ライフサイエンスコーパス: FGF-5

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1 s overexpressing fibroblast growth factor-5 (FGF-5).

2 e growth factor, fibroblast growth factor-5 (FGF-5).

3 nized nonmutated fibroblast growth factor-5 (FGF-5).

4 FGF receptor, the preferred FGF receptor for FGF-5.

5 Exogeneous FGF-5 (0.37 nM) increased the growth of COLO-357 cells b

6 Neither alpha(2)M nor alpha(2)M* bind to FGF-5, -7, -9, or -10.

7 eficient adenoviral construct overexpressing FGF-5 (AdvFGF-5) to improve flow and function in swine w

8 ults demonstrate that N-CAM, tenascin-C, and FGF-5 are dispensable for major aspects of synaptic deve

9 thelial cells exhibit expression in vitro of FGF-5 at the messenger RNA and protein levels.

10 Expression of FGF-5 by BCME cells was studied by a combination of Nort

11 Northern blot analysis with a 306 bp FGF-5 cDNA revealed the presence of 4.0 kb and 1.6 kb FG

12 ransgenic mice was substantially reversed in FGF-5-deficient mice.

13 Because nonglycosylated recombinant FGF-5 does not appear to be a mitogen in BCME cells in v

14 FGF-5 expression by quantitative real-time reverse trans

15 Differentiated myofibers express FGF-5, FGF-7, and reduced levels of FGF-6 mRNA.

16 human pancreatic cancer was investigated, as FGF-5 has a classical signal sequence for secretion not

17 In this study the role of FGF-5 in human pancreatic cancer was investigated, as FG

18 and in situ hybridization demonstrated that FGF-5 localized in the cancer cells, stromal fibroblast

19 These observations suggest that FGF-5 may participate in autocrine and paracrine pathway

20 A revealed the presence of 4.0 kb and 1.6 kb FGF-5 mRNA transcripts in both normal and cancerous panc

21 ic analysis indicated that 4.0 kb and 1.6 kb FGF-5 mRNA transcripts levels were increased 2.4- and 2.

22 FGF-5 mRNA was also detected in COLO-357 human pancreati

23 the mitogenic activity of human recombinant FGF-5 on a variety of cell types was evaluated, using a

24 ygous mutant mice lacking N-CAM, tenascin-C, FGF-5, or both N-CAM and tenascin-C.

25 class I molecules presenting a nine-residue FGF-5 peptide generated by protein splicing.

26 Furthermore, secreted FGF-5 protein was present in conditioned medium of COLO-

27 cinomas and little normal tissue expression, FGF-5 represents an immunotherapy target with potential

28 expressing human fibroblast growth factor-5 (FGF-5) resulted in messenger RNA and protein expression

29 rivascular and endothelial cell staining for FGF-5 seen previously in choroidal neovascular membranes

30 ected against the aminoterminus of the human FGF-5 sequence was used in Western blot analyses to iden

31 ded evidence for the expression of two major FGF-5 transcripts at 4 kb and 3 kb and two minor transcr

32 y Northern blot analysis for the presence of FGF-5 transcripts, using a 1-kb human complemetary DNA.

33 ular proliferation assays, human recombinant FGF-5 was not mitogenic in BCME cells but exhibited an a

34 time reverse transcription PCR revealed that FGF-5 was overexpressed in the majority of renal cell ca

35 xpression of the differentiation marker gene FGF-5 whereas the expression of the stem cell marker gen

36 RS cells produce fibroblast growth factor-5 (FGF-5), which acts in a paracrine fashion to terminate p

37 Studies with fully glycosylated recombinant FGF-5 will be required, however, to assess the biologic

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