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1 olic analysis of the dominant oral bacterium Fusobacterium nucleatum.
2 ggregation with the anaerobic oral bacterium Fusobacterium nucleatum.
3 mediated adherence to a peridontal pathogen, Fusobacterium nucleatum.
4 ella parvula, Peptostreptococcus micros, and Fusobacterium nucleatum.
5 , Aggregatibacter actinomycetemcomitans, and Fusobacterium nucleatum.
6 xed infection with the otherwise stimulatory Fusobacterium nucleatum.
7 activation by another periodontal bacterium, Fusobacterium nucleatum.
8 bserved for Bacteroides thetaiotaomicron and Fusobacterium nucleatum.
9 llowing stimulation of epithelial cells with Fusobacterium nucleatum.
10 Actinomyces israelii with the coisolation of Fusobacterium nucleatum.
11 adA adhesin from the Gram-negative bacterium Fusobacterium nucleatum.
12 olymicrobial oral infections with or without Fusobacterium nucleatum.
13 cus gordonii and the opportunistic commensal Fusobacterium nucleatum.
14 symporter (NSS) family has been cloned from Fusobacterium nucleatum.
17 inations that resulted in tailing endpoints (Fusobacterium nucleatum, 86% agreement) or in cases of l
19 production of CCL20 and hBDs in response to Fusobacterium nucleatum, a commensal bacterium of the or
21 entified a cell wall-associated protein from Fusobacterium nucleatum, a Gram-negative bacterium of th
22 t evidence for the immunosuppressive role of Fusobacterium nucleatum, a gram-negative oral bacterium
24 Key quorum-sensing plaque bacteria, such as Fusobacterium nucleatum, act as bridging species between
25 monas gingivalis, Prevotella intermedia, and Fusobacterium nucleatum activated both TLRs, but TLR4 pl
26 helial cell response to the common bacterium Fusobacterium nucleatum, an important bridging species t
29 D-2 mRNA was induced by cell wall extract of Fusobacterium nucleatum, an oral commensal microorganism
30 eration sequencing implicated coinfection of Fusobacterium nucleatum and Actinomyces israelii, resolv
31 also significantly associated with pathogens Fusobacterium nucleatum and Aggregatibacter actinomycete
32 mixed infection with the periodontopathogens Fusobacterium nucleatum and Porphyromonas gingivalis.
34 treptococcus mitis, Veillonella parvula, and Fusobacterium nucleatum) and the same biofilm plus the p
35 ophilus aphrophilus, Actinomyces naeslundii, Fusobacterium nucleatum, and A. actinomycetemcomitans, a
36 as Prevotella intermedia, Selenomonas noxia, Fusobacterium nucleatum, and Actinobacillus actinomycete
37 hyromonas gingivalis, Prevotella intermedia, Fusobacterium nucleatum, and Aggregatibacter actinomycet
40 tella intermedia, Streptococcus intermedius, Fusobacterium nucleatum, and Peptostreptococcus micros,
44 gar Candida medium, coaggregation assay with Fusobacterium nucleatum, and sugar assimilation profiles
49 these findings by identifying the bacterium Fusobacterium nucleatum as a previously unrecognized che
50 cus anginosus, Porphyromonas gingivalis, and Fusobacterium nucleatum, as well as Campylobacter rectus
51 gregatibacter actinomycetemcomitans JP2, and Fusobacterium nucleatum ATCC 10953 were unable to grow a
52 s biofilm (Streptococcus sanguinis DSM20068, Fusobacterium nucleatum ATCC10953, and Porphyromonas gin
54 i, Actinobacillus actinomycetemcomitans, and Fusobacterium nucleatum) biofilm formation under anaerob
55 hyromonas gingivalis, Prevotella intermedia, Fusobacterium nucleatum, Campylobacter rectus, and Trepo
56 hyromonas gingivalis, Prevotella intermedia, Fusobacterium nucleatum, Campylobacter rectus, Eikenella
57 h extraction followed by oral infection with Fusobacterium nucleatum caused BONJ-like lesions and del
58 vivo experiments, the combination of Pam and Fusobacterium nucleatum caused the death of gingival fib
59 ous studies showed that hBD-2 was induced by Fusobacterium nucleatum cell wall extract without the in
62 omonas gingivalis, Campylobacter rectus, and Fusobacterium nucleatum, could cause localized bone reso
63 for the presence and amount of EBV, CMV, and Fusobacterium nucleatum DNA using real-time polymerase c
64 F together with either Escherichia coli DNA, Fusobacterium nucleatum DNA, or Porphyromonas gingivalis
66 s as potentiators of tumorigenesis-including Fusobacterium nucleatum, enterotoxigenic Bacteroides fra
67 wall extracts of Porphyromonas gingivalis or Fusobacterium nucleatum, Escherichia coli lipopolysaccha
69 eviously demonstrated that sonic extracts of Fusobacterium nucleatum FDC 364 were capable of inhibiti
73 ction was used for detecting and quantifying Fusobacterium nucleatum (Fn), Aggregatibacter actinomyce
77 (Sg)/S. oralis (So)/S. sanguinis (Ss) and Sg/Fusobacterium nucleatum (Fn)/Porphyromonas gingivalis (P
78 employed by the Gram-negative oral pathogen Fusobacterium nucleatum for cell death induction of huma
79 s were significantly higher for P. micra and Fusobacterium nucleatum for the screw-retained group.
81 monas gingivalis, Prevotella intermedia, and Fusobacterium nucleatum, have recently been shown to sec
82 report a case of Lemierre's syndrome due to Fusobacterium nucleatum in a previously healthy 19-year-
84 inoculation of Porphyromonas gingivalis and Fusobacterium nucleatum in young (4 to 5 mo) and aged (1
85 dentify an anaerobic Gram-negative bacillus, Fusobacterium nucleatum, in a patient with "culture-nega
86 otella intermedia, Campylobacter rectus, and Fusobacterium nucleatum, in subgingival dental plaque of
87 /+) mouse model of intestinal tumorigenesis, Fusobacterium nucleatum increases tumor multiplicity and
88 ed replication plan of key experiments from 'Fusobacterium nucleatum infection is prevalent in human
89 thelial cells reached its peak 2 h following Fusobacterium nucleatum infection whereas it rapidly dec
100 ristic of the suspected periodontal pathogen Fusobacterium nucleatum is its ability to adhere to a pl
101 and difficult-to-cultivate species, such as Fusobacterium nucleatum, Leptotrichia (Sneathia) spp., a
103 l as with Veillonella sp. (early colonizer), Fusobacterium nucleatum (middle colonizer), and Aggregat
105 ly significant, Porphyromonas gingivalis and Fusobacterium nucleatum occur in higher concentrations m
106 ella forsythia [previously T. forsythensis], Fusobacterium nucleatum, Parvimonas micra [previously Pe
107 pathogens, including Prevotella intermedia, Fusobacterium nucleatum, Peptostreptococcus micros, and
108 ection protocol using Prevotella intermedia, Fusobacterium nucleatum, Peptostreptococcus micros, and
109 their ability to coaggregate with strains of Fusobacterium nucleatum, Peptostreptococcus micros, Pept
110 inomycetemcomitans, Eikenella corrodens, and Fusobacterium nucleatum/periodonticum were statistically
111 alis (Pg); 4) group G-PgFn: oral gavage with Fusobacterium nucleatum + Pg; 5) group I-Pg: heat-killed
112 , Campylobacter curvus, Eikenella corrodens, Fusobacterium nucleatum, Porphyromonas gingivalis, and P
113 nisms (Actinobacillus actinomycetemcomitans, Fusobacterium nucleatum, Porphyromonas gingivalis, Pepto
114 ctinomycetemcomitans), Campylobacter rectus, Fusobacterium nucleatum, Porphyromonas gingivalis, Prevo
115 ntified virulence mechanisms of oral species Fusobacterium nucleatum, Porphyromonas gingivalis, Strep
116 imens yielded pathogenic bacteria, including Fusobacterium nucleatum, Prevotella heparinolytica, Prev
117 three orange-complex periodontal pathogens (Fusobacterium nucleatum, Prevotella intermedia, and Camp
118 phyromonas gingivalis, Tannerella forsythia, Fusobacterium nucleatum, Prevotella intermedia, and Camp
119 phyromonas gingivalis, Tannerella forsythia, Fusobacterium nucleatum, Prevotella intermedia, and tota
120 same double-labeling techniques to identify Fusobacterium nucleatum, Prevotella intermedia, oral Cam
121 thogens such as Porphyromonas gingivalis and Fusobacterium nucleatum produce five different short-cha
122 ere we present the crystal structures of the Fusobacterium nucleatum riboswitch bound to FMN, ribofla
125 anaerobic pathogens, Prevotella intermedia, Fusobacterium nucleatum, Streptococcus intermedius, and
126 nfections (endodontic pathogens [EP]), i.e., Fusobacterium nucleatum, Streptococcus intermedius, Parv
127 e draft genome sequence and its analysis for Fusobacterium nucleatum sub spp. vincentii (FNV), and co
130 e in corncob formation between S. crista and Fusobacterium nucleatum, this property was examined.
131 eponema denticola, Tannerella forsythia, and Fusobacterium nucleatum to colonize the periodontium and
132 ms enumerated were Porphyromonas gingivalis, Fusobacterium nucleatum, Veillonella sp., and total anae
133 givalis, whereas phagocytosis of heat-killed Fusobacterium nucleatum was augmented compared with that
135 omonas gingivalis, Tannerella forsythia, and Fusobacterium nucleatum were analyzed for prediction of
136 , Aggregatibacter actinomycetemcomitans, and Fusobacterium nucleatum were assessed in anaerobic condi
138 votella intermedia, Eikenella corrodens, and Fusobacterium nucleatum were determined by real-time pol
139 lla, Salmonella, Haemophilus influenzae, and Fusobacterium nucleatum, which share structural and func
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