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1 ntified a novel human FGF-7 homologue, named FGF-10.
2 cal to human FGF-10 and 91% identical to rat FGF-10.
3 nhibits expression of the critical morphogen FGF-10.
4 m is through secreted growth factors such as FGF-10.
5 posed to be typified by negligible levels of FGF-10.
6                                              FGF-10, a member of the fibroblast growth factor family,
7 dies with human cells indicated two modes of FGF-10 action: (i) translocation of rFGF-10 into urothel
8                                              FGF-10 alone did not stimulate prostatic bud formation i
9 he encoded protein is 92% identical to human FGF-10 and 91% identical to rat FGF-10.
10                                              Fgf-10 and Fgf-18 are expressed specifically within vent
11                                     Although FGF-10 and FGF-7 bind and activate the same resident epi
12   Cells expressing Wnt-2b are able to induce Fgf-10 and generate an extra limb when implanted into th
13                                              FGF-10 and its receptors, FGFR1 and FGFR2, have been imp
14                  These results indicate that FGF-10 and KGF have similar receptor binding properties
15 pe was partially reversed by the addition of FGF-10 and testosterone, resulting in the formation of p
16              Paracrine signaling mediated by FGF-10 and the FGF-R2IIIb receptor is required for forma
17 s expression of fibroblast growth factor-10 (FGF-10) and impairs epithelial-mesenchymal interactions
18 ctor I (IGF-I), fibroblast growth factor 10 (FGF-10), and Epithelial Growth Factor (EGF), which may b
19  of pro-proliferative signals such as IGF-1, FGF-10, and BMP4.
20 mal cells exhibit strictly a cell-associated FGF-10 antigen that correlates with an alternately trans
21                                        Thus, FGF-10 appears to act as a growth factor which is requir
22 -1), keratinocyte growth factor (FGF-7), and FGF-10 are homologues with distinct specificity.
23 ferred dual activities on both the FGF-7 and FGF-10 backbones.
24 ovalent cross-linking analysis revealed that FGF-10 binds with an affinity equal to FGF-7 to resident
25 tor Ptc, raising the possibility that WNT5a, FGF-10, BMP4, and SHH signaling pathways are functionall
26 utant lungs leads to increased expression of Fgf-10, Bmp4, Shh, and its receptor Ptc, raising the pos
27      These data suggested that inhibition of FGF-10 by inflammatory signaling involves the NF-kappaB-
28 clear translocation, prevented inhibition of FGF-10 by SP3.
29                                              FGF-10 can prevent or reduce lung specific inflammation
30                                     Although FGF-10 cDNA exhibits a signal sequence for secretion, cu
31  stromal cells in tissues, the expression of FGF-10 correlated with the presence of stromal cells of
32                    The mitogenic activity of FGF-10 could be distinguished from that of KGF by its di
33 developmental gene re-activation in IPF, and FGF-10 deficiency as a potentially critical factor in di
34                                              Fgf-10-deficient mice (Fgf-10(-/-)) were generated to de
35             Within this concentration range, FGF-10 did not stimulate DNA synthesis in NIH/3T3 mouse
36                                              FGF-10 does not seem to interfere with early epithelial
37                                              FGF-10 exerts a powerful chemoattractant effect on the d
38                                              FGF-10 exhibits a similar specificity but also binds the
39  to interfere with the normal stimulation of FGF-10 expression by Sp1.
40 ediators that inhibited airway branching and FGF-10 expression in LPS-resistant C.C3-Tlr4(Lpsd)/J fet
41 eat-shock induced Dkk1 blocked fgf-8 but not fgf-10 expression in the blastema.
42 b region, another Wnt gene, Wnt-8c, controls Fgf-10 expression, and is also capable of inducing ectop
43 d TNF-alpha) that activate NF-kappaB reduced FGF-10 expression, whereas chemokines that signal via ot
44 t of NF-kappaB and SP3 suppress SP1-mediated FGF-10 expression.
45                                  Strikingly, Fgf-10(-/-) fetuses continued to develop until birth, de
46 ype that synthesized FGF-10 RNA and (ii) the FGF-10 gene is located at the 5p12-p13 locus of chromoso
47       We maintained localized high levels of FGF-10 in cultured lungs using FGF-10-soaked heparin bea
48  NF-kappaB disrupts the normal expression of FGF-10 in fetal lung mesenchyme by interfering with the
49                The chemoattractant effect of FGF-10 in the lung epithelium is reminiscent of the patt
50                   Lastly, direct delivery of FGF-10 in the lungs of rats led to an increase of LR-MSC
51 ) were generated to determine the role(s) of Fgf-10 in vertebrate development.
52    BrdU incorporation analysis suggests that FGF-10, in contrast to FGF-7, is a modest proliferation
53 ments required for direct NF-kappaB-mediated FGF-10 inhibition.
54         In this study we investigate whether FGF-10 is able to direct lung epithelial buds to proper
55       Based on these results we propose that FGF-10 is an inductive signal that initiates ocular glan
56     In this report, we provide evidence that FGF-10 is both necessary and sufficient to initiate glan
57            During normal ocular development, FGF-10 is expressed in the perioptic mesenchyme adjacent
58 e induction of Fgf-8 in the limb ectoderm by FGF-10 is mediated by the induction of Wnt-3a.
59                                              Fgf-10 is necessary for apical ectodermal ridge (AER) fo
60 blast growth factor (FGF) family, designated FGF-10, is expressed during development and preferential
61 ults show that prostate stromal cell-derived FGF-10, like FGF-7, exhibits the properties of an androm
62 a-catenin act as key regulators of the FGF-8/FGF-10 loop.
63               Recent studies have shown that FGF-10 may be a key regulator of lung branching morphoge
64 ulation in wound repair, we cloned the mouse FGF-10 (mFGF-10) cDNA.
65                                              Fgf-10(-/-) mice exhibited perinatal lethality associate
66                   The pulmonary phenotype of Fgf-10(-/-) mice is strikingly similar to that of the Dr
67                     Prostatic rudiments from FGF-10(-/-) mice transplanted into intact male hosts gre
68 have examined the male reproductive tract of FGF-10(-/-) mice, and at birth, most of the male seconda
69 tatic anlagen, the urogenital sinus (UGS) of FGF-10(-/-) mice.
70         Limb bud initiation was abolished in Fgf-10(-/-) mice.
71 gs, suggesting that the protective effect of FGF-10 might be mediated, in part, by the mobilization o
72                Immunophenotyping analysis of FGF-10 mobilized and cultured cells revealed expression
73 crimal and Harderian glands were not seen in FGF-10 null fetuses.
74                      In cultures of UGS, the FGF-10 null phenotype was partially reversed by the addi
75         To better characterize the effect of FGF-10 on LR-MSCs, FGF-10 was intratracheally delivered
76                  Additionally, the effect of FGF-10 on lung-resident mesenchymal stem cells (LR-MSCs)
77 ween the fibroblast growth factors FGF-8 and FGF-10 plays a key role in limb initiation and AER induc
78 kappaB kinase beta mutant not only decreased Fgf-10 promoter activity but also increased RELA-SP3 nuc
79       SP3 co-expression reduced SP1-mediated Fgf-10 promoter activity, suggesting antagonistic intera
80                   Mutational analysis of the FGF-10 promoter failed to identify genetic elements requ
81 ased interactions between SP3, RELA, and the Fgf-10 promoter.
82 dent interactions between RELA, SP3, and the Fgf-10 promoter.
83 NF-kappaB and Sp1 physically interact at the FGF-10 promoter.
84                                The predicted FGF-10 protein is most related to keratinocyte growth fa
85                                              FGF-10 requires 1.4 times higher NaCl for elution from i
86                 In the absence of mesenchyme FGF-10 requires an associated proliferative signal to in
87 nces into the C-terminal portion of FGF-7 or FGF-10 revealed that substitution of GSCKRG for GIPVRG o
88       During proliferative phases, levels of FGF-10 rise at the urothelial cell surface and/or within
89  propria were the cell type that synthesized FGF-10 RNA and (ii) the FGF-10 gene is located at the 5p
90 igh levels of FGF-10 in cultured lungs using FGF-10-soaked heparin beads.
91               Epithelial buds grow toward an FGF-10 source within 24 h, and subsequently form concent
92                                              FGF-10(-/-) testes produced sufficient androgens to indu
93 s domain might underlie failure of FGF-7 and FGF-10 to bind to the FGFRIIIc isoforms.
94 mulate prostatic bud formation in control or FGF-10(-/-) UGS.
95            Similar to FGF-7, recombinant rat FGF-10 was a specific mitogen for prostate epithelial ce
96 haracterize the effect of FGF-10 on LR-MSCs, FGF-10 was intratracheally delivered into the lungs of r
97 ed that, similar to FGF-7, the expression of FGF-10 was responsive to androgen in stromal cells from
98                  Lens-specific expression of FGF-10 was sufficient to induce ectopic ocular glands wi
99    Importantly, fibroblast growth factor-10 (FGF-10) was markedly suppressed in IPF subjects with pro
100 ological and biochemical properties of human FGF-10, we isolated the cDNA and expressed its encoded p
101        Recombinant (r) preparations of human FGF-10 were found to induce proliferation of human uroth
102           Site-directed mutants of FGF-7 and FGF-10 were prepared to test whether this domain might u
103                       Fgf-10-deficient mice (Fgf-10(-/-)) were generated to determine the role(s) of
104                            Neither FGF-7 nor FGF-10 will bind to IIIc isoforms of FGFR.
105                      An understanding of how FGF-10 works in conjunction with these other processes w

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