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1 hosphorylate and inactivate the proapoptotic forkhead transcription factor.
2 gatively regulates the activity of DAF-16, a Forkhead transcription factor.
3 Daf-16 encodes a forkhead transcription factor.
4 at are efficiently and specifically bound by Forkhead transcription factors.
5 ypertrophic signaling through its effects on Forkhead transcription factors.
6 that involves regulation of the proapoptotic forkhead transcription factors.
7 activated by IGF/insulin, can phosphorylate forkhead transcription factors.
8 y gonadotrope cells is the FOXO subfamily of forkhead transcription factors.
9 e binding competition between POU5F1 and the forkhead transcription factors.
10 udies show FGF15 signaling decreases hepatic forkhead transcription factor 1 (FoxO1) activity through
13 ls induced by CpG ODN-activated PDCs express forkhead transcription factor 3 and produce IL-10, TGF-b
14 We suggest that insulin signaling and FOXO (forkhead transcription factor), a downstream molecule in
15 t KM-Brk, blocked the phosphorylation of the forkhead transcription factor, a downstream Akt target.
16 e to stress by regulating the FOXO family of Forkhead transcription factors, a family of proteins tha
20 eduction-of-function mutations in the daf-16/forkhead transcription factor, an effector of the DAF-2/
21 al changes in lipid absorption by regulating forkhead transcription factors and MTP and that inhibiti
24 tion and inhibition of FKHR and related FOXO forkhead transcription factors and that androgens provid
25 FOX:ETS motif, a composite binding site for Forkhead transcription factors and the Ets transcription
27 of Sir2 (SirT1) regulates p53, NF-kappaB and Forkhead transcription factors, and is implicated in str
28 g the pro-apoptotic Bc1-2 family member Bad, Forkhead transcription factors, and the cyclic AMP respo
37 ave identified three consensus sequences for forkhead transcription factor binding in transforming gr
38 e Isl1 SHF enhancer contains three consensus Forkhead transcription factor binding sites that are eff
40 3 partner molecule, Foxo3a, a subtype of the forkhead transcription factors, by inhibiting its associ
41 ations in Whn (Hfh11, Foxn1), a winged-helix/forkhead transcription factor, cause the nude phenotype,
43 e an evolutionary conserved subfamily of the forkhead transcription factors, characterized by the for
45 A), independent of p53, but dependent on the forkhead transcription factor class-O family member, FOX
46 fibroblast growth factor signaling and four Forkhead transcription factors consist the central part
47 The NAD-dependent deacetylase SIR2 and the forkhead transcription factor DAF-16 regulate lifespan i
48 in/IGF-1 signaling pathway, dependent on the forkhead transcription factor DAF-16, but independent of
49 ian homologues of the Caenorhabditis elegans forkhead transcription factor DAF-16, function in the in
55 dylinositol 3-OH kinase (age-1) regulating a forkhead transcription factor (daf-16) [6, 7], as well a
56 Downregulation of this pathway activates a forkhead transcription factor (daf-16), which may regula
58 overexpressing the phosphatase dPTEN or the forkhead transcription factor dFOXO, prevents the declin
65 contains Smad2 or Smad3, Smad4, and a novel forkhead transcription factor, FAST-1, and binds to an e
70 d Hi-C experiments on cells depleted for the Forkhead transcription factors, Fkh1 and Fkh2, previousl
74 Akt activation, is a potent inhibitor of the forkhead transcription factor FKHR (FOXO1), identifying
76 cascade resulting in phosphorylation of the forkhead transcription factor FKHR in a breast cancer ce
77 gen synthase kinase 3beta (GSK3beta) and the Forkhead transcription factor FKHR were phosphorylated a
78 d PDGF-BB-induced phosphorylation of BAD and forkhead transcription factor FKHR-L1, and these events
80 apoptosis by phosphorylating the substrates forkhead transcription factor (FKHR) and glycogen syntha
81 ylation of glycogen synthase kinase (GSK)-3, forkhead transcription factor (FKHR), p70(S6K), and ERK,
83 DAF-16, the closely-related FOXO subclass of forkhead transcription factors (FKHR/FOXO1, FKHRL1/FOXO3
84 K), -mammalian target of rapamycin (-mTOR), -forkhead transcription factor (-FKHR), -caspase-9, and -
85 synthase kinase--3 beta (GSK-3 beta), and a forkhead transcription factor, FKHR, in a human T-cell l
87 eins such as the BCL2 family member Bad, the forkhead transcription factor FKHRL-1, and caspase 9.
88 essed the phosphorylation and degradation of forkhead transcription factor FKHRL1 (FOXO3a), resulting
89 Galpha12QL stimulates the phosphorylation of forkhead transcription factor FKHRL1 via AKT in a PDGFRa
90 arly, induction of FasL by the Akt-regulated forkhead transcription factor FKHRL1 was dependent upon
92 hat Akt-mediated signals, acting through the forkhead transcription factor FKHRL1, can regulate colla
97 or proteins, glycogen synthase kinase-3, and forkhead transcription factors, FKHRL1/FOXO3 and FKHR/FO
99 revealed an inverse correlation between the Forkhead transcription factor Forkhead box class O (FOXO
100 ia complementation group D2 (FANCD2) and the forkhead transcription factor forkhead box O 3a (FOXO3a)
101 ance was mediated directly by inhibiting the Forkhead transcription factor Forkhead box O3a (FOXO3a).
102 oinsufficiency of the Foxo1 gene, encoding a forkhead transcription factor (forkhead box transcriptio
103 regeneration phenotypes and that RNAi of the forkhead transcription factor FoxA, which is expressed i
106 in normal mice, plasma insulin inhibits the forkhead transcription factor Foxa2 by nuclear exclusion
107 or Tbx1 failed to activate expression of the forkhead transcription factor Foxa2 in the pharyngeal me
110 ng, nonsense and frameshift mutations in the forkhead transcription factor FOXC2 in affected members
113 ctional and positional candidate gene, helix/forkhead transcription factor (FOXC2), by sequencing and
116 ies described in this report reveal that the forkhead transcription factor, Foxd1, is not expressed i
118 nic stem to epiblast cells and uncovered the forkhead transcription factor FOXD3 as a major regulator
119 sing a neural crest-specific deletion of the Forkhead transcription factor Foxd3, we ablated neural c
120 ment of RCs is additionally dependent on the forkhead transcription factor Foxd3, which is more broad
122 t fibroblast growth factor signaling and the forkhead transcription factor FoxF directly upregulate t
124 we show that the closely linked mesenchymal forkhead transcription factors Foxf1 and Foxl1 are part
125 vestigated how in intestinal fibroblasts the forkhead transcription factor Foxf2 controls Wnt signali
136 tify a novel interaction between SRF and the Forkhead transcription factor FOXK1 in human cells.
139 We find that mouse XX gonads lacking the forkhead transcription factor Foxl2 form meiotic prophas
140 c analysis identified a central role for the forkhead transcription factor FOXM1 and its targets, and
145 (PA) in mice increased the expression of the forkhead transcription factor FoxM1 in type II cells coi
147 nstrate that the expression of the oncogenic forkhead transcription factor FOXM1 is upregulated by GO
148 rongly correlated with the expression of the forkhead transcription factor FoxM1, which binds to the
151 the key nodes in the metabolic network, the forkhead transcription factor FOXO has been shown to int
154 ressed in adult worms, and this requires the forkhead transcription factor (FOXO) encoded by daf-16.
156 ans extends lifespan by activating DAF-16, a forkhead transcription factor (FOXO) repressed by insuli
158 factor 1 (HIF-1) through the inactivation of Forkhead transcription factors (FOXO) in PTEN-null cells
159 physiological roles in this process are the forkhead transcription factor FOXO1 (also known as FKHR)
162 ce, but Akt-dependent phosphorylation of the forkhead transcription factor FoxO1 at serine 256, which
164 ng evidence documents a key function for the forkhead transcription factor FoxO1 in cellular metaboli
168 We report that haploinsufficiency for the forkhead transcription factor Foxo1 reverses beta cell f
170 determined the molecular mechanisms whereby forkhead transcription factor Foxo1, a key downstream si
171 is associated with reduced expression of the forkhead transcription factor Foxo1, a substrate of the
176 an target of rapamycin (mTOR), the family of forkhead transcription factors (FOXO1, FOXO3a, and FOXO4
177 glycogen synthase kinase-3alpha/beta and the forkhead transcription factor, FoxO1, in protection of F
178 rmone deficiencies, we are investigating the forkhead transcription factor, FOXO1, which has been imp
180 D31 signals prevents the localization of the forkhead transcription factor FoxO3 to the nucleus, thus
181 demonstrated that Sirt6 can be recruited by forkhead transcription factor FoxO3 to the proximal prom
184 acetylates and represses the activity of the forkhead transcription factor Foxo3a and other mammalian
190 e the first linkage by the identification of Forkhead transcription factor FOXO3a, the PI3K/Akt downs
192 we show that such inflammation requires the forkhead transcription factor Foxo3a: Foxo3a-deficient m
193 vation of a ubiquitously expressed mammalian forkhead transcription factor, Foxo3a, induces cerebral
196 the in vitro interactions among 14-3-3, the Forkhead transcription factor FOXO4, and its target DNA,
199 pproaches were used to evaluate the roles of forkhead transcription factors (FoxOs) in endoplasmic re
204 strate that absence of the gene encoding the forkhead transcription factor Foxp1 resulted in a profou
213 alogous cooperative complex of NFAT with the forkhead transcription factor FOXP3, a lineage specifica
214 lls, both mRNA and protein expression of the forkhead transcription factor Foxp3, a specific marker o
215 , they efficiently induced expression of the forkhead transcription factor FoxP3, the master regulato
216 al circumstances, male mice deficient in the forkhead transcription factor FoxP3, which lack regulato
222 nd -gamma), members of the FoxA subfamily of forkhead transcription factors, function as initial chro
223 esenchymal markers, including members of the forkhead transcription factor gene family, have been det
231 onstrating a critical role for at least this forkhead transcription factor in the regulation of B lym
232 he phosphorylation and hence inactivation of forkhead transcription factors in a p66Shc-dependent man
234 he Forkhead box, class O (FOXO) subfamily of Forkhead transcription factors in the regulation of BIM
235 t Isl1 is a direct transcriptional target of Forkhead transcription factors in the SHF and establish
238 as a primary downstream target for Foxn4, a forkhead transcription factor involved in the genesis of
239 We show here that the Foxn4 winged helix/forkhead transcription factor is coexpressed with the bH
240 Here, we show that the Foxn4 winged helix/forkhead transcription factor is expressed in a subset o
241 Finally, a putative binding element for forkhead transcription factors is necessary for promoter
242 Forkhead box gene, group O (FoxO) family of Forkhead transcription factors is phopsphorylated and in
246 malian FOXO3, a member of the FOXO family of Forkhead transcription factors known to promote resistan
247 osphorylation of FOXO3a, a tumor suppressive forkhead transcription factor, leading to its cytoplasmi
248 ted a physical interaction of SMA-3 with the forkhead transcription factor LIN-31, which is enhanced
249 nase promoter and that increased activity of forkhead transcription factors may underlie the increase
250 iogenesis, transcriptionally suppresses AFX1 forkhead transcription factor-mediated apoptosis and inc
252 e Xenopus and zebrafish homologs of Foxj1, a forkhead transcription factor necessary for ciliogenesis
255 atic nuclear receptor 4alpha (HNF4alpha) and forkhead transcription factor O1 (FOXO1), two critical t
256 lycogen synthase kinase-3beta (GSK3beta) and forkhead transcription factor of the O class (FOXO)3A, k
261 es through the inhibitory phosphorylation of forkhead transcription factors, our results describe for
263 sure and was mediated through an AKT-1/PDK-1/forkhead transcription factor pathway overlapping with b
264 2 is a let-7 target in seam cells, while the forkhead transcription factor pha-4 is a target in the i
270 escribe a mechanistic model whereby Foxd3, a forkhead transcription factor, prevents neural crest-der
271 mitotic genes in close proximity to Fkh2, a forkhead transcription factor previously implicated in r
274 on mutations in Whn (Hfh 11), a winged-helix/forkhead transcription factor, result in the nude mouse
278 erior domain by activating expression of the forkhead transcription factors sloppy paired 1 and slopp
280 f FOXO4 (also known as AFX), a member of the forkhead transcription factor superfamily that is negati
281 ential display PCR, we have cloned an 85-kDa forkhead transcription factor (termed Mac-1-regulated fo
285 the nuclear accumulation of DAF-16/FOXO, the forkhead transcription factor that is the major downstre
286 ic protein consisting of MLL fused to AFX, a forkhead transcription factor that normally regulates ge
290 ar location of FoxO1 and FoxO3a, two Class O Forkhead transcription factors that mediate lymphocyte q
292 cyte nuclear factors-3 (Foxa-1-3) are winged forkhead transcription factors that regulate gene expres
293 homologue (CHES-1-like), two genes encoding forkhead transcription factors that we discovered utiliz
295 on and represses the expression of FOXP2, a forkhead transcription factor tightly associated with sp
297 Akt; induces phosphorylation of the FKHRL-1 Forkhead transcription factor; upregulates a series of i
299 FKHR is a member of the FOXO subfamily of Forkhead transcription factors, which are important targ
300 n by up-regulating the expression of Class-O Forkhead transcription factors, which play essential rol
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