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1                                              Fe3 Mo3 C supported IrMn as a bifunictional catalysts ex
2                                              Fe3+ and Pb2+ inhibited weakly, exhibiting Kis of 50 mic
3                                              Fe3+ was detected indirectly by reducing the nonabsorbin
4 econstituted to various core sizes (100-3500 Fe3+/HoSF) by depositing Fe(OH)3 within the hollow HoSF
5 ue, yolk-shell structured MPHNs comprising a Fe3 O4 core within a hollow cavity encircled by a porous
6 he [2Fe-2S]2+ cluster, with the release of a Fe3+ ion and two sulfide ions.
7 oxidized form of cytochrome oxidase (heme a3 Fe3+ and CuB2+, plus hydroxide).
8                                     Although Fe3+-sub-(Mn)SOD binds the small anions N3- and F-, the
9 implied the concerted involvement of both an Fe3+ reductase and FeoB in the uptake of Fe supplied as
10 site of bovine brain calcineurin contains an Fe3+-Zn2+ dinuclear metal center.
11 te labile cytoslic Fe2+, and calcein plus an Fe3+ chelator to estimate total cytosolic labile iron (F
12 f the plasmid was produced by exposure to an Fe3+/ascorbic acid free radical generating system.
13 rushes of different hydrophilicity on Au and Fe3 O4 surfaces separately.
14     In contrast, as estimated by calcein and Fe3+ chelator, total erythrocyte labile iron was similar
15 ssential to uptake of environmental Cu2+ and Fe3+.
16 d with adriamycin, dithiothreitol (DTT), and Fe3+ under aerobic, aqueous conditions yields double-str
17 2 is significantly enhanced by both Fe2+ and Fe3+ chelators.
18  tendency to form ordered arrays of Fe2+ and Fe3+ ions competes with the topological frustration of t
19 gly depend on the speciation of the Fe2+ and Fe3+ phases, although the underlying reasons remain uncl
20 e feoB mutation fully restored both Fe2+ and Fe3+ transport.
21             Ferrozine inhibition of Fe2+ and Fe3+ uptake implied the concerted involvement of both an
22  product phosphate to both the Fe3+-Fe2+ and Fe3+-Zn2+ forms of calcineurin led to perturbations of t
23 is quenched to different extents by Fe2+ and Fe3+.
24 cquisition systems, including both Fe2+- and Fe3+-citrate transporters.
25 des based on a polypyrrole gel framework and Fe3 O4 nanoparticles as a model system in this study dem
26 human holo-transferrin, holo-lactoferrin and Fe3+-dicitrate, but not by FeSO4.
27           MCD spectra of native Fe3+-LPO and Fe3+-CN--LPO are approximately 10 nm red shifted from an
28                              Cu2+, Zn2+, and Fe3+ association with Abeta explains the recently report
29  an iron oxide lowering EH values of aqueous Fe3+/Fe2+ redox couples.
30 nite-symplesite solid solution identified as Fe3(PO4)1.7(AsO4)0.3.8H2O.
31 embers of the spinel ferrite family, such as Fe3 O4 and CoFe2 O4 , which have similar crystallographi
32 o those on GC, but for redox systems such as Fe3+/2+, ascorbic acid, dopamine, and oxygen, the kineti
33 ase and FeoB in the uptake of Fe supplied as Fe3+.
34 eitol (DTT)/Fe3+/O2 but not by the ascorbate/Fe3+/O2 MCO system.
35  vesicle assembled from Janus amphiphilic Au-Fe3 O4 NPs grafted with polymer brushes of different hyd
36 nic behavior between Au NPs, Au NPPs, and Au-Fe3 O4 NPPs; magnetic studies found that the addition of
37 obate and bifunctional plasmonic-magnetic Au-Fe3 O4 @hexaniobate nanopeapods (NPPs).
38 esicle shell is composed of two layers of Au-Fe3 O4 NPs in opposite direction, and the orientation of
39 t was determined that some metal ions (Be2+, Fe3+, Al3+, Ru2+ and Dy2+) are extraordinarily disruptiv
40              Covalent bond formation between Fe3+ and His93(F8) provides an additional factor of 10(3
41 ults demonstrate that the reactivity between Fe3+ and dopamine quickly facilitates the degradation of
42 n the holo-protein conformation when binding Fe3+.
43 d amino acid analysis shows that Y447H binds Fe3+ in approximately 10 of the 12 active sites of 3,4-P
44 as been the endocytosis of transferrin-bound Fe3+ by the transferrin receptor.
45     A new metal-organic framework, Fe-BTTri (Fe3[(Fe4Cl)3(BTTri)8]2.18CH3OH, H3BTTri =1,3,5-tris(1H-1
46  Transcription of the ugd gene is induced by Fe3+ via the PmrA-PmrB two-component system and by low M
47               In vitro, NADH was oxidized by Fe3+ up to 16-fold faster than NADPH, despite their iden
48 alyst consisting of iron-molybdenum carbide (Fe3 Mo3 C) and IrMn nanoalloys is demonstratred.
49 ch issues, well-designed yolk-shelled carbon@Fe3 O4 (YSC@Fe3 O4 ) nanoboxes as highly efficient sulfu
50 h is a hydrocarbon analog of Dewar's classic Fe3(CO)12 complex.
51 oms are fused to afford the tri-iron cluster Fe3(COT)3, which is a hydrocarbon analog of Dewar's clas
52 f metal ions including Li+, Cs+, Cu2+, Co2+, Fe3+, and Al3+.
53 ely 100 times higher than that of commercial Fe3 O4 (Feridex, ILP = 0.15 nH m(2) kg(-1) ) at Happl .f
54   Reaction of a trinuclear iron(II) complex, Fe3 Br3 L (1), with KC8 under N2 leads to dinitrogen act
55 romatic ring cleavage dioxygenase containing Fe3+.
56  metal-organic frameworks (MOFs), containing Fe3-mu3-oxo clusters, are proposed as visible light phot
57                                 In contrast, Fe3+ induced the deposition of "fibrillar" amyloid plaqu
58 FeO2 compound with tetrahedrally coordinated Fe3+ is reported.
59 acity to provide a proton for proton-coupled Fe3+ reduction, and (3) strong hydrogen bond acceptance
60          These include the FRE1-encoded Cu2+/Fe3+ reductase and the CTR1 and CTR3-encoded membrane-as
61 ction in the context of periplasm-to-cytosol Fe3+ transport.
62 ter was not observed, indicating that the di-Fe3+ form of the center is stable.
63  was concluded that, in the presence of DNA, Fe3+ is reduced by some DNA radicals.
64 CO) system comprised of dithiothreitol (DTT)/Fe3+/O2 but not by the ascorbate/Fe3+/O2 MCO system.
65 res of DNA and adriamycin incubated with DTT/Fe3+, H2O2, or formaldehyde all show DS DNA bands on DPA
66 ling on the lithiation kinetics in epitaxial Fe3 O4 thin film on a Nb-doped SrTiO3 substrate is inves
67                               The equatorial Fe3+ coordination site within this cavity is unoccupied
68 m is activated in a pmrA mutant experiencing Fe3+ and low Mg2+, resulting in expression of both cps a
69 estimate total cytosolic labile iron (Fe2+ + Fe3+).
70 ized, reconstituted anaerobically with Fe2+, Fe3+, and S2-, and characterized by Mossbauer, EPR, and
71 rements at atmospheric pressure suggest Fe2+-Fe3+ hopping (small polaron) as the dominant conductivit
72 m (< 0.1 microM Mn2+), but not in iron (Fe2+/Fe3+)-limited medium, and was enhanced in the presence o
73 d a working electrode, we show that the Fe2+/Fe3+ couple in SWa-1 is redox-active over a large range
74 ing insect Rhodnius prolixus, uses a ferric (Fe3+) heme to deliver NO to its victims.
75 soils have favoured the formation of ferric (Fe3+)-rich minerals, such as goethite, rather than sider
76        CP450cam contains a high-spin ferric (Fe3+) heme in the resting state; the isotropic shift was
77 ylenetriaminepentaacetic acid and ferrozine, Fe3+- and Fe2+-specific chelators.
78 hexaniobate nanoscrolls and partially filled Fe3 O4 @hexaniobate NPPs were first fabricated.
79 ting different coordination environments for Fe3+.
80 0.6 x 10(-6) M for Fe2+ and 2 x 10(-6) M for Fe3+ were obtained with 300 nm thick films after 30 min
81 hat the transporter has high specificity for Fe3+ and selectivity for trivalent metals, including Ga3
82                               At 25 ms, four Fe3+-oxy species (three Fe dimers and one Fe trimer) wer
83 ection enables Fe2+ to be distinguished from Fe3+, which is the first step in developing a sensor for
84                                 Furthermore, Fe3+ released during this step is susceptible to reducti
85  the presence of high-spin metal ions (e.g., Fe3+), limits the use of NMR.
86  order: (Fe3+)-NO > (Fe2+)-NO > (Fe2+)-CO &gt; (Fe3+)-H2O > (Fe2+)-H2O.
87 rmed via the Fenton reaction (Fe2++H2O2+H+--&gt;Fe3++H2O+.OH)], interferon-gamma (IFN-gamma), and calciu
88 activity pattern for 2-H as compared to 1-H; Fe3(OCHO)3L (1-3CO2) was generated from 1-H, while 2-H a
89 e formation of a hexacoordinate aquomet (H2O.Fe3+) species has been proposed to drive mechanism (iii)
90  H2O2 + Fe(3+)-nitrilotriacetic acid (H2O2 + Fe3+) to produce hydroxyl radical, and xanthine oxidase+
91 results indicate that exposure to the H2O2 + Fe3+ free radical-generating system reduces activator Ca
92                 In muscles exposed to H2O2 + Fe3+ for 10 minutes, both twitch force and Ca2+ transien
93  176 mV for the cross-reaction couple, His82-Fe3+-His18 + e- --> Met80-Fe2+-His18.
94 -Fe3+/2+-His18 couple, E0' = 47 mV for His82-Fe3+/2+-His18 couple, and E0' = 176 mV for the cross-rea
95 b,AC = 17 s-1 for Met80-Fe3+-His18 --> His82-Fe3+-His18 and kf,BD > 10 s-1 for His82-Fe2+-His18 --> M
96  entropies indicates that the oxidized His82-Fe3+-His18 form is highly disordered.
97  net reduction/rearrangement reaction, His82-Fe3+-His18 + e- --> Met80-Fe2+-His18, and compared to th
98  form, while in the oxidized state the His82-Fe3+-His18 form predominates.
99           Using a combination of immobilized Fe3+-chelate affinity chromatography and reversed-phase
100                            More importantly, Fe3 Mo3 C is very active for oxygen reduction reaction (
101 ase activity is required for SFT function in Fe3+ transport and that Cu depletion reduces cellular ir
102               The non-native pKs observed in Fe3+-sub-(Mn)SOD and the differences in the Fe3+ coordin
103               We show that, for reactions in Fe3+-coordinating phosphate buffer, step 2 is enhanced t
104  the deformylase into catalytically inactive Fe3+ ion by atmospheric oxygen.
105  moles/mol of protein) were used to increase Fe3+ in mineral precursor forms.
106                             An intermediate, Fe3+/Fe4+ cluster (intermediate X), has been identified
107  reaction (ORR)-active ordered intermetallic Fe3 Pt NPs.
108 n in the basic magnetic unit, the intralayer Fe3(mu-OH)3 triangle.
109                 The effect of the metal ion (Fe3+ vs Co3+) on MOF structure was generally found to be
110                                         IrMn/Fe3 Mo3 C enables Zn-air batteries to achieve long-term
111 The extraordinarily high performance of IrMn/Fe3 Mo3 C bifunictional catalyst provides a very promisi
112 strict chemical requirements of ferric iron (Fe3+) binding and membrane transport.
113 itive ratios of oxidized iron to total iron (Fe3+/SigmaFe), determined with Fe K-edge micro-x-ray abs
114 tin H chain is critical to store iron in its Fe3+ oxidation state, while the L chain shows iron nucle
115 ancies of gamma-Fe2 O3 instead of well-known Fe3 O4 SPNPs.
116 apped with a thf-solvated Li cation [((tbs)L)Fe3(mu(3)-N)]Li(thf)3.
117  (Fe(IV))(Fe(III))2 (in the case of [((tbs)L)Fe3(mu(3)-N)]NBu4) are electrochemically accessible.
118 rum compared to previously reported [((tbs)L)Fe3(mu(3)-N)]NBu4, and can be directly synthesized by pr
119 ) yields triiron mu(3)-imido cluster ((tbs)L)Fe3(mu(3)-NH) and ammonia or aniline, respectively.
120                                      ((tbs)L)Fe3(mu(3)-NH) has a similar zero-field (57)Fe Mossbauer
121 tonation of the triiron parent imido ((tbs)L)Fe3(mu(3)-NH) with lithium bis(trimethylsilyl)amide resu
122 ydrazine by ((tbs)L)Fe3(thf) affords ((tbs)L)Fe3(mu(3)-NPh) and aniline.
123         The solid state structure of ((tbs)L)Fe3(mu(3)-NPh) is similar to the series of mu(3)-nitrido
124 ) to yield triiron bis-imido complex ((tbs)L)Fe3(mu(3)-NPh)(mu(2)-NPh), which has been structurally c
125    High-spin trinuclear iron complex ((tbs)L)Fe3(thf) ([(tbs)L](6-) = [1,3,5-C6H9(NC6H4-o-NSi(t)BuMe2
126 eduction of 1,2-diphenylhydrazine by ((tbs)L)Fe3(thf) affords ((tbs)L)Fe3(mu(3)-NPh) and aniline.
127 solation of homotrinuclear complexes ((tbs)L)Fe3(THF) and ((tbs)L)Mn3(THF), respectively.
128  is also achieved in the presence of ((tbs)L)Fe3(thf) to yield triiron bis-imido complex ((tbs)L)Fe3(
129 of hydrazine or phenylhydrazine with ((tbs)L)Fe3(thf) yields triiron mu(3)-imido cluster ((tbs)L)Fe3(
130 Q69E-FeSOD's apparent deviation from WT-like Fe3+ coordination in the oxidized state can be explained
131 the film and substrate, the existence of Lix Fe3 O4 rock-salt phase during lithiation consequently re
132                          Interaction of M3+ (Fe3+ or Ga3+) with the adenine ring would bring it into
133                                     Magmatic Fe3+/SigmaFe ratios increase toward subduction zones (at
134 mplexes on the surface of colloidal magnetic Fe3 O4 /SiO2 core/shell particles, a robust and recovera
135 h elongated prismatic crystals of magnetite (Fe3 O4 ).
136 re, we demonstrate that Ti-doped magnetites (Fe3 - xTixO4) reduce U(VI) to U(IV).
137 y, heme proteins with a pentacoordinate met (Fe3+) form might be expected to lack this pathway.
138 S is rapidly autooxidized to attain the met (Fe3+) form, whereas DosT exists in the O2-bound (oxy) fo
139 red to those for wild-type cytochrome, Met80-Fe3+-His18 + e- --> Met80-Fe2+-His18.
140 coordination forms: kb,AC = 17 s-1 for Met80-Fe3+-His18 --> His82-Fe3+-His18 and kf,BD > 10 s-1 for H
141 --> Met80-Fe2+-His18; E0' = 247 mV for Met80-Fe3+/2+-His18 couple, E0' = 47 mV for His82-Fe3+/2+-His1
142                                 Using 100 mM Fe3+ as the MHD-generating redox species at a 3-mm worki
143 T offers some signal enhancement with 100 mM Fe3+.
144                  Two different S = 5/2, mono-Fe3+ lambdaPP species were identified: the first with an
145  lambdaPP results in a decrease in both mono-Fe3+ species and the appearance of a new S = 5/2, Fe(3+)
146                    The first and second mono-Fe3+ species are thought to represent Fe present in the
147                 The addition of Zn2+ to mono-Fe3+ lambdaPP results in a decrease in both mono-Fe3+ sp
148       The subsequent decay of these multiple Fe3+-oxy species to the superparamagnetic mineral sugges
149 strated to facilitate the transport of naked Fe3+ across the periplasmic space of several Gram-negati
150                        MCD spectra of native Fe3+-LPO and Fe3+-CN--LPO are approximately 10 nm red sh
151 cted indirectly by reducing the nonabsorbing Fe3+-bipy complexes that accumulated in the film to abso
152 e Mn2+ and Ca2+ from the WOC and nonspecific Fe3+, a new EPR signal becomes visible upon binding of M
153 ons, the Ca2+ analogs Tb3+ and La3+ (but not Fe3+) significantly enhanced proteolytic activity, sugge
154    However, hydrophobic core relaxation--not Fe3+ loss--is rate-determining for RdPf unfolding.
155                                   Binding of Fe3+ by FbpA significantly decreases the ability of tryp
156 ng with reduction of a high concentration of Fe3+.
157 e better understood structural correlates of Fe3+ binding by the transferrins is the conformational s
158 rom the methionine ligand to the dxy-hole of Fe3+.
159 nd this rate difference, the interactions of Fe3+ and Ga3+ with NAD(P)H were examined by 1H, 13C, and
160 with [2-14C]bromopyruvate in the presence of Fe3+ ions resulted in the incorporation of 0.70 mol of 1
161  under aerobic conditions in the presence of Fe3+, Fe2+, Fe2+/NADH, or Fe3+/NADH with and without eth
162  to a much smaller extent in the presence of Fe3+, S2-, and dithiothreitol.
163 nstead, it is proposed that the reduction of Fe3+ in heme to Fe2+ oxidizes a peroxide to yield an ini
164 coli Fe-containing SOD (FeSOD), reduction of Fe3+ is accompanied by protonation of a coordinated OH-,
165 eine decay by cysteine-mediated reduction of Fe3+.
166  to that of blood is regulated by removal of Fe3+ by macrophages that accumulate in the thrombus duri
167 n sequential loss of sulfur and retention of Fe3+.
168          Here we report the sequestration of Fe3+ by FbpA in the presence of sulfate, at an assumed p
169 redox potentials are due to stabilization of Fe3+ in FeFbpA-X by X(n-).
170 dized) Fe3+-sub-(Mn)SOD differs from that of Fe3+-SOD with respect to the EPR signals produced at bot
171 d F-, the KD for N3- is tighter than that of Fe3+-SOD, suggesting that the (Mn)SOD protein favors ani
172 tein-protein recognition during transport of Fe3+ between membranes, and may explain how these protei
173                             In the uptake of Fe3+, Fre1p produces Fe2+ that is a substrate for Fet3p;
174 perties and iron content consistent with one Fe3+(SCys)4 site per polypeptide but is clearly distinct
175 oxorubicin (DOX) are coupled separately onto Fe3 O4 @SiO2 and polystyrene surfaces of a unique polyst
176 , in particular, the presence of Fe2+ and/or Fe3+ chelators can influence significantly the cluster c
177 site direction, and the orientation of Au or Fe3 O4 in the shell can be well controlled by exploiting
178                  The substitution of Ca2+ or Fe3+ for Mg2+ does not significantly alter the amount of
179 ta1-40 were not aggregated by Zn2+, Cu2+, or Fe3+, indicating that histidine residues are essential f
180 ve of whether Fe was supplied in the Fe2+ or Fe3+ form.
181 ron transporters specific for either Fe2+ or Fe3+.
182 in the presence of Fe3+, Fe2+, Fe2+/NADH, or Fe3+/NADH with and without ethanol.
183 ron oxidation state in the following order: (Fe3+)-NO > (Fe2+)-NO > (Fe2+)-CO > (Fe3+)-H2O > (Fe2+)-H
184 el-iron nitride (Ni3 FeN) supporting ordered Fe3 Pt intermetallic nanoalloy.
185  high activity for the OER while the ordered Fe3 Pt nanoalloy contributes to the excellent activity f
186                The active site of (oxidized) Fe3+-sub-(Mn)SOD differs from that of Fe3+-SOD with resp
187 rocytes in the thrombus) to its paramagnetic Fe3+ form.
188 ythrocyte lysis-derived iron to paramagnetic Fe3+, which causes thrombus T1 relaxation time shortenin
189 be the integrity of the functionally pivotal Fe3+-M80 linkage.
190 onductivity, the carbon shells and the polar Fe3 O4 cores facilitate fast electron/ion transport and
191 polystyrene surfaces of a unique polystyrene/Fe3 O4 @SiO2 Janus structure.
192 show that various metal cations (principally Fe3+/Fe2+, Ni2+, and Cr3+) released from acid corrosion
193 ws evaluation and refinement of the proposed Fe3+ dioxygenase mechanism.
194 enous anion can only stabilize oxidized Q69E-Fe3+SOD and, therefore, cannot account for the increased
195                         Using a radiolabeled Fe3+ transport assay, we established an apparent Km=0.9
196                  Both NADH, which can reduce Fe3+, and ethanol, which can scavenge some free radicals
197 tion of a ferrireductase that acts to reduce Fe3+ to Fe2+, with subsequent transport of the divalent
198 llular reductants such as NADH, which reduce Fe3+ to Fe2+ and allow the recycling of iron.
199 in the reaction cycle of 3,4-PCD and related Fe3+-containing dioxygenases.
200 F ratio, Fe2+ was oxidized and the resulting Fe3+ was deposited within HoSF but no H2O2 was detected
201 e ferritin which showed decreases in several Fe3+ intermediates and stabilization of Fe2+, emphasize
202               Taken together, the yolk-shell Fe3 O4 @Au NPs can be regarded as an ideal magnetic-plas
203                    In the oxidized (siroheme Fe3+, Fe4S42+) SiRHP crystal structure, the high density
204      In uncomplexed 3,4-PCD, the active site Fe3+ is bound at the bottom of the active site crevasse
205                    The essential active site Fe3+ of protocatechuate 3,4-dioxygenase [3, 4-PCD, subun
206 ally chelated complexes with the active site Fe3+ that result in dissociation of the endogenous axial
207  salts in water yields the trimeric species [Fe3(mu-L)6(H2O)6](6-).
208 s of 2.2 and 4.3 and attributed to high-spin Fe3+.
209 Anaerobic titrations of the iron-substituted Fe3+-Fe2+ enzyme with dithionite resulted in a gradual l
210                     Robust Ni3 FeN-supported Fe3 Pt catalysts show superior catalytic performance to
211 fluence oxygen atom transfer in tetranuclear Fe3 Mn clusters.
212       Although Fe2+ is far more soluble than Fe3+, it rapidly oxidizes aerobically at pH > or = 7.
213         This clearly rules out the fact that Fe3+ is solely responsible for the kinetic stability of
214                        Our results show that Fe3+-rich phyllosilicates probably precipitated under we
215  the superparamagnetic mineral suggests that Fe3+ species in different environments may be translocat
216                                          The Fe3 Pt/Ni3 FeN bifunctional catalyst enables Zn-air batt
217                                          The Fe3+ provides the high cathodic current necessary to pro
218  the covalent bond between His93(F8) and the Fe3+ atom, and (3) hydrogen bonding between distal resid
219 wed substantial dipolar coupling between the Fe3+ of FeEnt and the spin label and provided an iron-ni
220    Addition of product phosphate to both the Fe3+-Fe2+ and Fe3+-Zn2+ forms of calcineurin led to pert
221 hibitors (PHB, CHB, and FHB) coordinates the Fe3+ adjacent to Wat827, resulting in a shift in its pos
222 uces Fe2+ that is a substrate for Fet3p; the Fe3+ produced by Fet3p is a ligand for the iron permease
223 onded well to the formation constant for the Fe3+-tyrosinate complex (920 s(-1)) observed previously
224 gher for the Fe3+.NADPH complex than for the Fe3+.NADH complex.
225 fer species was significantly higher for the Fe3+.NADPH complex than for the Fe3+.NADH complex.
226 ve shown that Tyr447 is dissociated from the Fe3+ in the anaerobic 3,4-PCD complex with protocatechua
227              Once Tyr447 is removed from the Fe3+ in the final E.PCA complex by either dissociation o
228 udies found that the addition of gold in the Fe3 O4 @hexaniobate NPPs reduced interparticle coupling
229  Fe3+-sub-(Mn)SOD and the differences in the Fe3+ coordination indicated by the EPR spectra are consi
230                                  Iron in the Fe3+ form can also be determined, but it has a more comp
231 ronal enzyme (nNOSox), the Fe-NO bond in the Fe3+NO complex is weaker than in the wild type enzyme, c
232  which the C3 phenolic function occupies the Fe3+ ligand site opposite the endogenous ligand Tyr408(O
233 ly prevents the decline in relaxivity of the Fe3 O4 core caused by the Au layer.
234  The extraordinarily high performance of the Fe3 Pt/Ni3 FeN bifunctional catalyst makes it a very pro
235 ge neutrality and coordination number of the Fe3+ center.
236                             Oxidation of the Fe3+-Fe2+ cluster to the diferric state by hydrogen pero
237 ndicates that several or possibly all of the Fe3+-oxy species involve tyrosine.
238 e heme iron atoms from the Fe2+ state to the Fe3+ state.
239 than the wild type was to treatment with the Fe3+ chelator deferoxamine, indicating that it is defect
240                                        These Fe3+-oxy species were found to form at similar rates and
241  those observed upon binding the first F- to Fe3+-SOD, but the EPR spectrum obtained upon binding N3-
242 of the quench following oxidation of Fe2+ to Fe3+ at the ferroxidase center was not observed, indicat
243 t ferroxidase activity that converts Fe2+ to Fe3+ in the presence of molecular oxygen.
244 se activity catalyzing conversion of Fe2+ to Fe3+, by identification of yeast copper oxidases homolog
245 , at which Fe2+ is spontaneously oxidized to Fe3+ at 37 degrees C in 20 mM Bis-Tris buffer at pH 5.8,
246 tained high spin Fe2+ that, when oxidized to Fe3+, inactivated the enzyme.
247 reduction of a pyrazolate-bridged triangular Fe3 (mu3 -O) core induces a cascade wherein all three me
248      Human serum transferrin (hTF), with two Fe3+ binding lobes, transports iron into cells.
249 sociation of the endogenous axial tyrosinate Fe3+ ligand, Tyr447 (147beta).
250 n uptake was completely blocked by unlabeled Fe3+ and by other trivalent cations including Al3+, Ga3+
251 ent of Zn2+ with Fe2+ yields a mixed valence Fe3+-Fe2+ center that exhibits a characteristic EPR sign
252                       The effects of varying Fe3+ concentration (1-100 mM), working electrode size (1
253 er mantle phase magnesiowustite with varying Fe3+ content.
254 ts with itself and other byssal proteins via Fe3(+)-DOPA complexes, and the mannose-binding domain in
255 initrogen activation products (2) from which Fe3 (NH)3 L (2-1; L is a cyclophane bridged by three bet
256 ary iron sources, ferritin is a complex with Fe3+ iron in a mineral (thousands of iron atoms inside a
257 cated by the EPR spectra are consistent with Fe3+-sub-(Mn)SOD's inability to oxidize O2*- and suggest
258 ptide based on the His-rich decapeptide with Fe3+, Co2+, Ni2+, Zn2+, and Cu2+ indicates that only Cu
259  shells and strong chemical interaction with Fe3 O4 cores, this unique architecture immobilizes the a
260 fer (one beta and two alpha) mechanism, with Fe3+ acting as a buffer for the spin-forbidden two-elect
261 eted, and NADPH, which is less reactive with Fe3+, functions as the major nicotinamide nucleotide red
262                          Reconstitution with Fe3+ was unsuccessful.
263 ne moment developed from spin canting within Fe3(mu-OH)3 triangles.
264  or 2-H with CO2 at room temperature yielded Fe3(OCHO)(H)2L (1-CO2) or Co3(OCHO)(H)2L (2-CO2), respec
265 ell-designed yolk-shelled carbon@Fe3 O4 (YSC@Fe3 O4 ) nanoboxes as highly efficient sulfur hosts for
266                 With these merits, the S/YSC@Fe3 O4 cathodes support high sulfur content (80 wt%) and

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