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1 ction of neutrophils leading to neutropenia (Felty's syndrome).
2 isks, and clarify the optimum use of CFSs in Felty's syndrome.
3 use of colony-stimulating factors (CSFs) in Felty's syndrome.
4 -CSF appears well tolerated and effective in Felty's syndrome.
5 nia and decreasing the risk of infections in Felty's syndrome.
6 the manifestations observed in patients with Felty's syndrome.
7 c mechanism between LGL leukemia and typical Felty's syndrome.
8 nomegaly in the spectrum of LGL leukemia and Felty's syndrome.
11 ally high prevalence of DR4 in patients with Felty's syndrome and in those with LGL leukemia plus RA
12 c lupus erythematosus, rheumatoid arthritis, Felty's syndrome, and large granular lymphocyte (LGL) le
14 oid vasculitis or to undergo splenectomy for Felty's syndrome, cervical spine fusion for myelopathy,
18 thematosus (SLE), rheumatoid arthritis (RA), Felty's syndrome (FS), and antineutrophil cytoplasmic an
19 l of sera revealed that 66% of patients with Felty's syndrome had elevated levels of anti-eEF1A-1 ant
20 for rheumatoid vasculitis and splenectomy in Felty's syndrome have decreased over the past 19 years,
23 e risk of hospitalization for splenectomy in Felty's syndrome was 71% lower in 1998-2001 than in 1983
24 the manifestations observed in patients with Felty's syndrome, which is a rare complication of RA cha
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