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1 Fc gamma RIII cross-linking stimulated PBMC to release 5
3 Fc gamma RI (CD64), Fc gamma RII (CD32), and Fc gamma RIII (CD16), are expressed on blood leukocytes.
4 o additional IgG FcR, Fc gamma RI (CD64) and Fc gamma RIII (CD16), which may also participate in leuk
5 ain-deficient mice that lack Fc gamma RI and Fc gamma RIII, Fc gamma RII-deficient mice, and mice def
6 binding to all Fc gamma R; Fc gamma RII and Fc gamma RIII also utilize residues outside this common
11 IL-8 in response to immobilized IgG or anti-Fc gamma RIII Abs, we hypothesized that lymphocyte Fc ga
12 required for the enhanced Th2 responses, as Fc gamma RIII(-/-) DCs failed to augment Th2-mediated ai
14 hereas in CRPtg/Fc gamma RIIb(-/-) and CRPtg/Fc gamma RIII(-/-) neointimal thickness was equal to or
17 s thereby raising the threshold required for Fc gamma RIII activation and preventing autoantibody-tri
18 studies reveal a novel and specific role for Fc gamma RIII signaling in the regulation of Th cell res
20 ptors for the Fc domain of immunoglobulin G, Fc gamma RIII, are encoded by two genes (IIIA and IIIB)
21 e cell type-specific expression of the human Fc gamma RIII genes within the 5' flanking sequences and
22 ice with disruption of the genes encoding Ig Fc gamma RIII or both Fc gamma RIII and Fc epsilon RI.
23 s are accessible to circulating neutrophils, Fc gamma RIII-deficient mice had a significant reduction
28 L-8 in response to supernatants from IgG- or Fc gamma RIII-stimulated lymphocytes, suggesting that th
32 nd CRPtg lacking Fc gamma RI, Fc gamma RIIb, Fc gamma RIII, or the common gamma chain (FcR gamma) had
34 by differential reconstitution in vivo that Fc gamma RIII on mast cells is necessary for this inflam
35 cific for the tumor antigen HER2/neu and the Fc gamma RIII-activating receptor (CD16) found on NK cel
37 us, the interaction of immune complexes with Fc gamma RIII may mediate early neutrophil recruitment i
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