ライフサイエンスコーパス: ErbB3 receptor

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1 these cells to the bone marrow requires the ErbB3 receptor.

2 d phosphorylated Thr-871 and Ser-1120 in the ErbB3 receptor.

3 activation of the PI3-kinase pathway by the ErbB3 receptor.

4 neuregulin-1beta (NRG1beta) binding to ErbB2/ErbB3 receptors.

5 hat HRGbeta1 activates signaling through the ErbB3 receptor and influences receptor trafficking.

6 The ErbB3 receptor and the downstream signaling kinase Akt a

7 uronan to facilitate signaling through ErbB2/ErbB3 receptors and then is terminated by an increase in

8 ErbB2 heterodimerizes with and activates the ErbB3 receptor, and the two receptors synergize in promo

9 of EGF by up-regulating EGFR, c-ErbB2 and c-ErbB3 receptors, and by enhancing EGF-stimulated tyrosin

10 , are phenocopied by loss of oligodendrocyte ErbB3 receptors, and social isolation leads to reduced e

11 Our analysis also indicates that ERBB3 receptors are apparently segregated from ERBB2 rec

12 Ebp1, an ErbB3 receptor-binding protein, inhibits cell proliferat

13 Ebp1, an ErbB3 receptor-binding protein, inhibits cell proliferat

14 Ebp1, an ErbB3 receptor-binding protein, inhibits the proliferati

15 tein identified by its interactions with the ErbB3 receptor, down-regulates expression of AR and AR-r

16 (HRG) -mediated ErbB3 signaling and inducing ErbB3 receptor downregulation.

17 induces the relocalization of the ErbB2 and ErbB3 receptors from intracellular compartments to the p

18 cted with adenoviral particles containing an ErbB3 receptor gene or a vehicle beta-galactosidase gene

19 al is apparently transduced through an ErbB2/ErbB3 receptor heterodimer.

20 his work, we altered the levels of ErbB2 and ErbB3 receptors, individually and in combination, by usi

21 ity, heregulin-beta (HRGbeta) binding to the erbB3 receptor initiated rapid and potent induction of b

22 nds the soluble extracellular domains of the ERBB3 receptor into a targeted and highly specific cross

23 th ATFs, the effects of changes in ErbB2 and ErbB3 receptor levels were evaluated by using cell proli

24 Ebp1-knockdown significantly reduced ErbB3 receptor levels, yet over-expression of ErbB3 in E

25 inases, Schwann cells express both erbB2 and erbB3 receptors over the entire interval studied.

26 turally occurring secreted form of the human ErbB3 receptor, p85-soluble ErbB3 (sErbB3), is a potent

27 Stimulation of Schwann cell ErbB3 receptor phosphorylation by SMDF was not affected

28 howed a specific interaction of Shc with the ErbB3 receptor protein and demonstrated the importance o

29 In this study, mutant ErbB3 receptor proteins expressed in COS7 cells were use

30 and L3.6pl cells, which highly expressed the ErbB3 receptor, significant reduction in cell viability,

31 he tyrosine phosphorylation of the ErbB2 and ErbB3 receptors to similar extents, but only NRG1beta po

32 Our in vivo study used an ErbB3 receptor transfection strategy to determine if top

33 iology, we are studying the specific role of ErbB3 receptor tyrosine kinase (RTK) since it is the rec

34 The ErbB3 receptor tyrosine kinase contributes to a variety

35 Overexpression of the ErbB3 receptor tyrosine kinase protein in breast and oth

36 The ErbB2 and ErbB3 receptor tyrosine kinases act synergistically to p

37 the insulin-like growth factor-I (IGF-I) and ErbB3 receptor tyrosine kinases, and prolonged the assoc

38 D44 constitutively associated with erbB2 and erbB3, receptor tyrosine kinases that heterodimerize and

39 ee receptors were expressed, though only the erbB3 receptor was phosphorylated, suggesting that overe

40 ulate the trafficking or localization of the ErbB3 receptor, we have identified a tripartite or RBCC

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