ライフサイエンスコーパス: GABA cell

1 nsgenic mouse model that specifically labels GABA cells.

2 ated and regulated by neighboring inhibitory GABA cells.

3 ned excitatory plasticity in fluorescent VTA GABA cells.

4 th immunocytochemistry to distinguish DA and GABA cells.

5 y is far greater in excitatory cells than in GABA cells.

6 th GluR3 and GluR6 subunits not expressed by GABA cells.

7 influences on the serotonin-accumulating and GABA cells.

8 cally innervating aversion-encoding midbrain GABA cells.

9 adual transition of RG cells into CalR(+) or GABA(+) cells.

10 We show that GABA cell activation only promotes cataplexy attacks ass

11 lied Delta(9)-tetrahydrocannabinol depressed GABA cell activity, therefore downstream dopamine cells

12 alter behavior through regulating VTA DA and GABA cell activity.

13 tribute to the activity of dopamine (DA) and GABA cells and, hence, to the affective and cognitive fu

14 s a novel form of synaptic plasticity in VTA GABA cells, and the synaptic remodeling that can occur a

15 Ventral thalamus GABA cells are derived from a region connecting the vent

16 physiological studies indicate that cortical GABA cells are modulated by a variety of afferents.

17 not been possible to record selectively from GABA cells, because they have no defining morphological

18 tory influence on both mesoprefrontal DA and GABA cells but a divergent impact on mesoaccumbens neuro

19 d) cells were also found but no new Cr(+) or GABA(+) cells colabeled with a mature neuron marker, Neu

20 the trisynaptic pathway, so that subtypes of GABA cells could be defined by their location in various

21 we found that excitatory synapses on DA and GABA cells display several differences.

22 GABA cell dysfunction in both schizophrenia (SZ) and bip

23 In contrast, synapses on GABA cells exhibit a facilitation in response to repetit

24 yramidal cells and gamma-amino butyric acid (GABA) cells; GluR2/3 immunoreactivity is preferentially

25 mouse line that enables genetic targeting of GABA cells in orexin(-/-) mice.

26 y excitatory synapses that innervated DA and GABA cells in rough proportion to their representation w

27 s the first functional evidence to implicate GABA cells in the amygdala as regulators of cataplexy tr

28 stimuli may trigger cataplexy by activating GABA cells in the CeA.SIGNIFICANCE STATEMENT Although ca

29 bers from the MD and different subclasses of GABA cells in the PFC are not known.

30 n which excitatory synaptic inputs to DA and GABA cells in the VTA can be modulated have potentially

31 Previous studies demonstrated that the GABA cells intrinsic to the VTA receive insufficient syn

32 R4 immunoreactivity is largely restricted to GABA cells; NMDA receptor subunit immunoreactivity is fa

33 Intrinsic GABA cells of dorsal thalamus may, therefore, derive fro

34 w hypotheses regarding the regulation of the GABA cell phenotype in the hippocampus of SZ and BD.

35 ut affecting their duration, suggesting that GABA cells play a functional role in initiating but not

36 GABA+ cells represented a mean of 16.2% (14.8-17.2%) of

37 askets target the somas of certain GABA+ and GABA- cells, resembling cortical axosomatic synapses.

38 To manipulate GABA cells specifically, we developed a new mouse line t

39 A novel CB1-dependent LTD was induced in GABA cells that was dependent on metabotropic glutamate

40 some population of gamma-aminobutyric acid (GABA) cells that, in turn, innervates DA neurons.

41 tion and/or genomic integrity of hippocampal GABA cells varies according to diagnosis and their locat

42 The serotonin-accumulating and GABA cells were affected as they were drastically reduce

43 logical type, whereas VAChT(+) synapses onto GABA cells were more frequently symmetric (presumed inhi

44 CalR(+) and GABA(+) cells were apparent for the first time after 3 d

45 After 24 h, no CalR(+) or GABA(+) cells were seen in cultures, whereas 5-10% cells

46 um (ganglionic eminence) generate CalR(+) or GABA(+) cells, whereas this was not the case with RG cel

47 rs and altered integration among hippocampal GABA cells with extrinsic and intrinsic afferent fiber s

48 Then, we show that GABA cells within the CeA are responsible for mediating

49 ity of both dopaminergic (DA) and GABAergic (GABA) cells, yet little is known about the basic propert