ライフサイエンスコーパス: FPD

1 FPD/AML is a familial platelet disorder characterized by

2 FPD/AML patients have a bleeding disorder characterized

3 We corrected the RUNX1 mutation in 1 FPD iPSC line through gene targeting, which led to norma

4 e studied the in vitro megakaryopoiesis of 3 FPD/AML pedigrees.

5 Informative recombination events in 6 FPD/AML pedigrees with evidence of linkage to markers on

6 rrow or peripheral blood cells from affected FPD/AML individuals showed a decrement in megakaryocyte

7 let disorder with propensity to develop AML (FPD/AML).

8 Outcome measures were implant and FPD failures, biologic and prosthetic complications, and

9 milar to other viral FPDs, the putative ASLV FPD has been modeled as an amphipathic helix where most

10 Both FPD mutations accelerate formation of protofibrils, sugg

11 o 4.5 mm) supporting fixed partial dentures (FPDs) in posterior mandibular and maxillary jaws.

12 Fracture of ceramic fixed-partial dentures (FPDs) tends to occur in the connector area because of st

13 Footpad dermatitis (FPD) is used in the poultry industry as an animal welfar

14 an because of FPD are more likely to develop FPD in their own childbirth compared with women born vag

15 The 41 x 41-cm digital FPD is constructed on a single monolithic glass substrat

16 with autosomal-dominant Parkinson's disease (FPD).

17 high incidences of fetopelvic disproportion (FPD) in human childbirth.

18 gions referred to as fusion peptide domains (FPDs) at or near the amino terminus of the membrane-anch

19 tion of the familial paroxysmal dyskinesias (FPD) recognizes several distinct, although overlapping,

20 a nonfunctional version of the FP enhancer (FPD) that does not bind SF2/ASF also fails to block spli

21 nd the mononitrosyl adduct of the flavinated FPD (FDP(NO)) show nu(NO) at 1681 cm(-1), which is also

22 tal practitioners, and OPG was advocated for FPD planning, whereas CBCT was advocated for implant pla

23 No suitable animal models exist for FPD/AML, as Runx11/2 mice and zebra fish do not develop

24 Our findings support a model for FPD/AML in which haploinsufficiency of CBFA2 causes an a

25 that co-segregated with the disease in four FPD/AML pedigrees.

26 The same extraction procedure and a GC-FPD analysis were used to determine nitrile metabolites

27 The compounds were analysed by GC-FPD, GC-muECD or LC-MS/MS, with LOQs from 1 to 8 mug/kg.

28 tography with flame photometric detector (GC-FPD), respectively; was carried out.

29 toward the development of a miniaturized GC-FPD capable of ultrafast detection of low levels of OP a

30 In FPD/AML MKs, expression of MYL9 and MYH9 was decreased,

31 responsible for megakaryopoietic defects in FPD patients.

32 the defects in megakaryopoiesis observed in FPD/AML are, in part, related to a deregulation of myosi

33 tosomal dominant allele for non-kinesiogenic FPD.

34 edisposition to acute myelogenous leukaemia (FPD/AML, MIM 601399) is an autosomal dominant disorder c

35 edisposition for acute myelogenous leukemia (FPD/AML).

36 th predisposition to acute myeloid leukemia (FPD/AML) is an autosomal dominant disease of the hematop

37 rcurrent flame photometric detector (microcc-FPD) was adapted and optimized for ultrafast gas chromat

38 r and hydrogen are introduced to the microcc-FPD from opposite directions, creating a hydrogen-rich f

39 The microcc-FPD is capable of detecting very narrow (13 ms) chromato

40 In this microcc-FPD, combustion takes place between the burner tips with

41 , 71% birds in all treatments developed mild FPD and pens were top-dressed with dry litter to promote

42 of a microfabricated column and a miniature FPD is an important step toward the development of a min

43 scores during d21-42, HTM reduced the AUC of FPD lesion scores during d7-21 and d21-42.

44 M, LTM reduced area under the curve (AUC) of FPD lesion scores during d21-42, HTM reduced the AUC of

45 lies that women born by Caesarean because of FPD are more likely to develop FPD in their own childbir

46 FPD for mothers born by Caesarean because of FPD is 2.8 times the risk for mothers born vaginally.

47 by blebbistatin rescued the ploidy defect of FPD/AML MKs.

48 , which, in turn, has inflated incidences of FPD.

49 demonstrate successful in vitro modeling of FPD with patient-specific iPSCs and confirm that RUNX1 m

50 which we show here predicts that the risk of FPD for mothers born by Caesarean because of FPD is 2.8

51 strongly affects the fracture resistance of FPDs.

52 This study evaluated the impact of TM on FPD and consisted of 3 treatments supplemented with 0 (N

53 One FPD failed in the NDI group versus two FPDs in the SDI g

54 trogen phosphorous (NPD), flame photometric (FPD) detectors, as well as gas chromatography-olfactomet

55 greater for fixed partial denture planning (FPD) 59%, whereas CBCT was highly preferred for implant

56 were top-dressed with dry litter to promote FPD healing.

57 ut also reduced FPD development by promoting FPD wound healing.

58 mutations were introduced into the putative FPD.

59 improved growth performance but also reduced FPD development by promoting FPD wound healing.

60 Five-year data indicate that FPD treatment in posterior mandibular and maxillary jaws

61 The FPD provides radiographic images with excellent inherent

62 elope glycoprotein (EnvA) proposed to be the FPD is internal and contains a centrally located proline

63 Basic imaging characteristics of the FPD and associated image processing system were assessed

64 f curvature at the gingival embrasure of the FPD connector significantly affects the fracture resista

65 in a family with FPD/AML, and found that the FPD iPSCs display defects in megakaryocytic differentiat

66 Images obtained with the FPD demonstrated excellent uniformity, repeatability, an

67 Results with the FPD system were compared to those with a storage phospho

68 scatter content of images acquired with the FPD were equivalent to those acquired with the storage p

69 egated with the disease in the remaining two FPD/AML pedigrees at phylogenetically conserved amino ac

70 nite element models (FEMs), representing two FPD connector designs, were created in a manner correspo

71 One FPD failed in the NDI group versus two FPDs in the SDI group (P >0.99).

72 ffects the fracture resistance of three-unit FPDs.

73 Similar to other viral FPDs, the putative ASLV FPD has been modeled as an amphi

74 lls (iPSCs) from 2 patients in a family with FPD/AML, and found that the FPD iPSCs display defects in