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1 I-E CRISPR/Cas system of the model bacterium Escherichia coli K12.
2 (the Rut pathway) was recently discovered in Escherichia coli K12.
3 y running it over 2390 promoter sequences of Escherichia coli K12.
4 and transcriptional control of metabolism in Escherichia coli K12.
5 on, we identified recently formed operons in Escherichia coli K12.
6 e biology of the prototypical model organism Escherichia coli K12.
7 rovar Typhi (S. typhi), and 29% missing from Escherichia coli K12.
8 y against Listeria monocytogenes Scott A and Escherichia coli K12.
9 approximately 4.5% of Yersinia pestis, 5% of Escherichia coli K12, 6% of Archaeoglobus fulgidus, 8% o
13 arly, we examined a wild-type (WT) strain of Escherichia coli K12 and mutant strains with lesions aff
14 from a well-characterized list of operons in Escherichia coli K12 and perform comparative analysis of
15 ing the preponderance of LRCs in the GRNs of Escherichia coli K12 and several other organisms, we fin
16 PS to characterize the chemical stability of Escherichia coli K12 antibodies on the surfaces of diamo
17 d biosensor that can reliably detect a model Escherichia coli K12 bacterium using EIS, positioning th
18 s can escape type I-E CRISPR-Cas immunity in Escherichia coli K12 by making point mutations in the se
19 -A+/+ mice increased the permeability of the Escherichia coli K12 cell membrane to a greater extent t
20 yte cells and of the gyr B gene in bacterial Escherichia coli K12 cells establishes that SCGA will al
23 particular, we predicted 158 uber-operons in Escherichia coli K12 covering 1830 genes, and found that
24 Induction of antibacterial activity against Escherichia coli K12 D31 was 7.5 times greater in pupal
26 mulated reads from real genomes and a PacBio Escherichia coli K12 dataset demonstrate that Not-So-Gre
27 The basolateral addition of LPS (ultrapure Escherichia coli K12) decreased HCO(3)(-) absorption in
28 -SIGN) specifically binds to the core LPS of Escherichia coli K12 (E. coli), promoting bacterial adhe
29 Campylobacter jejuni, Enterococcus faecalis, Escherichia coli K12, E. coli O157:H7, Salmonella enteri
32 of almost all SlyA-activated genes from the Escherichia coli K12 genome suggests that the functional
33 to 94% of known cis-regulatory motifs in the Escherichia coli K12 genome, while achieving high predic
36 our prediction, we have tested the method on Escherichia coli K12, in which operon structures have be
40 teria tested, including both Gram-negatives (Escherichia coli K12, K1 and Salmonella typhimurium) and
41 1, and a nonmotile (with paralyzed flagella) Escherichia coli K12 MG1655 DeltamotA that is incapable
42 ity were selected, including a highly motile Escherichia coli K12 MG1655, an environmental strain Sph
44 mutations conferring rifampin resistance in Escherichia coli K12 (MG1655) and explores the nature of
45 s, phagocytosed PLA-expressing Y. pestis and Escherichia coli K12 more efficiently than PLA-negative
46 ated products of each of the 4,290 predicted Escherichia coli K12 open reading frames (ORFs) to measu
47 Here, the percent cell viability loss of Escherichia coli K12 resulting from the interaction with
51 are encoded in both the 16S and 23S rRNAs of Escherichia coli K12; that nucleotide sequences that cou
52 encing of bisulfite-treated genomic DNA from Escherichia coli K12 to describe, for the first time, th
53 pplied to gene expression data obtained from Escherichia coli K12 undergoing a carbon source transiti
54 igned and constructed a synthetic circuit in Escherichia coli K12, using glycolytic flux to generate
56 inding periplasmic binding protein (LivK) of Escherichia coli K12 was flanked with CFP (cyan fluoresc
59 of colonic epithelial cells challenged with Escherichia coli K12, we showed that inhibition of histo
61 if to an F(+), alpha-complementing strain of Escherichia coli K12, which expresses the omega-domain o
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