ライフサイエンスコーパス: GABA agonist

1 otentiates the memory-impairing effects of a GABA agonist.

2 eeded to impart full efficacy to the partial GABA agonists.

3 cell patch clamp recordings of responses to GABA agonists.

4 by GABA without altering the binding of the GABA agonist [3H]muscimol, indicating that this residue

5 hem with rates determined in the presence of GABA (agonist), 4-[6-imino-3-(4-methoxyphenyl)pyridazin-

6 ed both low- and high-affinity components of GABA agonist activity in alpha6betaY157Fdelta receptors,

7 Because the application of GABA agonists also decreases neuronal activity, we hypot

8 Although GABA agonists and antagonists have been shown to modulat

9 sought to test the hypothesis that selective GABA agonists as well as cell transplant-derived GABA ar

10 we examined effects of THP (100 nM) and full GABA agonists at alpha4beta2delta (gaboxadol, 10 muM, an

11 lective inactivation of OFC neurons with the GABA agonists baclofen + muscimol decreased cue-induced

12 of Pf inactivation, through infusion of the GABA agonists baclofen and muscimol, on place acquisitio

13 In contrast, GABA agonists did not escalate aggressive behaviors afte

14 e determined whether the administration of a GABA agonist, eszopiclone, was capable of preventing the

15 diated transmembrane currents in response to GABA agonist exposure.

16 ntagonist-opioid agonist and GABA antagonist-GABA agonist feeding interactions have been identified b

17 ynaptic receptors are transiently exposed to GABA agonists for several minutes.

18 mentation of tonic inhibitory tone using the GABA agonist gaboxadol (THIP).

19 anifested as decreased responsiveness to the GABA agonist gaboxadol at concentrations that are select

20 Muscimol, a GABA agonist, inactivated part of the OMV, whereas bicuc

21 ithin the VTA or NACs differentially affects GABA agonist-induced feeding elicited from the same site

22 al SCH23390 administration into the OFC plus GABA agonist-induced neural inactivation of the contrala

23 vity, we hypothesized that administration of GABA agonists into the AcbSh would stimulate feeding in

24 t, simultaneous unilateral microinjection of GABA agonists into the dPFC in one hemisphere and into t

25 In addition, the injection of muscimol, a GABA agonist, into motor cortex resulted in an increase

26 superior colliculus by injecting muscimol, a GABA agonist, into the substantia nigra pars reticulata

27 ions occur for opioid receptor modulation of GABA agonist-mediated feeding, the present study examine

28 eceptor specificity governing the ability of GABA agonist mediation of food intake, the present study

29 , nonaffective verb-generation task, and the GABA agonist midazolam (which increases neural inhibitio

30 Microinjection of the GABA agonist muscimol (250 pmol) into the caudal ventrol

31 s temporarily inactivated by infusion of the GABA agonist muscimol before acute stressor exposure.

32 s, the mPFC was bilaterally inactivated with GABA agonist muscimol before the stressor.

33 he effects of PL-IL or OFC infusion with the GABA agonist muscimol in the context of 2 flexible respo

34 The GABA agonist muscimol induced a pronounced feeding respo

35 re of slices to H(2)O(2), the ability of the GABA agonist muscimol to increase [Cl(-)]i was attenuate

36 ibly inactivated with microinjections of the GABA agonist muscimol.

37 ivated the stimulated motor cortex using the GABA agonist muscimol.

38 teral but not dorsomedial striatum using the GABA agonists muscimol and baclofen decreased context-in

39 of glucose with the gamma-aminobutyric acid (GABA) agonist muscimol into the medial septum on memory

40 We show that application of the GABA agonist, muscimol (10-200 microM), produces time- a

41 mbens shell (AcbSh) with local injections of GABA agonists or glutamate antagonists elicits an intens

42 e initially thought to affect the binding of GABA agonists, recent studies suggest an effect on recep

43 esearch is clearly needed, existing approved GABA agonists should be considered in the management of

44 ing inhibition of selected brain nuclei with GABA agonists show that the suppression of cocaine seeki

45 alpha4Fbeta2delta and treated 24h later with GABA agonists, THP, GABA plus THP or vehicle (0.01% DMSO

46 ve and respond with full efficacy to partial GABA agonists, to generate distinct ensembles of recepto

47 The effect of LHb injection of GABA agonists was mimicked by intra-LHb muscarinic choli

48 Muscimol, a GABA agonist, was used to inactivate the prelimbic and i

49 these acid salts, responses more typical of GABA agonists were seen.

50 ltiple administration of sodium valproate, a GABA agonist, would prevent the expression of sensitizat