ライフサイエンスコーパス: Fc gamma RIII

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1 Fc gamma RIII cross-linking stimulated PBMC to release 5

2 a RI cross-linking and indirectly through an Fc gamma RIII-stimulated soluble lymphocyte factor.

3 Fc gamma RI (CD64), Fc gamma RII (CD32), and Fc gamma RIII (CD16), are expressed on blood leukocytes.

4 o additional IgG FcR, Fc gamma RI (CD64) and Fc gamma RIII (CD16), which may also participate in leuk

5 ain-deficient mice that lack Fc gamma RI and Fc gamma RIII, Fc gamma RII-deficient mice, and mice def

6 binding to all Fc gamma R; Fc gamma RII and Fc gamma RIII also utilize residues outside this common

7 to the other Fc receptors, Fc gamma RII and Fc gamma RIII.

8 Fc epsilon RI, Fc gamma RII, and Fc gamma RIII expression was not affected.

9 Fc gamma Rs: Fc gamma RI, Fc gamma RII, and Fc gamma RIII.

10 -type, FcR gamma-chain-, Fc gamma RIIb-, and Fc gamma RIII-deficient mice from infection.

11 IL-8 in response to immobilized IgG or anti-Fc gamma RIII Abs, we hypothesized that lymphocyte Fc ga

12 required for the enhanced Th2 responses, as Fc gamma RIII(-/-) DCs failed to augment Th2-mediated ai

13 the genes encoding Ig Fc gamma RIII or both Fc gamma RIII and Fc epsilon RI.

14 hereas in CRPtg/Fc gamma RIIb(-/-) and CRPtg/Fc gamma RIII(-/-) neointimal thickness was equal to or

15 hat carry IIIA 5' flanking sequences express Fc gamma RIII in macrophages and NK cells.

16 mice that contain IIIB 5' sequences express Fc gamma RIII in neutrophils only.

17 s thereby raising the threshold required for Fc gamma RIII activation and preventing autoantibody-tri

18 studies reveal a novel and specific role for Fc gamma RIII signaling in the regulation of Th cell res

19 Further, Fc gamma RIII(-/-) mice had impaired Th2 cytokine produc

20 ptors for the Fc domain of immunoglobulin G, Fc gamma RIII, are encoded by two genes (IIIA and IIIB)

21 e cell type-specific expression of the human Fc gamma RIII genes within the 5' flanking sequences and

22 ice with disruption of the genes encoding Ig Fc gamma RIII or both Fc gamma RIII and Fc epsilon RI.

23 s are accessible to circulating neutrophils, Fc gamma RIII-deficient mice had a significant reduction

24 independent of the well-established role of Fc gamma RIII in augmenting antigen presentation.

25 The recent crystal structure of Fc gamma RIII in complex with IgG-Fc has provided detail

26 ntracellular signals mediated via CD2 and/or Fc gamma RIII (CD16).

27 Supernatants from IgG- or Fc gamma RIII -stimulated lymphocytes induced monocytes

28 L-8 in response to supernatants from IgG- or Fc gamma RIII-stimulated lymphocytes, suggesting that th

29 s specific for Fc gamma RI, Fc gamma RII, or Fc gamma RIII.

30 (ADCC) through Fc gamma RI, Fc gamma RII, or Fc gamma RIII.

31 Ligation of the low affinity receptor Fc gamma RIII was specifically required for the enhanced

32 nd CRPtg lacking Fc gamma RI, Fc gamma RIIb, Fc gamma RIII, or the common gamma chain (FcR gamma) had

33 e low-affinity IgG receptors Fc-gamma RIIb2, Fc-gamma RIII, and the FcR-associated gamma-chain.

34 by differential reconstitution in vivo that Fc gamma RIII on mast cells is necessary for this inflam

35 cific for the tumor antigen HER2/neu and the Fc gamma RIII-activating receptor (CD16) found on NK cel

36 hydrate moieties are on the periphery of the Fc gamma RIII-Fc interface.

37 us, the interaction of immune complexes with Fc gamma RIII may mediate early neutrophil recruitment i

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