ライフサイエンスコーパス: FAE

1 FAE cells also produce chemokines that attract Ag-presen

2 FAE decreased 5-HTT binding sites in the medial nucleus

3 FAE decreased 5-HTT binding sites temporarily in the ven

4 FAE increased 5-HTT binding sites in cortical layers 5,

5 FAE led to distinct effects on 5-HTT sites depending on

6 FAE(XynZ) had similar properties.

7 FAE, all the tested flavonoids except genistein, and two

8 FAE-CBD(XynZ) had a molecular mass of 45 kDa that corres

9 FAEs and ECs are synthesized by a series of endogenous e

10 FAEs are hydrolyzed intracellularly by either fatty acid

11 ranged between 3.9-7.6mug/g and 1299-1607mug FAE/g and was higher for high altitude cultivars.

12 nant feruloyl esterase domain of XynZ alone (FAE(XynZ)) and with the adjacent cellulose binding domai

13 These results show that an FAE M-cell targeting protein Ag delivery system facilita

14 -bound immunoglobulin M restored B cells and FAE development.

15 We show that TLR2 is expressed in both FAE and villus epithelium, but TLR2 activation by peptid

16 ed contextual fear memory deficit induced by FAE, and reversed the hippocampal expression changes in

17 In contrast, FAE increased 5-HTT sites in the lateral nucleus of the

18 vide new MS-based methodologies for in-depth FAE and bsAb formation monitoring.

19 with the adjacent cellulose binding domain (FAE-CBD(XynZ)) were characterized.

20 Flexible airway endoscopy (FAE) is an accepted and frequently performed procedure i

21 These innate responses to flagellin enhance FAE functions and may promote adaptive immune responses

22 vidence that formaldehyde-activating enzyme (FAE) homologs might be involved in methylotrophy.

23 ria/mm(2) of follicle-associated epithelial (FAE) surface were found in three-dimensional reconstruct

24 esent in the follicle-associated epithelium (FAE) above organized conjunctiva-associated lymphoid tis

25 to label the follicle-associated epithelium (FAE) almost exclusively.

26 ncluding the follicle-associated epithelium (FAE) and microfold (M) cells of Peyer's patches, but als

27 asion of the follicle associated epithelium (FAE) by an enteric pathogen and were responsible for the

28 Follicle-associated epithelium (FAE) in the intestinal Peyer's patches contains M cells

29 estricted to follicle-associated epithelium (FAE) of ileal Peyer's patches.

30 through the follicle-associated epithelium (FAE) of Peyer's patch (PP) is the critical first step in

31 s across the follicle-associated epithelium (FAE) of Peyer's patches.

32 pecialized follicular-associated epithelium (FAE) overlying mucosa-associated lymphoid tissues, their

33 The follicle-associated epithelium (FAE) plays key roles in antigen uptake and subsequent in

34 cells in the follicle-associated epithelium (FAE) region of Peyer's patches.

35 The follicle-associated epithelium (FAE) secretes chemokines important in the recruitment of

36 covered by a follicle-associated epithelium (FAE) specialized for the uptake of antigens.

37 specialized follicle-associated epithelium (FAE) that contains M cells, which mediate uptake and tra

38 gions of the follicle-associated epithelium (FAE), although the conditions responsible for expression

39 (BG-12, Tecfidera) is a fumaric acid ester (FAE) that was advanced as a multiple sclerosis (MS) ther

40 s to probe the active site of two esterases (FAE-III and CinnAE) from Aspergillus niger.

41 other unsaturated fatty acid ethanolamides (FAEs) (e.g. linoleoylethanolamide) without affecting tho

42 Fatty acid ethanolamides (FAEs) and endocannabinoids (ECs) have been shown to alle

43 The fatty acid ethanolamides (FAEs) are a family of bioactive lipid mediators that inc

44 nd monounsaturated fatty acid ethanolamides (FAEs), a family of endogenous lipid agonists of peroxiso

45 Amides of fatty acids with ethanolamine (FAE) are biologically active lipids that participate in

46 Fatal adverse events (FAEs) have been reported in cancer patients treated with

47 determine the risk of fatal adverse events (FAEs) in patients with cancer treated with VEGFR TKIs.

48 suprachoroidal space and fluid-air exchange (FAE) was started with sequential increases in infusion a

49 gG4 undergoes a process of Fab-arm exchange (FAE) in which the heavy chains of antibodies of differen

50 undergo a process known as Fab arm exchange (FAE).

51 ittle is known about fetal alcohol exposure (FAE) effects on adult infections.

52 mined the effects of fetal alcohol exposure (FAE) on serotonin transporter (5-HTT) binding sites in t

53 FASD), the result of fetal alcohol exposure (FAE), affects 2-11% of children worldwide, with no effec

54 The autocrine activation of RelB-expressing FAE enterocytes by RANKL/RANK induces the EMT-regulating

55 in Luria-Bertani (LB) broth induce extensive FAE loss and exhibit efficient M-cell invasion, whereas

56 erty of a flavonoid-rich apple peel extract (FAE), its constituents, selected flavonoids and some que

57 ollowing: a 3-methoxy group is essential for FAE-III activity, whereas a 3-methoxy group precluded ac

58 elate to k(cat) for CinnAE (r = 0.33) or for FAE-III (r = 0.43).

59 Differences in Km values for FAE-III were small (0.45-2.08 mM) but there were two ord

60 t conversely, were only able to bind to free FAE-III.

61 Using a human FAE model, we show that the tyrosine kinase HCK regulate

62 IgG4 FAE is suggested to be an important biological mechanism

63 ditions, the S228P mutation can prevent IgG4 FAE to undetectable levels both in vitro and in vivo.

64 ing the pocket were found to be conserved in FAE A from Orpinomyces sp., which further supports the p

65 expression in rhesus macaques was reduced in FAE and, significantly, absent in germinal centers.

66 t1 inhibitor to control neonates resulted in FAE-like deficits in fear memory and hippocampal allele-

67 t invasion of cultured cells, fail to induce FAE destruction and, when inoculated in LB, are attenuat

68 ity and the adult immune system, we infected FAE adult mice with influenza virus.

69 ot entirely understood and studies measuring FAE in ex vivo matrices have been hampered by the presen

70 eatment was higher in phenolic acid (7.36 mg FAE/g), flavonoid (206.74 mug catechin/g), anthocyanin (

71 In the HUVEC model, FAE, quercetin-3-O-glucoside and quercetin-3-O-glucuroni

72 FAE, suggesting that other TLRs may modulate FAE functions.

73 On the other hand, the monounsaturated FAE oleoylethanolamide (OEA) reduces feeding and body we

74 of bacteria increased to 579 +/- 44/mm(2) of FAE surface and they had moved 50% deeper into the folli

75 erminal amino acids, residues 247 to 286, of FAE(XynZ) resulted in protein without activity.

76 hoxy substitutions increased the activity of FAE-III, and decreased the activity of CinnAE; (c) 4-hyd

77 efore, to determine the long-term effects of FAE on disease susceptibility and the adult immune syste

78 n epithelial-mesenchymal transition (EMT) of FAE enterocytes into M cells.

79 We have also demonstrated that the extent of FAE destruction correlates with the extent of M-cell inv

80 ittee members highlighting the importance of FAE-specific airway management techniques and anesthesia

81 To date, the mechanism of FAE is not entirely understood and studies measuring FAE

82 for the first time for online monitoring of FAE and bsAb formation using Hz6F4-2v3 and natalizumab,

83 Null mutants of FAE and homologs revealed that FAE and FAE2 influence th

84 he activity of CinnAE, but decreased that of FAE-III; (d) the rate of hydrolysis with sodium hydroxid

85 The most common causes of FAEs were hemorrhage (23.5%), neutropenia (12.2%), and g

86 ast decades, increasing the concentration of FAEs and ECs through the inhibition of degrading enzymes

87 The incidence of FAEs related to VEGFR TKIs was 1.5% (95% CI, 0.8% to 2.4

88 The overall incidence of FAEs with bevacizumab was 2.5% (95% CI, 1.7%-3.9%).

89 group analysis, no difference in the rate of FAEs was found between different VEGFR TKIs or tumor typ

90 KIs was associated with an increased risk of FAEs compared with control patients.

91 mab was associated with an increased risk of FAEs in patients receiving taxanes or platinum agents (R

92 ut was not associated with increased risk of FAEs when used in conjunction with other agents (RR, 0.8

93 mab was associated with an increased risk of FAEs, with an RR of 1.46 (95% CI, 1.09-1.94; P = .01; in

94 Overall, FAE and most of the flavonoids tested showed ACE inhibit

95 d-type S. typhimurium on mouse Peyer's patch FAE is dependent on the inoculum composition.

96 Development of Peyer's patches, FAE, and M cells was found to be impaired in mice that h

97 of randomized controlled trials in pediatric FAE.

98 lished technical standards on how to perform FAE in children are lacking.

99 delineate technical standards for performing FAE in children.

100 The polyunsaturated FAE anandamide (arachidonoylethanolamide) increases food

101 atal administration of T4 and metformin post FAE affect memory via elevating Dnmt1 and consequently n

102 xine (T4) or metformin to neonatal rats post FAE and rats were tested in the hippocampus-dependent co

103 enyl-4-carboxamide (3) inhibit NAAA, prevent FAE hydrolysis in activated inflammatory cells, and redu

104 ll allow researchers to monitor and quantify FAE of their own IgG4 molecules in physiologically relev

105 evant matrices for assessing and quantifying FAE.

106 A novel method for quantifying FAE using a single MSD immunoassay is also reported and

107 unity, but their possible role in regulating FAE functions is unknown.

108 t male hippocampus, while metformin restored FAE-caused changes in Igf2 expression only.

109 lamide) without affecting those of saturated FAEs (e.g. palmitoylethanolamide).

110 e-buffered saline fail to induce significant FAE disruption and invade M cells at significantly lower

111 min-gamma-positive O55:H7 EPEC also targeted FAE.

112 bility to influenza virus infection and that FAE individuals could be at increased risk for severe an

113 Moreover, we demonstrate that FAE mice have impaired adaptive immune responses, includ

114 ll mutants of FAE and homologs revealed that FAE and FAE2 influence the growth rate and FAE3 influenc

115 Together, our results suggest that FAE induces significant and long-term defects in immunit

116 al neuroprotection as it was recognized that FAEs are capable of activating the antioxidative transcr

117 The FAE can transcytose a variety of luminal contents, inclu

118 d, also enhanced microparticle uptake by the FAE and induced DC migration into the FAE, suggesting th

119 nstrated that CCL9 mRNA was expressed by the FAE but not by the villus epithelium.

120 pithelial transport of microparticles by the FAE of mouse Peyer's patches in vivo.

121 By morphological criteria, the FAE contains >90% M cells.

122 vivo porcine model confirms that during the FAE in PPV, pressurized air from an infusion cannula mal

123 her chemokine specifically secreted from the FAE of mouse Peyer's patches, CCL9 (MIP-1gamma, CCF18, M

124 e resulted in receptor redistribution in the FAE only.

125 subepithelial dendritic cells (DCs) into the FAE, better positioning DCs for antigen capture.

126 dent migration of subepithelial DCs into the FAE, but not into villus epithelium of wild-type and TLR

127 by the FAE and induced DC migration into the FAE, suggesting that other TLRs may modulate FAE functio

128 ed dramatic morphological alterations of the FAE in rabbit PP.

129 Further confocal microscopy analysis of the FAE surface showed that a significant increase in the nu

130 his problem by evaluating the ability of the FAE to bind, internalize, and transport fluorescent poly

131 ls regulate the gatekeeping functions of the FAE to promote Ag capture by DCs in organized mucosal ly

132 were accompanied by enhanced ability of the FAE to transport antigens.

133 the protein level, CCL9 was detected on the FAE and on extracellular matrix structures within the do

134 lectin was found to specifically target the FAE, while the control lectin did not.

135 a provide the first direct evidence that the FAE-specific antigen sampling function may be manipulate

136 Thus, the FAE of Peyer's patches responds to TLR2 ligands in a man

137 endent on the expression of CD155 within the FAE, including the M cells, and on cells within Peyer's

138 cies, and special procedures associated with FAE in children.