ライフサイエンスコーパス: FUdR

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1 FUdR exposure caused elevation of RR activity at 10 and

2 of sequential treatment with Adriamycin and FUdR (MCF-7 A/F) release the most IL-8 and express the g

3 lls treated sequentially with Adriamycin and FUdR expressed a metastatic phenotype.

4 The effect of enhanced viral replication by FUdR was suppressed by hydroxyurea, a known inhibitor of

5 Adriamycin and/or 5-fluoro-2'-deoxyuridine (FUdR), induced changes in the expressed phenotype.

6 orouridine (FUrd), 5-fluoro-2'-deoxyuridine (FUdR), trifluorothymidine (TFT); to the nonpyrimidine TS

7 dine nucleoside analog 5-fluorodeoxyuridine (FUdR) would inhibit growth, PhtC and PhtD conferred a gr

8 ynthetase (TS) inhibitor fluorodeoxyuridine (FUdR) can alter expression of cellular RR and GADD34, we

9 synthase (TS) inhibitor fluorodeoxyuridine (FUdR) in human cancer cell lines.

10 Treatment of H630-C6 cells with 5-FU, FUrd, FUdR, TFT, AG331, AG337, ZD1694, and methotrexate up-reg

11 rent hepatic arterial fluorodeoxyuridine (HA FUdR).

12 nstrate that the growth advantage of G207 in FUdR-treated cells is primarily based on an RR-dependent

13 HSV-1 was impaired in the presence of 10 nM FUdR, whereas G207 demonstrated increased replication un

14 The amino acid phosphoramidate diesters of FUdR (2) and Ara-C (6), 5-fluoro-2'-deoxy-5'-uridyl N-(1

15 etabolism of the phosphoramidate diesters of FUdR in proliferating tissue proceeds through two separa

16 lar RR and GADD34, we examined the effect of FUdR on G207 bioactivity with the hypothesis that FUdR-i

17 monophosphate and a lag in the generation of FUdR.

18 beled 5b resulted in the rapid production of FUdR 5'-monophosphate and a lag in the generation of FUd

19 Combined use of FUdR and G207 resulted in synergistic cytotoxicity.

20 osure of MCF-7 cells to either Adriamycin or FUdR rapidly increases, in a dose-dependent manner, the

21 inhibitors of deoxyribonucleotide synthesis FUdR, methotrexate and hydroxyurea than cells with wild-

22 on G207 bioactivity with the hypothesis that FUdR-induced cellular changes will alter viral prolifera

23 accompanied by a significant decrease in the FUdR IC(50)(72h) ( approximately 75-fold for SW620 cells

24 TS inhibition impairs viral replication, the FUdR-induced RR elevation may overcome this disadvantage

25 at a concentration of 100 microM, while the FUdR conjugates 5a,b exhibited IC50 values within a rang

26 te synthase inhibition that is comparable to FUdR in thymidine kinase deficient cells.

27 inhibition studies show that, in contrast to FUdR, the nitrofuran compound 2a is of comparable potenc

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