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1 erently resistant to azoles (e.g., Mucor and Fusarium).
2 t has not always paralleled the evolution of Fusarium.
3 al reservoir of human-pathogenic isolates of Fusarium.
4 es indicate activity of posaconazole against Fusarium.
5 conserved across the phylogenetic breadth of Fusarium.
6 , including many plant pathogenic species of Fusarium.
7 l ulcers, particularly among those caused by Fusarium.
8 se(6), displayed enhanced resistance against Fusarium.
9                 Causative organisms included Fusarium (128 patients [40%]), Aspergillus (54 patients
10 = 14) was isolated in highest number whereas Fusarium (13.4%, N = 6) was the most common fungus speci
11  library alone (Aspergillus, 93% versus 69%; Fusarium, 84% versus 42%; and Scedosporium, 94% versus 1
12      These napins possessed significant anti-Fusarium activity (IC(50)=70 muM) and a compact secondar
13        Further results suggest that the anti-Fusarium activity of M6 may be transferable to maize and
14 lapse and black roots, symptoms similar to a Fusarium-Alternaria disease complex, recently characteri
15                                         Both Fusarium and Acanthamoeba at concentrations tested above
16 ded risk factor for case contamination among Fusarium and Acanthamoeba keratitis patients.
17 nduce and harbor dormant-resistant stages of Fusarium and Acanthamoeba.
18                                              Fusarium and Aspergillus species of mold are major cause
19                                              Fusarium and Aspergillus species were the two predominan
20  NAT enzymes in crop-compromising species of Fusarium and Aspergillus, identifying three groups of ho
21 unctional divergence of two beta-tubulins in Fusarium and identified type II variations in FgTub2 res
22  Seed-coated M6 swarms towards root-invading Fusarium and is associated with the growth of root hairs
23                     The genera Cladosporium, Fusarium and Penicillium were the fungi more frequently
24 (coagulase-negative Staphylococcus, Candida, Fusarium, and Propionibacterium species).
25 sporium, Alternaria, Penicillium, Ulocadium, Fusarium, Arthrinium, Epicoccum, Aureobasidium, Curvular
26 onale (46%), Fusarium cortaderiae (13%), and Fusarium austroamericanum (3%).
27                                              Fusarium biofilms exhibited species-dependent antifungal
28 ir influence on two native fungal pathogens, Fusarium brachygibbosum U4 and Alternaria sp. U10, and t
29 tracellular pH promotes infectious growth of Fusarium by stimulating phosphorylation of a conserved m
30                                              Fusarium cases fared better with natamycin than with vor
31 06; 95% CI, 0.01 to 0.28; P<.001), while non-Fusarium cases fared similarly (regression coefficient=0
32                         Subgroup analysis of Fusarium cases found improvement with natamycin compared
33  belief for natamycin treatment, whereas non-Fusarium cases had a 57.3% belief.
34                                          For Fusarium cases, the posterior demonstrated a 99.7% belie
35 fference attributable to improved results in Fusarium cases.
36 ost no difference between treatments for non-Fusarium cases.
37                 The Cyber infrastructure for Fusarium (CiF) was built to support archiving and utiliz
38 disease incited by the necrotrophic pathogen Fusarium circinatum.
39 ns, have established a robust foundation for Fusarium classification.
40                   A total of 297 isolates of Fusarium collected were subjected to MLST to identify th
41 ium Comparative Genomics Platform (FCGP) and Fusarium Community Platform (FCP).
42 a and knowledge and consists of Fusarium-ID, Fusarium Comparative Genomics Platform (FCGP) and Fusari
43 ss the RodA hydrophobin, and Aspergillus and Fusarium conidia from clinical isolates that were treate
44  has high levels of airborne Aspergillus and Fusarium conidia.
45 -glucan and alpha-mannose on Aspergillus and Fusarium conidia.
46 arum s.s. (37%), Fusarium meridionale (46%), Fusarium cortaderiae (13%), and Fusarium austroamericanu
47                                              Fusarium crown rot (FCR) of wheat and barley, predominan
48 ditions and following inoculation with fungi Fusarium culmorum.
49 % of buildings surveyed yielded at least one Fusarium culture.
50                        Protein extracts from Fusarium cultures were found to truncate ChitA and ChitB
51 ling, diverse fungicides and resistance to a Fusarium-derived antibiotic.
52 xins in wheat three standardized approaches (Fusarium disease severity, PCR assays for Fusarium spp.
53 ed Artemia survivorship and hyphal growth of Fusarium during the immature and mature stages.
54 genes in Magnaporthe, Ustilago, Aspergillus, Fusarium, Epichloe, and Penicillium species indicate tha
55          Fungal mutants lacking a functional Fusarium (F)-RALF peptide failed to induce host alkalini
56 s caused by a novel three-locus haplotype of Fusarium falciforme designated FSSC 3+4qqq.
57                   Three of the FSSC species (Fusarium falciforme, Fusarium keratoplasticum, and Fusar
58                    Conidia of representative Fusarium from the 2004-2006 keratitis outbreak and troph
59 een isolated from the rice pathogenic fungus Fusarium fujikuroi already more than 20 years ago.
60 is of gibberellic acid (GA(3)) by the fungus Fusarium fujikuroi is catalyzed by seven enzymes encoded
61              Sequencing of the rice pathogen Fusarium fujikuroi revealed the presence of far more SM-
62 ndary metabolites produced by members of the Fusarium fujikuroi species complex (FFSC) and rare strai
63 several fusaria, including the rice pathogen Fusarium fujikuroi.
64                                           In Fusarium, fumonisin biosynthetic genes (FUM) are cluster
65 ial to control the growth of Aspergillus and Fusarium fungi in the cornea.
66 d by the presence of mycotoxins derived from Fusarium fungus, and, in particular, by deoxynivalenol (
67 ted toxigenic species of the Aspergillus and Fusarium genera isolated from grapes and cereals.
68  Comparative analyses have revealed that the Fusarium genome is compartmentalized into regions respon
69                                              Fusarium graminearum (FG) is one of the major cereal inf
70 ina sp. LEB-18 phenolic extract (PE) against Fusarium graminearum (Fg) isolates.
71 ha-L-fucosidase from plant pathogenic fungus Fusarium graminearum (FgFCO1) actively releases fucose f
72                       Expression of HopAI in Fusarium graminearum also mainly affected the activation
73  Trichoderma atroviride, the plant pathogens Fusarium graminearum and Colletotrichum graminicola, the
74                                              Fusarium graminearum and F. sporotrichioides have tricho
75 (1)CFU g(-1) and the species most found were Fusarium graminearum and Fusarium verticillioides with 2
76                                              Fusarium graminearum and Fusarium verticillioides, couma
77 ly larger than that of two fungal pathogens, Fusarium graminearum and Magnaporthe grisea.
78  the plasma membrane of the ascomycete fungi Fusarium graminearum and Neurospora crassa and induces a
79                          Mycotoxin-producing Fusarium graminearum and related species cause Fusarium
80            The ubiquitous filamentous fungus Fusarium graminearum causes the important disease Fusari
81                             Infection of the Fusarium graminearum disruption mutant Deltafgl1, which
82 recovered via the heterologous expression of Fusarium graminearum GSK3 homolog gene FGK3, whose codin
83 ecific pollination and during infection with Fusarium graminearum In both Arabidopsis species, the la
84 most obvious seasonal input source, that is, Fusarium graminearum infected wheat crop areas.
85                                    Following Fusarium graminearum infection, we identified accumulati
86         Fusarium head blight (FHB) caused by Fusarium graminearum is a devastating disease of cereal
87                                              Fusarium graminearum is a filamentous fungus and causes
88                         Wheat scab caused by Fusarium graminearum is an important disease.
89                                              Fusarium graminearum is an important pathogen of wheat a
90                                              Fusarium graminearum is an important plant pathogen that
91               Fusarium head blight caused by Fusarium graminearum is one of the most destructive dise
92 nthesis of mycotoxin deoxynivalenol (DON) in Fusarium graminearum is regulated by two pathway-specifi
93 reticulum (ER) of the phytopathogenic fungus Fusarium graminearum is reorganized both in vitro and in
94 t and spiked into fungal culture filtrate of Fusarium graminearum respectively.
95                          Four members of the Fusarium graminearum species complex were isolated: F. g
96 he primary inoculum in the wheat scab fungus Fusarium graminearum that was recently shown to have sex
97                        Head blight caused by Fusarium graminearum threatens world-wide wheat producti
98          The role of the retromer complex in Fusarium graminearum was investigated using cell biologi
99 d blight (FHB) is a cereal disease caused by Fusarium graminearum, a fungus able to produce type B tr
100 c fungi B. cinerea, Alternaria brassicicola, Fusarium graminearum, and Sclerotinia sclerotiorum and y
101  the ascomycete fungi, Neurospora crassa and Fusarium graminearum, at micromolar concentrations.
102 mes of three phenotypically diverse species: Fusarium graminearum, Fusarium verticillioides and Fusar
103                        In axenic cultures of Fusarium graminearum, in vitro feeding of CYP3RNA, a 791
104        Fusarium head blight (FHB), caused by Fusarium graminearum, is a devastating disease of wheat
105 ly characterized the FgMCM1 MADS-box gene in Fusarium graminearum, the causal agent of wheat and barl
106 se gene important for sexual reproduction in Fusarium graminearum, we found that two tandem stop codo
107 nase Cel5A, from the plant-pathogenic fungus Fusarium graminearum, whose production was increased by
108  were discovered through characterization of Fusarium graminearum-induced responses.
109 ent infection with the hemibiotrophic fungus Fusarium graminearum.
110 ortant for pathogenesis and morphogenesis in Fusarium graminearum.
111 causal fungal agent of Fusarium head blight, Fusarium graminearum.
112 iological and genetic functions of FgSch9 in Fusarium graminearum.
113 -vis the archetypal galactose 6-oxidase from Fusarium graminearum.
114 own mutants also showed higher resistance to Fusarium graminearum.
115 lso in the phylogenetically unrelated fungus Fusarium graminearum.
116 estation with western corn rootworm (WCR) or Fusarium graminearum.
117 n wheat infected with the filamentous fungus Fusarium graminearum.
118  genes (ATGs) in the wheat pathogenic fungus Fusarium graminearum.
119 is phosphorylated at five conserved sites in Fusarium graminearum.
120  to pathogens including the toxigenic fungus Fusarium graminearum.
121                                              Fusarium grown as a biofilm on silicone hydrogel contact
122 ould be valuable efficient targets to reduce Fusarium growth and therefore to prevent food contaminat
123 by barley resistant genotype CI89831 against Fusarium head blight (FHB) based on metabolo-transcripto
124 by barley resistant genotype CI89831 against Fusarium head blight (FHB) based on metabolo-transcripto
125                                              Fusarium head blight (FHB) caused by Fusarium graminearu
126                                              Fusarium head blight (FHB) is a cereal disease caused by
127                                              Fusarium head blight (FHB) management is a great challen
128                                              Fusarium head blight (FHB), caused by Fusarium graminear
129 leaf volatile Z-3-HAC protects wheat against Fusarium head blight by priming for enhanced JA-dependen
130                                              Fusarium head blight caused by Fusarium graminearum is o
131 t FgATG17, prevented the fungus from causing Fusarium head blight disease.
132 sarium graminearum and related species cause Fusarium head blight on cultivated grasses, such as whea
133 ium graminearum causes the important disease Fusarium head blight on various species of cereals, lead
134 s for breeding programs intended to obtained Fusarium head blight resistance.
135 cide residues in wheat grains susceptible to fusarium head blight treated with fungicides, and to eva
136 actor produced by the causal fungal agent of Fusarium head blight, Fusarium graminearum.
137                                              Fusarium head blight, leaf spotting diseases, and, more
138 bally important wheat (T. aestivum) disease, Fusarium head blight.
139 inst the database which is Web accessible at FUSARIUM-ID and the Centraalbureau voor Schimmelcultures
140 ction allows users to collect sequences from Fusarium-ID and the FCGP and analyze them later using mu
141                                          The Fusarium-ID archives phylogenetic marker sequences from
142 ncreasing data and knowledge and consists of Fusarium-ID, Fusarium Comparative Genomics Platform (FCG
143               Our sampling suggests that the Fusarium incarnatum-equiseti species complex (FIESC), wi
144 ecies limits within the clinically important Fusarium incarnatum-F. equiseti and F. chlamydosporum sp
145                             The fungal genus Fusarium includes many plant and/or animal pathogenic sp
146 transferase (NAT) and has been identified in Fusarium infecting cereal plants as responsible for deto
147 ofilms is a key pathogenicity determinant of Fusarium, irrespective of the thickness of these biofilm
148                                              Fusarium is a genus of filamentous fungi that contains m
149                                              Fusarium is a major cause of microbial keratitis, and it
150                                              Fusarium is a ubiquitous mold that can cause superficial
151 tivity of a fungus from the ubiquitous genus Fusarium is related to the presence of nanometre-scale p
152                                   The fungus Fusarium is well known as a plant pathogen, but has rece
153 ed by mycotoxin-producing fungi of the genus Fusarium, is an economically important crop disease.
154      We investigated the ability of outbreak Fusarium isolates (F. solani species complex [FSSC] and
155 g (MLST) survey of plumbing drain-associated Fusarium isolates and comparing the diversity observed t
156 n elongation factor (TEF)-1alpha analysis of Fusarium isolates included FSSC 1-a, FOSC 33, and FDSC E
157          In this large susceptibility study, Fusarium isolates were least susceptible to voriconazole
158  observed to the known diversity of clinical Fusarium isolates.
159 90 demonstrated synergy against six of eight Fusarium isolates.
160 ocular sources of 52 patients diagnosed with Fusarium keratitis at the Bascom Palmer Eye Institute (M
161                                              Fusarium keratitis is common and often results in poor o
162 d not find a benefit for all corneal ulcers, Fusarium keratitis may benefit from the addition of oral
163 006 outbreak of contact lens (CL)-associated Fusarium keratitis, there may have been a rise in CL-ass
164 nt oral voriconazole on clinical outcomes in Fusarium keratitis.
165 ee of the FSSC species (Fusarium falciforme, Fusarium keratoplasticum, and Fusarium sp. FSSC 12) acco
166 ment of their antimicrobial activity against Fusarium langsethiae.
167 ex were isolated: F. graminearum s.s. (37%), Fusarium meridionale (46%), Fusarium cortaderiae (13%),
168 nge of endogeneous estrogens, androgens, and fusarium metabolites.
169 ng such threats, it is difficult to identify Fusarium morphologically.
170                  This study investigated the Fusarium mycotoxin Nivalenol (NIV) on the metabolism of
171                                     Emerging Fusarium mycotoxins were predominant in Italian samples
172 s study was concerned with the fate of these Fusarium mycotoxins within malting, brewing, milling and
173 thy and its cause is hypothesised to involve Fusarium mycotoxins.
174 which were lower than TDI for all legislated Fusarium mycotoxins.
175                                              Fusarium (n = 126) and Aspergillus species (n = 52) were
176 plants and can contribute to pathogenesis of Fusarium on some crop species.
177   It does not amplify Candida, Scedosporium, Fusarium or Rhizopus species and its clinical sensitivit
178 sing the same biocatalyst in both processes, Fusarium oxysporum 152B.
179 f the most widespread phytopathogenic fungi, Fusarium oxysporum and Botrytis cinerea, were chosen to
180 ainst Rhizopus stolonifer, Botrytis cinerea, Fusarium oxysporum and Colletotrichum gloeosporioides.
181     Formae speciales (ff.spp.) of the fungus Fusarium oxysporum are often polyphyletic within the spe
182  flavoprotein nitroalkane oxidase (NAO) from Fusarium oxysporum catalyzes the oxidation of primary an
183                                              Fusarium oxysporum f. lycopersici (Fol), the causal agen
184 mosomes within strains pathogenic to tomato (Fusarium oxysporum f. sp. lycopersici) and pea (Fusarium
185 ichum higginsianum, Cercospora beticola, and Fusarium oxysporum f. sp. lycopersici.
186 um graminearum, Fusarium verticillioides and Fusarium oxysporum f. sp. lycopersici.
187 of a set of strains of the melon wilt fungus Fusarium oxysporum f. sp. melonis (Fom), bioinformatics-
188            Identity of the effector genes of Fusarium oxysporum f. sp. niveum (Fon) races that affect
189 yses suggested that G-LSR2 was acquired from Fusarium oxysporum f. vasinfectum through horizontal gen
190                     Panama disease caused by Fusarium oxysporum f.sp. cubense infection on banana is
191                    Resistance to wilt fungus Fusarium oxysporum f.sp. matthioli (FOM) is a polygenic
192 of a set of strains of the melon wilt fungus Fusarium oxysporum f.sp. melonis (Fom), bioinformatics-b
193                         Genome sequencing of Fusarium oxysporum revealed that pathogenic forms of thi
194 eromone from the plant pathogenic ascomycete Fusarium oxysporum revealed the presence of a central be
195      When infecting a host plant, the fungus Fusarium oxysporum secretes several effector proteins in
196                                          The Fusarium oxysporum species complex (FOSC) comprises a mu
197 e Fusarium solani species complex (FSSC) and Fusarium oxysporum species complex (FOSC), the most comm
198  and one each within the FIESC (1-a) and the Fusarium oxysporum species complex (ST-33) were widespre
199 Here, we show that the root-infecting fungus Fusarium oxysporum uses a functional homologue of the pl
200 (Botrytis cinerea, Pseudomonas syringae, and Fusarium oxysporum) were used to demonstrate potential t
201 haracterized a PR-1-like protein, Fpr1, from Fusarium oxysporum, an ubiquitous fungal pathogen that c
202  capsulatum, Coccidioides immitis/posadasii, Fusarium oxysporum, Aspergillus spp., and Bipolaris spp.
203 t Aspergillus flavus, Aspergillus ochraceus, Fusarium oxysporum, Saccharomyces cerevisiae and Candida
204 i species complex (FFSC) and rare strains of Fusarium oxysporum.
205 to the root-infecting vascular wilt pathogen Fusarium oxysporum.
206 is and virulence in the vascular wilt fungus Fusarium oxysporum.
207 against several filamentous fungi, including Fusarium oxysporum.
208 tants, as shown for Pseudomonas syringae and Fusarium oxysporum.
209 orium prolificans, Scedosporium apiospermum, Fusarium oxysporum/Fusarium solani, Rhizopus arrhizus, R
210 y 20% to 30% and increased susceptibility to Fusarium pathogens.
211  and further elucidating the role of Six6 in Fusarium-plant interactions.
212 larity to the closely related partitiviruses Fusarium poae virus 1 and Penicillium stoloniferum virus
213 iun strains including Fusarium subglutinans, Fusarium proliferatum and Fusarium tricinctum.
214 on elongation factor coding genes identified Fusarium proliferatum, which was confirmed later by cult
215  predominantly caused by the fungal pathogen Fusarium pseudograminearum, is a disease of economic sig
216 e culture collections (e.g., CBS-KNAW or the Fusarium Research Center) cultures of novel mycosis-asso
217 ization of an orphan gene (Triticum aestivum Fusarium Resistance Orphan Gene [TaFROG]) as a component
218 portant clade of veterinary relevance within Fusarium Six of the multilocus STs within the FSSC (3+4-
219                                              Fusarium solani and F. oxysporum were the causative orga
220 -containing poly(butylene adipate) films and Fusarium solani cutinase (FsC).
221                                  Conversely, Fusarium solani Cutinase and lipases from Mycobacterium
222 polytic activity of three microbial lipases: Fusarium solani cutinase, Rv0183, and LipY from Mycobact
223 -adipate ratio by Rhizopus oryzae lipase and Fusarium solani cutinase.
224 in France suffered an outbreak of the fungus Fusarium solani in 2001.
225 crobial peptide (CRAMP) in a murine model of Fusarium solani keratitis.
226 e sources of human pathogenic strains in the Fusarium solani species complex (FSSC) and Fusarium oxys
227 lates (47/67 = 70.1%) were nested within the Fusarium solani species complex (FSSC), and these includ
228                              Three isolates (Fusarium solani, Aspergillus fumigatus, Candida albicans
229 Scedosporium apiospermum, Fusarium oxysporum/Fusarium solani, Rhizopus arrhizus, Rhizopus microsporus
230 d compare it to the well-studied enzyme from Fusarium solani.
231 arium oxysporum f. sp. lycopersici) and pea (Fusarium 'solani' f. sp. pisi).
232 ota showed a predominance of Alternaria sp., Fusarium sp. and Epicoccum sp. Microdochium nivale (23%)
233 ; however, strains of F. keratoplasticum and Fusarium sp. FSSC 12 were mostly (25/27) isolated from m
234 um falciforme, Fusarium keratoplasticum, and Fusarium sp. FSSC 12) accounted for four-fifths of the v
235 rowth of two fungal pathogens, Phoma sp. and Fusarium sp., and reduced survivorship of brine shrimp,
236 ipants, 72 (30.4%) were culture positive for Fusarium species (41 [56.9%] male and 31 [43.1%] female;
237 tification of Aspergillus, Scedosporium, and Fusarium species (n = 28) by matrix-assisted laser desor
238 ubgroup analyses looking at ulcers caused by Fusarium species and adjusting for baseline best spectac
239 mptomatic native grasses for the presence of Fusarium species and confirmed infected grasses as hosts
240 sses for the presence of mycotoxin-producing Fusarium species and evaluated the ability of these fung
241                                              Fusarium species are among the most important phytopatho
242                              Aspergillus and Fusarium species are important causes of fungal infectio
243 d for patients with Aspergillus or other non-Fusarium species as the causative organism (1.5 points [
244 conazole, and especially among patients with Fusarium species as the causative organism.
245 acilitate molecular identification of the 69 Fusarium species associated with human or animal mycoses
246                              Genomes of four Fusarium species have been published with more being cur
247  pathogens and is active with malonyl-CoA in Fusarium species infecting cereals.
248                 Compared to other organisms, Fusarium species isolates had the highest MICs to vorico
249                               However, these Fusarium species may have had a longer evolutionary hist
250 sm subgroups, there was some suggestion that Fusarium species might have a decreased rate of perforat
251        Fumonisins are mycotoxins produced by Fusarium species that commonly live in maize.
252                          Mycotoxin-producing Fusarium species were shown to be prevalent in phylogene
253 chygibbosum, F. flocciferum, and two unnamed Fusarium species within the F. tricinctum species comple
254 h American grasses are commonly inhabited by Fusarium species, but appear to accommodate these toxige
255 hows highly inhibitory activity towards some Fusarium species, but not to other fungi, indicating tha
256 t UBL1 has a broader role in virulence among Fusarium species.
257 itative disease resistance to plants against Fusarium species.
258                      Here, a newly developed Fusarium-specific monoclonal antibody (mAb ED7) was used
259 rder Mucorales, n=16; Aspergillus spp, n=16; Fusarium spp, n=9), commonly with multiple mold species
260 ere able to quantify mycotoxins and identify Fusarium spp.
261 1, B and B1 (ENs) are mycotoxins produced by Fusarium spp. and are normal contaminants of cereals and
262          Common causes include Candida spp., Fusarium spp. and Aspergillus spp..
263 s (Fusarium disease severity, PCR assays for Fusarium spp. identification and mycotoxin quantificatio
264 ns (ITS1 and -2) to detect and differentiate Fusarium spp. responsible for ocular infections.
265 ypes: order Mucorales, Aspergillus spp., and Fusarium spp.) significantly prolonged the time to wound
266 or non-Aspergillus molds (Mucoromycotina and Fusarium spp.).
267 evis, which was artificially inoculated with Fusarium spp., as well as their emission via drainage wa
268 versity and evolutionary relationships of 67 Fusarium strains from veterinary sources, most of which
269                                    Six major Fusarium STs were frequently isolated from plumbing drai
270 oduced by several Fusariun strains including Fusarium subglutinans, Fusarium proliferatum and Fusariu
271  secreted by multiple species from the genus Fusarium, that cmp from Fusarium verticillioides (Fv-cmp
272  evidence that horizontal gene transfer from Fusarium to Vd991 contributed significantly to its adapt
273  and classify three agriculturally important Fusarium toxins in wheat.
274 ed calibration curves were used to determine Fusarium toxins in wheat.
275 otoxin method was developed for 12 different Fusarium toxins including modified mycotoxins in beer (d
276                  None of the other monitored Fusarium toxins like 15-acetyldeoxynivalenol, HT2- and T
277                 Cut off levels for all three Fusarium toxins were validated using blank wheat and whe
278  assay was developed and validated for three Fusarium toxins, deoxynivalenol (DON), zearalenone (ZEA)
279 rium subglutinans, Fusarium proliferatum and Fusarium tricinctum.
280                                              Fusarium ulcers randomized to oral voriconazole had a 0.
281 though there may have been some effect among Fusarium ulcers.
282 ngal species from the genera Aspergillus and Fusarium using solid-state voltammetry is described.
283 ecies from the genus Fusarium, that cmp from Fusarium verticillioides (Fv-cmp) is a fungalysin metall
284                       The mycelial growth of Fusarium verticillioides (Sacc.) Nirenberg was reduced s
285 ber officinale Roscoe) was evaluated against Fusarium verticillioides (Saccardo) Nirenberg.
286 ATCC MYA-3631, A. terreus ATCC MYA-3633, and Fusarium verticillioides [moniliforme] ATCC MYA-3629) an
287 cally diverse species: Fusarium graminearum, Fusarium verticillioides and Fusarium oxysporum f. sp. l
288                                              Fusarium verticillioides produces fumonisin mycotoxins d
289 ies most found were Fusarium graminearum and Fusarium verticillioides with 26% and 12% of incidence,
290                     Fusarium graminearum and Fusarium verticillioides, coumarin derivatives were prov
291                      Pokkah boeng, caused by Fusarium verticillioides, is a serious disease in sugarc
292 d in the Fvmcm1 mutants of the heterothallic Fusarium verticillioides.
293                Corn plants contaminated with Fusarium verticilloides were harvested at two dates and
294                                              Fusarium virguliforme is a soil borne pathogen that caus
295 ophthora sojae and the fungal plant pathogen Fusarium virguliforme that are pathogenic to soybean (Gl
296 dying growth, germination and sporulation in Fusarium virguliforme that causes sudden death syndrome
297       Sudden death syndrome (SDS), caused by Fusarium virguliforme, is one of the top yield-limiting
298  underpinnings of pathogenicity in the genus Fusarium, we compared the genomes of three phenotypicall
299 ful to select melon cultivars to avoid melon Fusarium wilt, but also to monitor how quickly a Fom pop
300 ed distributions, notably tropical race 4 of Fusarium wilt, rival its impact.

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