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1 me mutation, a base pair substitution in the G protein-coupled inwardly rectifying potassium channel
2 (HEK) 293 cells expressing rat MOR1 as well G protein-coupled inwardly rectifying potassium channel
3 K+ selectivity, we randomly mutagenized the G protein-coupled inwardly rectifying potassium channel
4 ed inhibition occurs via the activation of a G-protein-coupled inwardly rectifying potassium channel
5 Receptor-mediated activation of a G-protein-coupled inwardly rectifying potassium channel
6 lves coordinated postsynaptic inhibition via G protein-coupled inwardly rectifying potassium channels
7 G protein-coupled inwardly rectifying potassium channels
8 nopus laevis oocytes coexpressing mGluR8 and G protein-coupled inwardly rectifying potassium channels
9 enosine A1 receptor and its downstream GIRK (G protein-coupled inwardly rectifying potassium channels
10 We sought to determine whether G protein-coupled inwardly rectifying potassium channels
11 effectors of Galpha(i/o)-coupled receptors, G protein-coupled inwardly rectifying potassium channels
12 P) kinase phosphorylation, and activation of G protein-coupled inwardly rectifying potassium channels
13 abeled antagonist [(3)H]CGP 54626A, activate G-protein coupled inwardly rectifying potassium channels
14 -HT1A -IPSCs) generated by the activation of G-protein-coupled inwardly rectifying potassium channels
15 hin from keratinocytes and the activation of G-protein-coupled inwardly rectifying potassium channels
16 G-protein-coupled inwardly rectifying potassium channels
17 hibition of firing through the activation of G-protein-coupled inwardly rectifying potassium channels
18 ium channel (BK) and specific members of the G-protein-coupled inwardly rectifying potassium channels
19 hannel and A1 receptor-mediated opening of a G-protein-coupled inwardly rectifying potassium channel.
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