ライフサイエンスコーパス: FSH receptor

1 The tumor lymphatic vessels did not express FSH receptor.

2 ly active by the mutation in the i3 from the FSH receptor.

3 splayed high-affinity binding to both CG and FSH receptors.

4 The small molecule FSH receptor agonists described here could lead to an or

5 ric receptor containing the EC region of the FSH receptor and the TM region of the LH receptor.

6 ctivated human TSH receptors, but not LH and FSH receptors, and showed high affinity to TSH receptors

7 xenograft tumors, after perfusion with anti-FSH-receptor antibodies coupled to colloidal gold, showe

8 that interactions between TM V and VI of the FSH receptor are essential for maintaining the receptor

9 FSH receptors are localized to Sertoli cells of the test

10 we mutated the follicle-stimulating hormone (FSH) receptor at the corresponding amino acids.

11 Lutropin (LH) and follitropin (FSH) receptors belong to a group of leucine-rich repeat-

12 ide mass increased from 1482 to 2327, LH and FSH receptor-binding affinities of the dual-specificity

13 eclined significantly, while the decrease in FSH receptor-binding affinity for eFSH was not significa

14 lpha subunit and a beta subunit, whereas the FSH receptor consists of two halves with distinct functi

15 The human follicle-stimulating hormone (FSH) receptor consists of two distinct domains of approx

16 noclonal antibodies that recognize different FSH receptor epitopes and in situ hybridization to detec

17 of FSH action through its ability to inhibit FSH receptor expression.

18 is, suppresses follicle-stimulating hormone (FSH) receptor expression, and stimulates kit ligand expr

19 rogens enhance follicle-stimulating hormone (FSH) receptor expression, which then augments FSH-mediat

20 ified and recombinant human FSH are the only FSH receptor (FSH-R) agonists available for infertility

21 ction, which causes the suppression of basal FSH receptor (FSH-R) expression, without affecting adeny

22 rminal hormone-binding fragment of the human FSH receptor (FSHR) extracellular domain (ECD).

23 Neither FSHbeta nor FSH receptor (FSHR) null mice have bone loss despite sev

24 ged AKT activation by FSH stimulation of the FSH receptor (FSHR) promotes gankyrin expression, which,

25 similarly to mesenchymal cells isolated from FSH receptor (FSHR)(-/-) mice.

26 FSH, a glycoprotein hormone, and the FSH receptor (FSHR), a G protein-coupled receptor, play

27 quired for this transition-and expression of FSH receptors (FSHR), which reflects the degree of bioch

28 regions in the follicle-stimulating hormone (FSH) receptor (FSHR) ECD that could bind its cognate lig

29 ollitropin, or follicle-stimulating hormone (FSH) receptor (FSHR), is a G protein-coupled receptor be

30 nocopying genetic haploinsufficiency for the Fsh receptor gene Fshr.

31 Immunoelectron microscopy was used to detect FSH receptor in mouse tumors.

32 epitopes and in situ hybridization to detect FSH receptor in tissue samples from patients with a wide

33 not to discriminate between the TSH and the FSH receptors, indicating for the first time that domain

34 FSH receptor integrity, and TGFbeta (transforming growth

35 nal stability which in turn is necessary for FSH-receptor interaction.

36 s coupled to colloidal gold, showed that the FSH receptor is exposed on the luminal endothelial surfa

37 FSH receptor is selectively expressed on the surface of

38 lt humans, the follicle-stimulating hormone (FSH) receptor is expressed only in the granulosa cells o

39 We next examined changes in FSH receptor mRNA expression.

40 ingly, BMP-15 severely reduced the levels of FSH receptor mRNA in both basal and FSH-stimulated cells

41 However, the FSH receptor mutants showed minimal increases in basal c

42 A truncated form of the Fsh receptor promoter served as the Cre driver.

43 m by which the follicle-stimulating hormone (FSH) receptor signals to promote activation of the p42/p

44 oblotting techniques involving four separate FSH-receptor-specific monoclonal antibodies that recogni

45 tropin (CG) or follicle-stimulating hormone (FSH) receptor, that at least two distinct bioactive popu

46 We fused the ectodomain of human LH or FSH receptors to the transmembrane region of fly LGR2.

47 In all 1336 patients examined, FSH receptor was expressed by endothelial cells in tumor

48 ring surgery performed to remove tumors, the FSH receptor was not expressed in the normal tissues loc

49 The endothelial cells that expressed FSH receptor were located at the periphery of the tumors

50 activity but was still capable of binding to FSH receptors, which are restricted to Sertoli cells in