic reticulum (ER) via the addition of an ER retention signal at the carboxyl terminus of UL20p fo

16 bstituting this sequence with KDEL, a robust ER retention signal, concentrated COX-2 in the ER where

17 DeltaE19, an E19 mutant lacking the ER-retention signal, delays maturation of class I molecu

18 g their cytoplasmic N-terminal tails with an ER retention signal derived from the cytoplasmic domain

19 ory function of TGD, whereas addition of the ER retention signal did not alter its function.

20 n of the NR1 C-terminal serine 897 (masks an ER retention signal), followed by a PKC-dependent phosph

21 2B and, M4 of NR1, are necessary for masking ER retention signals found in M3.

22 n glutamic and aspartic acid with a putative ER retention signal (HDEL) at the C terminus.

23 on, reminiscent of an endoplasmic reticulum (ER) retention signal; however, Spn4.1 and neuroserpin ha

24 Here we report the identification of a novel ER retention signal in the alternatively spliced C-termi

25 ult in C-terminal truncation and loss of the ER retention signal in the mutant protein.

26 BR1 and GBR2 masks an endoplasmic reticulum (ER) retention signal in the cytoplasmic region of GBR1 a

27 ts contain a dominant endoplasmic reticulum (ER) retention signal in their pore region, preventing su

28 the presence of novel endoplasmic reticulum (ER) retention signals in SUR1 and KIR6.2; incompletely a

29 Cis-inhibition is compromised when an ER retention signal is added to Serrate, or when the lev

30 C terminus of the NR2B subunit show that an ER retention signal is also present in the NR2B subunit.

31 focal microscopic imaging indicated that the ER retention signal is likely present within the C-termi

32 ting cells with brefeldin A or by fusing the ER retention signal KDEL to S1P obviates the SCAP requir

33 R with or without the endoplasmic reticulum (ER) retention signal (KDEL), using either a plasmid (p3S

34 MANF ("RTDL"), which resemble the canonical ER retention signal ("KDEL"), to study MANF regulation i

35 Having an inefficient ER retention signal leads to sluggish Golgi to ER transi

36 Suppressor mutations in a putative dibasic ER retention signal, located within the cytoplasmic C te

37 s harboring either an endoplasmic reticulum (ER) retention signal (NCT-ER) or a trans-Golgi network (

38 L4-3) was fused to an endoplasmic reticulum (ER) retention signal (NefKKXX).

39 The ER retention signal of GBR1 is not part of the core coil

40 ction is required to inactivate the adjacent ER retention signal of GBR1.

41 e expression in concert with other described ER retention signals of FcepsilonRI-alpha.

42 In assembled complexes, ER retention signals on the individual subunits must be

43 e C1 cassette has been identified as a major ER retention signal present in NR1 subunits, and the sur

44 ) include a potential endoplasmic reticulum (ER) retention signal, RGR, which when mutated to LGL (HE

45 We identified an ER retention signal (RRR) in the C1 cassette of NR1, whi

46 subunits fail to mature because of an "RXR" ER retention signal specific to the 1b N terminus of the

47 wild type TMEM97, but not TMEM97 missing an ER-retention signal suggesting that TMEM97 contributes t

48 the CAV1 P158 protein contains a functional ER-retention signal that inhibits ER exit and caveolae f

49 , in addition to cytosolic and transmembrane ER retention signals, the FcepsilonRI alpha-chain signal

50 function, we added an endoplasmic reticulum (ER) retention signal to TGD and, separately, deleted the

51 investigation revealed that mutation of the ER retention signal was able to partially restore surfac

52 a truncated eroA, missing the putative HEEL ER-retention signal was unable to complement as well as

53 -terminal 104 amino acids is to mask the RGR ER retention signal, which becomes exposed when mutation

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