ライフサイエンスコーパス: Fisher syndrome

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1 Infectious illnesses usually precede Miller Fisher syndrome.

2 nt to the clinical pathophysiology of Miller Fisher syndrome.

3 neuromyotonia, Morvan's syndrome and Miller-Fisher syndrome.

4 0 was isolated from a patient who had Miller-Fisher syndrome.

5 potential to ameliorate the course of Miller Fisher syndrome.

6 ies in the diagnosis and causation of Miller-Fisher syndrome?

7 BS (18 demyelinating, 8 axonal, and 1 Miller Fisher syndrome), 5 had encephalitis (3 with concomitant

8 transmission occurs in some cases of Miller Fisher syndrome and Guillain-Barre syndrome.

9 ater understanding of the spectrum of Miller Fisher syndromes and to refine clinical diagnoses in pat

10 Ganglioside complexes in GBS and Miller-Fisher syndrome are frequently present and are more path

11 Miller Fisher syndrome-associated anti-GQ1b ganglioside antibod

12 n-Barre syndrome and its variant, the Miller Fisher syndrome, during TNFalpha antagonist therapy.

13 f clinical features is reminiscent of Miller Fisher syndrome, in which acute-phase anti-disialylated

14 Miller Fisher syndrome is a localized variant of Guillain-Barre

15 Miller-Fisher syndrome is an autoimmune neuropathy characterize

16 o facilitates differentiation between Miller-Fisher Syndrome (MFS) and BBE in cases of diagnostic dou

17 of Guillain-Barre syndrome (GBS) and Miller-Fisher syndrome (MFS) following C. jejuni infection.

18 s are the serological hallmark of the Miller Fisher syndrome (MFS) variant of the paralytic neuropath

19 triggering of the Guillain-Barre and Miller-Fisher syndrome neuropathies after C. jejuni infection.

20 he Guillain-Barre syndrome, including Miller-Fisher syndrome, subsets of immunoglobulin M paraprotein

21 with rheumatoid arthritis in whom the Miller Fisher syndrome variant of the Guillain-Barre syndrome d

22 ncluded in the analyses, and cases of Miller Fisher syndrome were excluded.

23 with chronic ataxic neuropathies and Miller Fisher syndrome were studied.

24 f the role of anti-GQ1b antibodies in Miller Fisher syndrome with particular respect to the motor ner

25 persons had Brighton level 1, 2, or 3 GBS or Fisher Syndrome, with symptom onset 1-119 days after vac

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