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1 EtBr further reduced already low levels of mtDNA in polg
2 esults obtained on complexes consisting of 1 EtBr bound/10 bp leads to the following conclusions: (i)
3 nergy transfer (FRET) between the C-dots and EtBr was studied, in which the C-dots serve as an excita
4 nt nucleophilic reactions with Me(3)SiCl and EtBr to form the corresponding disubstituted products.
5 hannel opening was transient with astrocytic EtBr uptake observed near the abscess at day 3 but not d
8 and the intercalating drug ethidium bromide (EtBr) have been determined by use of a Raman microscope
9 2+) complexation inhibited ethidium bromide (EtBr) intercalation and stabilized FdU-substituted duple
10 ing the intercalating drug ethidium bromide (EtBr) to 160 base pair (bp) fragments of nucleosomal cal
11 e activity assay utilizing ethidium bromide (EtBr) to deplete mtDNA, showed that polg(+/-) and WT zeb
12 ity as evident by enhanced ethidium bromide (EtBr) uptake that could be blocked by several pharmacolo
18 measured using a small fluorescent molecule (EtBr), eliminating the possibility of steric and size ef
20 Quantification of structure parameters of EtBr and DNA in the complex were assessed using Raman te
24 NS inhibited the transport of some (pyronin, EtBr) but not other (ANS, Leu-Nap) substrates of the pum
26 t 752 nm, vibrational signatures of both the EtBr intercalant and DNA target have been identified in
27 ch were obtained from oriented fibers of the EtBr:DNA complex, are interpreted in terms of the relati
29 converted from the B type to the A type with EtBr binding, commensurate with the proportion of ethidi
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