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1 kinase activity is required for induction of FSHbeta and JunB protein.
2 c ratio and modulated the effects of GnRH on FSHbeta and LHbeta expression.
3 ependent secreted factors in regulating both FSHbeta and LHbeta gene expression.
4 ecific reduction of Homer1b/c increased both FSHbeta and LHbeta mRNA induction, whereas reduction of
5 mic neuron cells; CAMK2D, which promotes the FSHbeta and LHbeta secretion in pituitary cells; and OST
6 knockdown resulted in increased GnRH-induced FSHbeta and LHbeta transcript levels.
7       Transcriptional activation of both the FSHbeta and luteinizing hormone beta subunit (LHbeta) ge
8 eta-subunit of follicle-stimulating hormone (FSHbeta) are protected from bone loss despite severe est
9 ts preferential and paradoxical induction of FSHbeta by low frequency GnRH pulses are incompletely un
10 s of the dually active triple-domain chimera FSHbeta-CGbeta-alpha are synthesized, each corresponding
11                            Here we show that FSHbeta-deficient mice have lowered TNFalpha levels.
12 nto the regulated secretory pathway, whereas FSHbeta does not traverse the nuclear envelope region.
13                     These data identify that FSHbeta establishes a state of photosensitivity for the
14           Conversely, exogenous GDF9 induced FSHbeta expression in immortalized gonadotropes and in m
15 ular and secreted components, that regulates FSHbeta expression in response to activation of cell sur
16                                              FSHbeta expression increased in quail held in non-stimul
17 The presence of secreted factors influencing FSHbeta expression levels was tested by studying the eff
18                The independent regulation of FSHbeta expression provides an alternative pathway throu
19 ducing Galpha(s) signaling also disinhibited FSHbeta expression, suggesting the presence of a Galpha(
20 r inhibitor SB-505124, which also suppressed FSHbeta expression.
21  that contributes to feedback suppression of FSHbeta expression.
22 knockdown and cholera toxin-treated cells on FSHbeta expression.
23           Follicle-stimulating hormone-beta (FSHbeta) expression increased during the simulated sprin
24    Previously, we reported that GNRH induces FSHbeta (Fshb) transcription via induction of the AP-1 t
25 ne and MIS-mediated activation of LHbeta and FSHbeta gene expression, and suggest that MIS may modula
26 9) as a GnRH-suppressed autocrine inducer of FSHbeta gene expression.
27                In LbetaT2 gonadotrope cells, FSHbeta gene induction depended predominantly on Galpha(
28                       GnRH regulation of the FSHbeta gene occurs through induction of multiple Fos an
29 opin-releasing hormone (GnRH) agonist on the FSHbeta gene promoter and synergizes with the GnRH agoni
30 RH frequency-response characteristics of the FSHbeta gene.
31 s follicle-stimulating hormone beta-subunit (FSHbeta) gene expression in pituitary gonadotropes in a
32       Using Western blotting to assess human FSHbeta glycosylation inhibition revealed 30-47% nonglyc
33 onadotropes expressed alpha-GSU, LHbeta, and FSHbeta gonadotropin subunits while almost no cells expr
34  determined the effects of GDF9 knockdown on FSHbeta induction at different GnRH pulse frequencies us
35 oximal 398 bp, since its mutation eliminates FSHbeta induction by c-Fos and c-Jun.
36             Smad2/3 knockdown indicated that FSHbeta induction by GDF9 involved Smad2 and Smad3.
37 uction of Homer1a had the opposite effect on FSHbeta induction.
38 erodimers specified by unique beta subunits (FSHbeta/LHbeta).
39 resorption is decreased in haploinsufficient FSHbeta+/- mice with normal ovarian function, suggesting
40 F9 knockdown or immunoneutralization reduced FSHbeta mRNA expression.
41                                              FSHbeta mRNA induction by GDF9 and GnRH was synergistic.
42 , follicle-stimulating hormone beta subunit (FSHbeta) mRNA levels are significantly induced by additi
43 SH glycosylation varies due to inhibition of FSHbeta N-glycosylation, elaboration of 1-4 branches pos
44                                      Neither FSHbeta nor FSH receptor (FSHR) null mice have bone loss
45        Gonadotropes expressing alpha-GSU and FSHbeta only were detected in both male and female pitui
46                                         When FSHbeta or LHbeta genes were expressed, gonadotropes wer
47 exes exhibited kiss1 mRNA expression in most FSHbeta-positive cells and never in LHbeta-positive cell
48             The proximal 398 bp of the mouse FSHbeta promoter is sufficient for response to GnRH.
49      Here, we identify elements in the mouse FSHbeta promoter responsible for GnRH-mediated induction
50 reductions in serum FSH levels and pituitary FSHbeta subunit (Fshb) expression relative to controls,
51 ofluorescent localization as compared to the FSHbeta subunit and LHbeta mutants.
52                   In contrast, the wild-type FSHbeta subunit and the mutants LHbetaDeltaT and LHbetaL
53                                          The FSHbeta subunit is primarily regulated by gonadotropin-r
54 heptapeptide to the carboxyl terminus of the FSHbeta subunit resulted in an increased regulated secre
55 u-Ser-Gly-Leu-Leu-Phe-Leu), not found in the FSHbeta subunit, influences the intracellular behavior o
56 sted in two well-established activin assays: FSHbeta transcription and HepG2 cell apoptosis.
57 pression showed sustained elevated levels of FSHbeta under conditions of the spring equinox, compared