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1 t it encodes AGB1, a putative heterotrimeric G-protein beta subunit.
2 s one of the SDD products that encodes a rat G-protein beta subunit.
3 WD repeat structure similar to that of known G protein beta subunits.
4 ype cells overexpressing signaling-defective G protein beta subunits.
5 ranuclearly with DNA encoding five different G-protein beta subunits.
8 d to the zymogen granule membrane along with G-protein beta-subunit; all three alpha subunits were pr
9 ulatory process that blocks signaling from a G protein beta subunit and causes its relocalization wit
10 ise enhanced upon direct activation of Ste4 (G protein beta subunit) and Ste11 (Ste7 kinase) but not
11 nding in vitro of Ste5 to Ste11, Ste7, Ste4 (G protein beta subunit), and Fus3 (MAPK), confirmed that
12 ificity of interaction of RGS6 and RGS7 with G protein beta subunits, and certain biochemical propert
14 f this family include the signal-transducing G protein beta subunit, as well as other proteins that r
16 tes that the posttranslational processing of G protein beta subunits begins inside the protein-foldin
18 annel indicates that Ca2+ channel beta1b and G protein beta subunits bind to the alpha1 subunit at th
19 ion of the single gene for the Dictyostelium G protein beta-subunit blocks development at an early st
21 units, or antibodies to different regions of G-protein beta subunits established the involvement of a
24 with AC2, the structural determinants on the G protein beta subunit for interaction with various effe
26 with the short splice isoform of the type 5 G-protein beta subunit (G beta 5) and the RGS9 anchor pr
30 s where it forms complexes with the atypical G protein beta subunit, Gbeta(5), and transmembrane prot
32 ns form trimeric complexes with the atypical G protein beta subunit Gbeta5 and a membrane anchor, R7B
40 GS form trimeric complexes with the atypical G protein beta subunit, Gbeta5, and membrane anchor R7BP
44 signaling (RGS) protein, RGS9-1, and type 5 G protein beta-subunit, Gbeta5L, regulates the duration
46 accompanied by a parallel redistribution of G protein beta subunits; however, there was no increase
48 rms overexpressing gar-3 or lacking GPB-2, a G-protein beta-subunit involved in RGS-mediated inhibiti
49 olding of the first five beta strands in the G protein beta subunit is a requirement for appropriatel
52 single gcr1, gpa1, and agb1 (heterotrimeric G-protein beta-subunit) mutants are additive or synergis
53 characterized the modulatory domains of the G protein beta subunit on the recombinant P/Q-type chann
54 nalyzed the binding of Ca2+ channel beta and G protein beta subunits on the two separate binding site
57 structure of another WD repeat protein, the G-protein beta-subunit, predicts that esc protein adopts
58 elopment ensures expression of RGS9-2/type 5 G-protein beta subunit/R7BP complexes at postsynaptic si
59 family and the long splice variant of type 5 G protein beta subunit (RGS9-Gbeta5L) plays a critical r
62 d the physiological role of Gbeta5, a unique G protein beta subunit that dimerizes with regulators of
63 in beta-propeller enzymes and is used by the G protein beta subunit to bind the G protein alpha subun
65 at phenotypically resemble cells lacking the G-protein beta-subunit yielded the protein kinase YakA.
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