ライフサイエンスコーパス: GABA receptor

1 e action of inhibitory steroids on the rho 1 GABA receptor.

2 mily that depend on cholesterol, such as the GABA receptor.

3 re noncompetitive antagonists (NCAs) for the GABA receptor.

4 nit, can impart picrotoxin resistance to the GABA receptor.

5 ibitory GABA receptor and a novel excitatory GABA receptor.

6 s-talk between these two distinct classes of GABA receptor.

7 tions between these two different classes of GABA receptor.

8 (i.e. fiprole), high-affinity probe for the GABA receptor.

9 ytes and HEK-293 cells to form a heteromeric GABA receptor.

10 might coassemble to form a heteromultimeric GABA receptor.

11 e genes such as acetylcholinesterase and the GABA receptor.

12 oth small and large cholangiocytes expressed GABA receptors.

13 response to voltage pulses in cells lacking GABA receptors.

14 e VMH can be reversed by local modulation of GABA receptors.

15 impairing the inhibitory action of neuronal GABA receptors.

16 otoxin and thus not mediated by conventional GABA receptors.

17 or rho subunit-containing GABA(C) over other GABA receptors.

18 gy of native, bicuculline-insensitive insect GABA receptors.

19 ases of resistance to insecticides acting on GABA receptors.

20 tor (CB1R) but not metabotropic glutamate or GABA receptors.

21 receptors, and bicuculline, an antagonist of GABA receptors.

22 screen, the proteins of which interact with GABA receptors.

23 in and hot plate tests that are dependent on GABA receptors.

24 and by blockers of ionotropic glutamate and GABA receptors.

25 xin-sensitive nAChRs or picrotoxin-sensitive GABA receptors.

26 by picrotoxin-sensitive chloride conducting GABA receptors.

27 tes expressing recombinant alpha1beta2gamma2 GABA receptors.

28 ion of previously uncharacterized ionotropic GABA receptors.

29 inals where they are apposed by postsynaptic GABA receptors.

30 picrotoxin, indicating spontaneously opening GABA receptors.

31 M GABA-induced currents in alpha1beta2gamma2 GABA receptors.

32 e during simultaneous activation of NMDA and GABA receptors.

33 gher concentrations, the steroid also blocks GABA receptors.

34 ho1 M3-M4 intracellular loop along with rho1 GABA receptors.

35 eliminated by antagonists of either AMPA or GABA receptors.

36 t-term plasticity and the configuration with GABA receptors.

37 e propensity of caged GABAs to interact with GABA receptors.

38 ration by LS/MS and its bioactivity on human GABA receptors.

39 drin), gene encoding a subunit of inhibitory GABA receptors.

40 due to clathrin-dependent internalization of GABA receptors.

41 ty of granule cells by acting on presynaptic GABA receptors.

42 release is necessary for maintaining axonal GABA receptors.

43 rophysiological activity of the transplanted GABA receptors.

44 ticide targeting on gamma-aminobytyric acid (GABA) receptors.

45 nists of ionotropic gamma-aminobutyric acid (GABA) receptors.

46 Aergic neurons or disruption of metabotropic GABA receptor 1 and 2 (GABA(B)R1/2) signaling in astrocy

47 feedback pathway is mediated by metabotropic GABA receptors.(1,2,5,6-Tetrahydropyridine-4-yl)-methylp

48 t microinjection of gamma-aminobutyric acid (GABA) receptor A antagonist substance, bicuculline, into

49 identified M1 residues to inhibit the UNC-49 GABA receptor, a homomeric GABA receptor from Caenorhabd

50 ucing intrastriatal gamma-aminobutyric acid (GABA) receptor-A inhibition synchronizes striatal dynami

51 We saw no evidence for changes in GABA receptor abundance or the overall number of glutama

52 Potentiation of GABA receptor activation through an allosteric benzodiaz

53 oversy surrounding the role of Loop F during GABA receptor activation.

54 ation with propofol and other compounds with GABA receptor activity are frequently used in patients w

55 ic synaptic transmission but does not affect GABA receptor activity in cultured neurons.

56 ntrast, gaboxadol, a selective extrasynaptic GABA receptor agonist and late-stage investigational tre

57 Bilateral microinjection of the GABA receptor agonist muscimol centered in the basolater

58 nal nicotinic antagonist mecamylamine or the GABA receptor agonist muscimol, agents that reduce activ

59 isting of approximately 150-200 muscimols (a GABA receptor agonist) covalently joined to the qdot via

60 Baclofen, a metabotropic GABA receptor agonist, both enhanced and suppressed high

61 duration of sleep when the animals receive a GABA receptor agonist.

62 mol (2.5 microM), a gamma-aminobutyric acid (GABA) receptor agonist.

63 thalamus reproduce limbic cortex injury, and GABA-receptor agonist injections into anterior thalamus

64 b) reversible inactivation of the dlCPu with GABA receptor agonists (baclofen and muscimol) immediate

65 To test this hypothesis, GABA receptor agonists (baclofen/muscimol) were microinj

66 ext, we reversibly inactivated the vSub with GABA receptor agonists (muscimol+baclofen) before the co

67 In contrast to GABA receptor agonists which induce sleep by generalized

68 gents that influenced (lidocaine) or spared (GABA receptor agonists) fibers of passage blocked cocain

69 rescued by suppressing GABA reuptake and by GABA receptor agonists.

70 ex in adult rodents that can be prevented by GABA-receptor agonists and antipsychotics such as halope

71 receptors did not overlap the effect of the GABA receptor- allosteric modulator, diazepam.

72 Activation of ionotropic glutamate and/or GABA receptors along the GnRH neuron projection is capab

73 acetylcholine, serotonin Type 3, glycine and GABA receptors) along with the crystal structure of the

74 Candidate gene GABRA2, which encodes the GABA receptor alpha2 subunit, was evaluated independentl

75 s GABA neuron identity, a classic inhibitory GABA receptor and a novel excitatory GABA receptor.

76 acting as allosteric inhibitors of the rho 1 GABA receptor and support the hypothesis that divergent

77 n-presenting cells (APCs) express functional GABA receptors and respond electrophysiologically to GAB

78 Combination of the blockade of mNTS GABA receptors and spinal iGLURs also abolished PVN-indu

79 Also, transcription of mRNA for both GABA receptors and the GABA reuptake transporter was aff

80 underlying mechanisms, including the role of GABA receptors and their link to synaptic adhesion molec

81 tion, suggesting potential links between the GABA receptors and these conditions.

82 ors, transporters, and interacting proteins, GABA receptors and transporters, synaptic-related marker

83 did not lead to the expression of functional GABA receptors, and injected oocytes failed to generate

84 can be attributed to their effects at insect GABA receptors, and the presence of a Val at the 2' posi

85 y to VIP(-/-) SCN cultures during continuous GABA receptor antagonism but not during G(i/o) blockade.

86 In contrast to the relative inability of GABA receptor antagonism in both sites to alter 2-deoxy-

87 A site-specific effect of GABA receptor antagonism was observed for deprivation-in

88 Under GABA receptor antagonism, however, mean plateau activity

89 e further analyzed the response of NS to the GABA receptor antagonist bicuculline.

90 OB interneurons or locally introducing DA or GABA receptor antagonists alters kinship preference.

91 uring simultaneous microiontophoresis of the GABA receptor antagonists bicuculline and phaclofen.

92 ppressed transient output from bipolar cells.GABA receptor antagonists blocked the effect of ATPA.

93 Conversely, superfusion with GABA receptor antagonists decreased the threshold for ev

94 diated inhibitory synaptic transmission, and GABA receptor antagonists did not block leptin-mediated

95 Glutamate and GABA receptor antagonists did not block this effect.

96 nses and reduces the disinhibitory effect of GABA receptor antagonists on PNs.

97 In addition, focal application of GABA receptor antagonists to sensory ganglia triggered o

98 e presence of either gabazine or picrotoxin (GABA receptor antagonists), many action potentials appea

99 g persisted in the presence of glutamate and GABA receptor antagonists.

100 in the presence of ionotropic glutamate and GABA receptor antagonists.

101 nce of muscarinic, ionotropic glutamate, and GABA receptors antagonists were also reduced by NRG 1bet

102 GABA receptor are involved in a number of complex disord

103 Ionotropic GABA receptors are abundant in both vertebrate and inver

104 ficance and mechanism(s) of the UV effect on GABA receptors are discussed.

105 M1) and pre-M1 of alpha and beta subunits in GABA receptors are essential for positive modulation of

106 nts, indicating that a subset of presynaptic GABA receptors are tonically active.

107 Ionotropic GABA receptors are widely distributed throughout the ver

108 ctrophysiological properties of human native GABA receptors as a consequence of AD.

109 signaling by using a mimetic peptide of the GABA receptor associated protein-interacting domain of G

110 proteins N-ethylmaleimide-sensitive factor, GABA receptor-associated protein (GABARAP), and glutamat

111 ric binder of GABA receptors, GABARAPL1, the GABA receptor-associated protein, and SLC6A11, a postsyn

112 Lower levels of gamma-aminobutyric acid (GABA) receptor-associated protein (GABARAP) gene express

113 , widely diverse NCA structures fit the same GABA receptor beta subunit site with important implicati

114 procedures yield a good model of the insect GABA receptor binding site and the location of agonists

115 ns of 3 alpha 5 beta PC, we characterize the GABA receptor block in some detail.

116 tiation and NMDA receptor block and diminish GABA receptor block may lead to a clinically useful neur

117 ated the increase in 5-HT efflux produced by GABA receptor blockade in the DRN.

118 VLM can be activated in the presence of RVLM GABA receptor blockade, but sympathoinhibitory influence

119 ke propagation becomes stereotyped following GABA receptor blockade.

120 Depression was prevented by MOR- but not GABA-receptor blockade.

121 rainstem slice with ionotropic glutamate and GABA receptors blocked, whole-cell patch-clamp recording

122 Here we show that the application of the GABA receptor blocker picrotoxin unmasks a robust excita

123 uropil or of interneurons in the presence of GABA receptor blockers caused no alteration in granule c

124 Indeed, we found that application of GABA receptor blockers increased the sensitivity of most

125 In the presence of GABA receptor blockers, residual taurine currents averag

126 und effects on the EPSCs were insensitive to GABA receptor blockers.

127 dback is mediated by at least three types of GABA receptor, both metabotropic and ionotropic.In conju

128 PMA specifically caused internalization of GABA receptors, but not neuronal acetylcholine receptors

129 we investigate the inhibition of the UNC-49 GABA receptor by another sulfated neurosteroid, dehydroe

130 sponse augmentation and the direct gating of GABA receptors by 5alpha-reduced potentiating steroids.

131 bunit is capable of forming fully functional GABA receptors by itself in Xenopus oocytes and suggest

132 e, we find that co-activation of presynaptic GABA receptors by photolytic uncaging of RuBi-GABA has a

133 Ionotropic GABA receptors can mediate presynaptic and postsynaptic

134 Finally, blocking NMDA, but not GABA, receptors causes ATM levels to rise while ATR leve

135 Commonly used antagonists of metabotropic GABA receptors, CGP35348 and CGP55845, were more potent

136 GABA receptor/channel rho 1 subunit expression in the rh

137 We studied the expression localization of GABA receptor/channel rho 1 subunit in mouse spinal cord

138 To understand the functions of the GABA receptor/channel rho 1 subunit in these crucial sit

139 f sensory transmission in vivo, we generated GABA receptor/channel rho 1 subunit mutant mice (rho 1-/

140 These findings indicate that the GABA receptor/channel rho 1 subunit plays an important r

141 immunohistochemistry results indicated that GABA receptor/channel rho 1 subunits were expressed in m

142 gamma-Aminobutyric acid (GABA) receptor/channel rho 1 subunits are important comp

143 he discrete insertion and removal of AMPA or GABA receptor channels.

144 Therefore, the functional role of GABA receptor/channels in the brain, retina, and spinal

145 etaDB double mutants, the size and number of GABA receptor clusters are decreased at cerebellar inhib

146 Ionotropic gamma-amino butyric acid (GABA) receptors composed of heterogeneous molecular subu

147 Insect GABA receptors contain Ala at their 2' position in the p

148 Since the functional pentameric GABA receptor contains two alpha subunits, two beta subu

149 By recording endogenous GABA receptor currents directly from BC terminals in gol

150 Whole-cell GABA receptor currents were moderately sensitive to GABA

151 increased the rate and degree of macroscopic GABA receptor desensitization during prolonged GABA appl

152 eceptor-like family member GrlB as the major GABA receptor during early development, and either disru

153 GrlE but not GrlB is the GABA receptor during late development.

154 e beta8-sheet of the adjacent subunit in the GABA receptor (E175-K46 being the structurally equivalen

155 The changes in GABA receptor efficacy after UL are therefore not due to

156 The downregulation of GABA receptor efficacy in the ipsi-lesional MVN neurones

157 0-F227) in the N-terminal domain of the rho1 GABA receptor expressed in Xenopus oocytes using a site-

158 Thus, the GABA receptors expressed by the yeast mRNA retained all

159 Homomeric rho1 GABA receptors expressed in oocytes had a single channel

160 e in the activation of rat alpha1beta2gamma2 GABA receptors expressed in Xenopus laevis oocytes by si

161 wild-type (WT) alpha6beta3gamma2L or WT RDL GABA receptors expressed in Xenopus laevis oocytes were

162 nts underlying the ionic selectivity of rho1 GABA receptors expressed in Xenopus oocytes and human em

163 that proton alters the apparent affinity of GABA receptors for agonist.

164 activated current amplitude from recombinant GABA receptors (formed by rho1 or alphabetagamma subunit

165 nhibit the UNC-49 GABA receptor, a homomeric GABA receptor from Caenorhabditis elegans that is homolo

166 Furthermore, GABA receptors from AD brains were slightly, but signifi

167 onto SACs - removing either GABA release or GABA receptors from SACs.

168 We measured presynaptic GABA receptor function at parallel fibre synapses onto s

169 s and receptors [glutamate, aspartate, GABA, GABA receptor (GABA-R), NMDA-R, AMPA-R, and kainate-R] a

170 r loop (ICL) of the gamma2 subunit of type A GABA receptors (GABA(A)R), which is required to anchor G

171 A-type and rho-subunit-containing (C-type) GABA receptors (GABA(A)Rs and GABA(C)Rs) mediate both fo

172 xpression of type A gamma-aminobutyric acid (GABA) receptor (GABA(A)R) subunit genes plays a critical

173 o acid GABA via activation of two ionotropic GABA receptors, GABA(A) and GABA(C).

174 ynaptic levels after 48 h blockade of type A GABA receptor (GABAA R)-mediated inhibition with bicucul

175 GABA receptors (GABAARs) mediate inhibition in the adult

176 The GABA receptor (GABABR) is a class C G protein-coupled re

177 iched synapses, virtually devoid of the main GABA receptor (GABAR) subunits alpha1 and gamma2.

178 GABA receptor (GABAR) types C (GABACR) and A (GABAAR) ar

179 schizophrenia often involve potentiation of GABA receptors (GABAR) to augment antipsychotic therapy

180 These include DBI, an allosteric binder of GABA receptors, GABARAPL1, the GABA receptor-associated

181 link between PMCA2 and glutamate receptors, GABA receptors (GABARs), and glutamate transporters that

182 ively spliced in TS compared to HC including GABA receptors GABRA4 and GABRG1, the nicotinic ACh rece

183 of the approximately 1.2-Mb region from the GABA receptor gene cluster to the OCA2 locus was generat

184 esponse was abolished in animals lacking the GABA receptor gene unc-49.

185 There is a strong history of mutations in GABA receptor genes being involved in neurologic disease

186 ric acid (GABA), in this putative imbalance: GABA receptor genes have been associated with autism in

187 of variants and mutations have been found in GABA receptor genes in patients with autism, schizophren

188 No variants of the commonly studied GABA receptor genes that have been associated with subst

189 thways strongly dominated in HIVE, including GABA receptors, glutamate signaling, synaptic potentiati

190 BA(A) receptors, study of steroid actions at GABA receptors has been hampered by a lack of pharmacolo

191 of recombinant rho1 gamma-aminobutyric acid (GABA) receptors has previously identified five residues

192 Therefore, alpha4/6beta3delta GABA receptors have two distinct alcohol modulation site

193 Here, we describe a novel ionotropic GABA receptor in mouse cerebellar Purkinje cells (PCs) u

194 Here, we identify a GABA receptor in the nematode Caenorhabditis elegans tha

195 ping with the distinct expression pattern of GABA receptors in presynaptic cells, we detected no GABA

196 Activation of GABA receptors in rRPa does not mediate the cholinergic

197 he increase in BAT SNA evoked by blockade of GABA receptors in rRPa.

198 ChR in rRPa does not depend on activation of GABA receptors in rRPa.

199 The role of GABA receptors in synaptic transmission to neonatal rat

200 Little is known about the properties of GABA receptors in the amphibian brain.

201 nt NRHypo neurotoxicity mainly by activating GABA receptors in the anterior thalamus, diagonal band o

202 er, these results support the involvement of GABA receptors in the APC in feeding in general and the

203 Blockade of GABA receptors in the medial nucleus tractus solitarius

204 ion of agonists and antagonists, we compared GABA receptors in the medial vestibular nucleus of brain

205 ive regulation of the functional efficacy of GABA receptors in the MVN neurones may be an important c

206 ast inhibitory neurotransmission mediated by GABA receptors in the nervous system.

207 downregulation of the functional efficacy of GABA receptors in the rostral MVN cells did not occur.

208 rmed yeast strains produced fully functional GABA receptors in Xenopus oocytes.

209 kade of ionotropric gamma-aminobutyric acid (GABA) receptors in the LH elicits eating in satiated rat

210 ied in principal cells, but INs also express GABA receptors, in particular the GABAA type (GABAARs),

211 extrinsic inputs, but selective blockade of GABA receptors indicates that local inhibition is requir

212 by in vitro phosphorylation, did not prevent GABA receptor internalization, nor did coexpression of t

213 nfirm that desensitization reversibly shifts GABA receptors into a high-affinity state.

214 The rho 1 GABA receptor is inhibited by a number of neuroactive st

215 One of the most widely studied insect GABA receptors is constructed from RDL (resistance to di

216 The rho1 subunit of the ionotropic GABA receptors is thought to contribute to the formation

217 domain (TM2) of the gamma-aminobutyric acid (GABA) receptor lines the integral ion pore.

218 These data suggest that in the PVN GABA receptors may be important regulators of cardiopulm

219 terations in spinal gamma-aminobutyric acid (GABA) receptors may contribute to persistent pain states

220 We directly recorded dendritic GABA receptor-mediated inhibitory synaptic events in adu

221 abapentin increased the amplitudes of evoked GABA receptor-mediated IPSCs (GABA-IPSCs) in CeA neurons

222 GABA receptor-mediated IPSCs were recorded with whole-ce

223 tence of time-locked, glutamate receptor and GABA receptor-mediated mono synaptic responses evoked by

224 Also, we observed a decrease in NMDA and GABA receptor-mediated synaptic transmission in the pyra

225 pocampus, without similarly changing NMDA or GABA receptor-mediated synaptic transmission, and withou

226 psychoses, as well as a marked reduction in GABA-receptor-mediated currents in pyramidal neurons of

227 By contrast, the GABA receptor needed for locomotion in Caenorhabditis el

228 the effects were predominantly directly upon GABA receptors of the neurons being recorded from.

229 he relative expression of the two classes of GABA receptor on each bipolar cell type correlates with

230 Because we could not localize ionotropic GABA receptors on cone axon terminals using electron mic

231 The absence of ionotropic glutamate or GABA receptors on DA terminals indicates that modulatory

232 r, evidence of positive immunoreactivity for GABA receptors on the neurite, as well as evidence for g

233 pic GABA receptor pathways, the metabotropic GABA receptor pathways act to enhance bipolar cell trans

234 n, axon guidance, and signaling of Notch and GABA receptor pathways, as well as factors important for

235 However, unlike the ionotropic GABA receptor pathways, the metabotropic GABA receptor p

236 lend new insights into the structure of the GABA receptor pore.

237 ferent compositions of insect and vertebrate GABA receptor pores are responsible for the differing to

238 We further tested if spinal blockade of GABA receptors prevents the antihyperalgesia produced by

239 Prior studies have shown that the GABA receptor RDL inhibits aversive olfactory learning v

240 ified a single point mutation (A302S) in the GABA receptor RDL that has been identified previously in

241 atures during adolescence, with frontal lobe GABA receptors reaching adult levels late in adolescence

242 TP in mitral cells by enhancing postsynaptic GABA receptor responses.

243 Activation of presynaptic GABA receptors results in a transient ( approximately 10

244 membrane potential ( approximately 8 mV) to GABA receptor reversal potential ( approximately -81 mV)

245 CRF increased postsynaptic GABA receptor sensitivity selectively in 5-HT neurons, a

246 ely modulate GAT expression via metabotropic GABA receptor signaling and highlight the importance of

247 r photoactivation depended on glutamate- and GABA-receptor signaling, and not on dopamine-receptor si

248 in the spinal cord dorsal horn and activates GABA receptors spinally.

249 ptor subtype antagonist-induced mediation of GABA receptor subtype agonist-induced feeding elicited f

250 nt with previously demonstrated differential GABA receptor subtype antagonist-induced mediation of op

251 The distribution of gamma-aminobutyric acid (GABA) receptor subtype B1 (GABA(B1)) mRNA-containing cel

252 ions of agonists or antagonists to the major GABA receptor subtypes.

253 ion cells, all of which are known to express GABA receptor subtypes.

254 d the role of RVLM gamma-amino butyric acid (GABA) receptor subtypes and glycine receptors in mediati

255 beta1 subunits (SCN1A, SCN2A, SCN1B) and one GABA receptor subunit gene (GABRG2) have been identified

256 nd showed similar effects of sex steroids on GABA receptor subunit gene expression in the AMD and HPC

257 GABA receptor subunit gene expression was generally high

258 These results support the hypothesis that GABA receptor subunit genes are involved in autism, most

259 Fourteen known autosomal GABA receptor subunit genes were studied to look for the

260 ng, adds to the functional diversity of this GABA receptor subunit.

261 mmunopositive for a gamma-aminobutyric acid (GABA) receptor subunit (GABAA Ralpha1 ), and that a syna

262 ning the associated gamma-aminobutyric acid (GABA) receptor subunit gene, Resistance to dieldrin.

263 data demonstrate differential regulation of GABA receptor subunits and GABAergic system components i

264 In addition to expected components including GABA receptor subunits and gephyrin, several novel prote

265 transmission in cortical layer 4, including GABA receptor subunits and KCC2, and thus prevents the s

266 d copy number variation studies [3-7], fewer GABA receptor subunits have been observed in the post-mo

267 The EXP-1 protein resembles ionotropic GABA receptor subunits in almost all domains.

268 are the expression profiles of glutamate and GABA receptor subunits in three immortalized GnRH cell l

269 uctions of mRNA and protein of the principal GABA receptor subunits normally present in the temporal

270 AD groups and analyses of the proportion of GABA receptor subunits revealed down-regulation of alpha

271 s in gene expression levels of glutamate and GABA receptor subunits were compared between sedentary a

272 e evaluated the expression of genes encoding GABA receptor subunits, glutamic acid decarboxylase (GAD

273 udies of alpha and beta subunits of type "A" GABA receptors suggest that these linkers couple extrace

274 Thus, by regulating GABA receptor surface expression, PKC may play a key rol

275 dings reveal a novel "crosstalk" between the GABA receptor systems, which can be recruited under cond

276 sed in Xenopus laevis oocytes, EXP-1 forms a GABA receptor that is permeable to cations and not anion

277 , prevent the down-regulation of efficacy of GABA receptors that also occurs in these neurons after U

278 dulate postsynaptic excitability at NMDA and GABA receptors, the findings establish zinc as a cotrans

279 In addition to the synaptic GABA receptors, there is a group of GABAA receptors (GAB

280 non-NMDA receptor transmission and depressed GABA receptor transmission in the wild-type mice, both e

281 holine receptors, two glycine receptors, one GABA receptor, two AMPA-type glutamate receptors and one

282 The GABA receptor type C (GABA(C)) is a ligand-gated ion cha

283 We test this hypothesis in the GABA receptor using simultaneous electrophysiology and s

284 s and antagonists of mammalian glutamate and GABA receptors, using a specially developed 96-well micr

285 The wild-type GABA receptor was chloride selective, with a small but s

286 gative purinergic modulation of postsynaptic GABA receptors was accompanied by small presynaptic enha

287 n training, the sensitivity of glutamate and GABA receptors was unchanged.

288 ion pore and selectivity filter of the rho1 GABA receptor, we used the substituted cysteine accessib

289 Following evaluation at clinically relevant GABA receptors, we have identified a difluoromethyl keto

290 s laevis oocytes expressing recombinant rho1 GABA receptors, we identified agonist-mediated molecular

291 s action of the antagonist was occluded when GABA receptors were blocked, indicating that the reducti

292 c acetylcholine (nACh) and beta2alpha1gamma2 GABA receptors were constructed based on the torpedo neu

293 ergic currents and glia-driven modulation of GABA receptors were significantly reduced in the P2X4 KO

294 ed currents in individual oocytes expressing GABA receptors were tested by two-electrode voltage clam

295 e into Xenopus oocytes, expressed functional GABA receptors whose properties were investigated by usi

296 Thus, we describe a population of ionotropic GABA receptors with a mixed GABA(A)/GABA(C) pharmacology

297 Inhibiting GABA receptors with bicuculline increased NMDA receptor-

298 e may be two baclofen-sensitive metabotropic GABA receptors with opposing effects on calcium channel

299 Antagonism of gamma-aminobutyric acid (GABA) receptors with bicuculline (BIC) phenocopied the d

300 GABA receptors within the mesolimbic circuitry have been