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1 le in the protective immune response against Encephalitozoon cuniculi.
2 2-7 complex from the microsporidian parasite Encephalitozoon cuniculi.
3 structural components of these organelles in Encephalitozoon cuniculi.
4 m Encephalitozoon intestinalis (2.3 Mbp) and Encephalitozoon cuniculi (2.9 Mbp) revealed massive gene
5                                              Encephalitozoon cuniculi, a microsporidial species most
6 (delta) T cells during murine infection with Encephalitozoon cuniculi, an intracellular parasite, was
7             In this study, we show that both Encephalitozoon cuniculi and Encephalitozoon intestinali
8 e genomes are strikingly similar to those of Encephalitozoon cuniculi and Encephalitozoon intestinali
9  assembly components for the microsporidians Encephalitozoon cuniculi and Trachipleistophora hominis.
10 c acid, a TrpRS from the eukaryotic pathogen Encephalitozoon cuniculi bound to tryptophan, a HisRS fr
11 rystal structure of the N-terminal domain of Encephalitozoon cuniculi Cdc27 (Cdc27(Nterm)), revealing
12       The CTD of the microsporidian parasite Encephalitozoon cuniculi consists of 15 heptad repeats,
13                                              Encephalitozoon cuniculi continues to pose a problem for
14 al antibody, termed 3B6, strongly recognized Encephalitozoon cuniculi, E. hellem, and E. intestinalis
15 sis with oligonucleotide probes specific for Encephalitozoon cuniculi, E. hellem, and Encephalitozoon
16      MicrosporidiaDB contains the genomes of Encephalitozoon cuniculi, E. intestinalis and E. bieneus
17 evious mutational and structural analyses of Encephalitozoon cuniculi Ecm1, a prototypal cellular cap
18 In aged animals challenged with the parasite Encephalitozoon cuniculi, effector CD8+ T cell survival
19 osporidian species: Enterocytozoon bieneusi, Encephalitozoon cuniculi, Encephalitozoon hellem, and En
20 Here we show that the intracellular parasite Encephalitozoon cuniculi encodes a complete mRNA capping
21     Notably the genome of the microsporidian Encephalitozoon cuniculi has lost all of its genes for M
22 eir ability to prime an IEL response against Encephalitozoon cuniculi in vitro.
23 er animals exhibit greater susceptibility to Encephalitozoon cuniculi infection, and their ability to
24                           Effectively, after Encephalitozoon cuniculi infection, CD11b(-) CD8(+) DC w
25 to play an important role in defense against Encephalitozoon cuniculi infection.
26  response is critical for protection against Encephalitozoon cuniculi infection.
27                                              Encephalitozoon cuniculi infects a wide range of mammali
28                                              Encephalitozoon cuniculi is a protozoan parasite that ha
29                                              Encephalitozoon cuniculi is commonly found in domestic r
30         Because it is able to grow in vitro, Encephalitozoon cuniculi is currently the best-studied m
31 er (ITS) region of a recently cultured human Encephalitozoon cuniculi isolate was analyzed by gene am
32                                          The Encephalitozoon cuniculi mRNA cap (guanine N-7) methyltr
33 primary human macrophages were infected with Encephalitozoon cuniculi or Encephalitozoon intestinalis
34 t with culture-grown Encephalitozoon hellem, Encephalitozoon cuniculi, or Vittaforma corneae or with
35 n to examine the protein interactions of the Encephalitozoon cuniculi polar tube proteins (EcPTPs).
36 rformed a bioinformatic investigation of the Encephalitozoon cuniculi proteome.
37 n live (freshly harvested) and dead (boiled) Encephalitozoon cuniculi spores.
38 , E. intestinalis, and E. hellem, as well as Encephalitozoon cuniculi, spores in fecal samples and is
39 testinalis but not Encephalitozoon hellem or Encephalitozoon cuniculi was confirmed in 6 of 8 mammali
40                                              Encephalitozoon cuniculi was detected in three patients:
41 0 also demonstrated in vivo activity against Encephalitozoon cuniculi, with prolonged survival and th

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