ライフサイエンスコーパス: Enterobacteriaceae

1 n-essential outer membrane glycolipid of the Enterobacteriaceae.

2 ed polymyxin resistance mechanism, MCR-1, in Enterobacteriaceae.

3 organisms that thrive in similar locales as Enterobacteriaceae.

4 h extended-spectrum beta-lactamase-producing Enterobacteriaceae.

5 riction-modification role for the cluster in Enterobacteriaceae.

6 lexities of the T3SS effector repertoires of Enterobacteriaceae.

7 most recently caused by carbapenem-resistant Enterobacteriaceae.

8 umoniae, and an update on cephalosporins for Enterobacteriaceae.

9 fficient to limit colonization of pathogenic Enterobacteriaceae.

10 ded-spectrum beta-lactamase (ESBL)-producing Enterobacteriaceae.

11 negative bacteria Pseudomonas aeruginosa and Enterobacteriaceae.

12 ncluding 25 isolates of carbapenem-resistant Enterobacteriaceae.

13 lers (0.5%) acquired carbapenemase-producing Enterobacteriaceae.

14 actamase-producing, and carbapenem-resistant Enterobacteriaceae.

15 th increased proportions of Enterococcus and Enterobacteriaceae.

16 MIC of >/=4 mug/ml for both A. baumannii and Enterobacteriaceae.

17 d, including both Enterobacteriaceae and non-Enterobacteriaceae.

18 of the major peptidoglycan synthase PBP1b in Enterobacteriaceae.

19 isodes among elderly patients were caused by Enterobacteriaceae.

20 ance phenotypes against carbapenem-resistant Enterobacteriaceae.

21 perior to that of the MHT for non-Klebsiella Enterobacteriaceae.

22 for cephalosporins and aztreonam for testing Enterobacteriaceae.

23 species, can also be found in members of the Enterobacteriaceae.

24 idues are not conserved in homologues of the Enterobacteriaceae.

25 e, mediated by analogous systems, across the Enterobacteriaceae.

26 ted to the emergence of carbapenem-resistant Enterobacteriaceae.

27 and third-generation cephalosporin-resistant Enterobacteriaceae.

28 bsiella spp and from 30.4% to 71.9% in other Enterobacteriaceae.

29 ebsiella spp and from 5.9% to 68.8% in other Enterobacteriaceae.

30 with the dissemination of the mcr-1 genes in Enterobacteriaceae.

31 difficult to contain carbapenemase-producing Enterobacteriaceae.

32 housand; p < 0.001), ciprofloxacin-resistant Enterobacteriaceae (0.8 per thousand vs 2.5 per thousand

33 d infections caused by ceftazidime-resistant Enterobacteriaceae (0.8 per thousand vs 3.6 per thousand

34 ncluded 4,427 isolates: 2,065 ESBL-producing Enterobacteriaceae, 1,105 A. baumannii, 888 susceptible

35 derly patients was more frequently caused by Enterobacteriaceae (24% in middle-aged, 32% in old, and

36 ird-generation cephalosporin (3GC)-resistant Enterobacteriaceae (3GC-R EB), Dutch guidelines recommen

37 Enterobacteriaceae (42%), Enterococcus spp. (24%), and C

38 uding 95% of Enterococcus faecium and 55% of Enterobacteriaceae; 82% of deep SSIs were caused by bact

39 The study tested 100 well-characterized Enterobacteriaceae, Acinetobacter baumannii, and Pseudom

40 identified 21 621 non-duplicate isolates of Enterobacteriaceae, Acinetobacter spp, and Pseudomonas a

41 y restored the activity of meropenem against Enterobacteriaceae, Acinetobacter spp. and Pseudomonas s

42 Eighty Gram-negative bacilli (54 Enterobacteriaceae and 26 nonfermenting Gram-negative ba

43 n of inflammation in Il10(-/-) mice supports Enterobacteriaceae and alters a small subset of microbia

44 ded-spectrum beta-lactamase (ESBL)-producing Enterobacteriaceae and CRE carriage among solid organ tr

45 eened IncX4 plasmids among 2,470 isolates of Enterobacteriaceae and determined the mcr-1 positive iso

46 reduced in previous HE, along with increased Enterobacteriaceae and Enterococcaceae.

47 rulence determinants in carbapenem-resistant Enterobacteriaceae and enterohemorrhagic Escherichia col

48 is moiety seems unique to amylomaltases from Enterobacteriaceae and folds into two distinct subdomain

49 Enterobacteriaceae and in particular the pathobiont Prot

50 anisms included potential pathogens from the Enterobacteriaceae and Listeriaceae and others, includin

51 ic Endo(hi) microbiota (a high proportion of Enterobacteriaceae and low proportion of Bacteroidetes)

52 interactions between the trimeric porins of Enterobacteriaceae and LPS.

53 ded-spectrum beta-lactamase (ESBL)-producing Enterobacteriaceae and MDR Acinetobacter baumannii.

54 essed 402 Gram-negative rods, including both Enterobacteriaceae and non-Enterobacteriaceae expressing

55 m-negative rods (GNR) tested, including both Enterobacteriaceae and non-Enterobacteriaceae.

56 enems in patients with ceftazidime-resistant Enterobacteriaceae and P aeruginosa.

57 iscordant development of bacterial genera of Enterobacteriaceae and Parabacteroides species in the fi

58 actobacillales members were positively while Enterobacteriaceae and Porphyromonadaceae were negativel

59 Widespread resistance in Enterobacteriaceae and Pseudomonas aeruginosa to cephalo

60 wofold and 2 twofold modal MIC decreases for Enterobacteriaceae and Pseudomonas aeruginosa, respectiv

61 of infections caused by carbapenem-resistant Enterobacteriaceae and Pseudomonas aeruginosa, which are

62 esistance to beta-lactam antibiotics in many Enterobacteriaceae and Pseudomonas aeruginosa.

63 in intrinsic antibacterial activity against Enterobacteriaceae and restoration of beta-lactam activi

64 common errors included misinterpretation of Enterobacteriaceae and Staphylococcus genus results.

65 the healthy status by increased abundance of Enterobacteriaceae and Streptococcus spp. and, functiona

66 relative abundances of Enterobacteriales and Enterobacteriaceae and the interferon gamma level; and t

67 Gammaproteobacteria, Enterobacteriales, and Enterobacteriaceae and the interleukin 1beta (IL-1beta)

68 Institute (CLSI) carbapenem breakpoints for Enterobacteriaceae and the lack of active surveillance t

69 relative abundances of Enterobacteriales and Enterobacteriaceae and the sCD14 level; the relative abu

70 ition (e.g., Lactobacillus, Bacteroides, and Enterobacteriaceae) and approximately 9% higher beta-glu

71 riaceae, 1,105 A. baumannii, 888 susceptible Enterobacteriaceae, and 369 CRE isolates.

72 cinetobacter baumannii, carbapenem-resistant Enterobacteriaceae, and Candida species, are a major cau

73 quent pathogens were Pseudomonas aeruginosa, Enterobacteriaceae, and methicillin-sensitive and methic

74 g resistant strains and carbapenem-resistant Enterobacteriaceae, and most anaerobic pathogens.

75 methicillin-resistant Staphylococcus aureus, Enterobacteriaceae, and Pseudomonas aeruginosa) in popul

76 ded-spectrum beta-lactamase (ESBL)-producing Enterobacteriaceae, and susceptible Enterobacteriaceae i

77 microbial counts (total aerobic mesophiles, enterobacteriaceae, and yeasts/moulds) while at 100/150

78 The Enterobacteriaceae are a family of Gram-negative bacteri

79 Escherichia coli and other Enterobacteriaceae are among the most common pathogens o

80 Carbapenemase-producing Enterobacteriaceae are an emerging threat to hospitals w

81 Extended-spectrum beta-lactamase-producing Enterobacteriaceae are difficult-to-treat pathogens like

82 Infections caused by carbapenemase-producing Enterobacteriaceae are increasing worldwide, especially

83 Carbapenem-resistant Enterobacteriaceae are resistant to most beta-lactam ant

84 We identified 510 patients with Enterobacteriaceae bacteremia and sepsis, severe sepsis,

85 such as Burkholderia spp. and members of the Enterobacteriaceae bacteria family and reduced colonizat

86 Penicillium (fungi) and Pseudomonadaceae and Enterobacteriaceae (bacteria) were among the most abunda

87 342 health-care-associated infections, with Enterobacteriaceae being the most frequently found (113

88 se-producing organisms, carbapenem-resistant Enterobacteriaceae, carbapenem-resistant Pseudomonas aer

89 ified the effects of carbapenemase-producing Enterobacteriaceae carriage on patient outcome in two Gr

90 notype that we observed among members of the Enterobacteriaceae Characterization of S. flexneri 2457T

91 In patients with ESBL-producing Enterobacteriaceae, clinical cure rates were 95.8% (23/2

92 r extended-spectrum beta-lactamase-producing Enterobacteriaceae colonization as a predictor of extend

93 hazard ratios, with carbapenemase-producing Enterobacteriaceae colonization as time-varying covariat

94 d 96 (9.5%) acquired carbapenemase-producing Enterobacteriaceae colonization during ICU stay, and 301

95 Carbapenemase-producing Enterobacteriaceae colonization was associated with a st

96 Carbapenemase-producing Enterobacteriaceae colonization was established through

97 r extended-spectrum beta-lactamase-producing Enterobacteriaceae colonization, and colonization was as

98 e independent risk factors for KPC-producing Enterobacteriaceae colonization.

99 that of BMD for testing a limited number of Enterobacteriaceae commonly isolated by clinical laborat

100 f patients were colonized with KPC-producing Enterobacteriaceae, compared to 3.3% (30 of 910) of shor

101 nserved regulatory network across the family Enterobacteriaceae comprised of 10 genes associated with

102 Enterococci and Enterobacteriaceae counts were very low or zero.

103 carbapenemase-producing carbapenem-resistant Enterobacteriaceae (CP-CRE) are a significant clinical a

104 carbapenemase-producing carbapenem-resistant Enterobacteriaceae (CP-CRE) is an important element of t

105 n of 166 isolates of carbapenemase-producing Enterobacteriaceae (CPE) and 82 isolates of Enterobacter

106 our understanding of carbapenemase-producing Enterobacteriaceae (CPE) and which will be valuable for

107 Carbapenemase-producing Enterobacteriaceae (CPE) are a significant threat to pub

108 Carbapenemase-producing Enterobacteriaceae (CPE) are emerging worldwide, limitin

109 bal dissemination of carbapenemase-producing Enterobacteriaceae (CPE) is a more recent problem that,

110 ly identification of carbapenemase-producing Enterobacteriaceae (CPE) is essential to prevent their d

111 le treatment against carbapenemase-producing Enterobacteriaceae (CPE) is unknown.

112 Rapid detection of carbapenemase-producing Enterobacteriaceae (CPE) represents a major challenge fo

113 for the detection of carbapenemase-producing Enterobacteriaceae (CPE).

114 alyzed for ESBL- and carbapenemase-producing Enterobacteriaceae (CPE).

115 and 161 (64.7%) had carbapenemase-producing Enterobacteriaceae (CPE).

116 nic probes specific for carbapenem-resistant Enterobacteriaceae (CRE) and they were designed based on

117 Carbapenem-resistant Enterobacteriaceae (CRE) are a concern for health care i

118 Carbapenem-resistant Enterobacteriaceae (CRE) are a serious public health thr

119 Carbapenem-resistant Enterobacteriaceae (CRE) are among the most severe threa

120 Carbapenem-resistant Enterobacteriaceae (CRE) are associated with considerabl

121 Carbapenem-resistant Enterobacteriaceae (CRE) are emerging global pathogens.

122 Carbapenem-resistant Enterobacteriaceae (CRE) are high-priority bacterial pat

123 Carbapenem-resistant Enterobacteriaceae (CRE) are increasingly reported world

124 month delay in changing carbapenem-resistant Enterobacteriaceae (CRE) breakpoints might have resulted

125 Detecting carbapenem-resistant Enterobacteriaceae (CRE) can be difficult.

126 gy for the isolation of carbapenem-resistant Enterobacteriaceae (CRE) from 300 rectal swab specimens

127 2010, the incidence of carbapenem-resistant Enterobacteriaceae (CRE) has been increasing in Singapor

128 ibactam (CAZ-AVI) among carbapenem-resistant Enterobacteriaceae (CRE) has infrequently been reported

129 Carbapenem-resistant Enterobacteriaceae (CRE) have spread globally and repres

130 nd clinical outcomes of carbapenem-resistant Enterobacteriaceae (CRE) in sentinel US hospitals.

131 Carbapenem-resistant Enterobacteriaceae (CRE) is an emergent microorganism of

132 Two collections of carbapenem-resistant Enterobacteriaceae (CRE) isolates were evaluated, includ

133 Carbapenem-resistant Enterobacteriaceae (CRE) producing the New Delhi metallo

134 Carbapenem-resistant Enterobacteriaceae (CRE) spread regionally throughout he

135 Carbapenem-resistant Enterobacteriaceae (CRE), a group of pathogens resistant

136 d fecal coliforms (FC), carbapenem-resistant Enterobacteriaceae (CRE), blaNDM-1, and selected extende

137 ICs among A. baumannii, carbapenem-resistant Enterobacteriaceae (CRE), extended-spectrum beta-lactama

138 ontaining the spread of carbapenem-resistant Enterobacteriaceae (CRE), primarily manifested by the ra

139 Thirty-seven carbapenem-resistant Enterobacteriaceae (CRE)-infected patients were treated

140 ive bacteria, including carbapenem-resistant Enterobacteriaceae (CRE).

141 ophilic bacteria, yeast, mold and pathogenic Enterobacteriaceae decreased as irradiation dose increas

142 ctale and Bifidobacterium and an increase of Enterobacteriaceae, Desulfovibrio sp., and mainly Akkerm

143 r extended-spectrum beta-lactamase-producing Enterobacteriaceae detection was routinely performed in

144 for the detection of polymyxin resistance in Enterobacteriaceae, developed by P.

145 owever, some non-pestis Yersinia strains and Enterobacteriaceae did elicit signals, albeit at highly

146 r extended-spectrum beta-lactamase-producing Enterobacteriaceae digestive colonization by weekly acti

147 h extended-spectrum beta-lactamase-producing Enterobacteriaceae during ICU-hospitalization.

148 two Greek ICUs with carbapenemase-producing Enterobacteriaceae endemicity.

149 creased whereas potentially pathogenic ones (Enterobacteriaceae, Enterococcaceae) increased in saliva

150 ria, Gammaproteobacteria, Enterobacteriales, Enterobacteriaceae, Erysipelotrichi, Erysipelotrichales,

151 f extended-spectrum beta-lactamase-producing Enterobacteriaceae (ESBL-E) in clinical stool samples.

152 f extended-spectrum beta-lactamase-producing Enterobacteriaceae (ESBL-E), compared with 2.9% of non-P

153 o extended-spectrum beta-lactamase-producing Enterobacteriaceae (ESBL-E).

154 s extended-spectrum beta-lactamase-producing Enterobacteriaceae (ESBL-PE) and may transmit the strain

155 of Firmicutes, Bacteroidetes, Enterococcus, Enterobacteriaceae, Escherichia coli, and Bifidobacteriu

156 All Enterobacteriaceae express a polysaccharide known as ent

157 s, including both Enterobacteriaceae and non-Enterobacteriaceae expressing IMP, VIM, KPC, NDM, and/or

158 cs approach, we found that E. coli and other Enterobacteriaceae expressing the Yersinia HPI also secr

159 Enterobacteriaceae family members are mainly transmitted

160 lateral flow assays developed for detecting enterobacteriaceae family members in food and water samp

161 However, only the Enterobacteriaceae family of human gut-associated Proteo

162 siella ictaluri is a primitive member of the Enterobacteriaceae family that causes enteric septicemia

163 Serratia marcescens, a member of the Enterobacteriaceae family, is a Gram-negative bacterium

164 ted P = 0.25] but decreased pro-inflammatory Enterobacteriaceae (FDR-corrected P = 0.25).

165 omycin-resistant Enterococcus (VRE), and MDR Enterobacteriaceae Fecal metagenomes were analyzed from

166 bactin is a secondary metabolite produced by Enterobacteriaceae for acquiring iron, an essential meta

167 mance was verified by testing members of the Enterobacteriaceae for susceptibility to ampicillin, cef

168 llance program collected 103,960 isolates of Enterobacteriaceae from 2008 to 2014.

169 e extended-spectrum beta-lactamase-producing Enterobacteriaceae from rectal swab culture.

170 ded-spectrum beta-lactamase (ESBL)-producing Enterobacteriaceae generally cannot be treated with peni

171 lly using this mode of expression amongst 36 Enterobacteriaceae genomes.

172 o reveal functionally unique features of the Enterobacteriaceae GusRs.

173 Transmissible carbapenem-resistance in Enterobacteriaceae has been recognized for the last 2 de

174 rbapenems in Klebsiella pneumoniae and Other Enterobacteriaceae) has contributed seminal multicenter

175 ients colonized with carbapenemase-producing Enterobacteriaceae have on average a 1.79 times higher h

176 Carbapenem-resistant Enterobacteriaceae have recently become an important cau

177 ber of microorganisms, yeasts and moulds and Enterobacteriaceae) have been determined during fermenta

178 Proteins of the Hha-family, conserved among enterobacteriaceae, have been implicated in dynamically

179 lebsiella pneumoniae carbapenemase-producing Enterobacteriaceae (hereafter "KPC") are an increasing t

180 nal Endo(lo) microbiota (a low proportion of Enterobacteriaceae, high proportion of Bacteroidetes, an

181 The global emergence of carbapenem-resistant Enterobacteriaceae highlights the urgent need to reduce

182 d chromogenic screening for highly resistant Enterobacteriaceae [HRE]); with contact precautions for

183 l early colonisers such as Streptococcus and Enterobacteriaceae, iii) domination of Bifidobacteriacea

184 d imipenem from 4 mg/liter to 1 mg/liter for Enterobacteriaceae in 2010.

185 nt on up to 86%, and Pseudomonas spp. and/or Enterobacteriaceae in 38% of positive cultures.

186 hothrix thermosphacta, Pseudomonas spp., and Enterobacteriaceae in AP meat compared to NAP meat.

187 s provides a brief summary of ESBL-producing Enterobacteriaceae in children, with a focus on recent c

188 bution to the dissemination of bla NDM among Enterobacteriaceae in China.

189 XA-48 is the most prevalent carbapenemase in Enterobacteriaceae in Europe and the Middle East, but it

190 ene in one isolate prompted inclusion of non-Enterobacteriaceae in our surveillance culture workup.

191 h extended-spectrum beta-lactamase-producing Enterobacteriaceae in previously noncolonized and noninf

192 ition, the CREST study (Carbapenem-Resistant Enterobacteriaceae in Solid Organ Transplant Patients) h

193 quate duration of therapy for ESBL-producing Enterobacteriaceae in solid organ transplants and MCS de

194 nter- and intraspecies competition among the Enterobacteriaceae in the inflamed gut.

195 ability of respiratory electron acceptors to Enterobacteriaceae in the lumen of the colon.

196 This correlates with a lower amount of Enterobacteriaceae in the midgut microbiota.

197 typic detection of carbapenemase activity in Enterobacteriaceae In this issue of the Journal of Clini

198 rsinia pestis is unique among the pathogenic Enterobacteriaceae in utilizing an arthropod-borne trans

199 ont closely related to Sodalis glossinidius (Enterobacteriaceae) in members of the ecologically succe

200 17 (EcN) to limit the expansion of competing Enterobacteriaceae (including pathogens and pathobionts)

201 roth microdilution (BMD) for 255 isolates of Enterobacteriaceae, including 25 isolates of carbapenem-

202 essed by Gram-negative bacteria belonging to Enterobacteriaceae, including emerging drug-resistant pa

203 ommon among pathogenic species in the family Enterobacteriaceae, including multidrug-resistant Klebsi

204 of several pathogenic species in the family Enterobacteriaceae, including UPEC, and is frequently as

205 relative abundance of facultative anaerobic Enterobacteriaceae increases.

206 n extended-spectrum beta-lactamase-producing Enterobacteriaceae infection (risk ratio, 49.62 [95% CI,

207 t extended-spectrum beta-lactamase-producing Enterobacteriaceae infection were 95.1% (95% CI, 54.7-99

208 Patients with Enterobacteriaceae infection with a carbapenem MIC of 2

209 Carbapenems resistant Enterobacteriaceae infections are increasing worldwide r

210 Recent outbreaks of carbapenem-resistant Enterobacteriaceae infections associated with duodenosco

211 tched-cohort analysis in adult patients with Enterobacteriaceae infections treated with meropenem, im

212 f extended-spectrum beta-lactamase-producing Enterobacteriaceae involvement in ventilator-associated

213 escens, a member of the carbapenem-resistant Enterobacteriaceae, is an important emerging pathogen th

214 identified by evaluating the antibiograms of Enterobacteriaceae isolated in the UCLA Health System fr

215 Here, blind analysis of 140 characterized Enterobacteriaceae isolates using two protein extraction

216 roducing Enterobacteriaceae, and susceptible Enterobacteriaceae isolates were determined by Etest, BM

217 Of 15,839 nonduplicate clinical Enterobacteriaceae isolates, 115 (0.73%) met the study d

218 Of 132 carbapenemase-producing Enterobacteriaceae isolates, 125 (94.7%) were Klebsiella

219 e developed a validation panel comprising 10 Enterobacteriaceae isolates, 5 Gram-positive cocci, 5 Gr

220 8 K-SeT) were compared by using a set of 166 Enterobacteriaceae isolates, including isolates producin

221 se control set of bla(KPC)-negative clinical Enterobacteriaceae isolates.

222 mal contact and conjunctival samples showing Enterobacteriaceae like colonies with positive oxidase t

223 mcr-1-positive Enterobacteriaceae (MCRPE) have attracted substantial me

224 o extended-spectrum beta-lactamase-producing Enterobacteriaceae, methicillin-resistant Staphylococcus

225 Multidrug-resistant Enterobacteriaceae (MRE) are widespread in the community

226 OXA-48-like carbapenemases (OXA-48 K-SeT) in Enterobacteriaceae (n = 82).

227 y extended-spectrum beta-lactamase-producing Enterobacteriaceae; of whom, 17 were previously detected

228 Although Enterobacteriaceae often comprise less than 1% of a heal

229 ibiotic, fosfomycin, to treat ESBL-producing Enterobacteriaceae (one that has completed enrollment an

230 al infection caused by ceftazidime-resistant Enterobacteriaceae or Pseudomonas aeruginosa.

231 g extended-spectrum beta-lactamase-producing Enterobacteriaceae outbreaks or data on pediatric popula

232 Overall, CR rates increased for Enterobacteriaceae (P = 0.03) and decreased for Acinetob

233 ty (P = 0.03) and Gammaproteobacteria (e.g., Enterobacteriaceae; P = 0.04) and was negatively associa

234 ncreased abundance in bacteria which include Enterobacteriaceae, Pasteurellacaea, Veillonellaceae, an

235 ll; it should also be applicable to numerous Enterobacteriaceae pathogenic species as the amino acid

236 r extended-spectrum beta-lactamase-producing Enterobacteriaceae phenotypic confirmation.

237 udomonas aeruginosa, Acinetobacter spp., and Enterobacteriaceae pose urgent public health threats.

238 families (autochthonous taxa negatively and Enterobacteriaceae positively) correlated with MR spectr

239 y extended-spectrum beta-lactamase-producing Enterobacteriaceae predominated.

240 ded-spectrum beta-lactamase (ESBL)-producing Enterobacteriaceae present an ever-growing burden in the

241 precedented outbreak of carbapenem-resistant Enterobacteriaceae, primarily involving KPC-producing Kl

242 h extended-spectrum beta-lactamase-producing Enterobacteriaceae prior to the development of pneumonia

243 iofilm formation, Escherichia coli and other Enterobacteriaceae produce an extracellular matrix consi

244 Enterobacteriaceae produce antimicrobial peptides for su

245 m-negative pathogens (Staphylococcus aureus, Enterobacteriaceae, Pseudomonas aeruginosa, and Acinetob

246 f extended-spectrum beta-lactamase-producing Enterobacteriaceae ranged from 5% to 10%.

247 r extended-spectrum beta-lactamase-producing Enterobacteriaceae rectal carriage as a way to predict t

248 rveillance definition for CPE to include all Enterobacteriaceae resistant to any carbapenem tested.

249 reduction the acquisition of ESBL-producing Enterobacteriaceae (RR, 0.28 [95% CI, .11-.69] for STD+A

250 The new breakpoints did little to the Enterobacteriaceae %S, but for P. aeruginosa, a profound

251 In Enterobacteriaceae, several membrane transporters are in

252 Most Enterobacteriaceae showed high rates of susceptibility (

253 concerning type of AMR, carbapenem-resistant Enterobacteriaceae, significantly declined after the pro

254 s called serine protease autotransporters of Enterobacteriaceae (SPATEs) are secreted by Shigella, Sa

255 elated to those on IncX3 plasmids in various Enterobacteriaceae species in China.

256 We included 219 MGEs described in 7 Enterobacteriaceae species isolated from human, animal a

257 bacter cloacae (n = 62, 14.4%) were the main Enterobacteriaceae species.

258 New Delhi metallo-beta-lactamase in diverse Enterobacteriaceae species.

259 nd can selectively recognize a wide range of Enterobacteriaceae species.

260 ral analyses and screening of ECA(LPS) among Enterobacteriaceae species.

261 ctive analysis of an additional 720 clinical Enterobacteriaceae spectra found an approximately 11,109

262 ed piglets, whereas in milk formula-fed pigs Enterobacteriaceae spp was 5-fold higher.

263 h tests were challenged with 294 isolates of Enterobacteriaceae spp., Pseudomonas aeruginosa, and Aci

264 f extended-spectrum beta-lactamase-producing Enterobacteriaceae subsequent infection and increased mo

265 The CLSI reduced the cefepime Enterobacteriaceae susceptibility breakpoint and introdu

266 l10(-/-) mice maintain a higher abundance of Enterobacteriaceae than healthy wild-type mice.

267 ted between CP-CRE and members of the family Enterobacteriaceae that do not produce carbapenemases.

268 is also active against carbapenem-resistant Enterobacteriaceae that produce Klebsiella pneumoniae ca

269 Enterobacteriaceae (CPE) and 82 isolates of Enterobacteriaceae that produced other beta-lactamases w

270 e practice of screening clinical isolates of Enterobacteriaceae that test as susceptible to carbapene

271 for 1.4% (1,493/103,960) of all isolates of Enterobacteriaceae The most frequently identified carbap

272 Comparative genomic analysis shows that, in Enterobacteriaceae, the cluster is a genomic island inte

273 However, compared to Enterobacteriaceae, the CR levels were 73-fold and 210-f

274 enem-resistance in Acinobacter baumannii and Enterobacteriaceae, there are limited treatment options

275 ramework for colonization resistance against Enterobacteriaceae, these mechanistic insights point to

276 For Enterobacteriaceae, this resulted in <2% reduction in su

277 Carbapenem-resistant Enterobacteriaceae threaten human health, since carbapen

278 otics reduce colonization resistance against Enterobacteriaceae to pinpoint possible control points f

279 he fecal relative abundance of MRE among all Enterobacteriaceae was determined in each carrier.

280 eview of infections caused by ESBL-producing Enterobacteriaceae was performed in heart transplant (HT

281 o carbapenemase PCR, for a collection of 125 Enterobacteriaceae We also investigated the impact of an

282 Enterobacteriaceae were isolated 11 times more frequentl

283 tapenem for carbapenemase activity among non-Enterobacteriaceae were low, but imipenem demonstrated a

284 cterial load, Bacteroidetes, Firmicutes, and Enterobacteriaceae were mostly similar, aged mice had a

285 Enterobacteriaceae were positively linked with serum inf

286 uminococcacae and higher Enterococcaceae and Enterobacteriaceae were seen in hospitalized patients.

287 bly distinct bacterial profile, dominated by Enterobacteriaceae, when compared to healthy milk.

288 inflamed intestine by suppressing commensal Enterobacteriaceae, which are susceptible to the antimic

289 Representatives of the Bacillaceae and Enterobacteriaceae, which contain many known facultative

290 ically important Gram-negative member of the Enterobacteriaceae, which has increasingly been recogniz

291 fferential abundance, including increases in Enterobacteriaceae, which have been associated with infl

292 interpretation associated with the mCIM for Enterobacteriaceae will likely lead to its adoption by c

293 Patients infected with Enterobacteriaceae with a carbapenem MIC of 2, 4, or 8 m

294 The proportions of non-Salmonella Enterobacteriaceae with extended spectrum beta-lactamase

295 dly detect 16S methyltransferase activity in Enterobacteriaceae with high specificity using the stand

296 and it is unknown if patients infected with Enterobacteriaceae with reduced susceptibility are more

297 popolysaccharide (LPS), we compared LPS from Enterobacteriaceae with that of Bacteroidetes.

298 ted resistance mechanism in human and animal Enterobacteriaceae, with a wide geographical distributio

299 e mCIM was easy to perform and interpret for Enterobacteriaceae, with results in less than 24 h and e

300 ource driving a post-antibiotic expansion of Enterobacteriaceae within the large bowel.