ライフサイエンスコーパス: Fallot

1 ptal defect, atrioventricular septal defect, Fallot, and truncus arteriosus operations were 1.1%, 0.6

2 found the following anomalies: tetralogy of Fallot (15 patients, 25%), tricuspid atresia (12 patient

3 Most patients had tetralogy of Fallot (18/31).

4 The most common diagnoses were tetralogy of Fallot (19%) and hypertrophic cardiomyopathy (14%).

5 ion (25%) or control cases with tetralogy of Fallot (30%).

6 Diagnoses included tetralogy of Fallot (51%), septal defects (20%), (congenitally correc

7 ), pulmonary atresia (11%), and tetralogy of Fallot (8%).

8 ty-seven patients with repaired tetralogy of Fallot (age >/=16 y; mean age, 35.8+/-10.1 y; 38 male) u

9 eurological outcomes, including tetralogy of Fallot (AOR 2.4, 95% CI: 1.1, 5.4), anencephaly (AOR 2.9

10 1.19; 95% CI: 1.00, 1.43), and tetralogy of Fallot (aOR = 1.44; 95% CI: 1.01, 2.19) in the various o

11 ricular septal defect (n = 11), tetralogy of Fallot (n = 3), atrioventricular canal (n = 3), and othe

12 great arteries (TGA) (n = 69), tetralogy of Fallot (n = 66), hypoplastic left heart syndrome (n = 51

13 /hypoplastic aortic arch (n=5), tetralogy of Fallot (n=1), hypoplastic right heart (n=1), and common

14 ension (PAH; n=10) and repaired tetralogy of Fallot (n=10) underwent MR-augmented cardiopulmonary exe

15 46), tricuspid atresia (n=103), tetralogy of Fallot (n=127), pulmonary atresia (n=177), heterotaxy sy

16 Patients with repaired tetralogy of Fallot (n=143; 12.5+/-3.2 years) had an echocardiogram a

17 es after Mustard/Senning (n=2), tetralogy of Fallot (n=2), aortic valve disease (n=2), and other bive

18 The majority had a variant of tetralogy of Fallot (n=36), or transposition of the great arteries, v

19 ons between carbon monoxide and tetralogy of Fallot (odds ratio = 2.04, 95% confidence interval: 1.26

20 to coarctation of the aorta and tetralogy of Fallot (P=0.002; Fisher exact test).

21 ardia (VT) late after repair of tetralogy of Fallot (rTOF).

22 alifornia carrying infants with tetralogy of Fallot (TOF) (n = 55), dextrotransposition of the great

23 late after total correction of tetralogy of Fallot (TOF) are at risk for major complications.

24 rly primary repair of acyanotic tetralogy of Fallot (ToF) can be performed safely with low requiremen

25 Patients after repair of tetralogy of Fallot (ToF) frequently have right ventricular (RV) dysf

26 ge versus "two-stage" repair of tetralogy of Fallot (TOF) in infants younger than 1 year of age.

27 ation in patients with repaired tetralogy of Fallot (TOF) is a risk factor for malignant ventricular

28 Tetralogy of Fallot (TOF) is the commonest cyanotic form of congenita

29 Tetralogy of Fallot (TOF) is the most common cyanotic congenital hear

30 Tetralogy of Fallot (TOF) is the most common cyanotic congenital hear

31 Tetralogy of Fallot (ToF) is the most common form of complex congenit

32 apt to hypoxia in children with tetralogy of Fallot (TOF) is unknown.

33 Individuals with tetralogy of Fallot (TOF) now routinely survive to reproductive age a

34 rtic root is a known feature in tetralogy of Fallot (TOF) patients with pulmonary stenosis (PS) or pu

35 e replacement (PVR) in repaired tetralogy of Fallot (TOF) reduces pulmonary regurgitation and decreas

36 The timing of repair of tetralogy of Fallot (TOF) remains controversial.

37 cal status in late survivors of tetralogy of Fallot (TOF) repair.

38 Tetralogy of Fallot (TOF) typically results in clinical cyanosis or v

39 Diagnoses included tetralogy of Fallot (TOF) with pulmonary atresia (n=13), TOF with pul

40 om 16 infants with nonsyndromic tetralogy of Fallot (TOF) without a 22q11.2 deletion, 3 fetal heart s

41 21.1 locus in 948 patients with tetralogy of Fallot (TOF), 1488 patients with other forms of CHD and

42 age, ventricular septal defect, tetralogy of Fallot (TOF), and an aortic arch abnormality.

43 e and 36% (n=326) of these have tetralogy of Fallot (TOF), comprising the largest subset of severe co

44 Disease entities included were tetralogy of Fallot (TOF), double outlet right ventricle (DORV), tran

45 ial recurrence of non-syndromic Tetralogy of Fallot (TOF), implicating genetic factors that remain la

46 Tetralogy of Fallot (TOF), the most common severe congenital heart ma

47 or risk alleles associated with Tetralogy of Fallot (TOF), using a northern European discovery set of

48 ohort of patients with repaired tetralogy of Fallot (TOF).

49 ain problematic after repair of tetralogy of Fallot (TOF).

50 nancy outcomes in patients with tetralogy of Fallot (TOF).

51 e complications after repair of Tetralogy of Fallot (ToF).

52 outflow tract of postoperative tetralogy of Fallot (TOF).

53 ion factor NKX2.5 as a cause of tetralogy of Fallot (TOF).

54 p of preoperative patients with tetralogy of Fallot (TOF).

55 atients after primary repair of tetralogy of Fallot (TOF).

56 arteriosus (TA), and 15.9% with tetralogy of Fallot (TOF).

57 to be a single-gene defect and tetralogy of Fallot a polygenic disorder with a small number of inter

58 ed pilot study in patients with tetralogy of Fallot aged 10 to 25 years.

59 lot with pulmonary atresia than tetralogy of Fallot alone.

60 agille syndrome, which includes tetralogy of Fallot among its more severe cardiac pathologies.

61 Eight patients with tetralogy of Fallot and 7 patients with transposition of the great ar

62 Of the 34 patients with tetralogy of Fallot and abnormal CA, 7 (21%) demonstrated CA compress

63 onary artery anomalies, such as tetralogy of Fallot and double outlet right ventricle.

64 uman congenital heart diseases, tetralogy of Fallot and double outlet right ventricle.

65 inically significant portion of tetralogy of Fallot and idiopathic AV block.

66 rwent surgical intervention for tetralogy of Fallot and major aortopulmonary collateral arteries at L

67 native and previously operated tetralogy of Fallot and major aortopulmonary collateral arteries.

68 laterals (APCs) in infants with tetralogy of Fallot and pulmonary atresia (TOF/PA) and to prospective

69 in 12 of 25 patients (48%) with tetralogy of Fallot and pulmonary atresia, and 4 of 54 (8%) with pros

70 Adults with repaired tetralogy of Fallot and RV dysfunction (RV ejection fraction [EF] <50

71 dian 12.0 years after repair of tetralogy of Fallot and similar lesions were studied echocardiographi

72 f abnormalities associated with tetralogy of Fallot and the DiGeorge syndrome.

73 We detected associations for tetralogy of Fallot and the upper exposure categories for TCAA, DCAA,

74 of PR in patients with repaired tetralogy of Fallot and warrants further investigation.

75 diac death late after repair of tetralogy of Fallot are devastating complications in adult survivors

76 ction in patients with repaired tetralogy of Fallot are incompletely understood.

77 Patients with repaired tetralogy of Fallot are monitored for pulmonary regurgitation (PR) an

78 rs after surgical correction of tetralogy of Fallot are nonexistent.

79 tcomes of transatrial repair of tetralogy of Fallot are unknown.

80 with Abernethy malformation and tetralogy of Fallot associated with nodular regenerative hyperplasia

81 f PVR in adults after repair of tetralogy of Fallot at a single tertiary center were retrospectively

82 ergoing a transatrial repair of tetralogy of Fallot between 1980 and 2005 were reviewed, their follow

83 p 2 (n = 22) received repair of tetralogy of Fallot by the traditional technique with ventricular sep

84 r diagnosis and first repair of Tetralogy of Fallot compared to natural progression.

85 llar infarct and a history of a tetralogy of Fallot corrected during childhood.

86 Patients with repaired tetralogy of Fallot experience variable outcomes for reasons that are

87 ar tachycardia (VT) in repaired Tetralogy of Fallot focuses on isthmuses in the right ventricle but m

88 and trended toward lower in the tetralogy of Fallot group (13.5+/-1.29 mL/kg per minute; P=0.06) comp

89 PVR after repair of tetralogy of Fallot has a low and improving mortality, with a low nee

90 e third of adults with repaired tetralogy of Fallot have an aortic root diameter >/=40 mm, the preval

91 placement (PVR) after repair of tetralogy of Fallot have recently been broadened to include asymptoma

92 ied (atrial septal defect in 6, tetralogy of Fallot in 4, and Fontan procedure in 6).

93 patient died after surgery for tetralogy of Fallot in situs inversus.

94 sex-specific controls, repaired tetralogy of Fallot in women had larger right ventricular end-systoli

95 Tetralogy of Fallot is a congenital heart disease that requires surgi

96 placement (PVR) after repair of tetralogy of Fallot is commonly required and is burdensome.

97 linical outcome after repair of tetralogy of Fallot is determined by the adaptation of the right vent

98 atrioventricular canal, and the tetralogy of Fallot malformation.

99 ection." Patients with repaired tetralogy of Fallot often have pulmonary regurgitation, which is freq

100 ptal defect was associated with tetralogy of Fallot or double-outlet right ventricle in 3 individuals

101 years) with surgically repaired tetralogy of Fallot or pulmonary atresia with ventricular septal defe

102 rome and some cases of isolated tetralogy of Fallot or pulmonic stenosis.

103 my, especially in patients with tetralogy of Fallot or transposition of the great arteries.

104 We included 413 tetralogy of Fallot patients (age, 36 +/- 13 years; QRS duration, 148

105 e (ACHD) centres that follow up Tetralogy of Fallot patients and Great Ormond Street Hospital (GOSH),

106 , this physiology is present in tetralogy of Fallot patients at mid-term follow-up and whether it is

107 ds from 28 consecutive repaired Tetralogy of Fallot patients from 2 centers who underwent VT ablation

108 Both the PAH and tetralogy of Fallot patients had blunted exercise-induced increases i

109 Although tetralogy of Fallot patients had the largest increase in cardiac outp

110 hich, when defective, cause the tetralogy of Fallot phenotype.

111 (PVR) in patients with repaired tetralogy of Fallot provides symptomatic benefit and right ventricula

112 In the first 24 hours after tetralogy of Fallot repair (n=11 patients), serial prospective measur

113 inical outcomes in adults after tetralogy of Fallot repair (rTOF).

114 V) dysfunction are common after tetralogy of Fallot repair (rTOF).

115 al cavopulmonary connection and tetralogy of Fallot repair and may prove to be an important therapeut

116 ive patients receiving standard tetralogy of Fallot repair in a single institution from 1964 to 2009.

117 n patients, 3 to 35 years after tetralogy of Fallot repair or pulmonary valvotomy, had PR measured by

118 V) restrictive physiology after tetralogy of Fallot repair results in low cardiac output and a prolon

119 After tetralogy of Fallot repair, acute restrictive RV physiology is associ

120 ate the long-term outcome after tetralogy of Fallot repair, yet the diagnostic and predictive value o

121 ion and exercise capacity after tetralogy of Fallot repair.

122 oing Fontan-type operations and tetralogy of Fallot repair.

123 be present early and late after tetralogy of Fallot repair.

124 patients require PVR late after tetralogy of Fallot repair.

125 g a nontransannular approach to tetralogy of Fallot repair.

126 Most patients with repaired tetralogy of Fallot require pulmonary valve replacement (PVR), but th

127 rmed a cross-sectional study of tetralogy of Fallot subjects who were tested for 22q11.2 deletion, an

128 rd deviation scores in repaired tetralogy of Fallot suggest that women perform poorer than men in ter

129 f PVR in patients with repaired tetralogy of Fallot that developed pulmonary insufficiency, until Dec

130 tory follow-up of patients with Tetralogy of Fallot to model the health-related costs and outcomes ov

131 to cardiac defects ranging from tetralogy of Fallot to transposition of the great arteries and that d

132 t of 252 patients with repaired tetralogy of Fallot undergoing programmed ventricular stimulation was

133 d on 205 patients with repaired tetralogy of Fallot undergoing PVR at our institution between 1988 an

134 Patients with repaired tetralogy of Fallot undergoing PVR with history of ventricular tachyc

135 nge, 8-20 years]) with repaired tetralogy of Fallot underwent cardiac magnetic resonance for ventricu

136 ollow-up, transatrial repair of tetralogy of Fallot was associated with a minimal risk of sudden deat

137 significantly increased risk of tetralogy of Fallot was observed (7 cases in fluconazole-exposed preg

138 ost per patient of a repair for Tetralogy of Fallot was pound26,938 (SE = pound4,140).

139 Tetralogy of Fallot was present in 23 subjects and was accompanied by

140 lation was 21.5+/-11 years, and tetralogy of Fallot was the cardiac condition in 59%.

141 secutive patients with repaired tetralogy of Fallot were analyzed (median age at follow-up 23 years [

142 gurgitation following repair of tetralogy of Fallot were considered benign.

143 Autopsy hearts with tetralogy of Fallot were evaluated to clarify the pathological substr

144 en hearts with the diagnosis of tetralogy of Fallot were examined.

145 Infants with tetralogy of Fallot were smaller in all measured dimensions, but they

146 ior history was significant for tetralogy of Fallot which was repaired at nine months of age.

147 (PVR) in patients with repaired tetralogy of Fallot who develop pulmonary insufficiency remains uncle

148 nsists of the 144 patients with tetralogy of Fallot who underwent surgical repair at <15 years of age

149 the human birth defect syndrome tetralogy of Fallot with complete pulmonary artery atresia.

150 Four groups of tetralogy of Fallot with confluent central pulmonary arteries were st

151 Tetralogy of Fallot with cyanosis requiring surgical repair in early

152 Tetralogy of Fallot with diminutive pulmonary arteries and severe pul

153 catheter management approach in tetralogy of Fallot with diminutive pulmonary arteries.

154 Tetralogy of Fallot with major aortopulmonary collateral arteries is

155 w tract stenting of symptomatic tetralogy of Fallot with poor anatomy (small pulmonary arteries) and

156 lagille syndrome suffering from tetralogy of Fallot with pulmonary atresia and multiple aortopulmonar

157 ic features, and more common in tetralogy of Fallot with pulmonary atresia than tetralogy of Fallot a

158 stenosis is rare and resembles tetralogy of Fallot with pulmonary atresia: There is a high incidence

159 cribed as a phenotypic mimic of Tetralogy of Fallot with pulmonary atresia; however, subsequent repor

160 p 1 (n = 20) received repair of tetralogy of Fallot with the modified technique with transatrial vent

161 s had undergone total repair of tetralogy of Fallot within 2 wks after admission for RSV infection.

162 eripheral pulmonic stenosis and tetralogy of Fallot) in the absence of liver dysfunction.

163 years; range, 8.5 to 64.9; 72% tetralogy of Fallot) underwent PVR for severe pulmonary regurgitation

164 maller placental size at birth: tetralogy of Fallot, -0.45 (95% confidence interval, -0.58 to -0.31);

165 ctive endocarditis was 1.3% for tetralogy of Fallot, 2.7% for isolated ventricular septal defect, 3.5

166 ), including 9 of 201 (4%) with tetralogy of Fallot, 3 of 71 (4%) with a secundum ASD, one each with

167 7.8%; 2) paternal anesthesia in tetralogy of Fallot, 3.6%; 3) painting in atrioventricular septal def

168 sition of the great arteries or tetralogy of Fallot, an event rate of 1/454 patients-years.

169 atent ductus arteriosus, 2 with tetralogy of Fallot, and 1 with pulmonary valve dysplasia.

170 position of the great arteries, tetralogy of Fallot, and anomalous pulmonary venous return, were also

171 ect, ventricular septal defect, tetralogy of Fallot, and transposition of the great arteries decrease

172 ing ventricular septal defects, tetralogy of Fallot, and tricuspid atresia, defects that resemble tho

173 right shunts, valvular disease, tetralogy of Fallot, and truncus arteriosus.

174 nes in mice are involved in the tetralogy of Fallot, and, using zebrafish phenotypes, propose the hyp

175 ly 20 years after operation for tetralogy of Fallot, aortic stenosis and coarctation.

176 n of the great arteries, and 81 tetralogy of Fallot, as well as their unaffected parents.

177 l patients after correction for tetralogy of Fallot, BNP levels were elevated and correlated signific

178 l defect, atrial septal defect, tetralogy of Fallot, coarctation of the aorta, cleft lip, cleft palat

179 aberrant subclavian artery and Tetralogy of Fallot, demonstrating that Hoxa1 is required for pattern

180 position of the great arteries, tetralogy of Fallot, double-chamber right ventricle, interrupted aort

181 ect, ventricular septal defect, tetralogy of Fallot, Ebstein anomaly, transposition of the great vess

182 abnormalities, five with severe tetralogy of Fallot, four with Ebstein's anomaly and two with single

183 or =10 mm, diagnosis other than tetralogy of Fallot, Genesis stent, higher prestent RV:aortic pressur

184 In tetralogy of Fallot, heart defects occurred in seven (3.1%) of 223 of

185 ified included heart failure in tetralogy of Fallot, mechanical circulatory support/transplantation,

186 fic malformations that resemble tetralogy of Fallot, overriding aorta with ventricular septal defect,

187 ventricular septal defects, and tetralogy of Fallot, resembling some of the most common congenital ma

188 After repair of tetralogy of Fallot, right ventricular (RV) dilation has been associa

189 In patients with tetralogy of Fallot, severe pulmonary stenosis and diminutive pulmona

190 nts with atrial septal defects, tetralogy of Fallot, single ventricle physiology, and following cardi

191 ongenital heart defects such as tetralogy of Fallot, systemic right ventricle, and univentricular hea

192 phic ventricular tachycardia in tetralogy of Fallot, the anatomy and histology of these regions have

193 been described in patients with tetralogy of Fallot, the scope of the problem remains poorly defined.

194 In patients with tetralogy of Fallot, the strongest correlations were observed with le

195 sus, valvar pulmonary stenosis, tetralogy of Fallot, transposition of the great arteries, muscular ve

196 rdiovascular disease, including tetralogy of Fallot, truncus arteriosus and interruption of the aorti

197 Severe CHD was defined as tetralogy of Fallot, truncus arteriosus, transposition complexes, end

198 in adult patients with repaired tetralogy of Fallot.

199 RVOT-sparing strategy to treat tetralogy of Fallot.

200 tenosis, pulmonic stenosis, and tetralogy of Fallot.

201 atifying patients with repaired tetralogy of Fallot.

202 ongenital cardiac defect termed tetralogy of Fallot.

203 nction in adults after repaired tetralogy of Fallot.

204 ae for patients after repair of tetralogy of Fallot.

205 block, idiopathic AV block, or tetralogy of Fallot.

206 er transannular patch repair of tetralogy of Fallot.

207 ntricular septal defect or with tetralogy of Fallot.

208 ly, of patients after repair of tetralogy of Fallot.

209 nant of outcome after repair of tetralogy of Fallot.

210 ive failure than in hearts with tetralogy of Fallot.

211 well as in surgically repaired tetralogy of Fallot.

212 ition of the great arteries and tetralogy of Fallot.

213 al heart defects in general, or tetralogy of Fallot.

214 he clinical variability seen in tetralogy of Fallot.

215 ance in patients with corrected tetralogy of Fallot.

216 outcome parameters in repaired tetralogy of Fallot.

217 and young adults with repaired tetralogy of Fallot.

218 -D deletions had CTDs including tetralogy of Fallot.

219 tcome in patients with repaired tetralogy of Fallot.

220 es ablation outcome in repaired Tetralogy of Fallot.

221 nd among patients with repaired tetralogy of Fallot.

222 f the great arteries; and 8 for tetralogy of Fallot.

223 nt class to have offspring with tetralogy of Fallot.

224 atation in adults with repaired tetralogy of Fallot.

225 may confer an increased risk of tetralogy of Fallot.

226 utations accounting for 2.3% of Tetralogy of Fallot.

227 art defects such as in repaired tetralogy of Fallot.

228 onary artery stenosis (n = 94), tetralogy of Fallot/pulmonary atresia (n = 72), congenital branch pul

229 ight Ventricular Dysfunction in Tetralogy of Fallot: Inhibition of the Renin-Angiotensin-Aldosterone

230 ed by supplement use except for tetralogy of Fallot; among supplement users, those in the Western cla

231 osis; coarctation of the aorta; tetralogy of Fallot; and D-transposition of the great arteries.