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1 er inoculation of their endophytic bacteria (Enterobacter cloacae).
2 hich is identical to plasmid pNDM-HF727 from Enterobacter cloacae.
3 gle D(H) gene segment (D-limited mice), with Enterobacter cloacae.
4 onse to alpha-1,3 dextran (DEX) expressed on Enterobacter cloacae.
5 ence analysis revealed that the organism was Enterobacter cloacae.
6 antibiotic for treatment of infections with Enterobacter cloacae.
7 robacter koseri but 97.0% similar to that of Enterobacter cloacae.
8 d by blood feeding, restored the immunity to Enterobacter cloacae.
9 illus, which most laboratories recognized as Enterobacter cloacae.
10 escens after injection of prepupae with live Enterobacter cloacae.
11 m patients were Pseudomonas aeruginosa (22), Enterobacter cloacae (21), Acinetobacter spp. (13), Ente
12 coli (18.8%), Klebsiella pneumoniae (14.2%), Enterobacter cloacae (9.1%), Acinetobacter spp. (6.2%),
13 hogens with interpretive criteria, excluding Enterobacter cloacae (98.3% S) and E. faecalis (86.0% S)
14 inst Escherichia coli, Klebsiella pneumonia, Enterobacter cloacae, Acinetobacter baumannii, and methi
15 linically important enzymes CTX-M-15, KPC-2, Enterobacter cloacae AmpC, Pseudomonas aeruginosa AmpC,
19 microbial bacteremia, and seven of these had Enterobacter cloacae and S. marcescens in the same cultu
20 stant (IC(50), approximately 10,000 nM), and Enterobacter cloacae and Serratia marcescens were highly
21 348 Klebsiella pneumoniae, one (<1%) of 890 Enterobacter cloacae, and one (1%) of 162 Enterobacter a
24 L49 antibodies were chemically conjugated to Enterobacter cloacae beta-lactamase (bL), and their abil
25 nd the increasing clinical importance of the Enterobacter cloacae complex have often been discussed.
26 nt Escherichia coli, Enterobacter aerogenes, Enterobacter cloacae complex, Klebsiella pneumoniae, or
27 m Staphylococcus aureus, and aac(3)-VIa from Enterobacter cloacae (conferring resistance to kanamycin
28 oci from isolates of Serratia marcescens and Enterobacter cloacae, demonstrating the presence of in-f
29 y Citrobacter freundii, Clostridium species, Enterobacter cloacae, Enterococcus faecalis, Klebsiella
30 fied in the Enterobacteria Escherichia coli, Enterobacter cloacae, Erwinia herbicola, and Salmonella
31 ing probes for pathogenic bacteria including Enterobacter cloacae, Escherichia coli J96, Pseudomonas
32 isolates of important Gram-negative species-Enterobacter cloacae, Escherichia coli, Klebsiella pneum
35 structure of the nitroreductase enzyme from Enterobacter cloacae has been determined for the oxidize
38 rganisms, intraocular infection secondary to Enterobacter cloacae infection is a devastating disease
41 iae isolates, 3 Escherichia coli isolates, 5 Enterobacter cloacae isolates, 2 S. marcescens isolates,
42 the x-ray structures of the D305A mutant of Enterobacter cloacae MurA and the D313A mutant of Escher
43 tured and depicted the Cys-115-PEP adduct of Enterobacter cloacae MurA in various reaction states by
44 ablished that Cys115 of Escherichia coli and Enterobacter cloacae MurA is the active site nucleophile
45 s been determined to be 8.3, by titration of Enterobacter cloacae MurA with the alkylating agent iodo
46 d the x-ray structure of the C115S mutant of Enterobacter cloacae MurA, which was crystallized in the
49 1.9%), Escherichia coli (n = 129, 30.0%) and Enterobacter cloacae (n = 62, 14.4%) were the main Enter
50 ies of the flavin mononucleotide cofactor of Enterobacter cloacae nitroreductase (NR), determined und
51 The oxygen-insensitive nitroreductase from Enterobacter cloacae (NR) catalyzes two-electron reducti
53 ed reaction, cephalosporin hydrolysis by the Enterobacter cloacae P99 cephalosporinase (beta-lactam h
54 dase of Streptomyces sp. R61, a PBP, and the Enterobacter cloacae P99 cephalosporinase, a class C bet
57 to react with the class C beta-lactamase of Enterobacter cloacae P99 in two ways, by acylation and b
58 stants for hydrolysis by beta-lactamase from Enterobacter cloacae P99 indicated kcat values of 476 +/
62 cts on V/K for the class C beta-lactamase of Enterobacter cloacae P99 suggest an acyl-transfer transi
64 hibited typical class A (TEM-2) and class C (Enterobacter cloacae P99) beta-lactamases in a time-depe
65 , catalyzed by the class C beta-lactamase of Enterobacter cloacae P99, have been studied in order to
66 -2 antibody detected Escherichia coli CMY-2, Enterobacter cloacae P99, Klebsiella pneumoniae ACT-1, a
71 several gram-negative bacteria, specifically Enterobacter cloacae, Pseudomonas aeruginosa, and Pantoe
73 , invasive aspergillosis (20%, 3 of 15), and Enterobacter cloacae, Serratia marcescens, Pneumocystis
74 mis, E. coli O157:H7, Klebsiella pneumoniae, Enterobacter cloacae, Shigella dysenteriae, Salmonella e
76 The membrane-bound selenate reductase of Enterobacter cloacae SLD1a-1 is purified in low yield an
77 erial species such as Klebsiella pneumoniae, Enterobacter cloacae, Stenotrophomonas maltophilia, and
79 C beta-lactamase from a clinical isolate of Enterobacter cloacae strain GC1 with improved hydrolytic
80 fication of a Shiga toxin 1 (Stx1)-producing Enterobacter cloacae strain, M12X01451, from a human cli
81 nterobacter aerogenes and 13,954 isolates of Enterobacter cloacae tested using a Vitek system; for th
82 ing the melibiose-H(+) symporter (MelY) from Enterobacter cloacae that had enhanced fermentation on 1
83 t on four cases of endophthalmitis caused by Enterobacter cloacae: two in patients with acute postope
84 rates higher than the totals were noted with Enterobacter cloacae versus ampicillin-sulbactam, aztreo
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