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1 ding domain of ACE (adhesin of collagen from Enterococcus faecalis).
2 ) pilus is an important virulence factor for Enterococcus faecalis.
3 ement commonly found in clinical isolates of Enterococcus faecalis.
4 Hymeglusin blocks growth of Enterococcus faecalis.
5 aphylococcus aureus and vancomycin-resistant Enterococcus faecalis.
6 ion system is a major virulence regulator in Enterococcus faecalis.
7 role of sigma(54) in the nosocomial pathogen Enterococcus faecalis.
8 vents involving nosocomial pathogens such as Enterococcus faecalis.
9 aphylococcus aureus and vancomycin-resistant Enterococcus faecalis.
10 nsporter), for subspecies differentiation of Enterococcus faecalis.
11 important gram-positive nosocomial pathogen Enterococcus faecalis.
12 etween secretion and cell wall attachment in Enterococcus faecalis.
13 translocation for the Gram-positive bacteria Enterococcus faecalis.
14 ance of the plasmid pAD1 in its native host, Enterococcus faecalis.
15 niae, Salmonella typhimurium, S. aureus, and Enterococcus faecalis.
16 fected with the human opportunistic pathogen Enterococcus faecalis.
17 to linezolid is rare in clinical isolates of Enterococcus faecalis.
18 its N terminus to NADH peroxidase (Npx) from Enterococcus faecalis.
19 ike genes, salA and salB, were identified in Enterococcus faecalis.
20 ly identified as a general stress protein of Enterococcus faecalis.
21 the diversity of capsular polysaccharides in Enterococcus faecalis.
22 leukemia cells, yeast, Escherichia coli and Enterococcus faecalis.
23 ion of IL-10 deficient (Il10(-/-)) mice with Enterococcus faecalis.
24 itogenic, protease-secreting enteric microbe Enterococcus faecalis.
25 ainst an intestinal opportunistic bacterium, Enterococcus faecalis.
26 estinal bacterial growth, mainly E. coli and Enterococcus faecalis.
27 Escherichia coli, Klebsiella pneumoniae, and Enterococcus faecalis.
28 tivity of LmPC as well as PC from pathogenic Enterococcus faecalis.
29 survival signals modulated by the bacterium Enterococcus faecalis.
30 sion in the opportunistic bacterial pathogen Enterococcus faecalis.
31 ginosa but not to the Gram-positive pathogen Enterococcus faecalis.
32 crobial treatment were notable for growth of Enterococcus faecalis.
33 positive results with vancomycin-susceptible Enterococcus faecalis.
34 89), Klebsiella pneumoniae (TOP52 1721), and Enterococcus faecalis (0852) were studied, and diabetic
35 . pyogenes, 226 Streptococcus pneumoniae, 93 Enterococcus faecalis, 1,356 Enterobacteriaceae, and 227
36 (predominant microorganism in institution A: Enterococcus faecalis, 18 cultures [51.4%]; institution
37 strain in the catalytic loop, alanine 110 of Enterococcus faecalis 3-hydroxy-3-methylglutaryl coenzym
38 ecium (VRE FCM) (16), vancomycin-susceptible Enterococcus faecalis (3), Aerococcus viridans (2), Baci
40 d 5 methods for testing daptomycin versus 48 Enterococcus faecalis, 51 Enterococcus faecium, and 50 S
41 Herein, we describe mechanisms that link Enterococcus faecalis, a bacterium known to produce extr
42 in the recognition of muramyl dipeptide and Enterococcus faecalis, a commensal bacterium that is a c
48 al instability and colon cancer triggered by Enterococcus faecalis, a human intestinal commensal bact
49 associated inflammation impacts infection by Enterococcus faecalis, a leading cause of catheter-assoc
50 ence attribute of the opportunistic pathogen Enterococcus faecalis, a leading cause of hospital-acqui
56 accounted for 34.5% of fecal coliforms, and Enterococcus faecalis accounted for 32% of enterococci.
57 two-chamber system to study translocation of Enterococcus faecalis across monolayers of polarized hum
58 high activity against the oral key pathogens Enterococcus faecalis, Actinomyces naeslundii, Streptoco
59 eant dye, YOYO-1, were first developed using Enterococcus faecalis, an organism that has previously b
61 he BC-GP correctly identified 14/15 cases of Enterococcus faecalis and Enterococcus faecium bacteremi
62 m for identification of vancomycin-resistant Enterococcus faecalis and Enterococcus faecium, VRESelec
63 coated quartz sand (IOCS) to remove two FIB (Enterococcus faecalis and Escherichia coli) suspended in
69 amma-GCS domain flexible loop sequences from Enterococcus faecalis and Pasteurella multocida gamma-GC
70 , involved in polysaccharide biosynthesis of Enterococcus faecalis and showed that disruption of epaB
71 kinetics of the d-Ala:d-Ser ligase VanG from Enterococcus faecalis and solved its crystal structure i
73 -positive bacteria Staphylococcus aureus and Enterococcus faecalis and two Gram-negative bacteria Esc
75 of MetAPs from Mycobacterium tuberculosis , Enterococcus faecalis , and human, three hotspots have b
76 g a microbe with host-protective properties (Enterococcus faecalis) and a pathogen (Staphylococcus au
77 genic bacteria (Photorhabdus luminescens and Enterococcus faecalis) and two nonpathogenic bacteria (E
78 roscan, 87% of Staphylococcus aureus, 90% of Enterococcus faecalis, and 88% of Enterococcus faecium i
79 us aureus (MRSA), Listeria monocytogenes and Enterococcus faecalis, and against the Gram-negative bac
80 Bacillus anthracis, Propionibacterium acnes, Enterococcus faecalis, and both Methicillin-sensitive an
81 ve bacteria, including Bacillus subtilis and Enterococcus faecalis, and drug-sensitive and drug-resis
82 survival of non-pathogenic Escherichia coli, Enterococcus faecalis, and E. coli O157:H7 were compared
83 occus epidermidis, Streptococcus pneumoniae, Enterococcus faecalis, and Enterococcus faecium) and thr
85 tion by a pure facultative anaerobic strain, Enterococcus faecalis, and fresh mixed anaerobic sludge,
88 9.65, and 100.00% for Staphylococcus aureus, Enterococcus faecalis, and streptococci, respectively.
89 te genome plasticity in antibiotic resistant Enterococcus faecalis, and their involvement has been im
90 coccus aureus, Streptococcus pneumoniae, and Enterococcus faecalis, and thus is considered as a poten
91 tive transfer and virulence functions of the Enterococcus faecalis antibiotic resistance plasmid pCF1
92 resistant "superbugs." Bacteria of the genus Enterococcus faecalis are highly antibiotic-resistant no
95 d-scan EPR can detect superoxide produced by Enterococcus faecalis at rates that are too low for dete
96 s aureus ATCC 29213 (0.004 to 0.015 mug/ml), Enterococcus faecalis ATCC 29212 (0.015 to 0.06 mug/ml),
97 cus aureus ATCC 29213 (0.03 to 0.12 mug/ml), Enterococcus faecalis ATCC 29212 (0.03 to 0.12 mug/ml),
98 sk only), S. aureus ATCC 29213 (broth only), Enterococcus faecalis ATCC 29212 (broth only), Streptoco
99 eudomonas aeruginosa ATCC 27853, 9 to 13 mm, Enterococcus faecalis ATCC 29212, 0.03 to 0.12 microg/ml
101 4028, Staphylococcus epidermidis ATCC 12228, Enterococcus faecalis ATCC 29212, and Escherichia coli D
102 pecies, including Pseudomonas putida KT2440, Enterococcus faecalis ATCC 29212, Salmonella Typhimurium
103 tracts showed antimicrobial activity against Enterococcus faecalis, Bacillus subtilis, Escherichia co
104 nted here had either Enterococcus faecium or Enterococcus faecalis bacteremia caused by both vancomyc
107 vancomycin-resistant Enterococcus faecium or Enterococcus faecalis, based on distinct colony colors.
109 ers were then shown to inhibit the growth of Enterococcus faecalis biofilms that play a role in early
110 photoinactivation of a laboratory strain of Enterococcus faecalis, but depressed photoinactivation o
111 iorated sepsis-induced death associated with Enterococcus faecalis, but not Escherichia coli, infecti
112 and vanA or vanB in Enterococcus faecium and Enterococcus faecalis) by the BC-GP assay also was asses
114 A gene sequencing identified the isolates as Enterococcus faecalis, Cardiobacterium valvarum, and Str
115 tural snapshots from the type II-A system of Enterococcus faecalis Cas1 and Cas2 during spacer integr
118 Upon sensing of the peptide pheromone cCF10, Enterococcus faecalis cells carrying pCF10 produce three
120 alf-life of the metK transcript in vivo when Enterococcus faecalis cells were depleted for SAM, but n
123 ructure of the ligand binding segment of the Enterococcus faecalis collagen binding MSCRAMM ACE (micr
125 The ethanolamine utilization (eut) locus of Enterococcus faecalis, containing at least 19 genes dist
126 an SDMH is involved in biofilm formation in Enterococcus faecalis, contributing to oxidant productio
127 on from the antagonistic prgX/prgQ operon in Enterococcus faecalis controlling conjugative transfer o
129 nt bioinformatic studies have suggested that Enterococcus faecalis could serve as a model system to b
130 present in other pathogenic bacteria such as Enterococcus faecalis, Coxiella burnetii, and Clostridiu
132 In other gram-positive bacteria, such as Enterococcus faecalis, disulfide bonds are formed in sec
133 ngly difficult to treat infections caused by Enterococcus faecalis due to its high levels of intrinsi
134 port and retention behavior of two model FIB Enterococcus faecalis (E. faecalis) and Escherichia coli
135 us aureus, coagulase-negative staphylococci, Enterococcus faecalis, E. faecium, E. avium, E. durans,
136 ins of diverse origin, including the species Enterococcus faecalis, E. faecium, E. casseliflavus, and
137 We recently demonstrated that the ubiquitous Enterococcus faecalis ebp (endocarditis- and biofilm-ass
138 inosa (Pa), Legionella pneumophila (Lp), and Enterococcus faecalis (Ef) by using anti-infective, anti
140 The pheromone-responsive plasmid pCF10 of Enterococcus faecalis encodes a putative cell wall hydro
141 transposon Tn916 from the bacterial pathogen Enterococcus faecalis, encodes a putative ArdA (alleviat
142 Expression of ace (adhesin to collagen of Enterococcus faecalis), encoding a virulence factor in e
143 ated that the ebp operon and the ace gene of Enterococcus faecalis, encoding endocarditis- and biofil
145 re several species of enterococci, including Enterococcus faecalis, Enterococcus faecium, Enterococcu
146 dis, Staphylococcus aureus, Bacillus cereus, Enterococcus faecalis, Enterococcus faecium, Listeria mo
147 coccus pneumoniae, a viridans streptococcus, Enterococcus faecalis, Enterococcus faecium, Streptococc
148 d wild-type control mice monoassociated with Enterococcus faecalis , Escherichia coli , or Pseudomona
149 ides thetaiotaomicron, Campylobacter jejuni, Enterococcus faecalis, Escherichia coli K12, E. coli O15
150 ved with the type of bacterium in the order: Enterococcus faecalis, Escherichia coli O157:H7, and Esc
151 es to PG composition in vancomycin-resistant Enterococcus faecalis following the growth in presence o
152 al species (seven Escherichia coli and three Enterococcus faecalis ) for all ten patient samples.
153 TDB, Escherichia coli, Bacillus subtilis and Enterococcus faecalis, from the guts of the desert woodr
155 (MIC) against Staphylococcus aureus GC 1131, Enterococcus faecalis GC 2242, Streptococcus pneumoniae
159 ia coli (group A, 38.4%; group B, 39.3%) and Enterococcus faecalis (group A, 32.7%; group B, 33.2%).
160 ndocarditis due to vancomycin-resistant (VR) Enterococcus faecalis has only rarely been reported.
162 protein, Esp, enhances biofilm formation by Enterococcus faecalis in a glucose-dependent manner.
163 positive organisms Staphylococcus aureus and Enterococcus faecalis in comparison with known analogues
164 of the pheromone receptor protein PrgZ from Enterococcus faecalis in complex with the heptapeptide c
165 lar function in Streptococcus agalactiae and Enterococcus faecalis In conclusion, the elucidation of
166 pheromone plasmids increase the virulence of Enterococcus faecalis in experimental pathogenesis model
168 a, Salmonella enterica, Yersinia pestis, and Enterococcus faecalis, indicating that HSF-1 is part of
169 induce high levels of (p)ppGpp production in Enterococcus faecalis, indicating that this nosocomial p
170 e, infection of catheter-implanted mice with Enterococcus faecalis induced the specific expression of
171 icantly decreased the burden of a subsequent Enterococcus faecalis infection in the nematode intestin
172 us gentamicin (AG) combinations for treating Enterococcus faecalis infective endocarditis (EFIE).
173 study, we compare outcomes in patients with Enterococcus faecalis infective endocarditis treated in
186 One of the well-studied virulence factors of Enterococcus faecalis is a secreted bacterial protease,
187 e (SAS) RelQ from the Gram-positive pathogen Enterococcus faecalis is a sequence-specific RNA-binding
199 olling pheromone-induced plasmid transfer in Enterococcus faecalis is the most thoroughly studied gen
203 e of a clinical pair of vancomycin-resistant Enterococcus faecalis isolates from the blood of a patie
206 , Clostridium species, Enterobacter cloacae, Enterococcus faecalis, Klebsiella oxytoca, Klebsiella pn
207 ecent clinical isolates of Escherichia coli, Enterococcus faecalis, Klebsiella pneumoniae, and Pseudo
208 Staphylococcus aureus, Enterococcus faecium, Enterococcus faecalis, Klebsiella pneumoniae, Pseudomona
209 ntestinal microbiota (Lactobacillus reuteri, Enterococcus faecalis, Lactobacillus crispatus and Clost
211 creasing sensitivity in the following order: Enterococcus faecalis LDH2 </= Lactococcus lactis LDH2 <
213 a monoclonal antibody developed against the Enterococcus faecalis major pilus protein EbpC, we ident
217 otic, the structure of HMG-CoA synthase from Enterococcus faecalis (MVAS) was determined by selenomet
219 lope homeostasis), from daptomycin-resistant Enterococcus faecalis not only reversed resistance to 2
221 ed molecular patterns, including heat-killed Enterococcus faecalis or CpG DNA, led to increased Ikapp
222 those of mild SIRS mice after infection with Enterococcus faecalis or methicillin-resistant Staphyloc
230 onoclonal antibody to the major component of Enterococcus faecalis pili, EbpC, labels polymerized pil
233 ion of the conjugative transfer genes of the Enterococcus faecalis plasmid pCF10 in response to an in
236 e (AS), a plasmid-encoded surface protein of Enterococcus faecalis, plays important roles in virulenc
239 d the GIT microbiota of MAT mothers, whereas Enterococcus faecalis predominated within the MAT infant
240 EfbA is a PavA-like fibronectin adhesin of Enterococcus faecalis previously shown to be important i
242 ost & Microbe, Keogh et al. (2016) show that Enterococcus faecalis promotes Escherichia coli biofilm
243 uconostoc mesenteroides, Bacillus cereus and Enterococcus faecalis proving its antimicrobial action.
244 species such as Staphylococcus epidermidis, Enterococcus faecalis, Pseudomonas aeruginosa, and Klebs
245 the AMP-modifications with Escherichia coli, Enterococcus faecalis, Pseudomonas aeruginosa, Staphyloc
246 atural enterococci diversity, 11 isolates of Enterococcus faecalis recovered from freshwater watershe
251 polysaccharide antigen (epa) gene cluster of Enterococcus faecalis strain OG1RF was shown to be atten
254 level of expression of conjugation genes in Enterococcus faecalis strains carrying the pheromone-res
255 of five E. faecium strains but none of five Enterococcus faecalis strains consistently developed res
260 including E. coli, P. aeruginosa, S. aureus, Enterococcus faecalis, Streptococcus pyogenes, Streptoco
261 aive or inactivated Staphylococcus aureus or Enterococcus faecalis, suggesting that in vivo, Eater di
263 endocarditis- and biofilm-associated pili of Enterococcus faecalis that are also important in experim
265 y for growth of the human bacterial pathogen Enterococcus faecalis The final enzyme in this pathway,
267 pathogenic microorganisms: the Gram-positive Enterococcus faecalis, the Gram-negative Escherichia col
270 st infection with the opportunistic pathogen Enterococcus faecalis through promotion of host-microbio
273 on on the antibacterial host defense against Enterococcus faecalis translocation was investigated.
276 e setup was tested with Escherichia coli and Enterococcus faecalis, two bacterial strains that are co
277 usly, our research demonstrated that dietary Enterococcus Faecalis UC-100 substituting antibiotics en
278 ons were tested against Escherichia coli and Enterococcus faecalis urinary tract infection isolates.
279 e required for DAF-16-mediated resistance to Enterococcus faecalis using a C. elegans killing assay.
280 served that a female was mono-colonized with Enterococcus faecalis vaginally as tested in aerobic cul
281 cus anginosus group (</=0.06 microg/mL), and Enterococcus faecalis (vancomycin susceptible, </=0.25 m
282 tient with recalcitrant vancomycin-resistant Enterococcus faecalis (VRE) and 2 patients with infectio
288 l and pathogenic host-microbe interaction of Enterococcus faecalis was explored using a Caenorhabditi
291 omonas gingivalis and the endodontic species Enterococcus faecalis were grown to early log phase and
294 ies report that 80 to 90% of enterococci are Enterococcus faecalis, whereas E. faecium accounts for 5
295 vibrios, type E Clostridium perfringens, and Enterococcus faecalis, whereas the reverse was true for
296 adenovirus 41, Phi X 174) and the bacterium Enterococcus faecalis, which are relevant for water hygi
297 n Tn1546, acquired from vancomycin-resistant Enterococcus faecalis, which is known to alter cell wall
298 n pCF10, an antibiotic resistance plasmid of Enterococcus faecalis, which negatively regulates conjug
299 ms are absent from multidrug-resistant (MDR) Enterococcus faecalis, which only possess an orphan CRIS
300 g of one organism containing an orphan SelD, Enterococcus faecalis, with 75Se revealed a protein cont
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