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1 lla zoster virus, human cytomegalovirus, and Epstein-Barr virus.
2 sine patch, which is absent in EBNA-1 of the Epstein-Barr virus.
3 such as varicella zoster virus and possibly Epstein-Barr virus.
4 tation of epitopes from different strains of Epstein-Barr virus.
5 LTL attrition, with no association found for Epstein-Barr virus.
6 ovirus, adenovirus, human herpesvirus 6, and Epstein-Barr virus.
7 virus, norovirus, rotavirus, parvovirus, and Epstein-Barr virus.
8 %; human herpesvirus [HHV] 6, 18%; HHV8, 6%; Epstein-Barr virus, 3%; herpes simplex virus 1, 3%; vari
9 o prevent infection with and reactivation of Epstein-Barr virus and cytomegalovirus and their associa
10 a conserved domain of EBNA-3B and ICP4 from Epstein-Barr virus and herpes simplex virus 1, respectiv
12 mology to a conserved domain of EBNA-3B from Epstein-Barr virus and ICP4 from herpes simplex virus 1,
13 estricted (407)HPVGEADYFEY(417) epitope from Epstein-Barr virus and naturally occurring variants at p
16 simplex, varicella zoster, cytomegalovirus, Epstein-Barr virus and Toxoplasma gondii in patients wit
20 rkel cell carcinoma-associated polyomavirus, Epstein-Barr virus, and Kaposi's sarcoma-associated herp
24 atural killer (NK) cell lymphoma (NNL) is an Epstein-Barr virus-associated lymphoma of cytotoxic NK c
25 Lymphomatoid granulomatosis (LYG) is a rare Epstein-Barr virus-associated lymphoproliferative disord
28 , 1 CSF specimen was positive for EV-D68 and Epstein-Barr virus by real-time polymerase chain reactio
29 Many double-stranded DNA viruses, such as Epstein-Barr virus, can establish persistent infection,
31 at least 1 positive viremia during follow-up.Epstein-Barr virus D+/R- patients (P = 0.046) as well as
34 , TSPyV, HPyV9, HPyV10) and 5 herpesviruses (Epstein Barr virus (EBV), cytomegalovirus (CMV), herpes
35 nancy (mean gestational age = 11.1 weeks) to Epstein Barr virus (EBV), herpes simplex virus types 1 a
37 subgroup (HR = 1.80, 95% CI = 1.36-2.38) and Epstein-Barr virus (EBV) (+) > 5% subgroup (HR = 1.59, 9
38 of replication of cytomegalovirus (CMV) and Epstein-Barr virus (EBV) (the most frequently detected H
39 P1) expression as observed in tumor cells of Epstein-Barr virus (EBV) -associated HL, we analyzed 3-d
40 s (EBNAs) encoded by the lymphoma-associated Epstein-Barr virus (EBV) activate MYC and silence BCL2L1
42 uman herpes virus (HHV)-6, HHV-7, chlamydia, Epstein-Barr virus (EBV) and bacterial 16S ribosomal DNA
43 to CD8 T cells specific for control viruses, Epstein-Barr virus (EBV) and cytomegalovirus (CMV), and
47 Clinically available drugs active against Epstein-Barr virus (EBV) and other human herpesviruses a
48 Endemic Burkitt lymphoma is associated with Epstein-Barr virus (EBV) and Plasmodium falciparum coinf
53 In the oral epithelium, peripheral stores of Epstein-Barr virus (EBV) are transmitted from infiltrati
55 ted that infants in Kenya were infected with Epstein-Barr virus (EBV) at <6 months of age, suggesting
56 between polyomavirus JC (JC virus [JCV]) and Epstein-Barr virus (EBV) at sequences of JCV found infec
66 ived T cells specific for cytomegalovirus or Epstein-Barr virus (EBV) could effectively restore virus
67 We show that both 5' and 3' derivatives from Epstein-Barr virus (EBV) encoded miR-BART-18 precursor m
75 MP1) gene founder sequences and the level of Epstein-Barr virus (EBV) genome variability over time an
76 e bond (DB) C278/C335 in domain II (D-II) of Epstein-Barr virus (EBV) gH has an epithelial cell-speci
79 al cells in infection and persistence of the Epstein-Barr virus (EBV) have long been difficult to res
80 om anal samples; 2.7% (95% CI: 0.7-4.7%) for Epstein-Barr virus (EBV) immunoglobulin M (IgM) positivi
83 cells targeting viral antigens derived from Epstein-Barr virus (EBV) induce sustained complete tumor
86 essive head and neck cancer characterized by Epstein-Barr virus (EBV) infection and dense lymphocyte
87 al carcinoma (NPC) is highly associated with Epstein-Barr virus (EBV) infection and exhibits remarkab
92 HSCT) provides a unique opportunity to track Epstein-Barr virus (EBV) infection in the context of the
98 nked immunodeficiency with magnesium defect, Epstein-Barr virus (EBV) infection, and neoplasia' (XMEN
99 rine gammaherpesvirus 68 (MHV68), a model of Epstein-Barr virus (EBV) infection, and then after laten
100 , the patient developed primary asymptomatic Epstein-Barr virus (EBV) infection, followed by EBV+ B-c
105 R) capable of diagnosing different stages of Epstein-Barr virus (EBV) infections in clinical serum sa
112 erial artificial chromosome (BAC).IMPORTANCE Epstein-Barr virus (EBV) infects the majority of the wor
125 replication of the human gamma herpes virus Epstein-Barr virus (EBV) is an essential prerequisite fo
135 novel mechanisms of host control.IMPORTANCE Epstein-Barr virus (EBV) is transmitted orally, replicat
138 program and DNA methylation state.IMPORTANCE Epstein-Barr virus (EBV) latency and carcinogenesis invo
143 In this study, we provide evidence that Epstein-Barr virus (EBV) latent membrane protein 1 (LMP1
151 es reactivation of the oncogenic herpesvirus Epstein-Barr virus (EBV) out of latency into lytic repli
152 defects often can be overcome by adding the Epstein-Barr virus (EBV) partitioning element, FR (i.e.,
155 m patient samples the degree of diversity in Epstein-Barr virus (EBV) populations and the extent to w
157 rogenically impaired immune surveillance and Epstein-Barr virus (EBV) primary infection/reactivation
162 Recent studies reported that posttransplant Epstein-Barr virus (EBV) replication is frequent and ind
163 ect of subclinical cytomegalovirus (CMV) and Epstein-Barr virus (EBV) replication on CD4(+) and CD8(+
171 sequences from an enlarged set of about 200 Epstein-Barr virus (EBV) strains, including many primary
174 ecause of studies on the contribution of the Epstein-Barr virus (EBV) to the aggressiveness of nasoph
180 he oral cavity is a persistent reservoir for Epstein-Barr virus (EBV) with lifelong infection of resi
183 that recognized five common viral pathogens: Epstein-Barr virus (EBV), adenovirus (AdV), cytomegalovi
186 ons in humans such as cytomegalovirus (CMV), Epstein-Barr virus (EBV), and adenovirus, among others.
187 viral replication of cytomegalovirus (CMV), Epstein-Barr virus (EBV), and BK polyomavirus (BKV) at t
189 he viral reactivation rates of HHV-6, HHV-7, Epstein-Barr virus (EBV), and cytomegalovirus (CMV) in d
190 es, including human papillomaviruses (HPVs), Epstein-Barr virus (EBV), and hepatitis B and C viruses
191 ection of closely related DNA viruses: KSHV, Epstein-Barr virus (EBV), and herpes simplexvirus-2 (HSV
193 Most people in the world are infected by Epstein-Barr virus (EBV), and it causes several human di
194 ses, including human cytomegalovirus (HCMV), Epstein-Barr virus (EBV), and Kaposi's sarcoma-associate
195 HIV) subtypes (A, B, C, D, E, G, and panel), Epstein-Barr Virus (EBV), and Kaposi's Sarcoma-associate
196 iral response to HSV-1 and the related virus Epstein-Barr virus (EBV), as well as influenza A virus (
197 itative PCR assays of cytomegalovirus (CMV), Epstein-Barr virus (EBV), BK virus (BKV), adenovirus (AD
198 or herpes simplex virus (HSV) types 1 and 2, Epstein-Barr virus (EBV), cytomegalovirus (CMV), HHV-6A,
201 analysis for toxoplasmosis, cytomegalovirus, Epstein-Barr virus (EBV), herpes simplex virus I and II,
202 viremia episodes with cytomegalovirus (CMV), Epstein-Barr virus (EBV), human herpesvirus 6 (HHV-6), h
204 dings of previously identified infections by Epstein-Barr virus (EBV), Kaposi's sarcoma herpesvirus (
206 In B cells infected by the cancer-associated Epstein-Barr virus (EBV), RUNX3 and RUNX1 transcription
207 antigens from ubiquitous pathogens, such as Epstein-Barr virus (EBV), that persist in their host and
209 logic homology to the human gammaherpesvirus Epstein-Barr virus (EBV), which infects over 90% of huma
210 n follicular helper T cells (TFH cells), and Epstein-Barr virus (EBV), which persists in B cells.
212 's sarcoma-associated herpesvirus (KSHV) and Epstein-Barr virus (EBV)--is vital to capsid assembly, y
214 Furthermore, oncogenic viruses, including Epstein-Barr virus (EBV)-associated human cancers, were
215 ted B-cell immune dysregulation during acute Epstein-Barr virus (EBV)-associated infectious mononucle
218 t peripheral blood (PB) T cells to eliminate Epstein-Barr virus (EBV)-driven human B-cell lymphoma in
219 triggered by the gly-ala repeat sequence of Epstein-Barr virus (EBV)-encoded EBNA1, results in PI3Kd
223 s been found to regulate lytic activation of Epstein-Barr virus (EBV)-infected cells, we asked if STA
227 sed throughout infection, can be detected in Epstein-Barr virus (EBV)-positive tumors, and manipulate
229 vely, detected in cytomegalovirus (CMV)- and Epstein-Barr virus (EBV)-responsive CD4+ T cells followi
230 nt of LUBAC, interacts with LMP1 and IRF7 in Epstein-Barr virus (EBV)-transformed cells and that LUBA
239 osi's sarcoma-associated herpesvirus [KSHV], Epstein-Barr virus [EBV], and herpes simplex virus 1 [HS
240 titis B core [HBc], hepatitis C virus [HCV], Epstein-Barr virus [EBV], or cytomegalovirus [CMV]) in K
244 rom both adenovirus-associated (VA) RNAs and Epstein-Barr virus-encoded small RNAs (EBERs) with respe
245 lyssavirus, herpes simplex viruses 1 and 2, Epstein-Barr virus, enterovirus, cytomegalovirus, and ch
246 ctors included a high prevalence of previous Epstein-Barr virus exposure and a relatively low immunol
247 ere, we review our understanding of the path Epstein-Barr virus follows to move from a latent infecti
248 eveloped a novel helper-dependent adenovirus/Epstein-Barr virus (HDAd/EBV) hybrid reprogramming vecto
249 in Xenopus tropicalis, human cell lines, and Epstein-Barr virus; however, the biological significance
252 positive test results for cytomegalovirus or Epstein-Barr virus, indicating possible cross-reactivity
254 ll migration, the G protein-coupled receptor Epstein-Barr virus-induced gene 2 (EBI2 or GPR183) direc
257 se that expressed the extrafollicular marker Epstein-Barr virus-induced protein 2 (EBI2), but signifi
258 report that in human airway epithelial cells Epstein-Barr virus induces TRIM29, a member of the TRIM
261 g intensity of immunosuppression, control of Epstein-Barr virus infection among transplant recipients
264 cells from a Burkitt's lymphoma with latent Epstein-Barr virus infection showed dramatic proliferati
265 s, such as the incidence of acute rejection, Epstein-Barr virus infection, sepsis, biliary and vascul
270 nt for diverse functions of EBNA1.IMPORTANCE Epstein-Barr virus is a human gammaherpesvirus that is c
271 r efficient viral DNA replication.IMPORTANCE Epstein-Barr virus is the causative agent of infectious
273 ns can be modulated by viral proteins (e.g., Epstein-Barr virus LMPs) to favor spread of transformed
275 ent of the requirement for expression of the Epstein-Barr Virus nuclear antigen (EBNA), making it par
276 In contrast to homologous proteins such as Epstein-Barr virus nuclear antigen 1 (EBNA-1) of the rel
277 n vIRF1, which is identical to that found in Epstein-Barr virus nuclear antigen 1 (EBNA1) that intera
281 utologous VSTs specific for cytomegalovirus, Epstein-Barr virus, or adenovirus and genetically modifi
283 tional Cancer Institute describe a series of Epstein-Barr virus-positive (EBV+) diffuse large B-cell
286 Lee et al. show that a noncoding RNA from Epstein-Barr virus recruits a host transcription factor
288 bodies (aIRR = 2.03); in recipients who were Epstein-Barr virus-seronegative at the time of transplan
289 1 (EBNA-1) of the related gamma-herpesvirus Epstein-Barr virus, specific DNA recognition by LANA is
291 ibrary using phage display technology and by Epstein-Barr virus transformation of switched memory B c
292 T3) and mRNA levels of STAT3 gene targets in Epstein-Barr virus-transformed B (EBV) cells, human peri
293 HLA-A*02:01 and HLA-B*27:05 expressed on the Epstein-Barr virus-transformed B cell line Jesthom and M
294 eoplastic (human papillomavirus+ tumors 35%, Epstein-Barr virus+ tumors 4%), vascular/lymphatic (veno
295 HSV UL37 with the human cytomegalovirus and Epstein-Barr virus UL37 homologs revealed that Y480 was
298 tion to human cytomegalovirus, BK virus, and Epstein-Barr virus, while the importance of defining the
299 virus type 1, human herpesvirus type 6, and Epstein-Barr virus) with change in leukocyte telomere le
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