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1 ro (P0) and/or fatty acid binding protein 7 (Fabp7).
2 in with characteristics distinct from native FABP7.
3 in strongly reduced expression of Sox10 and Fabp7.
4 gh Sox10 levels and to trigger expression of Fabp7.
5 melanomas by immunohistochemistry with anti-FABP7 Ab showed 73 and 27% positivity, respectively (P<0
6 ic analysis as fatty acid binding protein 7 (FABP7), also known as brain lipid binding protein) which
7 he brain fatty acid-binding protein (B-FABP; FABP7) and glial fibrillary acidic protein (GFAP) genes
8 s, CSDE1 binds fatty acid binding protein 7 (FABP7) and vimentin (VIM) mRNAs, as well as transcripts
9 urther investigated one gene from the group, FABP7, and confirmed its association with survival in tw
22 astrocyte brain fatty acid binding protein (Fabp7) has previously been shown to have a coordinated d
23 ng proteins (FABPs), in particular FABP5 and FABP7, have recently been identified by us as intracellu
24 e expression in cancer and suggest that LTR2-FABP7 may contribute to the pathogenesis of DLBCL in a s
26 In this study, we confirmed an enrichment of Fabp7 mRNA and protein in the astrocytic perisynaptic co
29 lar trafficking and localized translation of Fabp7 mRNA in the tripartite synapse of mammalian brain.
30 e brain, the synchronized cycling pattern of Fabp7 mRNA is a novel discovery among known CPE-regulate
33 ving a fatty acid-binding protein gene (LTR2-FABP7), normally expressed in brain, that was ectopicall
34 d 12 (Cxcl12), fatty acid binding protein 7 (Fabp7), plasma membrane proteolipid (Pllp), and suppress
37 observations, we hypothesized that FABP5 and FABP7 would provide excellent pharmacological targets.
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