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1 versed shunting and associated cyanosis (ie, Eisenmenger syndrome).
2 ts with complex ACHD, Fontan physiology, and Eisenmenger syndrome.
3 graphs and computed tomography (CT) scans in Eisenmenger syndrome.
4 cardiac surgery (NCS) in patients (pts) with Eisenmenger syndrome.
5 h a high mortality rate in patients with the Eisenmenger syndrome.
9 athologic lung sections from 5 patients with Eisenmenger syndrome and from 3 patients with acyanotic
11 lesions on chest radiographs and CT scans in Eisenmenger syndrome appear to be correlated histologica
13 he general population, whereas patients with Eisenmenger syndrome, complex congenital heart disease,
16 re assessed in 181 consecutive patients with Eisenmenger syndrome, excluding those with complex conge
17 in the context of congenital heart disease (Eisenmenger syndrome) improves pulmonary hemodynamics, f
19 history, complications, and treatment of the Eisenmenger syndrome in adults were selected, and descri
21 ungs from control subjects and patients with Eisenmenger syndrome, IPAH lungs contained perivascular
25 D and especially its extreme expression, the Eisenmenger syndrome, is a chronic disease with slow pro
26 ar lesions were identified histologically in Eisenmenger syndrome: malformed, dilated, muscular arter
27 aphs, and clinical data were reviewed for 24 Eisenmenger syndrome patients subdivided into those with
28 s the numerous complications associated with Eisenmenger syndrome, representing a multisystem disorde
30 rity on chest radiographs was more common in Eisenmenger syndrome than acyanotic PAH, but differences
31 sion in a contemporary cohort of adults with Eisenmenger syndrome was associated with a lower risk of
32 tterns were characterized in adults with the Eisenmenger syndrome when two ventricles with a ventricu
34 disease, congenital diaphragmatic hernia and Eisenmenger syndrome who may be candidates for treatment
35 systemic collaterals were more prevalent in Eisenmenger syndrome, with severity greater in posttricu
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