ライフサイエンスコーパス: FFA

1 FFA concentration was measured in duplicate by the Wako

2 FFA content of the crude and stabilized bran fractions t

3 FFA produces a significantly greater enhancement of curr

4 FFAs increased MIR122 expression in livers of mice by ac

5 s of magnitude if an internal standard (15:0 FFA) was added.

6 ospholipid 17:0, phospholipid trans-16:1n-7, FFA 15:0, and FFA 17:0 were inversely associated with fa

7 the crystal structure of ADIPOR2 bound to a FFA molecule and show that ADIPOR2 possesses intrinsic b

8 HOMA-IR and positively with free fatty acid (FFA) and HDL after control for age and sex.

9 to analyze free (nonesterified) fatty acid (FFA) and triacylglycerol flux and lipogenesis.

10 opposed to low circulating free fatty acid (FFA) and triglyceride levels in patients with type 2 dia

11 Although plasma free fatty acid (FFA) concentrations have been associated with lipotoxici

12 weight and elevated plasma free fatty acid (FFA) concentrations.

13 ride and glycogen contents, free fatty acid (FFA) content and release, and cholesterol and cholestero

14 riacylglycerol composition, free fatty acid (FFA) content, peroxide index, thermal properties, meltin

15 zed for 3-MCPD esters, GEs, free fatty acid (FFA) contents, specific extinction at 232 and 268 nm (K2

16 -nitrogen (TVB-N) value and free fatty acid (FFA) formation.

17 icantly reduced insulin and free fatty acid (FFA) levels (P < 0.001) and ameliorated the oxidative da

18 lipolysis, elevated plasma free fatty acid (FFA) levels, and impaired insulin signaling.

19 id translocase and elevated free fatty acid (FFA) levels.

20 sed 'lyso'-lipid as well as free fatty acid (FFA) levels.

21 stance and dysregulation of free fatty acid (FFA) metabolism are core defects in type 2 diabetic (T2D

22 ctly, determine the rate of free fatty acid (FFA) oxidation.

23 d dysfunction in a model of free fatty acid (FFA) palmitate-induced oxidative stress.

24 hylomicrons into the plasma free fatty acid (FFA) pool is an important source of FFA and reflects ine

25 nses through members of the free fatty acid (FFA) receptor family, which includes FFA4.

26 triacylglyceride (TAG) and free fatty acid (FFA) species to be significantly increased.

27 6 facilitates cell membrane free fatty acid (FFA) transport, but its role in human metabolism is not

28 Alterations in hepatic free fatty acid (FFA) uptake and metabolism contribute to the development

29 p4/Fabp5) impairs exogenous free fatty acid (FFA) uptake by CD8(+) TRM cells and greatly reduces thei

30 When applied to free fatty acid (FFA) uptake in 3T3-L1 adipocytes, this muChopper permitt

31 membrane, and thus greater free fatty acid (FFA) uptake, in adipocyte cell models.

32 a (PPARalpha) activation by free fatty acid (FFA), and cAMP response element-binding protein (CREB) a

33 Neprilysin contributes to free fatty acid (FFA)-induced cellular dysfunction in nonislet tissues in

34 uld be exacerbated by acute free fatty acid (FFA)-induced insulin resistance.

35 Apoptosis and free fatty acid (FFA)-induced lipotoxicity are important features of NASH

36 calpain-1 degrades Erk5 in free fatty acid (FFA)-stressed cardiomyocytes, whereas the prevention of

37 o produce sphingosine and a free fatty acid (FFA).

38 into the crystallization of flufenamic acid (FFA) in a confined environment of mesoporous silica mate

39 idal anti-inflammatory drug flufenamic acid (FFA; 2-{[3-(trifluoromethyl)phenyl]amino}benzoic acid).

40 asting and mean OGTT plasma free fatty acid [FFA] x insulin concentrations), peripheral IR (1/[Matsud

41 was monitored by measuring the free acidity (FFA), peroxide (PV), p-anisidine (p-AV),) total polar co

42 cylglycerols, DAG, MAG and free fatty acids (FFA) and the concentration of saturated, mono- and polyu

43 hibits ATGL expression and free fatty acids (FFA) beta-oxidation.

44 l extraction, (ii) Omega-3 free fatty acids (FFA) concentration (low temperature winterization), (iii

45 Free fatty acids (FFA) content of beer affects the ability to form a stabl

46 nherent increase in plasma free fatty acids (FFA) in the HFD together with an HFD-induced alteration

47 incorporation of systemic free fatty acids (FFA) into circulating very low-density lipoprotein trigl

48 scence compounds (OFR) and free fatty acids (FFA) were evaluated throughout the storage for all sampl

49 e levels of triglycerides, free fatty acids (FFA), and leptin?

50 d content and composition, free fatty acids (FFA), thiobarbituric acid reactive substances (TBARS) an

51 a reduction in circulating free fatty acids (FFA).

52 in increased intracellular free fatty acids (FFAs) and elevated expression of uncoupling protein 2 (U

53 dance of saturated C16-C20 free fatty acids (FFAs) and long polyunsaturated complex lipids.

54 Free fatty acids (FFAs) are known to induce lipoapoptosis in liver cells i

55 podocytes, the presence of free fatty acids (FFAs) associated with serum albumin stimulated macropino

56 Nutrients such as free fatty acids (FFAs) contribute to precise regulation of beta cell mass

57 duced by extraction of the free fatty acids (FFAs) from flaxseed oil, concentration of PUFAs, and enz

58 Saturated free fatty acids (FFAs) have complex effects on the islet beta-cell, acute

59 l as increased circulating free fatty acids (FFAs) in NAFLD, we hypothesized the involvement of chola

60 The importance of free fatty acids (FFAs) in wort has been known for a long time because of

61 ons of the polyunsaturated free fatty acids (FFAs) linoleic and alpha-linolenic acid, which we detect

62 Serum free fatty acids (FFAs) profile is highlighted in its association with obe

63 ted the mechanism by which free fatty acids (FFAs) regulate MIR122 expression and the effect of MIR12

64 n elevated plasma ratio of free fatty acids (FFAs) to albumin when proteinuria reached nephrotic rang

65 l relations of serum total free fatty acids (FFAs) to insulin resistance (IR) and cardiovascular (CV)

66 Physiologically relevant free fatty acids (FFAs) were analyzed by UV-laser desorption/ionization or

67 Circulating free fatty acids (FFAs) were less suppressed in IR than IS subjects.

68 ons of intestinal origin), free fatty acids (FFAs), insulin, glucose, glucagon, glucagon-like peptide

69 or the slow suppression of free fatty acids (FFAs), which in turn is responsible for delayed suppress

70 n plasma phospholipids and free fatty acids (FFAs).

71 ia with elevated levels of free fatty acids (FFAs).

72 ated by nonesterified or "free" fatty acids (FFAs).

73 ents (hydrocarbons - HCs, free fatty acids - FFAs, free fatty alcohols - FALs and wax esters - WEs) o

74 lipid digestion products (free fatty acids, FFAs, and monoacylglycerides, MAGs) during in vitro dige

75 We find that fast flicker adaptation (FFAd) shifts the tuning of face perception to higher spa

76 Additional FFA analysis confirmed that the purified effector lipase

77 but are also effective in protecting against FFA-induced oxidative stress; thus, EMP function is refl

78 nd performed bioluminescence imaging with an FFA probe.

79 0, phospholipid trans-16:1n-7, FFA 15:0, and FFA 17:0 were inversely associated with fasting plasma g

80 cerides [SMD=0.97 (95% CI: 0.53, 1.40)], and FFA [SMD=0.86 (95% CI: 0.50, 1.22)], and a nonsignifican

81 tty acid acyl chains in the acylglycerol and FFA portions.

82 entify potential interactions between BA and FFA metabolism in NAFLD.

83 highlighting the specific roles of CD36 and FFA uptake.

84 rations indicated that lipid composition and FFA of hoki and saithe can be estimated by NIR with good

85 in beta-cells possibly comprise glycerol and FFA formation and release extracellularly and the divers

86 od levels, the interplay between insulin and FFA was studied with regard to hepatocyte proliferation

87 type 2 diabetes mellitus (T2DM), HOMA-IR and FFA.

88 is substantial disagreement between OCT and FFA findings in detecting active disease in patients wit

89 ificant changes in centerpoint thickness and FFA total lesion size over 12 months.

90 ing whole-body turnover rates of glucose and FFAs in L-AktFoxo1TKO mice also confirmed that hepatic E

91 Coadministration of insulin and FFAs, however, abolished hepatocyte proliferation and tr

92 NASH, based on a set of short-chain TAGs and FFAs.

93 aditionally, fundus fluorescein angiography (FFA) has been considered the reference standard to detec

94 graphy (FP), fundus fluorescein angiography (FFA), and optical coherence tomography (OCT).

95 Fundus fluorescence angiography (FFA), optical coherence tomography (OCT), and indocyanin

96 d temporal lobe, and the fusiform face area (FFA) and anterior temporal lobe play key roles in the re

97 sensory responses in the fusiform face area (FFA) and parahippocampal place area (PPA), respectively.

98 ural styles included the fusiform face area (FFA) in addition to several scene-selective regions.

99 tches in monkeys and the fusiform face area (FFA) in humans.

100 The fusiform face area (FFA) is thought to be a computational hub for face proce

101 the right face-selective fusiform face area (FFA) was closely associated with individual differences

102 ssing network comprising fusiform face area (FFA), superior temporal sulcus, amygdala, and intraparie

103 sulcus (pSTS) and to the fusiform face area (FFA), using a searchlight approach to reveal interaction

104 remains unclear whether fusiform face area (FFA)-the portion of fusiform gyrus that is functionally-

105 pecific regions like the fusiform face area (FFA).

106 d our first paper on the fusiform face area (FFA): how we chose the question, developed the methods,

107 ntation in the fusiform face-selective area (FFA).

108 adjacent regions (e.g., fusiform face area, FFA) within the temporal visual cortex.

109 Fenamates such as FFA (flufenamic acid; 2-{[3-(trifluoromethyl)phenyl]amin

110 oscillations with specialized areas such as FFA and PPA.

111 The relation between FFA and CV risk factors does not become significant unti

112 -entrant processing loop involving bilateral FFA and LOC.

113 isual cortical network composed of bilateral FFA and bilateral object-selective lateral occipital cor

114 1-dependent GPR40 signaling relative to both FFAs.

115 erence standard to detect nAMD activity, but FFA is costly and invasive.

116 ed in pancreatic beta cells and activated by FFAs.

117 red mitochondrial depolarization mediated by FFAs released as a result of lipolysis.

118 ong complex lipid subclasses and the C16-C20 FFAs but directly associated with short complex lipids w

119 hat increased abundance of saturated C16-C20 FFAs coupled with impaired beta-oxidation of FFAs and in

120 uptake, and 3) is needed for normal cardiac FFA uptake over a range of FFA concentrations from low t

121 , and micellar incorporation of carotenoids, FFAs and MAGs.

122 ocytes, this muChopper permitted single-cell FFA uptake rates to be quantified at 3.5 +/- 0.2 x 10(-1

123 le for the analysis of medium and long chain FFAs in beer.

124 Saturated long chain FFAs induced apoptosis and JNK activation in primary rat

125 Approximately 7% of circulating FFA was converted into VLDL-TG.

126 siderable evidence suggests that circulating FFAs promote beta cell expansion by direct and indirect

127 As enzymatic treatment, Omega-3 concentrate FFA (Omega-3>600mg Omega-3 per g oil) were esterified wi

128 quid-like layer besides crystalline confined FFA form I.

129 During one study day, FFA was elevated by infusion of Intralipid plus heparin.

130 acological blockade of Gq activity decreased FFA-induced insulin secretion.

131 diet, weight loss with an ADF diet decreases FFA concentrations through potentially different mechani

132 A novel diphenylalaninamid (FFA) based peptide nanoparticles (PNPs) modified pencil

133 e tested this hypothesis by comparing direct FFA storage in subcutaneous adipose tissue during insuli

134 We measured direct FFA storage in abdominal and femoral subcutaneous fat in

135 d and were positively correlated with direct FFA storage rates.

136 and subcortical face-selective areas (i.e., FFA and amygdala) remained intact.

137 Early FFA activity (50-75 ms) contained information regarding

138 e inferior frontal junction, IFJ, and either FFA or PPA, depending on which object was attended.

139 Although obesity is manifested as elevated FFA levels, the degree of EMT was not associated with th

140 irect evidence associating CD36 and elevated FFAs with HCC progression.

141 Defective suppression of endogenous FFA is one common link between impaired potentiation and

142 riven structural modifications to endogenous FFAs, focused on breaking planarity and reducing lipophi

143 ock down increased the capacity of exogenous FFAs to increase energy expenditure.

144 tive metabolism in the presence of exogenous FFAs; this increase was not seen in Fabp4/Fabp5 double-k

145 suggest that CD8(+) TRM cells use exogenous FFAs and their oxidative metabolism to persist in tissue

146 FABP5 expression and enhanced extracellular FFA uptake were also demonstrated in human CD8(+) TRM ce

147 ed UCP2 expression, suggesting that the FABP-FFA equilibrium controls UCP2 expression.

148 We conclude that CD36 1) facilitates FFA transport into muscle and adipose tissue in humans w

149 low-density lipoprotein triacylglycerol from FFAs (4.06 +/- 2.57 mumol/min vs 4.34 +/- 1.82 mumol/min

150 Whether insulin stimulates greater FFA clearance into adipose tissue in vivo is unknown.

151 In the ADF-HF group, FFA concentrations were positively correlated with waist

152 associated with changes in FAT%, TC, HbA1c, FFA and HDL-c.

153 mponents dictating the gel strength are HCs, FFAs and WEs in a descending order of importance.

154 th FALs and a positive correlation with HCs, FFAs and WEs.

155 Hepatic FFA uptake was similar in all participants regardless of

156 levated, 2) is not rate limiting for hepatic FFA uptake, and 3) is needed for normal cardiac FFA upta

157 for studying the dynamic changes in hepatic FFA flux in models of liver disease.

158 Throughout the two study days, higher FFA levels were significantly associated with lower (inc

159 system, beyond the putative homolog of human FFA.

160 otonic code of face orientation in the human FFA, in line with primate electrophysiology studies that

161 ys of shelf life without a notable change in FFA content of rice bran fraction which was obtained fro

162 pproach enabled us to observe the changes in FFA hepatic uptake under different physiological conditi

163 However, detecting dynamic changes in FFA uptake by the liver in live model organisms has prov

164 using this method, we detected a decrease in FFA accumulation in the liver after mice were given inje

165 Despite the postprandial decrease in FFA-driven esterification and oxidation, VLDL-TAG secret

166 associated with a progressive impairment in FFA suppression during OGTT, whereas the rise in mean pl

167 is associated with a progressive increase in FFA and fasting Adipo-IR.

168 e found the representation of orientation in FFA to be compatible with a linear angle code.

169 nalysis revealed that multivoxel patterns in FFA-but not other face-selective brain regions, other ca

170 l dynamics of face information processing in FFA remains unclear.

171 membrane content and improving viability in FFA producing E. coli while maintaining FFA titers.

172 wever, the restored fitness did not increase FFA productivity, indicating the existence of additional

173 In conclusion, insulin does not increase FFA storage in adipose tissue compared with niacin, whic

174 ed an intravenous lipid emulsion to increase FFA concentrations during infusion of linoleate and palm

175 sociated with hyperinsulinemia and increased FFA-blood levels, the interplay between insulin and FFA

176 ates a metabolic reprogramming by increasing FFA and glucagon levels.

177 Total and individual FFA and plasma lipid concentrations were measured before

178 consistent with two interrelated influences: FFAd reduces the responsiveness of magnocellular neurons

179 Cooking, however, inhibited FFA formation and induced formation of PV and TBARS.

180 t FABP4/aP2 directly regulates intracellular FFA levels and indirectly controls macrophage inflammati

181 opinocytosis through a pathway that involves FFA receptors, the Gbeta/Ggamma complex, and RAC1.

182 ode were determined for mixtures of isolated FFAs to values in the low 10 pmol range.

183 scle (truncal postural and thigh locomotive) FFA uptake using [(11)C]palmitate positron emission tomo

184 y in FFA producing E. coli while maintaining FFA titers.

185 ations in the zonation of proteins mediating FFA uptake or triglyceride release as very low density l

186 ly diminished by inhibition of mitochondrial FFA beta-oxidation in vivo.

187 FFAs but not to protective, monounsaturated FFAs.

188 group, decreases were found in several more FFAs than in the ADF-HF group.

189 palmitate concentrations, whereas myocardial FFA uptake was diminished in the Pro90Ser homozygotes du

190 In this structure, no FFA is observed and the ceramide binding pocket and puta

191 ces of FFA: the first principal component of FFA shows differential connectivity with occipital and p

192 To enable noninvasive real-time imaging of FFA flux in the liver, we generated transgenic mice with

193 n, WD-feeding results in increased levels of FFA and microbiota that, even in absence of hyperglycaem

194 This panel of FFA ratios could be used for identification and early in

195 liquid cartridge extraction, purification of FFA fraction by solid phase extraction, boron trifluorid

196 or normal cardiac FFA uptake over a range of FFA concentrations from low to slightly elevated.

197 addition, we observed diurnal regulation of FFA hepatic uptake in living mice.

198 Replacement of FFA by OCT can be justified if there is a substantial ag

199 ty acid (FFA) pool is an important source of FFA and reflects inefficiency in dietary fat storage.

200 with different representational subspaces of FFA: the first principal component of FFA shows differen

201 Values of FFA, p-AV, TPC using TBHQ-SO with traditional frying wer

202 In myotubes, UT attenuated the ability of FFAs to induce insulin resistance and PP2A hyperactivity

203 gels increased with the increasing amount of FFAs and HCs and the decreasing amount of WEs and FALs.

204 iable procedure for quantitative analysis of FFAs in wort was developed and validated.

205 tion, which led to a higher concentration of FFAs in the mixed micelles.

206 was regulated by insulin-mediated control of FFAs.

207 n analytical method for the determination of FFAs in beer.

208 The determination of FFAs in wort was achieved via liquid-liquid cartridge ex

209 are more susceptible to the toxic effect of FFAs.

210 The acute effects of FFAs remain incompletely defined.

211 The extraction of FFAs in beer was achieved via Liquid-Liquid Cartridge Ex

212 e observed between groups in adipose flux of FFAs (414 +/- 195 mumol/min for HighLF vs 358 +/- 105 mu

213 ydrolysis of triacylglycerides, formation of FFAs and MAGs, and micellar incorporation of carotenoids

214 was a positive correlation between level of FFAs and level of MIR122 in plasma samples from 6 health

215 D also had higher nocturnal plasma levels of FFAs and did not suppress the contribution from de novo

216 (18.4% +/- 3.6%) had higher plasma levels of FFAs during the nighttime and higher concentrations of i

217 C to remove glycerol completely and most of FFAs; and the second distillation at optimized TE 155 de

218 FFAs coupled with impaired beta-oxidation of FFAs and inverse partitioning into complex lipids may be

219 proliferative insulin effect in presence of FFAs and prevented EGFR/CD95 association, CD95 tyrosine

220 Likewise, in presence of FFAs insulin increased apoptosis in hepatocytes from wil

221 g tolerance towards endogenous production of FFAs was implemented by modulating acyl-ACP pools with a

222 The release of FFAs and MAGs from TAGs proceeded faster than their inco

223 ocessing using electrodes placed directly on FFA in humans.

224 cts murine and human adipocytes from HFD- or FFA-elicited cell death through NF-kappaB-dependent upre

225 d cardiomyocytes challenged with TNFalpha or FFA, we demonstrate that 2-AG improves insulin sensitivi

226 tly greater for linoleate than for the other FFAs.

227 nd release and lipid synthesis (particularly FFA, triglycerides, and cholesterol), whereas glycogen p

228 -months of DAPA therapy, HbA1c, FBG, 2h-PBG, FFA, TG, blood pressure, BMI, WHR, body weight, FAT%, FI

229 To summarize, pharmacological FFA reduction improves insulin signalling in muscle from

230 e tested the hypothesis that pharmacological FFA reduction enhances insulin action by reducing local

231 t, despite a broad spectrum of physiological FFA concentrations, VLDL-TG SRs did not vary based on di

232 Plasma FFA and LPS levels were assessed, in addition to colonic

233 high (HF) or low (LF) in fat affects plasma FFA profiles in the context of weight loss, and changes

234 pimox 1) markedly reduced the fasting plasma FFA concentration and enhanced suppression of plasma FFA

235 al metabolites and concomitant higher plasma FFA.

236 ongly correlated with the decrease in plasma FFA and increase in insulin-mediated glucose disposal (b

237 We examined whether reduction in plasma FFA concentration with acipimox improved ATP synthesis r

238 osely correlated with the decrease in plasma FFA in obese NGT (r = 0.81) and T2DM (r = 0.76) subjects

239 Reduction in plasma FFA in obese NGT and T2DM individuals improves mitochond

240 entration and enhanced suppression of plasma FFA during oral glucose tolerance tests and insulin clam

241 mulated total glucose disposal (TGD), plasma FFA species, muscle insulin signalling, IBalpha protein,

242 Preventing the fall in plasma FFAs during insulin infusion either by administering int

243 oreover, mice with elevated levels of plasma FFAs as the result of a high-fat diet were more suscepti

244 Inhibiting the fall of plasma FFAs in these mice prevented the suppression of EGP duri

245 ctively analyzed 24 [product FFA]/[precursor FFA] ratios in fasting sera and clinical data from 481 i

246 als, we retrospectively analyzed 24 [product FFA]/[precursor FFA] ratios in fasting sera and clinical

247 the SCFA receptor free fatty acid receptor (FFA)3, one of the free fatty acid receptor family member

248 an SCFA receptor, free fatty acid receptor (FFA)3, to the enteric nervous system is unknown.

249 rved by neural computations within the right FFA as well as a re-entrant processing loop involving bi

250 lts elucidate the dynamic computational role FFA plays in multiple face processing stages and indicat

251 of a protective mechanism against saturated FFA-derived toxic metabolites that drive endoplasmic ret

252 ) coefficients for the analysis of saturated FFAs were found to be generally close to 0.98 over about

253 Our data show that the saturated FFAs palmitate and stearate induced cholangiocyte lipoap

254 Here, we demonstrate that the saturated FFAs palmitate and stearate induced robust and rapid cel

255 odocytes are highly susceptible to saturated FFAs but not to protective, monounsaturated FFAs.

256 Serum FFA, BA, and M30 were increased in NASH versus simple st

257 insulinemia or after oral niacin to suppress FFA compared with 11 saline control experiments.

258 pproximately 38% decrease in meal-suppressed FFA concentration (P < 0.0001) and an approximately 23%

259 e spherical nanostructure of the synthesized FFA based PNPs while attenuated total reflectance-fourie

260 tate to determine the proportion of systemic FFA that is converted to VLDL-TG.

261 This study finds that FFA is associated with IR starting in young adulthood.

262 It is concluded that FFAs can shift insulin-induced hepatocyte proliferation

263 and adipose tissues from mice, we found that FFAs increase hepatic expression and secretion of MIR122

264 The FFA family of receptors is a recently deorphanized set o

265 The FFA formed were transferred to the free lipid fraction,

266 ng-range functional connectivity between the FFA and the rest of the cortex during the same paradigm.

267 e methods, and followed the data to find the FFA and subsequently many other functionally specialized

268 nd adipose tissues of C57BL/6 mice given the FFA-inducer CL316243.

269 on: with increasing neural activation in the FFA, direct-gaze faces entered awareness more readily th

270 This simplifies screening of the FFA composition in crude tissue extracts.

271 We conclude that the FFA is involved in fine-grained neural encoding of scene

272 of error patterns further revealed that the FFA participated to a much larger extent in the neural e

273 hat local functional connectivity within the FFA was also reduced in individuals with ASD when viewin

274 quid-Liquid Cartridge Extraction (LLCE), the FFAs extract was purified by Solid Phase Extraction (SPE

275 edominantly molecular [M + K](+) ions of the FFAs, whereas other alkali metal adducts can be generate

276 The results showed that the % FFA was reduced by 44.3, PV by 50.2, and FOS reading by

277 These FFAs not only inhibited plant callose biosynthesis in vi

278 d extrarenal pools of Angptl4 reduced tissue FFA uptake in skeletal muscle, heart and adipose tissue,

279 es were advanced by 20 ms in IFJ compared to FFA or PPA.

280 ctions carrying face information from EVC to FFA and EVC to pSTS.

281 from EVC to the occipital face area, EVC to FFA, and EVC to posterior superior temporal sulcus (pSTS

282 ion presented a higher conversion of MAGs to FFAs during digestion, which led to a higher concentrati

283 The response to FFAs may function in the development of nephrotic syndro

284 diated hydrolysis of plasma triglycerides to FFAs.

285 Total FFA concentrations also decreased (P < 0.001).

286 difference in postprandial triacylglycerol, FFA, insulin, glucose, glucagon, or GIP related to prote

287 We concluded that these two FFAs have a major function in the suppression of the inn

288 analyses of mixtures containing unsaturated FFAs are also possible but require more effort on the ca

289 tably, both saturated and (poly-)unsaturated FFAs are detected sensitively in the presence of relativ

290 -beta signaling pathways were activated upon FFA treatment, potentially acting as upstream activators

291 te appreciable amounts of neutral lipid upon FFA treatment.

292 en magnocellular responses are mitigated via FFAd, human form perception is transiently sharpened bec

293 the disruption of the filtration barrier via FFAs bound to albumin and respond by enhancing fluid-pha

294 or-symmetric coding cannot be ruled out when FFA mean activity levels are considered as a dimension o

295 oronary heart disease, it is unclear whether FFA levels are associated with heart failure (HF).

296 fluence the risk of HF and determine whether FFAs could serve as a novel pharmacological target for H

297 However, the molecular mechanism by which FFA reduction improves insulin sensitivity in human subj

298 ed to explore biological mechanisms by which FFAs may influence the risk of HF and determine whether

299 atment of human liver cancer cell lines with FFAs exacerbated the EMT phenotype, whereas chemical inh

300 ally segregated functional subdomains within FFA.