ライフサイエンスコーパス: G

1 G protein-coupled receptors (GPCRs), the largest family

2 G-Au modified GCE exhibited an enhanced electrocatalytic

3 G-MDSCs, made of immature and mature granulocytes expres

4 G-overhang length increases with time after CTC1 disrupt

5 G-quadruplex (G4)-containing substrates mimicking the ma

6 G. bethesdensis remains, and in some cases appears to di

7 ncluding three novel mutations of c.1059 + 1 G > T, c.2002dupC and c.2236_2237del CT, as well as a pr

8 nsense mutation within the SYNE1 gene (23560 G>T) encoding Nesprin-1 [15, 16].

9 The difference between G 2W/G and G W/W uncovers the finite thermal resistance induc

10 ar mechanics calculations at the mPW1N/6-311+G(2d,2p):FF99SB//mPW1N/6-31G(d):FF99SB level.

11 tions were performed using the M06-2X/6-311++G(d,p) level of theory and CPCM solvation model.

12 utation in the X-chromosome gene FIGF (c.352 G>A) associated with early childhood respiratory deficie

13 everal aspects of metabolism by activating 4 G-protein-coupled receptors, the A1, A2A, A2B, and A3 ad

14 : G-1106A, A-1018T, T-1014C, T-988G, G-513A, G-462A, T-415C, C-89A, and C-57T.

15 + 2 T > G mutation in HSD17B3, and the c.544 G > A, c.548-44 T > G and c.278delG mutations in SRD5A2.

16 modification distinct from the classical m(7)G cap that promotes rather than inhibits RNA decay.

17 s (SNPs): G-1106A, A-1018T, T-1014C, T-988G, G-513A, G-462A, T-415C, C-89A, and C-57T.

18 Family A G protein-coupled receptors (GPCRs) control diverse biol

19 These studies show that T-oligo can form a G-quadruplex and that the antitumor effects of T-oligo m

20 maize (Zea mays), several genes, including a G-BOX BINDING FACTOR 3 (GBF3) were identified as candida

21 Proteinase-Activated Receptor-2 (PAR2 ) is a G protein-coupled receptor activated by serine proteinas

22 e type 1 taste receptor member 3 (T1R3) is a G protein-coupled receptor involved in sweet-taste perce

23 GPR151 is a G-protein coupled receptor for which the endogenous liga

24 nding sites on the A2A adenosine receptor, a G-protein-coupled receptor that is a target for the trea

25 A novel peptide substrate (A G G P L G P P G P G G) was developed for quantifying the

26 quent (10(-5)-10(-6)), whereas C-->G, U-->A, G-->C, and C-->A errors from purine-purine and pyrimidin

27 ymium magnetic sticks that capture protein A/G-coated paramagnetic beads bound to antibody-luciferase

28 4 (also known as TEM5/ADGRA2) is an adhesion G protein-coupled receptor family member that plays a pi

29 sets based on the expression of the adhesion G protein-coupled receptor GPR56, and GPR56(+) TEMRA cel

30 We present ALFRED-G, a greedy alignment-free distance estimator for phylog

31 efficiently to both double-stranded DNA and G-quadruplex (G4) DNA.

32 eening methods for estimating the MCPD-E and G-E contents in the fish products.

33 synthetic intermediate containing the EF and G rings of the target.

34 didate and that the expression of both F and G proteins of AMPV-C induces a protective response again

35 air (TC-NER) (category 1: XP-A, B, D, F, and G) and preserved TC-NER (category 2: XP-C, E, and V).

36 mone receptor farnesoid X receptor (FXR) and G protein-coupled membrane receptor TGR5 that demonstrat

37 The difference between G 2W/G and G W/W uncovers the finite thermal resistance induced by

38 peptide ligands from platelet GP1balpha and G-protein-coupled receptor MAS effectively bound Ig21 by

39 Sensors consist of a GPCR and G protein tethered by an ER/K linker flanked by FRET pro

40 NDPK isoforms and between NDPK isoforms and G proteins.

41 phorylation, thereby bridging MAP kinase and G-Protein-Coupled Receptor signaling.

42 otably including growth factor receptors and G protein-coupled receptors, control myelination.

43 Under basal conditions, receptors and G proteins form activity-dependent complexes that last f

44 However, it is unclear how receptors and G proteins meet, interact and couple.

45 ort that certain RNA template structures and G-rich sequences, ahead of diverse reverse transcriptase

46 eted-small-molecules to include both A.T and G.C base pairs, we recently discovered that the heterocy

47 Validation of probe targets and "G" and "H" site specificity was carried out using a seri

48 s revealed submembranous localization of Ank-G at nodes of Ranvier and AIS.

49 sis and 3D reconstructions revealed that Ank-G colocalized with TH only at the AIS.

50 ollowing administration of neutralizing anti-G-CSF antiserum.

51 ly stable species (parallel and antiparallel G-quadruplex in K+ and Na+, respectively).

52 specifically to human telomeric antiparallel G-quadruplex.

53 AP2-G expression during this 'commitment cycle' prepares gen

54 opment, we show that sexually committed, AP2-G(+) mature schizonts specifically upregulate additional

55 rasite transcriptomes from a conditional AP2-G knockdown line and NF54 wild-type parasites at multipl

56 een TMLE and 2 common estimation approaches (G-computation and inverse probability weighting) and pre

57 residues that are subsequently translated as G.

58 pair (MMR) pathways to generate mutations at G-C and A-T base pairs, respectively.

59 The RSV F protein and attachment (G) protein were found to be internalized in both infecte

60 Class B G protein-coupled receptors (GPCRs) respond to paracrine

61 The difference between G 2W/G and G W/W uncovers the finite thermal resistance

62 ST active site is composed of a GSH binding "G site" and a substrate binding "H site".

63 Inspired by the hydrogen-bonded G-quadruplexes found frequently in guanine-rich DNA, her

64 Our detailed investigation of Mg(2+)-bound G:C W:W Trans pairs occurring in high-resolution RNA cry

65 Goldin-Meadow & Brentari (G-M&B) challenge the traditional separation between gest

66 ition presented by Goldin-Meadow & Brentari (G-M&B) indicates a more complex picture.

67 weather variability and markedly affected by G x E interactions.

68 activation of a heterotrimeric G-protein by G-protein coupled receptors.

69 Acute hormone secretion triggered by G protein-coupled receptor (GPCR) activation underlies m

70 mperature from 90 degrees C to 10 degrees C, G' increased to reach a plateau at 10 degrees C.

71 ith rates of approximately 10(-4) The A-->C, G-->A, A-->U, C-->U, G-->U, U-->C, and U-->G errors most

72 ng lead for further development of cathepsin G inhibitors targeting chronic inflammatory disorders.

73 bitor-1 (SFTI-1) produced a potent cathepsin G inhibitor (Ki = 0.89 nM).

74 Substituting preferred cathepsin G substrate sequences into sunflower trypsin inhibitor-1

75 n pulmonary microvascular endothelial cells, G was 20.4 +/- 12 Pa and decreased by 20% and 22% with i

76 aracteristic N-terminal domain and a central G domain that are common to all Obg proteins.

77 the R-spondin/leucine-rich repeat-containing G protein-coupled receptor 4 (LGR4) axis in driving aber

78 two guanine electron donors into crystalline G-quadruplex-based organic frameworks, wherein the elect

79 ersity, on chromosomes 1 and 2 in cultivated G. hirsutum as compared with low nucleotide diversity on

80 Five cytokines (G-CSF, GM-CSF, IL-1-ra, IL-2 and IL-16) were significant

81 cantly different between women with detected G vaginalis or Bacteroides spp morphotypes and those wit

82 .1 9 nA/mug mL(-1)), low limit of detection (G, A = 0.5 mug mL(-1); T, C = 1.0 mug mL(-1)), and high

83 e property of individual human telomeric DNA G-quadruplexes.

84 n the activation loop signature sequence S-E-G.

85 l-tRNA in the small subunit P site during EF-G-catalyzed translocation.

86 y sequence also helps dissociation of the EF-G by providing alternative base-pairing options.

87 shift promoting signals mostly impair the EF-G-catalyzed translocation step of the two tRNALys and th

88 increase of curd firmness and both elastic (G') and viscous (G'') moduli.

89 an intracellular binding site that enhances G-protein coupling.

90 ross-order recombination between enterovirus G (order Picornavirales) and torovirus (order Nidovirale

91 scular signalling components, and especially G protein-coupled receptors (GPCRs), as next-generation

92 The average EVAN-G score was 84.3 (standard deviation, SD, 6.4) after VR,

93 These results suggest that the rLS/AMPV-C F&G recombinant virus is a safe and effective bivalent vac

94 or more subtypes, including subtypes A1, F, G, H, J, and K and unclassified fragments, including one

95 nit head domain within the elongation factor G (GDP)-bound ribosome complex.

96 The translation factors, elongation factor G and ribosome recycling factor, are known to be require

97 , and granulocyte colony-stimulating factor (G-CSF) levels in the amniotic fluid of ZIKV-positive pre

98 okine granulocyte colony-stimulating factor (G-CSF) through complex mechanisms.

99 IL-8, granulocyte-colony stimulating factor (G-CSF), IL-33, IL-11, IL-1alpha, and IL-1beta.

100 on of granulocyte-colony stimulating factor (G-CSF); these effects are reversed following administrat

101 -regulated kinase (ERK) activation following G-protein coupled receptor (GPCR) activation.

102 al latency that can be reactivated following G-CSF treatment.

103 Our investigation has been performed for G-quadruplexes formed by folding of GGG(TTAGGG)3 single

104 Guanine-rich oligonucleotides can form G-quadruplexes (G4), which are stabilized by the hydroge

105 with strong G/C skew and propensity to form G-quadruplex in non-template DNA, corroborating with all

106 vated receptors are largely sequestered from G proteins upon internalization.

107 ing component with a refractory period (e.g. G protein), and 3) inactivation of a factor needed for F

108 ding SNVs found in SCZ subjects in the GIT1 (G protein-coupled receptor kinase interacting ArfGAP 1)

109 loropropane-1,2-diol (MCPD-E), and glycidol (G-E).

110 F) or the RSV major attachment glycoprotein (G) between the hemagglutinin-neuraminidase (HN) and RNA-

111 VSVs that include the VSV glycoprotein (G) gene, even in most recombinant attenuated strains, ca

112 ilised for the development of graphene-gold (G-Au) nanocomposite.

113 On the other hand, GPCR-G protein fusions have been used extensively to understa

114 re, we present evidence that individual GPCR-G-protein interactions can reinforce each other to enhan

115 stablished for detecting ligand induced GPCR-G protein interactions in cells.

116 0(-)(8)) in an intron of the adhesion GPR98 (G-protein-coupled receptor V1) gene on chromosome 5q14.3

117 (1) a GSH-based photoaffinity probe (GSTABP-G) to target the "G site", and (2) an ABP designed to mi

118 The GSTABP-G features a photoreactive moiety for UV-induced covalen

119 lly pathogenic, homozygous variant, c.240C > G (p.Phe80Leu).

120 The m.3243A > G mitochondrial DNA mutation was originally described in

121 The phenotypic spectrum of the m.3243A > G mutation has since expanded to include a spectrum of n

122 rlie the tissue specificity of the m.3243A > G mutation, and importantly, allow the future testing of

123 specific markers indicated that c.254-649T > G CLRN1 represents a founder allele that may significant

124 139309795, p.Arg405*) and missense (c.701A > G, rs143439626, p.Lys234Arg) mutations of the alkylglyce

125 AS) syndrome, however, the common m.3243 A > G mutation was excluded.

126 n HSD17B3, and the c.544 G > A, c.548-44 T > G and c.278delG mutations in SRD5A2.

127 We evaluated the c.277 + 2 T > G mutation in HSD17B3, and the c.544 G > A, c.548-44 T >

128 ith a known genetic cause for RP) and c.124A>G, p.K42E in dehydrodolichol diphosphate synthase (DHDDS

129 d a novel splice mutation in IKBKG (c.518+2T>G, resulting in an in-frame deletion: p.DelQ134_R256).

130 oration opposite dT, predicting frequent A-->G errors in RNA with rates of approximately 10(-4) The A

131 tively frequent (10(-5)-10(-6)), whereas C-->G, U-->A, G-->C, and C-->A errors from purine-purine and

132 , G-->A, A-->U, C-->U, G-->U, U-->C, and U-->G errors mostly due to pyrimidine-purine mismatches were

133 ng loop adenines (A/AP) and tetrad guanines (G/AP) in quadruplexes formed by the human telomere d[AG3

134 for detecting activation of a heterotrimeric G-protein by G-protein coupled receptors.

135 he involvement of tolerogenic molecules (HLA-G, TGF-beta, and IL-10) were tested on a mixed lymphocyt

136 ve seen an explosion in our knowledge of HLA-G biology.

137 bulin and native sulfur phasing of the human G protein-coupled adenosine receptor.

138 (3D) ordered arrays of human immunoglobulin G (IgG) were fabricated using well-defined full-length a

139 fined as a 4-fold increase in immunoglobulin G directed against Cryptosporidium gp15 and/or Cp23 anti

140 an unexpected persistence of immunoglobulin G almost until weaning, potentially indicating prolonged

141 colonization and induction of immunoglobulin G and immunoglobulin A to all antigens tested, while cau

142 bic acid human serum protein, immunoglobulin G, and immunoglobulin M), and demonstrated a high correl

143 e for both Chlamydia-specific immunoglobulin G (IgG) and polymorphonuclear neutrophils and show the i

144 Total immunoglobulin G (IgG) from individuals residing in malaria-endemic reg

145 faces in the GB1 complex with immunoglobulin G (IgG).

146 Both in G protein-mediated pathways and in beta-arrestin 2 recru

147 s from PKC-(-/-) mice were also defective in G-BMDC induced Treg proliferation ex vivo, this defect c

148 062 differentially expressed genes (DEGs) in G. arboreum.

149 cted by fluorescent in situ hybridization in G. f.

150 and a 2.55% (95% CI, 0.93, 4.21) increase in G/I ratio.

151 hroughput acquisition of biomarker levels in G. fossarum exposed in four reference and 13 contaminate

152 lesions, hypohomocysteinaemia and increased G-6-P-dehydrogenase activity will facilitate early diagn

153 nsitions from inhibiting Raf-1 to inhibiting G-protein-coupled receptor kinase 2 upon phosphorylation

154 replaced with (th) G, an emissive isomorphic G surrogate, is reported.

155 xperiments with inhibitors of protein kinase G (PKG), protein kinase A (PKA), phosphodiesterase 3B (P

156 A novel peptide substrate (A G G P L G P P G P G G) was developed for quantifying the activit

157 e diversity on these chromosomes in landrace G. hirsutum.

158 al species, and also at the aggregate level (G); however, there is ambiguous evidence for the existen

159 versely showed upregulation of GO terms like G-protein coupled receptor pathway, membrane potential a

160 nd commercially important members of the low G + C Firmicutes.

161 % to 99.2%; kappa, 0.89), with the Lumipulse G TP-N having a shorter time to first and subsequent res

162 rated from the Genomes to Fields (G2F) Maize G x E project to assess the effect of selection on G x E

163 Many G protein-coupled receptors require association with oth

164 is apparent, indicating telomerase-mediated G-strand extension.

165 iferation and myelination through modulating G-protein expression and interacting with SOX10, respect

166 We confirm the inability of MojV-G to interact with known paramyxoviral receptors in vitr

167 60 binds nucleosome-free regions of multiple G box-containing genes, opposing in cis the promoting ef

168 ter Mtb infection, and inhibition of NO by N(G)-monomethyl-L-arginine enhanced intracellular survival

169 for infection of CNS neurons (SAD-G and N2C-G).

170 after CTC1 disruption and at early times net G-strand growth is apparent, indicating telomerase-media

171 t this theory may be applied to nonolfactory G protein-coupled receptors (GPCRs), including those ass

172 in (POMC) neurons leads to the activation of G-protein-coupled inward rectifier potassium (GIRK) chan

173 Preferential binding of G-quadruplex RNA is conserved, surprisingly using differ

174 e method in different structural contexts of G-quadruplexes and their complexes.

175 nd a highly cell-type-specific expression of G-protein-coupled receptors, implying that ligand-recept

176 mbers of the secretin-like class B family of G-protein-coupled receptors (GPCRs) and have opposing ph

177 plicational or co-transcriptional folding of G-quadruplex inside the polymerase machinery in cells.

178 y transduces signals from a diverse group of G protein-coupled receptors (GPCRs).

179 ; i < 5) are designed to study the impact of G-repeats on the formation of tetrameric i-motifs.

180 ontrolled by a GPCR-independent mechanism of G protein activation mediated by cytoplasmic factors.

181 cribe not only the basic structural motif of G-quadruplexes formed by, e.g., telomeric DNA sequences,

182 Signaling properties of G protein complexes carrying mutant Gbeta1 subunits were

183 R2), a classical chemoattractant receptor of G-protein-coupled receptors, is reported to be involved

184 , representing a novel mode of regulation of G protein-coupled receptor signaling by scaffolding prot

185 duration are controlled by the regulator of G protein signaling (RGS) proteins.

186 lished that in Arabidopsis, the regulator of G-protein signaling (RGS1) protein and a lipid-hydrolyzi

187 Regulator of G-protein signaling Gbeta5-R7 is a crucial activator of

188 hibitors, and most notably by stimulation of G protein-coupled receptors (GPCR).

189 cally involved in the signal transduction of G protein-coupled receptors (GPCRs) at the plasma membra

190 hese receptors co-localize with a variety of G proteins even before receptor activation, and activate

191 randomized 1:1 to receive 12 or 16 weeks of G/P (300 mg/120 mg) once daily.

192 Cs are preferentially mobilized to the PB on G-CSF treatment.

193 ligomers impart unique kinetic properties on G-protein-activated Kir3 currents.

194 project to assess the effect of selection on G x E variation and characterize polymorphisms associate

195 Three representative MPs, including one G-protein-coupled receptor, were successfully incorporat

196 only A:T base pairs than for those with only G:C base pairs.

197 C4a against a panel of both known and orphan G protein-coupled receptors and now provide evidence tha

198 or the V2R without any activity on 155 other G-protein-coupled receptors or on 15 ionic channels.

199 novel peptide substrate (A G G P L G P P G P G G) was developed for quantifying the activities of bac

200 A novel peptide substrate (A G G P L G P P G P G G) was developed for quantifying the activities of

201 units of intramuscular benzathine penicillin G (BPG) is recommended for the treatment of early syphil

202 r, few receive optimal benzathine penicillin G (BPG) therapy to prevent disease progression.

203 e, flufenamic acid, the K salt of penicillin G, and form 4 of the drug 4-[4-(2-adamantylcarbamoyl)-5-

204 s were randomized to receive 4 mg of PENNVAX-G DNA delivered intramuscularly by Biojector or electrop

205 r and unite into mirror-symmetric pentamers (G-R-B-R-G) by adhesion.

206 PG-ethanolamide (PG-EA) and PG-glycerol (PG-G), respectively.

207 To identify the endogenous receptor for PGE2-G, we performed a subtractive screening approach where m

208 tive screening approach where mRNA from PGE2-G response-positive and -negative cell lines was subject

209 The effects of PGE2-G required its hydrolysis into PGE2, were not observed w

210 Both PIV5/F and PIV5/G were also able to boost neutralization titers in RSV-p

211 natal Growth and Retinopathy of Prematurity (G-ROP) Study (a multicenter retrospective cohort study).

212 surface in an optimal orientation by protein G interaction.

213 were pre-incubated with alpha-gal or protein G to deplete IgG Ab. alpha-Gal-specific IgG1-4 Ab in ind

214 cro opioid receptor (microR), a prototypical G-protein-coupled receptor that is a physiologically rel

215 ite into mirror-symmetric pentamers (G-R-B-R-G) by adhesion.

216 OCT), and immunohistochemistry analysis of R/G opsin and rhodopsin.

217 ith untreated animals, animals that received G-CSF following radiation injury exhibited enhanced func

218 lmitoylation, prevents the enhanced receptor G-protein association and blocks acute analgesic toleran

219 nulocyte colony-stimulating factor receptor (G-CSFR) in approximately 80% of patients.

220 GABAB receptors that differentially regulate G-protein signaling of the receptor.

221 We find that PARP3 regulates G quadruplex (G4) DNA in response to DNA damage, which s

222 Members of the Mas-related G protein-coupled receptor (Mrgpr) family demarcate an i

223 idence that SP functions through Mas-related G protein-coupled receptors (Mrgprs) in addition to its

224 nosine receptor, a pharmaceutically relevant G protein-coupled receptor.

225 otential channel 1 (TRPC1) proteins requires G protein alpha q subunit (Galphaq)/phospholipase C (PLC

226 Microarray analyses revealed G-protein-coupled receptor (GPR) signaling as a prominen

227 The leucine-rich G protein-coupled receptor-5 (LGR5) is expressed in adul

228 as sensitive to triacsin C and rosiglitazone G.

229 nd the increased inhibition by rosiglitazone G confirmed the sensitivity of the bacterial acyl-ACP sy

230 ve commercial rice protein (RP) products, RP-G, RP-O, RP-RM, RP-RS1, and RP-RS2, were analyzed.

231 The minor allele of rs1057233 (G), within the previously reported CELF1 AD risk locus,

232 The rs10995 G-allele was associated with better BP response to hydro

233 two independent SNPs (i.e., WNT2B rs1175649 G>T and BTRC rs61873997 G>A) that showed a predictive ro

234 .e., WNT2B rs1175649 G>T and BTRC rs61873997 G>A) that showed a predictive role in CM-specific surviv

235 In vivo, mutations in RSV G and PIV5 L were found in individual isolates of PIV5-R

236 ere found in individual isolates of PIV5-RSV-G (HN-L), but plaque isolates of PIV5-RSV-F (HN-L) had n

237 PIV5 genome [PIV5-RSV-F (HN-L) and PIV5-RSV-G (HN-L), respectively].

238 Gervais & Fessler's (G&F's) Attitude-Scenario-Emotion (ASE) model reduces sen

239 inely used for infection of CNS neurons (SAD-G and N2C-G).

240 ical sensor exhibited excellent sensitivity (G = 178.8 nA/mug mL(-1), A = 92.9 nA/mug mL(-1), T = 1.4

241 Cdc42 is a Rho-family small G protein that has been widely studied for its role in c

242 nine single-nucleotide polymorphisms (SNPs): G-1106A, A-1018T, T-1014C, T-988G, G-513A, G-462A, T-415

243 erving differences between nonhuman species (G), the second that concerns observing individual differ

244 ly modified RNA constructs in which specific G residues are replaced with (th) G, an emissive isomorp

245 op formation near gene promoters with strong G/C skew and propensity to form G-quadruplex in non-temp

246 Chemokines and their cell surface G protein-coupled receptors are critical for cell migrat

247 and that bile salt receptors VDR and Takeda G-protein coupled receptor5 (TGR5) were highly expressed

248 Targeting G-quadruplex DNAs for cancer treatment is a very promisi

249 h specific G residues are replaced with (th) G, an emissive isomorphic G surrogate, is reported.

250 ersus 14%; log-rank P=0.14), but higher than G- HCM (22% versus 6%; log-rank P<0.001) and FG+ relativ

251 Bioinformatics studies have revealed that G-rich sequences with the potential to adopt these struc

252 Phylogenetic analyses suggested that G-LSR2 was acquired from Fusarium oxysporum f. vasinfect

253 The G allele of the TCN2 c.776G>C (rs1801198) polymorphism h

254 The G alleles of rs4293393 was associated with higher serum

255 The G-protein-coupled receptors LGR4, LGR5 and LGR6 are Wnt

256 The G-ROP Study enrolled all infants undergoing ROP examinat

257 tein pathway mediating cell survival and the G protein-coupled receptor --> Gs --> adenylate cyclase

258 subfamily RGS proteins are stabilized by the G-protein subunit Gbeta5, such that the knockout of the

259 Beyond those conditions, the G-quartet stacks dissociate laterally into monomer stack

260 Patients homozygous for the G allele responded significantly better to the selective

261 nds of MELD change over time (p=0.55 for the G-CSF group vs standard care and p=0.75 for the G-CSF pl

262 SF group vs standard care and p=0.75 for the G-CSF plus stem-cell infusion group vs standard care).

263 w pathway is distinctly parcellated from the G protein-coupled receptor --> Gs --> adenylate cyclase

264 vations and reveal allosteric changes in the G receptor binding and F-activating stalk domains, provi

265 induce targeted mechanical unfolding of the G-quadruplex while leaving the nanocage unperturbed.

266 eport a high-resolution NMR structure of the G-rich element within the KRAS NHE.

267 mly assigned to the standard care, 26 to the G-CSF group, and 28 to the G-CSF plus stem-cell infusion

268 d care, 26 to the G-CSF group, and 28 to the G-CSF plus stem-cell infusion group.

269 ision than previously accomplished using the G-band shift with charge.

270 tautp decreased roughly linearly with the G:C content of the hairpin helix, being 50% longer for h

271 hotoaffinity probe (GSTABP-G) to target the "G site", and (2) an ABP designed to mimic a substrate mo

272 We further find that this G-actin pool functions in spine development and its modi

273 lular purinergic signals that signal through G protein-coupled receptors.

274 tinct periodontal profile classes (PPCs A to G) and seven distinct tooth profile classes (TPCs A to G

275 en distinct tooth profile classes (TPCs A to G) ranging from health to severe periodontal disease sta

276 Introducing the term 'clustered damage to G-quadruplexes' we report here on the structural effects

277 Human colonic biopsy specimens exposed to G. duodenalis were depleted of mucus, and in vivo mice i

278 euron-microglia interaction that responds to G-CSF by engaging Cathepsin S-CX3CR1-inducible NOS signa

279 within the BM HSC compartment in response to G-CSF treatment.

280 lity in FG+ probands with HCM was similar to G+ HCM (22% versus 14%; log-rank P=0.14), but higher tha

281 Thus, the truncated G-CSFRs associated with SCN/AML may protect myeloid prec

282 ontaining five F-actin-binding sites and two G-actin-binding sites, and interacts with wheat (Triticu

283 sequence preference for incision between two G residues that reside in a T-rich region of DNA.

284 ately 10(-4) The A-->C, G-->A, A-->U, C-->U, G-->U, U-->C, and U-->G errors mostly due to pyrimidine-

285 Thus, how detection of U:G mismatches is translated into the single-strand nick r

286 al photoreceptor rhodopsin (Rho) is a unique G protein-coupled receptor as it utilizes a covalently t

287 ld of the kinase is embellished by a unique 'G-loop' element that accounts for guanine nucleotide spe

288 B (CTSB), which can be induced directly via G protein-coupled receptors on acinar cells or through i

289 The Tre1-NRY domain via G protein signaling is required for reading and respondi

290 a membrane-bound respiratory syncytial virus G rapidly recycles from the membrane via clathrin-mediat

291 firmness and both elastic (G') and viscous (G'') moduli.

292 lar signal-regulated kinase 1/2 (ERK1/2) was G protein-, but not beta-arrestin-, dependent.

293 All but one base substitutions were G:C to A:T, their distribution between coding and non-co

294 ic demonstrated that a recombinant VSV where G protein is replaced with EBOV GP (rVSV-EBOV) is safe a

295 s are elevated in spines upon activity, with G-actin immobilized by the local enrichment of phosphati

296 obacillus women, negatively correlating with G. vaginalis and other anaerobic bacteria, which deplete

297 ted of mucus, and in vivo mice infected with G. duodenalis had a thinner mucous layer and demonstrate

298 serine/threonine phosphatases integrate with G protein signaling pathways is less understood.

299 als to the cell interior by interacting with G proteins.

300 esses occurring at specific locations within G-quadruplex nucleic acids, providing valuable probes fo