ライフサイエンスコーパス: FBS

1 FBS (1%-8%) increased cell numbers in a dose-dependent m

2 FBS also influenced the mode of degradation by limiting

3 FBS also significantly decreased the lipid accumulation

4 FBS and IL-5 failed to inhibit or suppress the CD30 agon

5 FBS and RD were rich in Lys, Glu, Gly, Pro, Asp, Leu, Hi

6 FBS had little impact on mass loss of pure Mg during imm

7 FBS hydrolysates showed higher antioxidant activity base

8 FBS or IUPT resulted in a relatively high complication r

9 FBS reduced the mass loss of Mg-Yttrium (MgY) alloy with

10 FBS was hydrolysed to a greater extent than RD regardles

11 FBS, Fn, and Lm stimulated invasion of the M1(+) strain

12 FBS-associated RNA is co-isolated with cell-culture deri

13 FBS-cultured cells also showed higher MsgA and NanA acti

14 s were incubated for various periods in 0.1% FBS (a concentration that maintains cell health, but doe

15 In cells kept in 0.1% FBS, there is DIO2 inhibition via FOXO1 binding to the D

16 r (2) 10% FBS with FGF and heparin or (3) 1% FBS or (4) 1% FBS with TGF-beta.

17 with FGF and heparin or (3) 1% FBS or (4) 1% FBS with TGF-beta.

18 ize keratocan when reseeded on plastic in 1% FBS or on AM.

19 In 1% FBS, AM-expanded keratocytes rapidly became alpha-SMA-ex

20 absence of FBS but not in the presence of 1% FBS.

21 s generated by shifting HAECs from 10% to 1% FBS.

22 and 1000 pg/mL TGF-beta1 or in DMEM with 1% FBS and 10 ng/mL TGF-beta1.

23 ls were grown in DMEM/F12 containing (1) 10% FBS or (2) 10% FBS with FGF and heparin or (3) 1% FBS or

24 n DMEM/F12 containing (1) 10% FBS or (2) 10% FBS with FGF and heparin or (3) 1% FBS or (4) 1% FBS wit

25 ChIP studies indicate that 4 h after 10% FBS-containing medium, FOXO1 binding markedly decreases,

26 n in response to endothelin-1, PDGF, and 10% FBS by 62%, 51%, and 29%, respectively (P<0.01), without

27 Both 10% FBS and PDGF stimulated significant keratocyte migration

28 rough the uncompressed outer matrix, but 10% FBS produced more cell-induced collagen matrix reorganiz

29 ists (by 30% to 60%, P<0.01) but also by 10% FBS (by 35%, P<0.05).

30 y inhibited SMC proliferation induced by 10% FBS as well as the mitogens PDGF, bFGF, and EGF without

31 e cultured in DMEM/F12 medium containing 10% FBS and 5 microg/mL insulin at 37 degrees C in 5% CO(2).

32 , cells were cultured in DMEM containing 10% FBS and a) vehicle only; b) ascorbic acid (50 micrograms

33 te medium and DMEM/F12 medium containing 10% FBS in a 37 degrees C incubator supplied with 5% CO(2),

34 e cultured in DMEM/F12 medium containing 10% FBS in a 37 degrees C incubator supplied with 5% CO(2).

35 In the media containing 10% FBS, forskolin also stimulated proliferation of Schwann

36 owever, proliferated in media containing 10% FBS.

37 MEM/ITS) or 10% fetal bovine serum (DMEM/10% FBS), or in a defined keratinocyte serum-free medium (KS

38 downregulated in KSFM compared with DMEM/10% FBS.

39 d by switching KSFM to DMEM/ITS and DMEM/10% FBS.

40 ays using 0.1% fetal bovine serum (FBS), 10% FBS +/- 10 microM SB, or 20 ng/ml PDGF +/- 10 microM SB.

41 cells appeared dendritic on AM, even in 10% FBS but fibroblastic on plastic.

42 In contrast, cells in 10% FBS developed a bipolar fibroblastic morphology.

43 ucleotides followed by postincubation in 10% FBS lowers endogenous p27(kip1) protein levels and promo

44 (20 ng/ml) plus heparin (5 microg/ml) in 10% FBS medium had decreased expression of alpha-SM actin pr

45 Keratocytes cultured in 10% FBS proliferated, appeared fibroblastic, and synthesized

46 ued at least to passage 6 on AM, even in 10% FBS, but was rapidly lost each time when cells on AM wer

47 hen comparing levels of cellular fill in 10% FBS, GF displayed greater wound fill than the PDL.

48 levels of wound fill for PDLs and GFs in 10% FBS.

49 active when cultured in the presence of 10% FBS (fetal bovine serum), with a replication time of 1-

50 as a negative control, and a culture of 10% FBS served as a positive control.

51 ease of [Ca2+] to 1.8 mM and addition of 10% FBS synergistically downregulated the keratocan promoter

52 In the presence of 10% FBS, rBPI(21) and XMP.Z increased BRP growth by 91% +/-

53 response of either cell type compared to 10% FBS alone.

54 The addition of SB to 10% FBS did not significantly affect the wound fill response

55 Relative to 10% FBS, PDLs stimulated with PDGF showed significantly (P <

56 CGF and in positive controls exposed to 10% FBS.

57 eic acid but did not reduce responses to 10% FBS.

58 n a cell model transitioning from 0.1 to 10% FBS.

59 l was conferred by incubating cells with 10% FBS and IL-5.

60 Stimulation of Rb cells with 10% FBS resulted in an increase in CTCF expression and a dec

61 washed twice with MEM supplemented with 10% FBS, and fibroblasts in treatment and control groups wer

62 re either scrape wounded or treated with 10% FBS, PDGF, or FGF-2 for 6 hours.

63 orneal and 293T fibroblasts treated with 10% FBS, PDGF, or FGF-2.

64 er serum-starved cells were treated with 10% FBS, PDGF, or FGF-2.

65 hen incubated in media supplemented with 10% FBS, TGFbeta1, TGFbeta2, platelet-derived growth factor

66 ane matrix (AM) in DMEM, with or without 10% FBS, in serum-free DMEM containing insulin-transferrin-s

67 A culture of 0.2% FBS alone served as a negative control, and a culture of

68 by growth for 48 hours in DMEM/F12 with 0.2% FBS and subsequently were either scrape wounded or treat

69 In differentiation-promoting media (2% FBS), IUGR and control myoblasts had similar percentages

70 esponded to fluid flow in the presence of 2% FBS, confirming that P2Y2 purinoceptors are responsible

71 creases in [Ca(2+)](i) in the presence of 2% FBS.

72 DMEM) plus 2% fetal bovine serum (FBS) or 2% FBS plus EMD (100 microg/ml), with and without ascorbic

73 e effect on SVF cells, when compared with 2% FBS control.

74 owth of BRPs, even above that induced by 20% FBS.

75 ells were seeded, maintained overnight in 4% FBS to permit cell attachment, washed, and incubated for

76 itated by 0.9 mM Ca(2+) but suppressed by 5% FBS in KSFM at passage 0.

77 concentration to 0.9 mM, with or without 5% FBS.

78 that VEGF protects MM patient cells against FBS starvation-induced apoptosis.

79 In addition, both humic acid and FBS protein effectively lowered the amounts of fullerene

80 Humic acid and FBS significantly changed the characteristics of fullere

81 an mRNAs were downregulated by TGF-beta1 and FBS.

82 In contrast, both EGF and FBS significantly increased [(3)H]thymidine incorporatio

83 ne, whereas in the presence of CGF + EGF and FBS they remained elevated for up to 20 h.

84 to activate ERK1 and ERK2; however, EGF- and FBS-induced activation of ERKs was not different from th

85 ptor, integrin alpha5beta1, inhibited Fn and FBS-mediated invasion but did not specifically inhibit L

86 Proteins found in human and FBS bound exogenous HNP-2 and retrocyclin-1, and compete

87 Insulin-like growth factor I and FBS activate NADPH oxidase through transcriptional up-re

88 With poor ICHS and FBS as references, individuals with ideal ICHS and FBS s

89 was to compare the effectiveness of ICHS and FBS in predicting the presence and extent of subclinical

90 references, individuals with ideal ICHS and FBS showed lower adjusted odds of having atherosclerotic

91 criminating accuracy were found for ICHS and FBS with respect to the presence of plaques (C-statistic

92 e between 20 and 400 nm from human serum and FBS using ultracentrifugation and sucrose gradient centr

93 f the antenatal management strategy applied; FBS, IUPT, or IVIG with or without corticosteroids.

94 -ATP channel opener) administered 1 h before FBS addition restored the FBS response in propofol treat

95 consistent effect of cAMP was to block both FBS- and DeltaRaf-1:ER-induced expression of Cdc25A and

96 house screening programs that performed both FBS of 8800 fragments and screens of the full library.

97 nterference and misinterpretations caused by FBS-derived RNA.

98 Smaller fragments identified by FBS occupy both these pockets independently and suggest

99 e development of a potential fourth chelator FBS 0701 and the combined use of oral chelators may furt

100 Many evolutionally conserved FBS-derived RNA species can be falsely annotated as huma

101 dependent fashion and binds to the conserved FBS.

102 ycated FBS (experimental) or RPMI-1% control FBS, and cells were incubated for 1 or 4 d.

103 ation of a new shoulder site in deafferented FBS antidromically-activated a cell in the former forepa

104 that the new shoulder input in deafferented FBS is relayed from cells in the former forepaw region i

105 d to a new shoulder site in the deafferented FBS, we examined the thalamocortical pathway in 2 foreli

106 an L-type Ca(2+) channel blocker, decreased FBS-induced Ca(2+) response of control cells to a level

107 reporter plasmids by both estrogen-deficient FBS (ED-FBS) and EGF.

108 tion in the preparations of vesicle-depleted FBS (vdFBS) commonly utilized in the studies of extracel

109 growing them in culture media with different FBS content.

110 culture supplement), or heat-denatured DMEM-FBS.

111 MEM-AH differed from those incubated in DMEM-FBS.

112 the more spindle-shaped cells grown in DMEM-FBS.

113 atured DMEM-AH, 10% fetal bovine serum (DMEM-FBS, the standard culture supplement), or heat-denatured

114 d cell proliferation when compared with DMEM-FBS (11% vs. 141%).

115 medium isolated from cells treated with DMEM-FBS (442%), but only a 10% increase in myocilin was obse

116 same degree as the wild-type receptor by ED-FBS and EGF in both HSVSMC and PVEC.

117 plasmids by both estrogen-deficient FBS (ED-FBS) and EGF.

118 ion of primary cultures stimulated by either FBS or angiotensin II.

119 0(-3) s-1 for Ee AChE and 8 x 10(-3) s-1 for FBS AChE.

120 for Ee AChE and pK1 4.8-5.0 and pK2 5.8 for FBS AChE.

121 for in this study (heritability estimate for FBS is 68%).

122 tiple significant epistatic interactions for FBS, which accounts for half (14.6%) of F2 variance comp

123 ltures can be continually passaged in fresh, FBS-free F-12 medium at an initial inoculum of only appr

124 Nanobacteria were previously isolated from FBS after prolonged incubation in DMEM.

125 ntified putative nanobacteria, not only from FBS, but also from human saliva and dental plaque after

126 Furthermore, FBS transcripts can be taken up by cultured cells and af

127 Glycated FBS resulted in a 1.8-fold increase in 3H-thymidine upta

128 The medium was changed to RPMI-1% glycated FBS (experimental) or RPMI-1% control FBS, and cells wer

129 d by 550% and to albumin by 320% in glycated FBS-treated monolayers compared with controls.

130 The media of glycated FBS-treated cells contained 45% lower collagenase activi

131 Exposing cells to glycated FBS changed the distribution of cadherin from a linear t

132 However, FBS and RD hydrolysates protected HepG2 cells against te

133 Pepsin-hydrolysed FBS, at a 5% degree of hydrolysis (DH), showed the highe

134 ty lipoprotein secretion, which is absent in FBS-grown cells, is restored in Huh7.5 cells that are cu

135 Notably, specific miRNAs abundant in FBS, such as miR-122, miR-451a and miR-1246, have been p

136 city of S. intermedius toward HepG2 cells in FBS, and a higher concentration of human plasma was nece

137 , addition of human plasma to the culture in FBS appeared to inhibit the stimulatory effect of FBS on

138 e widely used, the use of (19)F detection in FBS has been only recently introduced with the aim of ta

139 activity, although overproduction of ILY in FBS was undetectable in mutants nanA-null and msgA-null.

140 those of the wild type; however, survival in FBS was reduced but not to the levels in CSF.

141 x-loaded TSL (TSLDox) was stable in vitro in FBS, BALB/c-nu plasma and human plasma, although release

142 dy directed against integrin beta1 inhibited FBS-, Fn-, and Lm-mediated invasion but did not abrogate

143 Ca2+ chelator BAPTA only partially inhibited FBS-stimulated LC20 phosphorylation and did not signific

144 LXA4 also significantly inhibited FBS-dependent proliferation and TGF-beta1-dependent coll

145 These results suggest that monomeric FBS AChE is trimmed at the C-terminus, and the results a

146 Moreover, FBS are very sensitive to strain, which induces an impor

147 ased >2.5 times when grown in the absence of FBS but not in the presence of 1% FBS.

148 hat oscillatory fluid flow in the absence of FBS failed to increase [Ca(2+)](i) in MC3T3-E1 cells.

149 reased in SK-Mel-110 cells in the absence of FBS.

150 n turn depends on the presence or absence of FBS.

151 AM) in DMEM with different concentrations of FBS.

152 gical effects associated with RNA content of FBS on cell cultures.

153 ppeared to inhibit the stimulatory effect of FBS on ILY, MsgA, and NanA, although there were individu

154 We studied the effect of FBS substitutes on both human BMSC proliferation in vitr

155 did not significantly alter the magnitude of FBS-stimulated isometric contraction.

156 ilable exRNA datasets supports the notion of FBS contamination.

157 One mutation altered the upstream portion of FBS 1, whereas the other, originally created to improve

158 dvertently altered the downstream portion of FBS 2.

159 Analysis of purified preparations of FBS AChE by gel permeation chromatography revealed the p

160 This did not require the presence of FBS and was associated with continued expression of the

161 in SK-Mel-110 in the absence or presence of FBS, respectively.

162 ing their differentiation in the presence of FBS.

163 increases in [Ca(2+)](i) in the presence of FBS.

164 d ERK1/2 dephosphorylation and repression of FBS-induced cell proliferation.

165 The use of FBS in hybridoma culture media is examined here, with re

166 This is the first study on FBS showing this behaviour in the full temperature, fiel

167 Of the stimuli tested only FBS and thrombin were able to stimulate a sustained acti

168 growth enhancement in F-12 containing BSA or FBS.

169 h recovery of Hg(2)(+) spiked in the BSA- or FBS-containing medium, and high stability of fluorescenc

170 In the presence of apoptotic cells or FBS, elicited expression of IL-1 beta by NOD macrophages

171 ytes to protein coronas preformed of EfCP or FBS.

172 , NGF, PDGF-BB, bovine pituitary extract, or FBS; or a combination of factors.

173 further show that epidermal growth factor or FBS stimulation induces association of endogenous RSK2 w

174 lth based on the number of favorable ICHS or FBS.

175 g/ml), beta-cyclodextrin (200 microg/ml), or FBS (2 to 4%) was added; addition of urea and phenol all

176 Serial fetal blood sampling (FBS) and intrauterine platelet transfusions (IUPT), as w

177 ], alimentation [A], and tobacco [T]) score (FBS), are also available.

178 ton-based NMR methods of fragment screening (FBS) have been well documented and are widely used, the

179 hich insulin-like growth factor I and serum (FBS) activate NADPH oxidase in pancreatic cancer (PaCa)

180 inesterase isolated from fetal bovine serum (FBS AChE) was previously characterized as a globular tet

181 viated in AM without 10% fetal bovine serum (FBS) [AM(-S)].

182 of Electric eel (Ee) and fetal bovine serum (FBS) acetylcholinesterase (AChE) inactivated with P(-)C(

183 growth factor (EGF) and fetal bovine serum (FBS) also increased Src activity in Vector cells, but no

184 in media containing 0.1% fetal bovine serum (FBS) and 1 of 5 concentrations of PDGF-BB.

185 ured in media containing fetal bovine serum (FBS) and a glycogen synthase kinase-3 (GSK3) inhibitor,

186 a native repertoire and fetal bovine serum (FBS) as a non-native reference.

187 membrane inserts and 20% fetal bovine serum (FBS) as chemoattractant.

188 e in media containing 2% fetal bovine serum (FBS) but not those from P30 mice, which, however, prolif

189 n presence or absence of fetal bovine serum (FBS) can provide reliable cryopreservation of various ki

190 days in media containing fetal bovine serum (FBS) concentrations (0, 0.1, 1, 5, 10, and 20%) as appro

191 Fetal bovine serum (FBS) has been used in eukaryotic cell cultures for decad

192 tion, we have shown that fetal bovine serum (FBS) induces Yes auto-phosphorylation and activation.

193 , 6, and 10 days in 0.2% fetal bovine serum (FBS) media containing different concentrations of either

194 the media containing 1% fetal bovine serum (FBS) on the 4 DIV, surface galC could be reexpressed in

195 's medium (DMEM) plus 2% fetal bovine serum (FBS) or 2% FBS plus EMD (100 microg/ml), with and withou

196 in medium containing 8% fetal bovine serum (FBS) plus additional growth factors.

197 tarved ME-180 cells with fetal bovine serum (FBS) resulted in a rapid increase in steady-state levels

198 Cells incubated with 10% fetal bovine serum (FBS) served as positive controls.

199 d mammalian cell growth, fetal bovine serum (FBS) supplemented media are still commonly used: a pract

200 treated (TCT) plastic in fetal bovine serum (FBS) supplemented medium.

201 us PC574 was cultured in fetal bovine serum (FBS) than when it was grown in the standard culture medi

202 of human ferritin in 10% fetal bovine serum (FBS) to mimic a real detection environment in complex me

203 n antagonized effects of fetal bovine serum (FBS) to stimulate cell proliferation, whereas siRNA-medi

204 moral immune response to fetal bovine serum (FBS) was detected in all animals following the second ad

205 harcoal-dextran-stripped fetal bovine serum (FBS) was found to be more efficient for serum supplement

206 r lavage (BAL) fluid and fetal bovine serum (FBS), (ii) survival in macrophages, and (iii) virulence

207 5, and 7 days using 0.1% fetal bovine serum (FBS), 10% FBS +/- 10 microM SB, or 20 ng/ml PDGF +/- 10

208 e reports the effects of fetal bovine serum (FBS), a physiologically relevant mixture of proteins, on

209 ure condition containing fetal bovine serum (FBS), Cdc25C protein levels were similar in these PCa ce

210 covered during growth in fetal bovine serum (FBS), elicit a robust increase in the amount of ACT, and

211 Fetal bovine serum (FBS), fibronectin (Fn), the extracellular matrix protein

212 fied Eagle's medium, 10% fetal bovine serum (FBS), then for an additional 3-10 days in the presence a

213 ia supplemented with 10% fetal bovine serum (FBS), to media supplemented with 2% HS.

214 1) in the presence of 5% fetal bovine serum (FBS), whereas XMP.Z enhanced BRP growth by 27% +/- 7% (P

215 ffects of humic acid and fetal bovine serum (FBS), which are ubiquitous in aquatic environments and r

216 ulture medium containing fetal bovine serum (FBS), which forms a protein corona on the surface of the

217 is a major component of fetal bovine serum (FBS), which is commonly used as a culture medium during

218 eletion of Mcl-1 reduces fetal bovine serum (FBS)-, VEGF-, and IL-6-induced proliferation.

219 cAMP blocked the fetal bovine serum (FBS)-induced degradation of p27(KIP1); however, loss of

220 ative role of [Ca2+]i in fetal bovine serum (FBS)-stimulated LC20 phosphorylation and force developme

221 te (HS)-, TGF-beta1-, or fetal bovine serum (FBS)-supplemented DMEM/F12 medium.

222 days in the presence of fetal bovine serum (FBS).

223 the G6P concentration in fetal bovine serum (FBS).

224 butylxanthine (IBMX) and fetal bovine serum (FBS).

225 (2+) response induced by fetal bovine serum (FBS).

226 medium with and without fetal bovine serum (FBS).

227 ay in the presence of 2% fetal bovine serum (FBS).

228 ining either 0.1% or 10% fetal bovine serum (FBS).

229 d ex vivo in medium with fetal bovine serum (FBS).

230 bsence or presence of 1% fetal bovine serum (FBS).

231 (DMEM), with and without fetal bovine serum (FBS).

232 PMI media containing 10% fetal bovine serum (FBS).

233 upon incubation in pure fetal bovine serum (FBS).

234 (F-12) in the absence of fetal bovine serum (FBS); this represents a breakthrough for studies in whic

235 ays in growth media (20% fetal bovine serum, FBS), myoblasts from IUGR fetuses had 34% fewer (P < 0.0

236 uture eCO2 using global food balance sheets (FBS).

237 volutionary conserved forkhead binding site (FBS).

238 erichia coli contains two Fur-binding sites (FBS) offset by 6bp.

239 Genetic analysis suggests that, of these six FBS QTL, three influence BMD, two influence bone quality

240 rimi wastes, including frame, bone and skin (FBS) and refiner discharge (RD), were investigated.

241 cells are grown in HS, compared to standard FBS culture conditions.

242 ic loci influencing femur-breaking strength (FBS), which was measured by three-point bending using an

243 e reorganization in forepaw barrel subfield (FBS) cortex.

244 organization in the forepaw barrel subfield (FBS) of primary somatosensory cortex (SI) that follows f

245 ost tunable of the Fe-based superconductors (FBS) in terms of acceptance of high densities of self-as

246 Fe-based superconductors (FBS) present a large variety of compounds whose properti

247 d significantly higher than for cells in TCT/FBS condition.

248 expansion medium supplementation (bFGF, TFP, FBS) and self-assembled construct seeding density (2, 3,

249 Addition of human aqueous humor rather than FBS to trabecular monolayer cell cultures triggers signi

250 e, using RNA sequencing, we demonstrate that FBS contains a diverse repertoire of protein-coding and

251 stimulation on TFPI secretion and found that FBS induced a 5-fold increase in TFPI antigen and activi

252 Eliminating the FBS-stimulated rise in [Ca2+]i with the Ca2+ chelator BA

253 input from the shoulder first appears in the FBS 4 weeks after amputation, and by 6 weeks, the new sh

254 strate for large-scale reorganization in the FBS.

255 ilar accuracy, highlighting the value of the FBS as a simpler and more affordable score for evaluatin

256 ossibly due to the confounding effect of the FBS.

257 w shoulder input comes to occupy most of the FBS.

258 istered 1 h before FBS addition restored the FBS response in propofol treated cells to a level simila

259 he shoulder becomes expressed throughout the FBS that quite likely has a subcortical origin.

260 Addition of purified MsgA and NanA to the FBS resulted in a release of 2.8 mM galactose and 4.3 mM

261 Therefore, FBS and RD hydrolysates have a potential as antioxidativ

262 ted Fbs, regardless of the HLA type of these FBS: CD8(+) T cell stimulation was inhibited by pretreat

263 Here we show that in contrast to FBS, addition of increasing amounts of human serum from

264 However, in contrast to FBS, PDGF-BB, or basic fibroblast growth factor (bFGF),

265 ely robust proliferative response of HCEC to FBS may involve stimulation of multiple downstream signa

266 shorter lasting rise in [Ca2+]i relative to FBS.

267 ration/proliferation in vitro in response to FBS or BDNF compared with WT astrocytes.

268 unique concentration-dependent responses to FBS (P<0.0025).

269 However, a comparison of responses to FBS, thrombin, TRPs, LPA and EGF revealed that Fra-1 and

270 Identification of the genes unique to FBS may have an impact on prediction of osteoporosis in

271 supplementation than dialyzed or undialyzed FBS, due to lower contaminating unlabeled pantothenate c

272 rb any detectable serum protein in undiluted FBS.

273 In solutions of 10% (vol/vol) FBS, we find rapid endonuclease hydrolysis at specific s

274 % confidence interval [CI]: 0.31 to 0.55 vs. FBS OR: 0.49; 95% CI: 0.36 to 0.66), coronary artery cal

275 applications, however, contact of BMSCs with FBS should be minimized.

276 crease by incubation of the COS-7 cells with FBS and to decrease upon adding the ERK pathway inhibito

277 e formed by BMSCs cultured continuously with FBS, bone formed by cells cultured with HS, or with FBS

278 ve, while bone formed by cells cultured with FBS switched to serum-free medium (SFM) was considerably

279 x vivo expansion of human BMSCs, medium with FBS remains most effective.

280 Medium with FBS was more effective in stimulating BMSC proliferation

281 In medium with FBS, colistin was less efficacious in the 3-D cell model

282 ne formed by cells cultured with HS, or with FBS switched to HS, was considerably less extensive, whi

283 med in human cell cultures supplemented with FBS instead of human serum (HS).

284 al cells cultured in media supplemented with FBS, bFGF/HS, or TGF-beta1.

285 into astrocytes in medium supplemented with FBS.

286 an virus produced in media supplemented with FBS.

287 nce and absence of rHuOP-1, with and without FBS.

288 ization under oscillatory fluid flow without FBS.

289 ed to the supernatant, unlike growth without FBS, in which >/=90% is associated with the bacterium.

290 Adding ATP or UTP to flow medium without FBS restored the ability of fluid flow to increase [Ca(2