ライフサイエンスコーパス: Enterococcus

1 r flagella), and a motile Streptococcus (now Enterococcus).

2 s aureus infections and vancomycin-resistant enterococcus.

3 Prevotella, and reduction of Escherichia and Enterococcus.

4 This isothermal assay targets the same Enterococcus 23S rRNA gene region as the existing quanti

5 95% CI 9.4% to 25.4%), vancomycin-resistant Enterococcus, 25 of 180 (13.9%, 95% CI 8.9, 18.9%) and m

6 iotic for patients with vancomycin-resistant Enterococcus (4.2 versus 43.7 h; P=0.006) and viridans g

7 01), enriched in Bacteroides and depleted in Enterococcus, Acinetobacter, Pseudomonas, and Hydrogenop

8 li, Klebsiella spp., Pseudomonas aeruginosa, Enterococcus aerogenes, Proteus vulgaris and Enterobacte

9 rculating seawater carries fecal indicators, Enterococcus and bird-associated Catellicoccus, through

10 11 genera and with increased proportions of Enterococcus and Enterobacteriaceae.

11 uid cultures were preliminarily positive for enterococcus and gram-negative rods.

12 ing the Vitek-2 system with the GPI card for Enterococcus and the API system for staphylococci.

13 wo gram positive (Staphyllococcus aureus and Enterococcus) and two gram negative pathogens (E. coli a

14 fections (CAUTI) caused by Escherichia coli, Enterococcus, and Staphylococcus aureus we observed that

15 For patients with vancomycin-resistant Enterococcus bacteremia, the mean time to appropriate an

16 Vancomycin-resistant Enterococcus bloodstream infections (VRE-BSIs) are assoc

17 Vancomycin-resistant Enterococcus bloodstream infections (VRE-BSIs) are becom

18 bility to C. difficile, vancomycin-resistant Enterococcus, carbapenem-resistant Klebsiella pneumoniae

19 ecium (14%), Enterococcus mundtii (13%), and Enterococcus casseliflavus (13%) were frequently detecte

20 of environmentally adapted species including Enterococcus casseliflavus.

21 d-type (ATCC 29212) and vancomycin-resistant Enterococcus cells were incubated at five different vanc

22 sfer protein TraM of the G+ broad host range Enterococcus conjugative plasmid pIP501.

23 dentified as a source of the fecal indicator Enterococcus ; correlations between fines and enterococc

24 Enterococcus counts exceeded the federal recommended nat

25 ive abundances of Firmicutes, Bacteroidetes, Enterococcus, Enterobacteriaceae, Escherichia coli, and

26 eroides HF183 16S rRNA, M. smithii nifH, and Enterococcus esp gene targets that have been proposed as

27 ecium (VRE FCM) (16), vancomycin-susceptible Enterococcus faecalis (3), Aerococcus viridans (2), Baci

28 Enterococcus faecalis (48%), Enterococcus faecium (14%),

29 inosa (Pa), Legionella pneumophila (Lp), and Enterococcus faecalis (Ef) by using anti-infective, anti

30 ia coli (group A, 38.4%; group B, 39.3%) and Enterococcus faecalis (group A, 32.7%; group B, 33.2%).

31 occus (n = 5), and daptomycin-nonsusceptible Enterococcus faecalis (n = 6).

32 cus anginosus group (</=0.06 microg/mL), and Enterococcus faecalis (vancomycin susceptible, </=0.25 m

33 tient with recalcitrant vancomycin-resistant Enterococcus faecalis (VRE) and 2 patients with infectio

34 Vancomycin-resistant Enterococcus faecalis (VREfs) is an important nosocomial

35 Vancomycin-resistant Enterococcus faecalis (VREfs) is an important nosocomial

36 al species (seven Escherichia coli and three Enterococcus faecalis ) for all ten patient samples.

37 of MetAPs from Mycobacterium tuberculosis , Enterococcus faecalis , and human, three hotspots have b

38 accounted for 34.5% of fecal coliforms, and Enterococcus faecalis accounted for 32% of enterococci.

39 acterial biofilm and faecal samples included Enterococcus faecalis and Enterobacter hormaechei.

40 he BC-GP correctly identified 14/15 cases of Enterococcus faecalis and Enterococcus faecium bacteremi

41 m for identification of vancomycin-resistant Enterococcus faecalis and Enterococcus faecium, VRESelec

42 coated quartz sand (IOCS) to remove two FIB (Enterococcus faecalis and Escherichia coli) suspended in

43 kinetics of the d-Ala:d-Ser ligase VanG from Enterococcus faecalis and solved its crystal structure i

44 The Gram-positive bacterium Enterococcus faecalis and the fungus Candida albicans ar

45 -positive bacteria Staphylococcus aureus and Enterococcus faecalis and two Gram-negative bacteria Esc

46 tive transfer and virulence functions of the Enterococcus faecalis antibiotic resistance plasmid pCF1

47 resistant "superbugs." Bacteria of the genus Enterococcus faecalis are highly antibiotic-resistant no

48 d-scan EPR can detect superoxide produced by Enterococcus faecalis at rates that are too low for dete

49 cus aureus ATCC 29213 (0.03 to 0.12 mug/ml), Enterococcus faecalis ATCC 29212 (0.03 to 0.12 mug/ml),

50 sk only), S. aureus ATCC 29213 (broth only), Enterococcus faecalis ATCC 29212 (broth only), Streptoco

51 4028, Staphylococcus epidermidis ATCC 12228, Enterococcus faecalis ATCC 29212, and Escherichia coli D

52 pecies, including Pseudomonas putida KT2440, Enterococcus faecalis ATCC 29212, Salmonella Typhimurium

53 nted here had either Enterococcus faecium or Enterococcus faecalis bacteremia caused by both vancomyc

54 of synergistic gentamicin for uncomplicated Enterococcus faecalis bacteremia in children.

55 ective endocarditis (IE) among patients with Enterococcus faecalis bacteremia.

56 ers were then shown to inhibit the growth of Enterococcus faecalis biofilms that play a role in early

57 The bacterium Enterococcus faecalis cannot synthesize heme but can acq

58 tural snapshots from the type II-A system of Enterococcus faecalis Cas1 and Cas2 during spacer integr

59 Enterococcus faecalis Cas1-Cas2 selectively binds to a s

60 The Enterococcus faecalis cell wall-anchored protein Ace is

61 Upon sensing of the peptide pheromone cCF10, Enterococcus faecalis cells carrying pCF10 produce three

62 The pheromone-responsive plasmid pCF10 of Enterococcus faecalis encodes a putative cell wall hydro

63 es to PG composition in vancomycin-resistant Enterococcus faecalis following the growth in presence o

64 positive organisms Staphylococcus aureus and Enterococcus faecalis in comparison with known analogues

65 of the pheromone receptor protein PrgZ from Enterococcus faecalis in complex with the heptapeptide c

66 lar function in Streptococcus agalactiae and Enterococcus faecalis In conclusion, the elucidation of

67 us gentamicin (AG) combinations for treating Enterococcus faecalis infective endocarditis (EFIE).

68 study, we compare outcomes in patients with Enterococcus faecalis infective endocarditis treated in

69 Enterococcus faecalis is a commensal and pathogen of hum

70 Enterococcus faecalis is a commensal bacterium found in

71 Enterococcus faecalis is a commensal bacterium of the hu

72 Enterococcus faecalis is a human intestinal commensal th

73 Enterococcus faecalis is a member of the intestinal and

74 Since Enterococcus faecalis is a natural heme auxotroph and ca

75 e (SAS) RelQ from the Gram-positive pathogen Enterococcus faecalis is a sequence-specific RNA-binding

76 Enterococcus faecalis is an established nosocomial patho

77 Enterococcus faecalis is an opportunistic pathogen respo

78 The Gram-positive bacterium Enterococcus faecalis is both a colonizer of the gastroi

79 Enterococcus faecalis is both a common commensal of the

80 Enterococcus faecalis is frequently associated with poly

81 Enterococcus faecalis is one of the most frequently isol

82 Enterococcus faecalis is part of the human intestinal mi

83 Expansion of intestinal Enterococcus faecalis is sufficient to exacerbate ethano

84 Enterococcus faecalis is unusual in that it encodes two

85 Enterococcus faecalis isolates (n = 1,112) were highly s

86 Eighty-one endocarditis-derived Enterococcus faecalis isolates that were collected from

87 creasing sensitivity in the following order: Enterococcus faecalis LDH2 </= Lactococcus lactis LDH2 <

88 Enterococcus faecalis may contribute to periodontal brea

89 lope homeostasis), from daptomycin-resistant Enterococcus faecalis not only reversed resistance to 2

90 ed molecular patterns, including heat-killed Enterococcus faecalis or CpG DNA, led to increased Ikapp

91 Enterococcus faecalis pCF10 transfers at high frequencie

92 onoclonal antibody to the major component of Enterococcus faecalis pili, EbpC, labels polymerized pil

93 Multidrug-resistant Enterococcus faecalis possess numerous mobile elements t

94 d the GIT microbiota of MAT mothers, whereas Enterococcus faecalis predominated within the MAT infant

95 EfbA is a PavA-like fibronectin adhesin of Enterococcus faecalis previously shown to be important i

96 The Enterococcus faecalis prg and pcf genes of plasmid pCF10

97 ost & Microbe, Keogh et al. (2016) show that Enterococcus faecalis promotes Escherichia coli biofilm

98 uconostoc mesenteroides, Bacillus cereus and Enterococcus faecalis proving its antimicrobial action.

99 atural enterococci diversity, 11 isolates of Enterococcus faecalis recovered from freshwater watershe

100 Enterococcus faecalis resistance to aminopenicillins, va

101 occi, as homologs from Bacillus subtilis and Enterococcus faecalis retain this ability.

102 R (D191N) first identified from the pathogen Enterococcus faecalis S613.

103 Overexpression of the Fst toxin in Enterococcus faecalis strain OG1X leads to defects in ch

104 Enterococcus faecalis strains are commensal bacteria in

105 of five E. faecium strains but none of five Enterococcus faecalis strains consistently developed res

106 have been shown to opsonize nonencapsulated Enterococcus faecalis strains.

107 ve Escherichia coli Symbio and gram-positive Enterococcus faecalis Symbio or its placebo.

108 In this study, we show in Enterococcus faecalis that SecA and Sortase A, required

109 y for growth of the human bacterial pathogen Enterococcus faecalis The final enzyme in this pathway,

110 st infection with the opportunistic pathogen Enterococcus faecalis through promotion of host-microbio

111 usly, our research demonstrated that dietary Enterococcus Faecalis UC-100 substituting antibiotics en

112 ons were tested against Escherichia coli and Enterococcus faecalis urinary tract infection isolates.

113 served that a female was mono-colonized with Enterococcus faecalis vaginally as tested in aerobic cul

114 l and pathogenic host-microbe interaction of Enterococcus faecalis was explored using a Caenorhabditi

115 Under sterile conditions, 1 muL Enterococcus faecalis was inoculated inside the implants

116 omonas gingivalis and the endodontic species Enterococcus faecalis were grown to early log phase and

117 However, the AcpAs of Lactococcus lactis and Enterococcus faecalis were inactive.

118 g a microbe with host-protective properties (Enterococcus faecalis) and a pathogen (Staphylococcus au

119 genic bacteria (Photorhabdus luminescens and Enterococcus faecalis) and two nonpathogenic bacteria (E

120 and vanA or vanB in Enterococcus faecium and Enterococcus faecalis) by the BC-GP assay also was asses

121 Expression of ace (adhesin to collagen of Enterococcus faecalis), encoding a virulence factor in e

122 (predominant microorganism in institution A: Enterococcus faecalis, 18 cultures [51.4%]; institution

123 d 5 methods for testing daptomycin versus 48 Enterococcus faecalis, 51 Enterococcus faecium, and 50 S

124 Enterococcus faecalis, a Gram-positive bacterium, and Ca

125 associated inflammation impacts infection by Enterococcus faecalis, a leading cause of catheter-assoc

126 Enterococcus faecalis, a leading cause of hospital-acqui

127 EF1143 from Enterococcus faecalis, a life-threatening pathogen that

128 The genome of Enterococcus faecalis, a low-GC Gram-positive opportunis

129 high activity against the oral key pathogens Enterococcus faecalis, Actinomyces naeslundii, Streptoco

130 eant dye, YOYO-1, were first developed using Enterococcus faecalis, an organism that has previously b

131 roscan, 87% of Staphylococcus aureus, 90% of Enterococcus faecalis, and 88% of Enterococcus faecium i

132 us aureus (MRSA), Listeria monocytogenes and Enterococcus faecalis, and against the Gram-negative bac

133 ve bacteria, including Bacillus subtilis and Enterococcus faecalis, and drug-sensitive and drug-resis

134 survival of non-pathogenic Escherichia coli, Enterococcus faecalis, and E. coli O157:H7 were compared

135 occus epidermidis, Streptococcus pneumoniae, Enterococcus faecalis, and Enterococcus faecium) and thr

136 lyzed, in particular, Staphylococcus aureus, Enterococcus faecalis, and Escherichia coli.

137 tion by a pure facultative anaerobic strain, Enterococcus faecalis, and fresh mixed anaerobic sludge,

138 9.65, and 100.00% for Staphylococcus aureus, Enterococcus faecalis, and streptococci, respectively.

139 photoinactivation of a laboratory strain of Enterococcus faecalis, but depressed photoinactivation o

140 The ethanolamine utilization (eut) locus of Enterococcus faecalis, containing at least 19 genes dist

141 present in other pathogenic bacteria such as Enterococcus faecalis, Coxiella burnetii, and Clostridiu

142 In other gram-positive bacteria, such as Enterococcus faecalis, disulfide bonds are formed in sec

143 ides thetaiotaomicron, Campylobacter jejuni, Enterococcus faecalis, Escherichia coli K12, E. coli O15

144 ved with the type of bacterium in the order: Enterococcus faecalis, Escherichia coli O157:H7, and Esc

145 TDB, Escherichia coli, Bacillus subtilis and Enterococcus faecalis, from the guts of the desert woodr

146 , Clostridium species, Enterobacter cloacae, Enterococcus faecalis, Klebsiella oxytoca, Klebsiella pn

147 ecent clinical isolates of Escherichia coli, Enterococcus faecalis, Klebsiella pneumoniae, and Pseudo

148 ntestinal microbiota (Lactobacillus reuteri, Enterococcus faecalis, Lactobacillus crispatus and Clost

149 In Enterococcus faecalis, lateral transfer of conjugative p

150 species such as Staphylococcus epidermidis, Enterococcus faecalis, Pseudomonas aeruginosa, and Klebs

151 the AMP-modifications with Escherichia coli, Enterococcus faecalis, Pseudomonas aeruginosa, Staphyloc

152 We studied the survival of Enterococcus faecalis, Salmonella spp., E. coli O157 and

153 In Enterococcus faecalis, the regulatory nucleotides pppGpp

154 e setup was tested with Escherichia coli and Enterococcus faecalis, two bacterial strains that are co

155 vibrios, type E Clostridium perfringens, and Enterococcus faecalis, whereas the reverse was true for

156 adenovirus 41, Phi X 174) and the bacterium Enterococcus faecalis, which are relevant for water hygi

157 n Tn1546, acquired from vancomycin-resistant Enterococcus faecalis, which is known to alter cell wall

158 n pCF10, an antibiotic resistance plasmid of Enterococcus faecalis, which negatively regulates conjug

159 ms are absent from multidrug-resistant (MDR) Enterococcus faecalis, which only possess an orphan CRIS

160 itogenic, protease-secreting enteric microbe Enterococcus faecalis.

161 ainst an intestinal opportunistic bacterium, Enterococcus faecalis.

162 estinal bacterial growth, mainly E. coli and Enterococcus faecalis.

163 Escherichia coli, Klebsiella pneumoniae, and Enterococcus faecalis.

164 tivity of LmPC as well as PC from pathogenic Enterococcus faecalis.

165 survival signals modulated by the bacterium Enterococcus faecalis.

166 sion in the opportunistic bacterial pathogen Enterococcus faecalis.

167 ginosa but not to the Gram-positive pathogen Enterococcus faecalis.

168 crobial treatment were notable for growth of Enterococcus faecalis.

169 positive results with vancomycin-susceptible Enterococcus faecalis.

170 ) pilus is an important virulence factor for Enterococcus faecalis.

171 ement commonly found in clinical isolates of Enterococcus faecalis.

172 Hymeglusin blocks growth of Enterococcus faecalis.

173 aphylococcus aureus and vancomycin-resistant Enterococcus faecalis.

174 leukemia cells, yeast, Escherichia coli and Enterococcus faecalis.

175 ion of IL-10 deficient (Il10(-/-)) mice with Enterococcus faecalis.

176 cocci (16.9%), Escherichia coli (11.8%), and Enterococcus faecium (11.4%).

177 Enterococcus faecalis (48%), Enterococcus faecium (14%), Enterococcus mundtii (13%),

178 g a predicted fibronectin-binding protein of Enterococcus faecium (fnm), a homologue of Streptococcus

179 stream infection caused by a DAP-susceptible Enterococcus faecium (minimum inhibitory concentration,

180 ine and linezolid were highly active against Enterococcus faecium (n = 267) globally (100% and 98% su

181 ee important pathogens: vancomycin-resistant Enterococcus faecium (n=19), methicillin-resistant Staph

182 ococcus pneumoniae (6), vancomycin-resistant Enterococcus faecium (VRE FCM) (16), vancomycin-suscepti

183 lococcus aureus (MRSA), vancomycin-resistant Enterococcus faecium (VRE), and beta-lactam-resistant Kl

184 lococcus aureus (MRSA), vancomycin-resistant Enterococcus faecium (VRE), Escherichia coli SMS-3-5, an

185 occus aureus (MRSA) and vancomycin-resistant Enterococcus faecium (VREF) with MIC values of 1.4 and 2

186 Vancomycin-resistant Enterococcus faecium (VREfm) is a leading cause of nosoc

187 Vancomycin-resistant Enterococcus faecium (VREfm) is an important cause of he

188 due to rESKAPE strains (vancomycin-resistant Enterococcus faecium [0], methicillin-resistant Staphylo

189 rcent of bacteria were MDR, including 95% of Enterococcus faecium and 55% of Enterobacteriaceae; 82%

190 aphylococcus epidermidis and vanA or vanB in Enterococcus faecium and Enterococcus faecalis) by the B

191 bacteria, including human pathogens such as Enterococcus faecium and Listeria monocytogenes.

192 ia (high priority) were vancomycin-resistant Enterococcus faecium and meticillin-resistant Staphyloco

193 With the exception of 2 isolates, Enterococcus faecium and Nocardia exalbida, all the othe

194 drug-sensitive and drug-resistant strains of Enterococcus faecium and Staphylococcus aureus.

195 ied 14/15 cases of Enterococcus faecalis and Enterococcus faecium bacteremia and 9 cases of Streptoco

196 ront-line antibiotic for multidrug-resistant Enterococcus faecium bloodstream infections (BSIs).

197 Enterococcus faecium encodes a triple-specific aaPGS (Ra

198 The incidence of multidrug-resistant Enterococcus faecium hospital infections has been steadi

199 rted to be important for the pathogenesis of Enterococcus faecium in a rat infective endocarditis mod

200 In particular, vancomycin-resistant Enterococcus faecium infections have been increasing in

201 Enterococcus faecium is a common cause of nosocomial inf

202 Enterococcus faecium is an important cause of hospital-a

203 nical settings, non-susceptibility to DAP by Enterococcus faecium is correlated frequently with a mut

204 We show that secreted antigen A (SagA) from Enterococcus faecium is sufficient to protect Caenorhabd

205 estinal colonization by antibiotic-resistant Enterococcus faecium is the first step in a process that

206 us, 90% of Enterococcus faecalis, and 88% of Enterococcus faecium isolates were interpreted as suscep

207 n of the vanA gene in 81 cultures containing Enterococcus faecium or E. faecalis was 100% sensitive a

208 The four patients presented here had either Enterococcus faecium or Enterococcus faecalis bacteremia

209 Here, we identify and characterize 6 Enterococcus faecium proteins containing the WxL domain

210 Human epidemic clones ( E. coli ST131 and Enterococcus faecium ST17) were identified for the first

211 re induced in one Campylobacter coli and one Enterococcus faecium strain, while these strains plus th

212 precedes infection with antibiotic-resistant Enterococcus faecium We used a mouse GIT colonization mo

213 occus pneumoniae, Enterococcus faecalis, and Enterococcus faecium) and three associated genetic resis

214 ined individually and in combination against Enterococcus faecium, Acinetobacter baumannii and Klebsi

215 ptomycin versus 48 Enterococcus faecalis, 51 Enterococcus faecium, and 50 Staphylococcus aureus isola

216 Staphylococcus aureus, vancomycin-resistant Enterococcus faecium, and beta-lactam-resistant Klebsiel

217 that the silver/platinum combination against Enterococcus faecium, and silver/copper combination agai

218 ation demonstrated platinum and gold against Enterococcus faecium, platinum against Klebsiella pneumo

219 ncomycin-resistant Enterococcus faecalis and Enterococcus faecium, VRESelect, was compared to bile es

220 patients were Clostridium sp. 7 2 43FAA and Enterococcus faecium.

221 of the bacteria such as Escherichia coli and Enterococcus faecium.

222 idermidis in synthetic urine also containing Enterococcus faecium.

223 aphylococcus aureus and vancomycin-resistant Enterococcus faecium.

224 s Lactococcus casei, Lactococcus lactis, and Enterococcus faecium.

225 neonate colonized with vancomycin-resistant Enterococcus faecium.

226 aphylococcus aureus and vancomycin-resistant Enterococcus faecium.

227 The median Enterococcus fecal abundance was 24% (range, 8%-95%) in

228 s belonging to the phylum Proteobacteria and Enterococcus genus have also been linked to increased tr

229 Among 41 episodes in which enterococcus, group B streptococci, or both were found i

230 performance, we conclude that the developed Enterococcus HDA assay has great potential as a qualitat

231 The developed Enterococcus HDA assay successfully discriminated 15 ent

232 Enterococcus hirae accounted for 92% of the fecal isolat

233 Here, we identified two bacterial species, Enterococcus hirae and Barnesiella intestinihominis that

234 d the crystal structures of the V1 moiety of Enterococcus hirae V-ATPase (EhV1) and proposed a model

235 irst high-resolution view of ATP activity in Enterococcus hirae V1-ATPase.

236 structure of the related A-ATP synthase from Enterococcus hirae, the arrangements of the ScDF molecul

237 served on the spread of vancomycin-resistant Enterococcus in an intensive care unit.

238 atients infected with vancomycin-susceptible Enterococcus isolates (P = 0.1145).

239 In all, 120 (57%) Enterococcus isolates from 20 rainwater tank samples har

240 In this study, 212 Enterococcus isolates from 23 rainwater tank samples in

241 ) were detected in 61%, 43%, 43%, and 23% of Enterococcus isolates, respectively.

242 d dense colonization by vancomycin-resistant Enterococcus, K. pneumoniae, and E. coli.

243 faecalis (48%), Enterococcus faecium (14%), Enterococcus mundtii (13%), and Enterococcus casseliflav

244 us (n = 37), high-level gentamicin-resistant Enterococcus (n = 15), linezolid-resistant Enterococcus

245 esistant MRSA (n = 10), vancomycin-resistant Enterococcus (n = 37), high-level gentamicin-resistant E

246 t Enterococcus (n = 15), linezolid-resistant Enterococcus (n = 5), and daptomycin-nonsusceptible Ente

247 f eight strains (four Campylobacter and four Enterococcus) obtained macrolide-resistant mutants, incl

248 olically active cells (E. coli, B. subtilis, Enterococcus, P. aeruginosa and Salmonella typhi) to ant

249 r 4 wk significantly increased the number of Enterococcus, Prevotella, Bacteroides, Bifidobacterium,

250 cillus anthracis, and a vancomycin-resistant Enterococcus sp. with MIC or IC50 values in the 0.25-4 m

251 al inoculation consisting of twelve cultured Enterococcus species combined with conventional intestin

252 UTIs caused by Enterococcus species were higher in Group 2 (28.6% vs. 4

253 Escherichia coli and Enterococcus species, both indicators of fecal contamina

254 cal indicator bacteria measured in seawater (Enterococcus species, fecal coliforms, total coliforms)

255 e bacteria such as Staphylococcus aureus and Enterococcus species.

256 on by clinically vexing vancomycin-resistant Enterococcus species.

257 beta-glucosidase (beta-gluco) is produced by Enterococcus spp.

258 ound to be much higher compared to the other Enterococcus spp.

259 razone and promote increased colonization by Enterococcus spp.

260 in mice by increasing numbers of intestinal Enterococcus spp.

261 Enterobacteriaceae (42%), Enterococcus spp. (24%), and Candida spp. (15%) were pre

262 faecal indicator bacteria (FIB; E. coli and Enterococcus spp. (ENT)), Pseudomonas spp., and ARGs (bl

263 . numbers as measured by EPA Method 1600 and Enterococcus spp. 23S rRNA gene qPCR assay, respectively

264 s observed by flow cytometry, and inoculated Enterococcus spp. and Salmonella typhimurium during the

265 ) was much lower for Gram positive bacteria (Enterococcus spp. and Staphylococcus spp., including two

266 Among these tank water samples, Enterococcus spp. from 5 (25%) samples harbored a single

267 phylococcus aureus, and vancomycin-resistant Enterococcus spp. in the environment.

268 r human-specific Bacteroidales, E. coli, and Enterococcus spp. in treated source waters were only det

269 Enterococcus spp. isolates (n = 286) collected from six

270 larly, 49 (98%) and 47 (94%) samples yielded Enterococcus spp. numbers as measured by EPA Method 1600

271 47, P = 0.0009), and EPA Method 1600 and the Enterococcus spp. qPCR assay (r = 0.42, P = 0.002).

272 und between the culture-based method and the Enterococcus spp. qPCR assay in terms of detecting fecal

273 a greater abundance of Escherichia spp. and Enterococcus spp. than healthy controls (n = 26).

274 oli vs P. aeruginosa tau = 0.090, p = 0.027; Enterococcus spp. vs P. aeruginosa tau = 0.126, p = 0.00

275 resistant S. aureus and vancomycin resistant Enterococcus spp. was completed an average of 41.8 to 42

276 acteria (FIB) Escherichia coli (E. coli) and Enterococcus spp. were enumerated using culture-based me

277 Enterococcus spp. were the most frequent bacterial isola

278 ia (e.g., Staphylococcus, Streptococcus, and Enterococcus spp.), and thus the enzymes of the mevalona

279 pp., Bacteroides fragilis, Escherichia coli, Enterococcus spp., Pseudomonas aeruginosa, and Serratia

280 of a gene specific to a vancomycin-resistant Enterococcus strain was performed on the developed micro

281 ly encountered dominating organisms included Enterococcus, Streptococcus, and various Proteobacteria.

282 er, swarm cells rarely tumbled, and cells of Enterococcus tended to swim in loops when moving slowly.

283 Indeed, a delayed vancomycin-resistant Enterococcus transmission component was identified in th

284 bundance of Enterococcus was associated with Enterococcus urinary tract infection (UTI).

285 TI compared to 0% in the 23 patients without Enterococcus UTI (interquartile range, 0.00%-0.08%) (P=0

286 % (range, 8%-95%) in the three patients with Enterococcus UTI compared to 0% in the 23 patients witho

287 pients with posttransplant diarrhea, AR, and Enterococcus UTI.

288 tic-resistant bacterium vancomycin-resistant Enterococcus (VRE) can exceed 10(9) organisms per gram o

289 ccus aureus (MRSA), and vancomycin-resistant Enterococcus (VRE) in patients with and without penicill

290 infection (BSI) to due vancomycin-resistant Enterococcus (VRE) is an important complication of hemat

291 ections attributable to vancomycin-resistant Enterococcus (VRE) strains have become increasingly prev

292 tal swabs collected for vancomycin-resistant Enterococcus (VRE) surveillance as well as from clinical

293 lococcus aureus (MRSA), vancomycin-resistant Enterococcus (VRE), and MDR Enterobacteriaceae Fecal met

294 estinal domination with vancomycin-resistant Enterococcus (VRE), leading to bloodstream infection in

295 occus aureus (MRSA) and vancomycin-resistant Enterococcus (VRE), while also exhibiting a minimal pote

296 occus aureus (MRSA) and vancomycin-resistant enterococcus (VRE).

297 Fecal abundance of Enterococcus was associated with Enterococcus urinary tr

298 Enterococcus was measured both by culture (cENT) and DNA

299 Concentrations of the bacterial genus Enterococcus were measured as an indicator of fecal cont

300 appears to promote overgrowth of intestinal Enterococcus, which promotes liver disease, based on dat