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1 on of pannier, a cardiogenic gene encoding a Gata factor.
2 X17CNXC zinc fingers typical of a vertebrate GATA factor.
3 ) and the C-terminal zinc finger of the same GATA factor.
4 ied by the activity of the divergent MED-1,2 GATA factors.
5 tors, the hypoxia-regulated factor HIF1, and GATA factors.
6 ells can be manipulated by the expression of GATA factors.
7 x5/Dlx6 function, it may require one or more GATA factors.
8 binding to DNA and physical association with GATA factors.
9  regulators, Mix-like paired-homeodomain and GATA factors.
10 evelopment through physical interaction with GATA factors.
11 transcriptional activation mediated by other GATA factors.
12 at found with the vertebrate two zinc finger GATA factors.
13 e specification of hematopoietic mesoderm by GATA factors.
14  at the seventh position of the Zn finger of GATA factors.
15 with the differentiation-promoting action of GATA factors.
16 fect is mediated in part by interaction with GATA factors.
17       Here we report the identification of a GATA factor (AaGATAa) that is synthesized after a blood
18                                   However, a GATA factor (AaGATAr) likely acts as a repressor, preven
19                                      A novel GATA factor (AaGATAr) that recognizes GATA binding motif
20  previously shown that the MED-1,2 divergent GATA factors act apparently zygotically to specify the f
21                   Our findings indicate that GATA factors act sequentially to regulate lineage determ
22  genetic and molecular analysis that the two GATA factors act upstream from the flowering time regula
23                                              GATA factors activate expression of nitrogen catabolic p
24       Friend of GATA (FOG) proteins regulate GATA factor-activated gene transcription.
25 e show how integration of Bmp4 signaling and Gata factor activity controls the progression of hematop
26  gene expression through their modulation of GATA factor activity.
27                                              GATA factors also contribute to heart formation indirect
28 demonstrate that PBP interacts with all five GATA factors analyzed, GATA-1, GATA-2, GATA-3, GATA-4, a
29 x between the DNA binding domain of a fungal GATA factor and a 13 base-pair oligonucleotide containin
30       This work establishes a link between a GATA factor and inflammatory genes, mechanistic insights
31     Several lines of evidence suggested that GATA factors and AR act cooperatively to activate Pp tra
32 ctors share highly specific association with GATA factors and are substantially interchangeable with
33 n of mouse embryonic stem cells deficient of GATA factors and conclude that GATA-4 is required for ES
34  in tumor cells, suggesting that the loss of GATA factors and dedifferentiation are irreversible proc
35 ior heart field and support a model in which GATA factors and ISL1 serve as the earliest transcriptio
36                   This critical link between GATA factors and SNCA may enable therapies designed to l
37 CF3/ATOH1 heterodimers as well as motifs for GATA factors and SP1.
38 is mediated by the C-terminal zinc finger of GATA factors and the homeodomain of HNF-1alpha.
39 nues with Zfh1 activation of Serpent (Srp; a GATA factor), and terminates with Srp activation of U-sh
40 is enhancer has been shown to rely on PDX-1, GATA factors, and Cdx factors for its function.
41                         FOG-2 interacts with GATA factors, and interaction of GATA-4 and FOG-2 result
42 portant functional interplay between Ikaros, GATA factors, and the NOTCH signaling pathway in specifi
43  activities of both mammalian and Drosophila GATA factors are controlled in part by physical interact
44 a4 in mouse embryonic pancreas and show that GATA factors are essential regulators of the proliferati
45 ndings demonstrate that the EGL-18 and ELT-6 GATA factors are essential, genetically redundant regula
46        Binding sites for FoxH1, scl, ets and gata factors are found in the zebrafish flk1 endothelial
47                                              GATA factors are fundamental components of developmental
48 trophy, demonstrating that cardiac-expressed GATA factors are necessary mediators of this process.
49                                        Since GATA factors are not chamber-restricted, these findings
50 ypertrophy-associated genes, suggesting that GATA factors are sufficient regulators of cardiomyocyte
51                                              GATA factors are transcriptional regulatory proteins tha
52 t that elt-5 and -6, adjacent genes encoding GATA factors, are essential for the development of the l
53 e to valine in the DNA-binding domain of the GATA factor AreA results in inability to activate some A
54               To evaluate the requirement of GATA factors as downstream transcriptional mediators of
55                  A new gene encoding a novel GATA factor, ASD4, of Neurospora crassa was isolated and
56                              We suggest that GATA factors at target sites in chromatin may generally
57                            Our results place GATA factors at the top of the transcriptional network h
58 ng gene duplication and modular evolution of GATA factors based upon inclusion of a class IV zinc fin
59 ese factors activate a sequential cascade of GATA factors, beginning with their immediate targets, th
60                                         Both GATA factors bind the PU.1/Sfpi1 gene at 2 highly conser
61                       Although hematopoietic Gata factors bind with Scl to both activated and repress
62 t-expressed genes possess a preponderance of GATA factor binding sites and one GATA factor, ELT-2, fu
63               Two highly conserved consensus GATA factor binding sites within the 275 bp region inter
64                                  Analysis of GATA factor binding to additional loci also revealed FOG
65                    ELT-2 interacted with the GATA factor-binding motif in vitro and was also capable
66 tors regulate GnRH transcription through two GATA factor-binding motifs that occur in a tandem repeat
67                                          The GATA factor-binding region exhibited cell-type-specific
68 oform accounts for about half of the nuclear GATA-factor-binding activity in the endoderm.
69  in animals and fungi and are referred to as GATA factors by virtue of their affinity for promoter el
70 scriptional regulators, including Sp1, AP-2, GATA factors, c-Myb, and NF-IL-6.
71 njection ventrally of a dominant-interfering GATA factor (called G2en) induced the formation of secon
72                                        Thus, GATA factors can function in ESC derivatives upstream of
73                          We demonstrate that GATA factors can interact with Gli factors and can recru
74  of a single transcription factor, the ELT-7 GATA factor, can convert the identity of fully different
75  similar to that of vertebrates but only one GATA factor, Ci-GATAa, is expressed in the heart progeni
76 e element-binding protein pathways and other Gata factors, compared with Gata2-dependent HPCs.
77        These results indicate that dispersed GATA factor complexes function via long-range chromatin
78                                              GATA factors constitute a family of transcriptional regu
79                  GATA-1 and other vertebrate GATA factors contain a DNA binding domain composed of tw
80 the potential for a regulatory loop in which GATA factors control the expression of their partner pro
81 rated micro RNA expression and function into GATA factor coordinated networks and provided mechanisti
82 ies establish a transcriptional hierarchy of Gata factor dependence during hematopoiesis and demonstr
83 of these and previous reports, we infer that GATA factor dependence is a critical aspect of FOG prote
84 evealed unique +9.5 site activity to mediate GATA factor-dependent chromatin structural transitions.
85 erable progress has been made in elucidating GATA factor-dependent genetic networks that control bloo
86 erable progress has been made in elucidating GATA factor-dependent genetic networks that control red
87 he Friend of GATA (FOG) coregulator mediates GATA factor-dependent transcription is unknown.
88 ference to poor ones (proline) by repressing GATA factor-dependent transcription of the genes needed
89                  In trophoblast progenitors, GATA factors directly regulate BMP4, Nodal and Wnt signa
90  dHAND as a direct transcriptional target of GATA factors during right ventricle development.
91           In line with the essential role of GATA factors, ectopic expression of miR-27a or miR-24 pr
92               The second class truncates the GATA factor either within or upstream of the putative Le
93                                  Moreover, a GATA factor, either GATA-1 or GATA-2, is required to ini
94 derance of GATA factor binding sites and one GATA factor, ELT-2, fulfills the expected characteristic
95     We have discovered a second gut-specific GATA factor, ELT-7, that profoundly synergizes with ELT-
96 eplace the complete set of endoderm-specific GATA factors: END-1, END-3, ELT-7 and (the probably non-
97 ntaining transcriptional regulators known as GATA-factors ensures efficient utilization of available
98 partially redundant, adjacent genes encoding GATA factors essential for viability, seam cell developm
99 chanisms underlying this restriction and how GATA factors establish genetic networks, we used ChIP-se
100                                              GATA factors establish transcriptional networks that con
101 cantly, only NIT2, of the several Neurospora GATA factors examined, interacts with NIT4.
102                       This work shows that a GATA factor exchange reconfigures higher-order chromatin
103 scriptional regulators and is the only known GATA factor expressed in the distal epithelium of the lu
104 we have demonstrated that down-regulation of GATA factor expression leads to dedifferentiation.
105 lasmids with wild-type and dominant negative GATA factor expression vectors demonstrated that both GA
106                               Restoration of GATA factors expression by ectopic transfection suppress
107 ng that the MEDs are atypical members of the GATA factor family that do not recognize GATA sequences.
108                       SRE is a member of the GATA factor family, which is comprised of transcription
109 e controlled by MED-1 and -2, members of the GATA factor family.
110 c-finger/basic domains characteristic of the GATA factor family.
111 elucidated mechanisms by which FOGs regulate GATA factor function and discuss how these factors use t
112               Finally, a functional role for GATA factor function in alveolar epithelial type 1 cell
113 lation is leukemogenic, and its influence on GATA factor function is unknown, this mechanistic link h
114 at it acts as a transcriptional repressor of GATA factor function.
115 modifying enzyme mediates cell-type-specific GATA factor function.
116 ive to consider mechanisms underlying normal GATA factor function/regulation and how dissecting such
117 n a genome wide scale that the hematopoietic GATA factors GATA-1 and GATA-2 bind overlapping sets of
118 ion directly at promoters, the hematopoietic GATA factors GATA-1 and GATA-2 often assemble dispersed
119                            The hematopoietic GATA factors GATA-1 and GATA-2, which have distinct and
120     Herein, we discuss how the hematopoietic GATA factors (GATA-1-3) function via a battery of mechan
121 In this study, we report that a heterologous GATA factor, GATA-4, was competent in supporting the dev
122   We also demonstrate that a closely related GATA factor, GATA4, is expressed transiently in the preh
123        Our experiments also demonstrate that GATA factor gene expression is highly regulated by the G
124                             In addition, the GATA factor gene pannier (pnr) and the homeobox gene tin
125                                          The GATA factor gene pannier is similarly expressed in the d
126 ta support the contention that regulation of GATA-factor gene expression is tightly and dynamically c
127 is, we identified the functionally redundant GATA factor genes egl-18 and elt-6 as Wnt pathway target
128    GATA5, the only one of the six vertebrate GATA factor genes not yet inactivated in mice, is expres
129 laps with but is distinct from that of other GATA factor genes.
130  Ure2, the protein that negatively regulates GATA factor (Gln3, Gat1)-mediated transcription in Sacch
131 charomyces cerevisiae requires activation by GATA factor Gln3p or Nil1p and is prevented by the prese
132 y does not interfere with the binding of the GATA factor Gln3p to GATAAG sites but acts directly on G
133 ger region highly homologous to those of the GATA factors Gln3p and Nil1p as an antagonist of Nil1p a
134 f a cross-repressive interaction between the GATA factors GNC and GNL and the MADS box transcription
135                       Here, we show that the GATA factor HANABA TARANU (HAN) is required to position
136                                    The END-1 GATA factor has been implicated in specifying endoderm i
137 n Drosophila, the existence of a cardiogenic GATA factor has been implicated through the analysis of
138                                           As GATA factors have been implicated in endoderm developmen
139                                 Both Ets and Gata factors have been shown to have important roles in
140                          Although vertebrate GATA factors have two highly conserved zinc finger motif
141                                      Several GATA factors have two promoters directing distinct tissu
142           GAT1 of Candida albicans encodes a GATA-factor homologous to the AREA protein of Aspergillu
143 ily conserved domain that determines B-class GATA factor identity and provides a further subclassific
144 servations identify GATA6 as the predominant GATA factor in the maintenance of endodermal gene expres
145           The transcriptional targets of the GATA factors in early embryonic development include Disa
146 of late endoderm development, but a role for GATA factors in establishing the endoderm is unknown.
147          Our data highlight a novel role for GATA factors in fine tuning Notch signaling during intes
148                    The roles of scl, ets and gata factors in hemangioblasts have been well defined, b
149 o participate in differentiation mediated by GATA factors in several tissues.
150 omplexes that are thought to sequester these GATA factors in the cytoplasm of cells cultured in exces
151 sible loss of expression of HNF3 (Foxa2) and GATA factors in the endoderm and the absence of factors
152                                        Thus, GATA factors in the endoderm are among the first to bind
153 for heart development and acts downstream of GATA factors in the pre-cardiac mesoderm to specify line
154      Herein, we analyzed the role of several GATA factors in the regulation of the erythropoietin gen
155 e first direct demonstration of a target for GATA factors in the vertebrate intestinal epithelium.
156 eracts specifically with the amino finger of GATA factors in the yeast two-hybrid system and in mamma
157 2 in hematopoietic cells are not occupied by GATA factors in trophoblast cells.
158 omous roles performed by multiple, redundant GATA factors in vertebrate cardiogenesis.
159  heart function and redundancy among various GATA factors in vertebrates.
160 sites within the 275 bp region interact with GATA factors in vitro.
161  suggesting that FOG proteins might act in a GATA factor-independent manner.
162 rker genes in animal cap assays, while other GATA factors induce these genes only weakly, if at all.
163                                              GATA factors interact with simple DNA motifs (WGATAR) to
164 ing modes on the affinity and specificity of GATA factors is discussed.
165 rm cells to show that a DNA-binding site for GATA factors is occupied on a liver-specific, transcript
166                              The activity of GATA factors is regulated, in part, at the level of prot
167 attern, indicating that a normal function of GATA factors is to limit the boundary of the Nkx2.5 expr
168 tional program, mediated at least in part by GATA factors, is critical in presumptive foregut endoder
169                                  Analysis of GATA factors isolated from vertebrates suggests that the
170                   These results suggest that GATA factors may function sequentially to regulate endod
171  genitourinary system and suggest that other GATA factors may have functions overlapping those of GAT
172  establish fundamental principles underlying GATA factor mechanisms in chromatin and illustrate a com
173                      This article highlights GATA factor mechanistic principles, with a heavy emphasi
174 anisms were also leveraged to discover novel GATA factor-mediated cell regulatory pathways.
175  However, our experiments also indicate that GATA factors might normally antagonize transcription of
176                                 Studies with GATA factor mutants and novel chimeric GATA factors prov
177    In addition, elt-3, which encodes another GATA factor normally expressed in non-seam epidermis, is
178               We found that knockdown of the GATA factors not only alters the expression of genes hav
179                                              GATA factors occupied loci encoding multiple components
180               These results demonstrate that GATA factors occupy a central position between canonical
181               However, the precise impact of GATA factors on vertebrate cardiogenesis is masked by fu
182 at are independent of those regulated by the GATA factors or TAL1.
183                                              GATA factors orchestrate hematopoiesis via multistep tra
184                       We also identified the GATA factor pannier (pnr) as a downstream target of Stat
185                                          The GATA factor Pannier (Pnr) is required for eye and heart
186 ring the regulation of nmr, we find that the GATA factor Pannier is essential for cardiac expression,
187       The homeodomain protein Tinman and the GATA factors Pannier and Serpent directly activate Hand
188 of Friend of GATA proteins and the role that GATA factors play during cell fate choice, these factors
189              These observations suggest that GATA factors positively regulate slow MyHC 3 gene expres
190 t assembly of a protein complex containing a GATA-factor, presumably GATA-1 or GATA-2, is critical to
191  with GATA factor mutants and novel chimeric GATA factors provided evidence that both GATA-1 and GATA
192                     In order to test whether GATA factors regulated TTF-1 gene transcription, GATA-5
193 ver, accumulating evidence now suggests that GATA factor regulation may occur by two separate pathway
194 ating that these genes are direct targets of GATA factor regulation.
195 eases during normal aging, and both of these GATA factors repress expression of elt-3, which shows a
196 derm differentiation, including a cascade of GATA factors required for development and maintenance of
197 els determine the threshold concentration of GATA factors required for PrE-like differentiation, and
198 ith the Pp in vitro, (iii) overexpression of GATA factors rescued expression from mutant Pp construct
199 entation of signals calling for cessation of GATA factor sequestration in the cytoplasm.
200 on occurs through specific expression of the GATA factor Serpent (Srp) in the lymph-gland primordium.
201                                          The GATA factor Serpent (Srp) is essential for hh activation
202                                          The GATA factor Serpent (Srp) is required for hemocyte precu
203 rough Ush interaction with the hematopoietic GATA factor Serpent (Srp).
204                               The Drosophila GATA factor Serpent interacts with the RUNX factor Lozen
205 mbryogenesis and larval development, and the GATA factor Serpent is essential for Ush embryonic expre
206 g proteins, Dorsal, Dif, and Relish, and the GATA factor Serpent.
207 es overlapped significantly with that of the GATA factor Serpent.
208                                          The GATA factor, Serpent, directly regulated the activity of
209 , or chick embryos depleted specifically for GATA factors, show in addition abnormal foregut developm
210            URE-BF appears to interact with a GATA factor, since formation of the URE-BF complex can b
211  pathway, including positive feedback loops, GATA factors, SoxB, Brachyury and a previously underemph
212              We report that the MED-1 and -2 GATA factors specify the entire fate of EMS, which other
213 hromatin sites, we propose that differential GATA factor stability is an important determinant of chr
214 ry of mechanistic permutations, which can be GATA factor subtype, cell type, and locus specific.
215       Other cardiovascular tissue-restricted GATA factors, such as GATA-5 and GATA-6, were equivalent
216 both Gata1 and Gata2, or independent of both Gata factors, suggesting that multiple pathways regulate
217              This is followed by genome-wide GATA factor switching that mediates further induction of
218 ults indicate that FOG-1 is a determinant of GATA factor target gene sensitivity by either facilitati
219 re identify a novel small-molecule-activated GATA factor that is required to regulate the cell type-s
220 ine, as well as to overexpression of the two GATA factors that are normally involved in intestinal di
221                  These data suggest that two GATA factors that are required for seam cell specificati
222 One mode involves cooperative binding by two GATA factors that interact with each other through prote
223 These results indicate that similar to other GATA factors, the GATA-3 gene can be controlled by two p
224 r gene expression is highly regulated by the GATA factors themselves in an interdependent manner.
225 or negative coregulators that cooperate with GATA factors to control right ventricular-specific gene
226 vidual contributions of the three vertebrate GATA factors to endoderm formation have been unclear.
227 FOG-like proteins are corepressors that link GATA factors to histone deacetylation and nucleosome rem
228 nt, indicating differential contributions of GATA factors to pancreas formation.
229                      The capacity of related GATA factors to promote cardiogenesis is untested.
230 In contrast, FOG1 antagonizes the ability of GATA factors to promote mast cell (MC) development.
231        Friend of GATA proteins interact with GATA factors to regulate development in a variety of tis
232    Knockdown of FOG1 reverses the ability of GATA factors to repress Gli1 expression.
233 ed by anthracyclines, the ability of ectopic GATA factors to rescue anthracycline-induced apoptosis w
234   The discovery of the GATA binding protein (GATA factor) transcription factor family revolutionized
235      FOGs can both facilitate and antagonize GATA factor transcriptional regulation depending on the
236 regulation of GATA-2, an essential mast cell GATA factor, via switching of GATA-1 for GATA-2 at a key
237 ave shown that the combined absence of these GATA factors virtually ablates primitive erythroid cell
238           Induction of HNF3beta by all three GATA factors was abolished when protein synthesis was in
239                                      Loss of GATA factors was also found in an in vitro model for spo
240                        Altered expression of GATA factors was found and proposed as the underlying me
241 5, a target of the MED-1 and MED-2 divergent GATA factors, was previously found to result in a profou
242                        In seeking additional GATA factors, we have cloned areB, which was originally
243 d differentiation can be replaced by another GATA-factor, we generated a knock-in mutation of the GAT
244                                    All three GATA factors were induced by activin, although GATA4 and
245         Multiple binding sites for PDX-1 and GATA factors were previously identified within the appro
246 nsidering control of the nitrogen-responsive GATA factors when studying the regulation of the protein
247 Neurospora possesses at least five different GATA factors which control different areas of cellular f
248 nal signaling regulates the translation of a GATA factor, which is the specific transcriptional activ
249  C4-type zinc finger to those of other known GATA factors, which recognize the consensus HGATAR.
250 y increasing the expression of GATA-binding (Gata) factors, which suppressed expression of the microR
251 otein-protein interactions of the individual GATA factors with additional pathway-specific regulatory
252  with previous studies linking regulation by GATA factors with c-Jun and BRG1, provides genome-wide e
253 mma transcriptional activity, interaction of GATA factors with C/EBP is necessary for their ability t
254 eable and interact with different endodermal GATA factors with only modest differences in affinity.
255                                     Multiple GATA factors, zinc finger DNA binding proteins that reco

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